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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2014, Volume 111, Issue 3, pp. 1084 - 1089
Journal Article
The Prostate, ISSN 0270-4137, 09/2018, Volume 78, Issue 13, pp. 970 - 980
BackgroundInflammation plays a key role in the etiology of benign prostatic hyperplasia (BPH) through multiple pathways involving the stimulation of... 
growth factor | targeted therapy | epithelial‐to‐mesenchymal transition | neuropeptide | benign prostatic hyperplasia | epithelial-to-mesenchymal transition | PROLIFERATION | HYPERPLASIA | CANCER | OSTEOPONTIN | THERAPY | PATHWAY | TRANSACTIVATION | ANDROGEN-RECEPTOR | ENDOCRINOLOGY & METABOLISM | UROLOGY & NEPHROLOGY | STROMAL CELLS | EXPRESSION | Cell Line | Epithelial Cells - metabolism | Cytokines - metabolism | Rats | Epithelial Cells - pathology | Male | Prostatitis - metabolism | Rats, Sprague-Dawley | Prostatic Hyperplasia - chemically induced | Prostatitis - drug therapy | Prostatic Hyperplasia - metabolism | Animals | Prostatitis - chemically induced | Signal Transduction - physiology | Carrageenan | Growth Hormone-Releasing Hormone - antagonists & inhibitors | Prostatic Hyperplasia - drug therapy | Hormone antagonists | Somatotropin | Carrageenin | Somatotropin releasing hormone | Analysis | Stem cells | Bone morphogenetic proteins | Prostatitis | Inflammation | Transforming growth factors | Health aspects | Epidermal growth factor receptors | Mesenchyme | Cytokines | Hyperplasia | Stat3 protein | Extracellular signal-regulated kinase | AKT protein | Antagonists | Insulin-like growth factors | Signal transduction | Growth hormone-releasing hormone | Etiology | Growth hormone | Autoimmune diseases | Growth factors | Prostate
Journal Article
Journal of Urology, The, ISSN 0022-5347, 2012, Volume 187, Issue 4, pp. 1498 - 1504
Journal Article
Journal Article
Investigational New Drugs, ISSN 0167-6997, 10/2014, Volume 32, Issue 5, pp. 871 - 882
Growth hormone-releasing hormone (GHRH) and its receptors have been implicated in a variety of cellular phenotypes related with tumorigenesis process. Human... 
Medicine & Public Health | HER | GHRH antagonists | GHRH | Cross-talk | Oncology | Pharmacology/Toxicology | Transactivation | ACTIVATION | SRC | CELL LINES | PROTEIN-COUPLED RECEPTORS | SPLICE VARIANTS | EGFR | PEPTIDE | ONCOLOGY | PATHWAY | PHARMACOLOGY & PHARMACY | HETERODIMER | EXPRESSION | Prostatic Neoplasms - metabolism | Receptor, Epidermal Growth Factor - genetics | Receptor, ErbB-2 - genetics | Humans | Receptor, ErbB-2 - metabolism | Receptors, Neuropeptide - genetics | Male | Growth Hormone-Releasing Hormone - pharmacology | RNA, Messenger - metabolism | Receptor, Epidermal Growth Factor - metabolism | Animals | Mice, Nude | Sermorelin - pharmacology | src-Family Kinases - metabolism | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Sermorelin - analogs & derivatives | Growth Hormone-Releasing Hormone - analogs & derivatives | Receptor, ErbB-2 - antagonists & inhibitors | Epidermal Growth Factor - pharmacology | Growth Hormone-Releasing Hormone - antagonists & inhibitors | Receptors, Pituitary Hormone-Regulating Hormone - genetics | Development and progression | Genetic aspects | Research | Somatotropin releasing hormone | Prostate cancer | Xenotransplantation | Studies | Genotype & phenotype | Growth hormones | Pharmacology | Epidermal growth factor | Analysis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 01/2014, Volume 111, Issue 3, p. 1084
  Advanced hormone-sensitive prostate cancer responds to androgen-deprivation therapy (ADT); however, therapeutic options for recurrent castration-resistant... 
Androgens | Castration | Growth hormones | Prostate cancer | Cancer therapies | Cells
Journal Article
Hormone and Metabolic Research, ISSN 0018-5043, 10/2011, Volume 43, Issue 11, pp. 816 - 820
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2010, Volume 107, Issue 51, pp. 22272 - 22277
Journal Article
Journal Article
Journal Article
The Prostate, ISSN 0270-4137, 09/2018, Volume 78, Issue 12, pp. 915 - 926
BackgroundTherapeutic strategies should be designed to transform aggressive prostate cancer phenotypes to a chronic situation. To evaluate the effects of the... 
antagonists | GHRH‐R | prostate cancer | cell proliferation | angiogenesis | cell adhesion | GHRH-R | HUMAN BREAST | CELL-ADHESION | E-CADHERIN | MESENCHYMAL TRANSITION | BETA-CATENIN | SPLICE VARIANTS | P21 | INHIBITION | GHRH | ENDOCRINOLOGY & METABOLISM | UROLOGY & NEPHROLOGY | EXPRESSION | Cell Survival - drug effects | Prostatic Neoplasms - pathology | PC-3 Cells | Apoptosis - drug effects | beta Catenin - analysis | Humans | Receptors, Neuropeptide - antagonists & inhibitors | Male | Vascular Endothelial Growth Factor A - analysis | Resting Phase, Cell Cycle | Vascular Endothelial Growth Factor A - metabolism | Cell Adhesion - drug effects | Sermorelin - pharmacology | Cell Line, Tumor | Sermorelin - analogs & derivatives | Cell Proliferation - drug effects | Cell Cycle - drug effects | Receptors, Pituitary Hormone-Regulating Hormone - antagonists & inhibitors | Wound Healing - drug effects | Prostatic Neoplasms - drug therapy | Cell proliferation | Phenotypes | Bax protein | p53 Protein | Malignancy | E-cadherin | Gelatinase B | Cell adhesion & migration | Gelatinase A | Angiogenesis | Growth hormone-releasing hormone | Cell lines | Cell adhesion | Proliferating cell nuclear antigen | Growth hormones | Growth hormone | Vascular endothelial growth factor | Prostate cancer | Prostate | Cell migration | Apoptosis
Journal Article