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Annals of Oncology, ISSN 0923-7534, 2014, Volume 25, Issue 1, pp. 126 - 131
Background: There is scarce data available about epidermal growth factor receptor (EGFR) mutations other than common exon 19 deletions and exon 21 (L858R)... 
Non-small-cell lung cancer | Exon 20 mutations | Tyrosine-kinase inhibitors | Epidermal growth factor receptor mutations | Exon 18 mutations | epidermal growth factor receptor mutations | GEFITINIB | axon 18 mutations | exon 20 mutations | tyrosine-kinase inhibitors | TKIS | SENSITIVITY | CLASSIFICATION | SOMATIC MUTATIONS | CHEMOTHERAPY | TYROSINE KINASE INHIBITORS | GROWTH-FACTOR RECEPTOR | ONCOLOGY | NEVER SMOKERS | CLINICAL-RESPONSE | non-small-cell lung cancer | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Exons | Lung Neoplasms - mortality | Humans | Middle Aged | Male | Antineoplastic Agents - therapeutic use | Young Adult | Aged, 80 and over | Adult | Female | Adenocarcinoma - genetics | Antineoplastic Agents - pharmacology | Lung Neoplasms - genetics | Genetic Association Studies | Carcinoma, Non-Small-Cell Lung - genetics | Gene Frequency | Proportional Hazards Models | Adenocarcinoma - drug therapy | Carcinoma, Non-Small-Cell Lung - mortality | Disease-Free Survival | Drug Resistance, Neoplasm - genetics | Protein Kinase Inhibitors - therapeutic use | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Aged | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Adenocarcinoma - mortality | Life Sciences | Cancer | exon 18 mutations | Original
Journal Article
Molecular Cell, ISSN 1097-2765, 2009, Volume 35, Issue 5, pp. 563 - 573
The target of rapamycin complex 1 (TORC1) is a central regulator of eukaryotic cell growth that is activated by a variety of hormones (e.g., insulin) and... 
CELLBIO | PROTEINS | SIGNALING | YEAST SACCHAROMYCES-CEREVISIAE | CELL-GROWTH CONTROL | SIGNALING PATHWAYS | FUNCTIONAL HOMOLOG | RAG GTPASES | VACUOLE FUSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMINO-ACID PERMEASE | COMPONENT | GTP-BINDING PROTEINS | GAP1 PERMEASE | CELL BIOLOGY | Vacuoles - enzymology | Intracellular Membranes - enzymology | Saccharomyces cerevisiae - genetics | Multiprotein Complexes | Adaptor Proteins, Vesicular Transport - genetics | Saccharomyces cerevisiae - drug effects | Guanosine Triphosphate - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Vacuoles - drug effects | DNA-Binding Proteins - metabolism | Amino Acids - metabolism | Time Factors | Guanine Nucleotide Exchange Factors - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Guanosine Diphosphate - metabolism | Protein Synthesis Inhibitors - pharmacology | Protein-Serine-Threonine Kinases - metabolism | Guanine Nucleotide Exchange Factors - genetics | Signal Transduction | Saccharomyces cerevisiae Proteins - antagonists & inhibitors | Monomeric GTP-Binding Proteins - genetics | Saccharomyces cerevisiae Proteins - genetics | Amino Acid Transport Systems - metabolism | Sirolimus - pharmacology | Cycloheximide - pharmacology | Protein Transport | Transcription Factors - metabolism | Monomeric GTP-Binding Proteins - metabolism | Saccharomyces cerevisiae Proteins - metabolism | Protein Binding | Saccharomyces cerevisiae - enzymology | Endosomes - enzymology | Intracellular Membranes - drug effects | Mutation | Saccharomyces cerevisiae - growth & development | Proteins | Purines | Amino acids | Gross domestic product | Guanosine
Journal Article
The Journal of Immunology, ISSN 0022-1767, 05/2009, Volume 182, Issue 9, pp. 5748 - 5756
IL-27 is secreted by APCs in response to inflammatory stimuli and exerts a proinflammatory Th1-enhancing activity but also has significant anti-inflammatory... 
EFFECTOR PHASE | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | TGF-BETA | INTERLEUKIN-27 | INFLAMMATION | HELPER-CELLS | REGULATORY T-CELLS | CENTRAL-NERVOUS-SYSTEM | CYTOKINE | INDUCTION | IMMUNOLOGY | Humans | Receptors, Retinoic Acid - biosynthesis | Encephalomyelitis, Autoimmune, Experimental - immunology | Receptors, Thyroid Hormone - antagonists & inhibitors | Receptors, Retinoic Acid - genetics | Cell Differentiation - genetics | Interleukins - genetics | Receptors, Thyroid Hormone - genetics | Interleukins - physiology | T-Lymphocytes, Helper-Inducer - immunology | T-Lymphocytes, Helper-Inducer - pathology | Encephalomyelitis, Autoimmune, Experimental - genetics | Protein Subunits - physiology | Growth Inhibitors - genetics | Cell Lineage - genetics | Protein Subunits - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 | Genetic Predisposition to Disease | Encephalomyelitis, Autoimmune, Experimental - pathology | T-Lymphocytes, Helper-Inducer - metabolism | Growth Inhibitors - deficiency | Gene Expression Regulation - immunology | Mice, Inbred C57BL | Receptors, Thyroid Hormone - biosynthesis | Cells, Cultured | Receptors, Retinoic Acid - antagonists & inhibitors | Interleukin-17 - biosynthesis | Mice, Knockout | Cell Differentiation - immunology | Animals | Interleukin-17 - antagonists & inhibitors | Protein Subunits - deficiency | Growth Inhibitors - physiology | Interleukins - deficiency | Mice | Cell Lineage - immunology
Journal Article
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 07/2011, Volume 31, Issue 14, pp. 3038 - 3051
Journal Article