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1. Full Text Oncogenic ras isoforms signaling specificity at the membrane
by Nussinov, Ruth and Tsai, Chung-Jung and Jang, Hyunbum
Cancer research (Chicago, Ill.), ISSN 0008-5472, 02/2018, Volume 78, Issue 3, pp. 593 - 602
Life Sciences & Biomedicine | Oncology | Science & Technology | ras Proteins - genetics | Neoplasms - genetics | Signal Transduction | Humans | Protein Isoforms | ras Proteins - metabolism | Cell Membrane - metabolism | Neoplasms - pathology | Oncogenes | TOR protein | H-Ras protein | Crosstalk | Extracellular signal-regulated kinase | MAP kinase | Raf protein | AKT protein | Activation | mRNA | Pharmacology | K-Ras protein | Signal transduction | Signaling | Pathways | Calcium-binding protein | Isoforms | Dimerization | Calmodulin | Cancer | Index Medicus | K-RAS4A | H-RAS | K-RAS | N-RAS | KRas4A | small molecule drug | K-RAS4B
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2. Full Text A novel role for flotillin‐1 in H‐Ras‐regulated breast cancer aggressiveness
by Koh, Minsoo and Yong, Hae‐Young and Kim, Eun‐Sook and Son, Hwajin and Jeon, You Rim and Hwang, Jin‐Sun and Kim, Myeong‐Ok and Cha, Yujin and Choi, Wahn Soo and Noh, Dong‐Young and Lee, Kyung‐Min and Kim, Ki‐Bum and Lee, Jae‐Seon and Kim, Hyung Joon and Kim, Haemin and Kim, Hong‐Hee and Kim, Eun Joo and Park, So Yeon and Kim, Hoe Suk and Moon, Woo Kyung and Choi Kim, Hyeong‐Reh and Moon, Aree
International journal of cancer, ISSN 0020-7136, 03/2016, Volume 138, Issue 5, pp. 1232 - 1245
lipid raft | flotillin‐1 | invasion | triple‐negative breast cancer | H‐Ras | triple-negative breast cancer | H-Ras | flotillin-1 | Life Sciences & Biomedicine | Oncology | Science & Technology | Phosphorylation | Signal Transduction | Neoplasm Invasiveness | Humans | Middle Aged | Proto-Oncogene Proteins c-akt - physiology | Receptor, Epidermal Growth Factor - metabolism | Animals | Breast Neoplasms - pathology | Membrane Proteins - physiology | Proteomics | Cell Line, Tumor | Phosphatidylinositol 3-Kinases - physiology | Adult | Breast Neoplasms - mortality | Female | Aged | Mice | Mice, Inbred BALB C | Genes, ras | Cell Movement | Proteins | Analysis | Breast cancer | Lipids | Epidermal growth factor | Metastasis | Aggressiveness | Cell culture | Biotechnology | Animal models | Correlation | H-Ras protein | Epithelial cells | Rafts | Staining | Lipid rafts | Metastases | Cell activation | Xenografts | Tumorigenesis | Inhibition | Aggressive behavior | Invasiveness | Tumorigenicity | Aberration | In vitro methods and tests | Cancer | Index Medicus
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3. Full Text H‐ras deletion protects against angiotensin II–induced arterial hypertension and cardiac remodeling through protein kinase G‐Iβ pathway activation
by Martín‐Sánchez, Paloma and Luengo, Alicia and Griera, Mercedes and Orea, María Jesús and López‐Olañeta, Marina and Chiloeches, Antonio and Lara‐Pezzi, Enrique and Frutos, Sergio and Rodríguez–Puyol, Manuel and Calleros, Laura and Rodríguez–Puyol, Diego
The FASEB journal, ISSN 0892-6638, 02/2018, Volume 32, Issue 2, pp. 920 - 934
CREB | cardiac hypertrophy | PKG | H‐ras | Cardiac hypertrophy | H-ras | Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Index Medicus
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4. Full Text Aspafilioside B induces G2/M cell cycle arrest and apoptosis by up‐regulating H‐Ras and N‐Ras via ERK and p38 MAPK signaling pathways in human hepatoma HepG2 cells
by Liu, Wei and Ning, Rui and Chen, Rui‐Ni and Huang, Xue‐Feng and Dai, Qin‐Sheng and Hu, Jin‐Hua and Wang, Yu‐Wen and Wu, Li‐Li and Xiong, Jing and Hu, Gang and Guo, Qing‐Long and Yang, Jian and Wang, Hao
Molecular carcinogenesis, ISSN 0899-1987, 05/2016, Volume 55, Issue 5, pp. 440 - 457
apoptosis | aspafilioside B | G2 arrest | N‐Ras | H‐Ras | H-Ras | N-Ras | Apoptosis | Aspafilioside B | Biochemistry & Molecular Biology | Oncology | Life Sciences & Biomedicine | Science & Technology | Spirostans - pharmacology | Cell Survival - drug effects | Liver Neoplasms - genetics | Saponins - administration & dosage | Humans | Liver Neoplasms - drug therapy | Hep G2 Cells | Xenograft Model Antitumor Assays | Antineoplastic Agents, Phytogenic - administration & dosage | Animals | Carcinoma, Hepatocellular - drug therapy | G2 Phase Cell Cycle Checkpoints - drug effects | MAP Kinase Signaling System - drug effects | Carcinoma, Hepatocellular - genetics | Spirostans - administration & dosage | Cell Line, Tumor | Female | Cell Proliferation - drug effects | Genes, ras - drug effects | Mice | Gene Expression Regulation, Neoplastic - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Saponins - pharmacology | Cysteine | Analysis | Cell cycle | Cytotoxicity | Phosphorylation | Chemotherapy | Kinases | Biotechnology | Reactive oxygen species | Animal models | H-Ras protein | G2 phase | Acetylcysteine | Raf protein | Activation | Hepatoma | Anticancer properties | Proteins | Transfection | Xenografts | Activation analysis | Asparagus | Extracellular signal-regulated kinase | MAP kinase | Pharmacology | Chemical compounds | Inhibitors | Lymphocytes B | Antitumor activity | N-Acetyl-L-cysteine | In vitro methods and tests | Index Medicus
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5. Full Text MageC2 protein is upregulated by oncogenic activation of MAPK pathway and causes impairment of the p53 transactivation function
by Pascucci, Franco Andrés and Ladelfa, María Fátima and Toledo, María Fernanda and Escalada, Micaela and Suberbordes, Melisa and Monte, Martín
Biochimica et biophysica acta. Molecular cell research, ISSN 0167-4889, 03/2021, Volume 1868, Issue 3, pp. 118918 - 118918
MAGE | H-Ras | Melanoma | B-Raf | MAPK | Oncogenes | p53 | Index Medicus
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6. Full Text H-Ras activation and fibroblast-induced TGF-β signaling promote laminin-332 accumulation and invasion in cutaneous squamous cell carcinoma
by Siljamäki, Elina and Rappu, Pekka and Riihilä, Pilvi and Nissinen, Liisa and Kähäri, Veli-Matti and Heino, Jyrki
Matrix biology, ISSN 0945-053X, 05/2020, Volume 87, pp. 26 - 47
TGF-β | Laminin-332 | H-Ras | Fibroblast | cSCC | Invasion | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Squamous cell carcinoma | Collagen (type I) | H-Ras protein | Gels | U.V. radiation | Invasiveness | Keratinocytes | Metastasis | Spheroids | Skin cancer | Metastases | Laminin | Fibroblasts | Cancer | Tumors | Index Medicus
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7. Full Text A comprehensive survey of ras mutations in cancer
by Prior, Ian A and Lewis, Paul D and Mattos, Carla
Cancer research (Chicago, Ill.), ISSN 0008-5472, 05/2012, Volume 72, Issue 10, pp. 2457 - 2467
Life Sciences & Biomedicine | Oncology | Science & Technology | Biological and medical sciences | Medical sciences | Antineoplastic agents | Pharmacology. Drug treatments | Tumors | Catalytic Domain | Signal Transduction | Epigenesis, Genetic | Humans | Molecular Conformation | Databases, Genetic | Models, Molecular | Protein Isoforms - physiology | Point Mutation | Codon | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | ras Proteins - chemistry | Genes, ras | Index Medicus | NRAS | KRAS | mutation hotspot | HRAS | GTPase | mutagenesis
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8. Full Text Selective cross-linking behavior of oligodeoxyribonucleotides containing 2'-O-[N-(4,5',8-trimethylpsoralen-4'-ylmethylcarbamoyl)]adenosine to mutant H-ras DNA
by Yamayoshi, Asako and Higuchi, Maiko and Sakai, Yui and Kobori, Akio and Yamamoto, Tsuyoshi and Shibata, Takayuki and Murakami, Akira
Nucleosides, nucleotides & nucleic acids, ISSN 1525-7770, 02/2020, Volume 39, Issue 1-3, pp. 119 - 130
Photo-responsive oligonucleotide | H-ras | psoralen | point mutation | Adenosine | H-Ras protein | Mutation | DNA probes | Ribonucleic acid--RNA | Deoxyribonucleic acid--DNA | Index Medicus
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9. Design graph theoretical analysis and In Silico modeling of Dunaliella bardawil biomass encapsulated N-succinyl chitosan nanoparticles for enhanced anticancer activity
by Kunjiappan, Selvaraj and Panneerselvam, Theivendren and Somasundaram, Balasubramanian and Sankaranarayanan, Murugesan and Parasuraman, Pavadai and Joshi, Shrinivas D and Arunachalam, Sankarganesh and Murugan, Indhumathy
Anti-cancer agents in medicinal chemistry, ISSN 1871-5206, 2018, Volume 18, Issue 13, pp. 1900 - 1918
Cytotoxicity | Graph theory | H-Ras P | N-succinyl | Dunaliella bardawil | Apoptosis | Pharmacology & Pharmacy | Oncology | Life Sciences & Biomedicine | Chemistry, Medicinal | Science & Technology
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10. Full Text Targeting the α4–α5 dimerization interface of K-RAS inhibits tumor formation in vivo
by Khan, Imran and Spencer-Smith, Russell and O’Bryan, John P
Oncogene, ISSN 0950-9232, 2018, Volume 38, Issue 16, pp. 2984 - 2993
Growth conditions | Transformation | H-Ras protein | Doxycycline | Dimerization | K-Ras protein | Tumors | Cancer | tumorigenesis | RAS GTPases | monobody | oncogenesis
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11. Full Text Oncogenic H-Ras expression induces fatty acid profile changes in human fibroblasts and extracellular vesicles
by Sagini, Krizia and Sagini, Krizia and Urbanelli, Lorena and Urbanelli, Lorena and Costanzi, Eva and Mitro, Nico and Caruso, Donatella and Caruso, Donatella and Emiliani, Carla and Buratta, Sandra
International journal of molecular sciences, ISSN 1661-6596, 11/2018, Volume 19, Issue 11, p. 3515
Acyl-coenzyme a synthetases | Elongases | H-Ras | Desaturases | Extracellular vesicles | Oncogene-induced senescence | Fatty acids | Biochemistry & Molecular Biology | Physical Sciences | Chemistry | Life Sciences & Biomedicine | Chemistry, Multidisciplinary | Science & Technology | Index Medicus | elongases | extracellular vesicles | acyl-coenzyme A synthetases | fatty acids | oncogene-induced senescence | desaturases
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