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European Journal of Medicinal Chemistry, ISSN 0223-5234, 11/2015, Volume 104, pp. 157 - 164
Journal Article
Journal Article
Archives of Toxicology, ISSN 0340-5761, 3/2014, Volume 88, Issue 3, pp. 659 - 671
Hypoxia-mediated stress responses are important in tumor progression, especially when tumor growth causes the tumor to become deprived of its blood supply. The... 
Biomedicine | Environmental Health | Occupational Medicine/Industrial Medicine | Hypoxia-inducible factor-1 alpha | Minocycline | Neovasculogenesis | Prolyl hydroxylase | Pharmacology/Toxicology | von Hippel–Lindau | Biomedicine general | Von Hippel-Lindau | PROTEIN | METASTASIS | ANGIOGENESIS | MECHANISMS | CELL-DEATH | P53 | von Hippel-Lindau | TETRACYCLINES | TOXICOLOGY | HIF-1-ALPHA | VHL | EXPRESSION | Cell Hypoxia - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Minocycline - pharmacology | Prolyl Hydroxylases - metabolism | Humans | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor C - genetics | Cell Movement - drug effects | Matrix Metalloproteinase 2 - genetics | Cell Line - drug effects | Glucose Transporter Type 1 - genetics | Signal Transduction - drug effects | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Neovascularization, Pathologic - drug therapy | Vascular Endothelial Growth Factor C - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Medical colleges | Endothelial growth factors | College graduates | Heat shock proteins | Drugstores | Development and progression | Glucose | Gene expression | Dextrose | Glucose metabolism | Resveratrol | Physiological aspects | Tumors | Oxidative stress | Hypoxia | Kinases | Vascular endothelial growth factor | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 2016, Volume 111, Issue 1, pp. 74 - 83
Stress response, in terms of activation of stress factors, is known to cause obesity and coronary heart disease such as atherosclerosis in human. However, the... 
Heat shock proteins | Obesity | Heat shock factor | Glycoproteins | Atherosclerosis | CARDIAC & CARDIOVASCULAR SYSTEMS | DIETARY-FAT | TRANSCRIPTION FACTOR-1 | MACROPHAGE | ALPHA | DEFICIENT MICE | RESISTANCE GENE | INFLAMMATION | PPAR-GAMMA | NF-KAPPA-B | HEAT-SHOCK-PROTEIN-27 | Carotid Arteries - metabolism | Up-Regulation | Atherosclerosis - genetics | Male | Aorta - metabolism | PPAR gamma - metabolism | Time Factors | Carotid Artery Diseases - pathology | Aortic Diseases - prevention & control | Transcription, Genetic | ATP Binding Cassette Transporter, Subfamily B - genetics | Binding Sites | Diet, Western | Disease Models, Animal | Atherosclerosis - pathology | Genetic Predisposition to Disease | Signal Transduction | Plaque, Atherosclerotic | ATP Binding Cassette Transporter, Subfamily B - metabolism | Mice, Knockout | Aorta - pathology | Cholesterol 7-alpha-Hydroxylase - genetics | Aortic Diseases - genetics | Phenotype | Carotid Artery Diseases - enzymology | Carotid Artery Diseases - genetics | Mutation | Carotid Artery Diseases - prevention & control | AMP-Activated Protein Kinases - metabolism | Liver - enzymology | Transcription Factors - deficiency | DNA-Binding Proteins - deficiency | Heat Shock Transcription Factors | Atherosclerosis - enzymology | Transfection | Receptors, LDL - deficiency | Female | Aortic Diseases - enzymology | Aortic Diseases - pathology | Promoter Regions, Genetic | Receptors, LDL - genetics | Mice, Inbred C57BL | Cells, Cultured | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Animals | Cholesterol 7-alpha-Hydroxylase - metabolism | Adipose Tissue - enzymology | Carotid Arteries - pathology | Atherosclerosis - prevention & control | ORIGINAL ARTICLES
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2016, Volume 100, pp. S184 - S185
Coronary heart disease and diabetes are highly prevalent among obese populations due to aberrant dietary cholesterol metabolism. Here we investigated the... 
Physiological aspects | Heat shock proteins | Coronary heart disease | Cholesterol | Atherosclerosis | RNA | Liver | Cytochrome P-450 | Blood lipids | Gene expression | Heart diseases | Deoxycholic acid
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2016, Volume 311, Issue 5, pp. L941 - L955
We previously showed that coincident exposure to heat shock (HS; 42 degrees C for 2 h) and TNF-alpha synergistically induces apoptosis in mouse lung... 
Heat shock factor-1 | Epithelial wound healing | Idiopathic pulmonary fibrosis | Fibroblast growth factor-1 | Heat shock | HYPERTHERMIA | APOPTOSIS | PHYSIOLOGY | fibroblast growth factor-1 | FACTOR MESSENGER-RNA | heat shock | KAPPA-B-ALPHA | MECHANISMS | idiopathic pulmonary fibrosis | epithelial wound healing | PROTEIN INDUCTION | RESPIRATORY SYSTEM | heat shock factor-1 | CELL-MIGRATION | A549 CELLS | IDIOPATHIC PULMONARY-FIBROSIS | ACUTE EXACERBATION | Idiopathic Pulmonary Fibrosis - genetics | Lung Injury - pathology | Epithelial Cells - metabolism | Humans | Apoptosis - genetics | Fibroblast Growth Factor 1 - genetics | Promoter Regions, Genetic - genetics | Heat Shock Transcription Factors | DNA-Binding Proteins - metabolism | Polymerase Chain Reaction | Lung - metabolism | Wound Healing - genetics | Binding Sites | Cell Line | Epithelium - pathology | Lung - pathology | Epithelium - metabolism | Fibroblast Growth Factor 1 - metabolism | Gene Expression Regulation | Epithelial Cells - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | HSP70 Heat-Shock Proteins - metabolism | Heat-Shock Response - genetics | Transcription Factors - metabolism | Lung Injury - genetics | Animals | Mice | Heat shock proteins | Fibroblast growth factors | Lungs | Epithelial cells | Health aspects | Call for Papers
Journal Article