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1. Full Text TCF-1 controls ILC2 and NKp46+ RORγt+ innate lymphocyte differentiation and protection in intestinal inflammation
by Mielke, Lisa A and Groom, Joanna R and Rankin, Lucille C and Seillet, Cyril and Masson, Frederick and Putoczki, Tracy and Belz, Gabrielle T
Journal of Immunology, ISSN 0022-1767, 10/2013, Volume 191, Issue 8, pp. 4383 - 4391
Innate lymphocyte populations play a central role in conferring protective immunity at the mucosal frontier. In this study, we demonstrate that T cell factor 1... 
BET | EFFECTOR | TISSUE-INDUCER CELLS | IMMUNITY | ROR-GAMMA-T | TRANSCRIPTION FACTOR GATA3 | NOTCH | EOMESODERMIN | RECEPTOR | IMMUNOLOGY | EXPRESSION | T-Box Domain Proteins - biosynthesis | T-Box Domain Proteins - immunology | Interleukin-5 - biosynthesis | Intestines - immunology | Enterobacteriaceae Infections - immunology | Interleukin-13 - biosynthesis | Mucous Membrane - cytology | T-Lymphocytes - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Antigens, Ly - metabolism | Interleukins - biosynthesis | T Cell Transcription Factor 1 - metabolism | GATA3 Transcription Factor - metabolism | Inflammation - microbiology | Citrobacter rodentium - immunology | Lymphocyte Activation | Hepatocyte Nuclear Factor 1-alpha | Receptor, Notch2 - metabolism | Inflammation - immunology | Mice, Knockout | T Cell Transcription Factor 1 - genetics | Cell Differentiation - immunology | Animals | Intestines - microbiology | Mucous Membrane - immunology | Natural Cytotoxicity Triggering Receptor 1 - metabolism | Mice | Killer Cells, Natural - metabolism | Life Sciences
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2. Full Text Wnt signaling arrests effector T cell differentiation and generates CD8 + memory stem cells
by Zhong, Xiao-Song and Gattinoni, Luca and Restifo, Nicholas P and Wrzesinski, Claudia and Ji, Yun and Palmer, Douglas C and Paulos, Chrystal M and Garvin, Lindsay M and Yu, Zhiya and Boni, Andrea and Cassard, Lydie and Muranski, Pawel and Hinrichs, Christian S
Nature Medicine, ISSN 1078-8956, 07/2009, Volume 15, Issue 7, pp. 808 - 813
Self-renewing cell populations such as hematopoietic stem cells and memory B and T lymphocytes might be regulated by shared signaling pathways. The... 
TRANSCRIPTION FACTORS | LONG-TERM | MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | SELF-RENEWAL | PROLIFERATION | BETA-CATENIN | CANCER | CELL BIOLOGY | MAINTENANCE | IN-VIVO | ADOPTIVE IMMUNOTHERAPY | CD8-Positive T-Lymphocytes - cytology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Lymphocyte Activation | Mice, Inbred C57BL | Hepatocyte Nuclear Factor 1-alpha | Glycogen Synthase Kinase 3 beta | L-Selectin - analysis | CD8-Positive T-Lymphocytes - physiology | Hyaluronan Receptors - analysis | T Cell Transcription Factor 1 - physiology | Animals | Hematopoietic Stem Cells - physiology | Immunologic Memory | Signal Transduction - physiology | Cell Differentiation | Mice | Wnt Proteins - physiology | beta Catenin - physiology | Signal transduction | Biomedical research | Immunology | Cellular biology | Stem cells
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3. Full Text The role of albumin and PPAR‐α in differentiation‐dependent change of fatty acid profile during differentiation of mesenchymal stem cells to hepatocyte‐like cells
by Esmaeli, Shahnaz and Allameh, Abdolamir and Soleimani, Masoud and Rahbarizadeh, Fatemeh and Frouzandeh‐Moghadam, Mehdi
Cell Biochemistry and Function, ISSN 0263-6484, 07/2014, Volume 32, Issue 5, pp. 410 - 419
Differentiation of mesenchymal stem cells (MSCs) to hepatocyte‐like cells is associated with morphological and biological changes. In this study, the effect of... 
hepatocyte‐like cells | fatty acid profile | reactive oxygen species | PPAR‐α | Hepatocyte-like cells | Fatty acid profile | Reactive oxygen species | PPAR-α | OXIDATIVE STRESS | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | MOUSE | CELL BIOLOGY | ACTIVATED-RECEPTOR-ALPHA | METABOLISM | PPAR-alpha | GENE-EXPRESSION | hepatocyte-like cells | Reactive Oxygen Species - metabolism | Albumins - metabolism | Humans | Umbilical Cord - cytology | Cells, Cultured | Fatty Acids - analysis | PPAR alpha - genetics | Chromatography, Gas | Hepatocytes - metabolism | RNA, Messenger - metabolism | Hepatocytes - cytology | Mesenchymal Stromal Cells - cytology | Cell Differentiation | PPAR alpha - metabolism | Lipid Peroxidation | Albumins - genetics | Fatty Acids - metabolism
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4. Full Text Royalactin induces queen differentiation in honeybees
by Kamakura, Masaki
Nature, ISSN 0028-0836, 05/2011, Volume 473, Issue 7348, pp. 478 - 483
The honeybee (Apis mellifera) forms two female castes: the queen and the worker. This dimorphism depends not on genetic differences, but on ingestion of royal... 
APIS-MELLIFERA-L | CULTURED RAT HEPATOCYTES | PROTEIN | DROSOPHILA-MELANOGASTER | CASTE DIFFERENTIATION | BODY-SIZE | PROTHORACIC GLAND | MULTIDISCIPLINARY SCIENCES | ENHANCES PROLIFERATION | JUVENILE-HORMONE | REGULATES GROWTH | Temperature | Drosophila melanogaster - physiology | Longevity - drug effects | Glycoproteins - metabolism | Body Weight - drug effects | Receptor, Epidermal Growth Factor - deficiency | Bees - growth & development | Glycoproteins - pharmacology | Ovary - growth & development | Receptor, Epidermal Growth Factor - metabolism | RNA Interference | Time Factors | Larva - drug effects | Larva - growth & development | Fertility - drug effects | Female | Glycoproteins - deficiency | Ovary - drug effects | Protein Stability | Fat Body - metabolism | Fatty Acids - metabolism | Insect Proteins - metabolism | Glycoproteins - genetics | Body Size - drug effects | Fatty Acids - chemistry | Insect Proteins - pharmacology | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Juvenile Hormones - metabolism | Cell Size - drug effects | Drosophila melanogaster - cytology | Insect Proteins - genetics | Social Dominance | Drosophila melanogaster - drug effects | Bees - physiology | Phenotype | Animals | Signal Transduction - drug effects | Body Size - physiology | Bees - drug effects | Drosophila melanogaster - enzymology | Bees - genetics | Fat Body - drug effects | Caseins - pharmacology | Fatty Acids - pharmacology | Mitogen-Activated Protein Kinases - metabolism | Mass spectrometry | Bees
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5. Full Text Mutant IDH inhibits HNF-4α to block hepatocyte differentiation and promote biliary cancer
by Saha, Supriya K and Saha, Supriya K and Parachoniak, Christine A and Parachoniak, Christine A and Ghanta, Krishna S and Ghanta, Krishna S and Fitamant, Julien and Fitamant, Julien and Ross, Kenneth N and Ross, Kenneth N and Najem, Mortada S and Najem, Mortada S and Gurumurthy, Sushma and Gurumurthy, Sushma and Akbay, Esra A and Akbay, Esra A and Sia, Daniela and Sia, Daniela and Cornella, Helena and Cornella, Helena and Miltiadous, Oriana and Miltiadous, Oriana and Walesky, Chad and Walesky, Chad and Deshpande, Vikram and Deshpande, Vikram and Zhu, Andrew X and Zhu, Andrew X and Hezel, Aram F and Heze, Aram F and Yen, Katharine E and Yen, Katharine E and Straley, Kimberly S and Straley, Kimberly S and Travins, Jeremy and Travins, Jeremy and Popovici-Muller, Janeta and Popovici-Muller, Janeta and Gliser, Camelia and Gliser, Camelia and Ferrone, Cristina R and Ferrone, Cristina R and Apte, Udayan and Apte, Udayan and Llovet, Josep M and Llovet, Josep M and Wong, Kwok-Kin and Wong, Kwok-Kin and Ramaswamy, Sridhar and Ramaswamy, Sridhar and Bardeesy, Nabeel and Bardeesy, Nabeel
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7516, pp. 110 - 114
Mutations in isocitrate dehydrogenase 1 (IDH1) and IDH2 are among the most common genetic alterations in intrahepatic cholangiocarcinoma (IHCC), a deadly liver... 
PROGENITORS | INTRAHEPATIC CHOLANGIOCARCINOMA | HOMEOSTASIS | LIVER-REGENERATION | MULTIDISCIPLINARY SCIENCES | MUTATION | GROWTH | MICE | PROLIFERATION | EXPRESSION | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Hepatocyte Nuclear Factor 4 - antagonists & inhibitors | Humans | ras Proteins - metabolism | Hepatocytes - pathology | Male | Hepatocytes - metabolism | Bile Duct Neoplasms - enzymology | Cell Differentiation - genetics | Neoplasm Metastasis | Hepatocyte Nuclear Factor 4 - biosynthesis | Female | Bile Duct Neoplasms - genetics | Cell Lineage - genetics | Cholangiocarcinoma - enzymology | Disease Models, Animal | Cell Division - genetics | Proto-Oncogene Proteins - metabolism | Hepatocyte Nuclear Factor 4 - metabolism | Bile Ducts, Intrahepatic - pathology | Mutant Proteins - genetics | Isocitrate Dehydrogenase - genetics | Mice, Transgenic | Mutant Proteins - metabolism | Proto-Oncogene Proteins - genetics | Hepatocyte Nuclear Factor 4 - genetics | Mutation - genetics | Bile Ducts, Intrahepatic - enzymology | Cholangiocarcinoma - pathology | Animals | Cholangiocarcinoma - genetics | Stem Cells - pathology | Isocitrate Dehydrogenase - metabolism | Glutarates - metabolism | Mice | Bile Duct Neoplasms - pathology | Hepatocytes - enzymology | Index Medicus | Càncer | Diferenciació cel·lular | Gallbladder diseases | Metabolisme | Cell diferentiation | Metabolism | Malalties de la vesícula biliar | Cancer
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6. Full Text Activin a-induced differentiation of embryonic stem cells into endoderm and pancreatic progenitors-the influence of differentiation factors and culture conditions
by Sulzbacher, Sabine and Schroeder, Insa S and Truong, Thuy T and Wobus, Anna M
Stem Cell Reviews and Reports, ISSN 1550-8943, 06/2009, Volume 5, Issue 2, pp. 159 - 173
The differentiation of murine and human embryonic stem (ES) cells into pancreatic cell types has been shown by several methods including spontaneous... 
Human | Activin A | Pancreatic endocrine lineage | Mouse | In vitro differentiation | Insulin-producing beta cells | Embryonic stem cells | Definitive endoderm | MEDICINE, RESEARCH & EXPERIMENTAL | DIRECTED DIFFERENTIATION | BETA-CELLS | FIBROBLAST-GROWTH-FACTOR | CELL & TISSUE ENGINEERING | CELL BIOLOGY | RETINOIC ACID | INSULIN-PRODUCING CELLS | BONE MORPHOGENETIC PROTEIN-4 | IN-VITRO DIFFERENTIATION | ES CELLS | HEPATOCYTE GROWTH | Animals | Embryonic Stem Cells - cytology | Activins - pharmacology | Embryonic Stem Cells - drug effects | Insulin-Secreting Cells - drug effects | Cell Differentiation - drug effects | Humans | Pancreas - cytology | Pancreas - drug effects | Cell Culture Techniques - methods | Cellular biology | Stem cells
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7. Full Text Differentiation and Transplantation of Human Embryonic Stem Cell–Derived Hepatocytes
by Basma, Hesham and Soto–Gutiérrez, Alejandro and Yannam, Govardhana Rao and Liu, Liping and Ito, Ryotaro and Yamamoto, Toshiyuki and Ellis, Ewa and Carson, Steven D and Sato, Shintaro and Chen, Yong and Muirhead, David and Navarro–Álvarez, Nalu and Wong, Ronald J and Roy–Chowdhury, Jayanta and Platt, Jeffrey L and Mercer, David F and Miller, John D and Strom, Stephen C and Kobayashi, Naoya and Fox, Ira J
Gastroenterology, ISSN 0016-5085, 2009, Volume 136, Issue 3, pp. 990 - 999.e4
Background & Aims The ability to obtain unlimited numbers of human hepatocytes would improve the development of cell-based therapies for liver diseases,... 
Gastroenterology and Hepatology | LONG-TERM | VIRUS | DEFICIENT RAT | HUMAN LIVER | MICE | INDUCTION | DRUG-METABOLIZING-ENZYMES | GASTROENTEROLOGY & HEPATOLOGY | FUNCTIONAL HEPATIC CELLS | RECONSTITUTION | EXPRESSION | Cell Line | Gene Expression | Embryonic Stem Cells - cytology | Humans | Fibroblast Growth Factor 2 - pharmacology | Male | Embryonic Stem Cells - ultrastructure | Hepatocyte Growth Factor - pharmacology | Mice, SCID | Microscopy, Electron | Cell Culture Techniques - methods | Stem Cell Transplantation | Hepatocytes - cytology | Phenotype | Animals | Dexamethasone - pharmacology | Activins - pharmacology | Cell Differentiation - drug effects | Glucocorticoid-Induced TNFR-Related Protein - pharmacology | Embryonic Stem Cells - transplantation | Mice, Inbred NOD | Mice | Cell Differentiation - physiology | Medical colleges | Alpha 1-antitrypsin | Embryonic stem cells | Cytochrome P-450
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8. Full Text Differentiation and Persistence of Memory CD8 + T Cells Depend on T Cell Factor 1
by Zhou, Xinyuan and Yu, Shuyang and Zhao, Dong-Mei and Harty, John T and Badovinac, Vladimir P and Xue, Hai-Hui
Immunity, ISSN 1074-7613, 2010, Volume 33, Issue 2, pp. 229 - 240
T cell factor 1 (TCF-1) is a transcription factor known to act downstream of the canonical Wnt pathway and is essential for normal T cell development. However,... 
MOLIMMUNO | CELLIMMUNO | Molimmuno | Cellimmuno | EFFECTOR | PATHWAY | IN-VIVO | GENE-EXPRESSION | C-MYC | PROLIFERATION | THYMOCYTE SURVIVAL | IMMUNOLOGY | BETA-CATENIN | WNT | CUTTING EDGE | CD8-Positive T-Lymphocytes - cytology | Regulatory Sequences, Nucleic Acid | Listeria monocytogenes - immunology | Wnt Proteins - metabolism | Interleukin-15 - immunology | T Cell Transcription Factor 1 - deficiency | CD8-Positive T-Lymphocytes - metabolism | Transcription, Genetic | Cell Differentiation | T Cell Transcription Factor 1 - metabolism | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Hepatocyte Nuclear Factor 1-alpha | T-Box Domain Proteins - genetics | beta Catenin - metabolism | T-Box Domain Proteins - metabolism | Mice, Knockout | Phenotype | Animals | Protein Binding | T Cell Transcription Factor 1 - immunology | Immunologic Memory | Mice | CD8-Positive T-Lymphocytes - immunology | Flow cytometry | Listeria | Lymphocytes | Rodents | Colleges & universities | Infections | Kinases | Gene expression | Apoptosis
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9. Full Text GATA2 regulates dendritic cell differentiation
by Onodera, Koichi and Fujiwara, Tohru and Onishi, Yasushi and Itoh-Nakadai, Ari and Okitsu, Yoko and Fukuhara, Noriko and Ishizawa, Kenichi and Shimizu, Ritsuko and Yamamoto, Masayuki and Harigae, Hideo
Blood, ISSN 0006-4971, 2016, Volume 128, Issue 4, pp. 508 - 518
Dendritic cells (DCs) are critical immune response regulators; however, the mechanism of DC differentiation is not fully understood. Heterozygous germ line... 
HEMATOPOIETIC-CELLS | STIMULATING FACTOR-RECEPTOR | SPORADIC MONOCYTOPENIA | C/EBP-ALPHA | TRANSCRIPTION FACTOR GATA-2 | GENOME-WIDE ANALYSIS | GENE-EXPRESSION | MACROPHAGE PROLIFERATION | AUTOSOMAL-DOMINANT | MOUSE BONE-MARROW | HEMATOLOGY | GATA3 Transcription Factor - genetics | Myeloid Cells - cytology | Hepatocyte Nuclear Factor 1-alpha - immunology | Hepatocyte Nuclear Factor 1-alpha - genetics | Dendritic Cells - immunology | GATA3 Transcription Factor - immunology | GATA2 Transcription Factor - immunology | Mice, Knockout | Cell Differentiation - genetics | Cell Differentiation - immunology | Animals | T-Lymphocytes - cytology | Myeloid Cells - immunology | T-Lymphocytes - immunology | Dendritic Cells - cytology | Mice | GATA2 Transcription Factor - genetics | Hematopoiesis and Stem Cells
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10. Full Text Multi-lineage differentiation of mesenchymal stem cells – To Wnt, or not Wnt
by Visweswaran, Malini and Pohl, Sebastian and Arfuso, Frank and Newsholme, Philip and Dilley, Rodney and Pervaiz, Shazib and Dharmarajan, Arun
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 11/2015, Volume 68, pp. 139 - 147
Mesenchymal stem cells (MSCs) are multipotent precursor cells originating from several adult connective tissues. MSCs possess the ability to self-renew and... 
Reactive oxygen species | Differentiation | Wnt | Cellular fate | Mesenchymal stem cells | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | HEPATIC PROGENITOR CELLS | OSTEOGENIC DIFFERENTIATION | CHONDROGENIC DIFFERENTIATION | BETA-CATENIN | CELL BIOLOGY | HUMAN ADIPOSE-TISSUE | RECEPTOR-RELATED PROTEIN-5 | ADIPOCYTE DIFFERENTIATION | FRIZZLED-RELATED PROTEIN-1 | WNT/BETA-CATENIN PATHWAY | Chondrocytes - cytology | Reactive Oxygen Species - metabolism | Humans | Adipocytes - cytology | Neurons - cytology | Hepatocytes - metabolism | Wnt Proteins - metabolism | Cell Differentiation - genetics | Hepatocytes - cytology | Mesenchymal Stromal Cells - cytology | Wnt Proteins - genetics | Adult | Neurons - metabolism | Cell Lineage - genetics | Chondrocytes - metabolism | Wnt Signaling Pathway | Myocytes, Cardiac - cytology | Osteocytes - metabolism | Osteocytes - cytology | Gene Expression Regulation | Muscle Cells - metabolism | Mesenchymal Stromal Cells - metabolism | beta Catenin - metabolism | beta Catenin - genetics | Muscle Cells - cytology | Adipocytes - metabolism | Myocytes, Cardiac - metabolism | Proteins | Stem cell research | Medical colleges | Therapeutics | Stem cells | Physiological aspects | Cellular signal transduction | Homeopathy | Materia medica and therapeutics | Oxidases | Cysteine | Glycogen | Superoxide | Cyclic adenylic acid | T cells | Lithium compounds | Synthesis | Casein | Nitric oxide | Bone morphogenetic proteins | Peroxides | Protein kinases | Protein binding
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