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Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 3050 - 3059
DJ‐1/Park7 is a redox‐sensitive chaperone protein counteracting oxidation and presumably contributing to the control of oxidative stress responses and thus... 
HEART | IMMUNITY | PHYSIOLOGY | PROTEIN | DENDRITIC CELLS | GENE | ROS | DJ-1 | EXPRESSION | PARKINSONS-DISEASE | PLASTICITY | CELL BIOLOGY | Up-Regulation | Cation Transport Proteins - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Protein-Tyrosine Kinases - metabolism | Protein Deglycase DJ-1 - genetics | RNA, Messenger - metabolism | Sodium-Hydrogen Exchangers - metabolism | Time Factors | Cation Transport Proteins - metabolism | Cation Transport Proteins - genetics | Sodium-Hydrogen Exchangers - genetics | Sodium-Hydrogen Exchangers - antagonists & inhibitors | Protein Deglycase DJ-1 - deficiency | Protein Deglycase DJ-1 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Genotype | Antioxidants - pharmacology | Mice, Knockout | Phenotype | Animals | Protein Kinase Inhibitors - pharmacology | Sodium-Hydrogen Exchanger 1 | CD4-Positive T-Lymphocytes - drug effects | Hydrogen-Ion Concentration | Protein-Tyrosine Kinases - antagonists & inhibitors | T cells | Cysteine | Oxidative stress | Ammonium | Parkinson's disease | Staurosporine | Acetylcysteine | Fluorescence | Lymphocytes T | Gene deletion | Kinases | Western blotting | Proteins | Clonal deletion | Lymphocytes | Rodents | Deletion | Oxidation | Protein-tyrosine kinase | Movement disorders | Tyrosine | Neurodegenerative diseases | Na+/H+-exchanging ATPase | Inflammation | CD4 antigen | Polymerase chain reaction | PARK7 protein | Mice
Journal Article
The Journal of Physiology, ISSN 0022-3751, 02/2019, Volume 597, Issue 3, pp. 661 - 662
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 11/2019, Volume 136, pp. 53 - 63
Heart failure is the leading cause of death among diabetic people. Cellular and molecular entities leading to diabetic cardiomyopathy are, however, poorly... 
Intracellular pH | Diabetic cardiomyopathy | Carbonic anhydrase II | NHE1 | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | HEART-FAILURE | II BINDS | PREVENTS | CELL BIOLOGY | INHIBITION | CARDIAC-HYPERTROPHY | COTRANSPORTER | NA+/H+ EXCHANGER
Journal Article
Glia, ISSN 0894-1491, 11/2018, Volume 66, Issue 11, pp. 2279 - 2298
Na+/H+ exchanger (NHE1) activation is required for multiple microglial functions. We investigated effects of selective deletion of microglial Nhe1 in... 
phagocytosis | macrophages | inflammation | microglia | white matter tissue repair | SURVIVAL | BRAIN-INJURY | FOCAL CEREBRAL-ISCHEMIA | RECEPTOR | CNS | NEUROSCIENCES | NEURONAL DEATH | GROWTH | MICE | EXPRESSION | Microglia - metabolism | Somatosensory Disorders - etiology | Brain - diagnostic imaging | Recovery of Function - drug effects | Male | CX3C Chemokine Receptor 1 - metabolism | CX3C Chemokine Receptor 1 - genetics | Infarction, Middle Cerebral Artery - complications | Recovery of Function - physiology | Female | Demyelinating Diseases - etiology | Infarction, Middle Cerebral Artery - drug therapy | Microfilament Proteins - metabolism | Infarction, Middle Cerebral Artery - diagnostic imaging | Demyelinating Diseases - drug therapy | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Macrophages - pathology | Microglia - drug effects | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Sodium-Hydrogen Exchanger 1 - genetics | Mice, Transgenic | Nerve Tissue Proteins - genetics | White Matter - pathology | Brain - drug effects | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Macrophages - metabolism | White Matter - diagnostic imaging | Animals | Tamoxifen - pharmacology | Sodium-Hydrogen Exchanger 1 - metabolism | Mice | Stroke (Disease) | Neurons | Analysis | Brain damage | Bone morphogenetic proteins | Inflammation | Macrophages | Transforming growth factors | Brain | CD86 antigen | Hydrogen | CX3CR1 protein | Activation | Recovery | Proteins | Cell activation | Corn oil | Clonal deletion | Ischemia | Demyelination | Rodents | Animal tissues | Deletion | Lesions | Repair | Stroke | CD11b antigen | Cell survival | Therapeutic applications | Astrocytes | Na+/H+-exchanging ATPase | Glial fibrillary acidic protein | Tamoxifen | Substantia alba | Survival | Microglia | Myelination | Brain injury | Ca2+/calmodulin-dependent protein kinase II
Journal Article
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 4/2009, Volume 185, Issue 1, pp. 163 - 176
Journal Article
British journal of pharmacology, ISSN 0007-1188, 2008, Volume 154, Issue 6, pp. 1266 - 1275
Journal Article