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index medicus (52) 52
hgf/c-met (46) 46
c-met (43) 43
humans (38) 38
hgf (29) 29
proto-oncogene proteins c-met - metabolism (27) 27
oncology (26) 26
animals (24) 24
cancer (22) 22
expression (22) 22
metastasis (20) 20
hepatocyte growth-factor (19) 19
hepatocyte growth factor - metabolism (18) 18
kinases (17) 17
cell biology (15) 15
cell line, tumor (15) 15
hepatocyte growth factor (15) 15
tumors (15) 15
angiogenesis (12) 12
mice (12) 12
cancer therapies (11) 11
male (11) 11
mutation (11) 11
signal transduction (11) 11
female (10) 10
gene expression (10) 10
proto-oncogene proteins c-met - genetics (10) 10
apoptosis (9) 9
growth factors (9) 9
medical prognosis (9) 9
biochemistry & molecular biology (8) 8
c-met protein (8) 8
growth (8) 8
hepatocellular carcinoma (8) 8
hgf/c-met signaling pathway (8) 8
invasion (8) 8
met (8) 8
proteins (8) 8
signal transduction - drug effects (8) 8
cabozantinib (7) 7
carcinoma, renal cell - drug therapy (7) 7
cell proliferation - drug effects (7) 7
hepatocellular-carcinoma (7) 7
hgf/c-met signaling (7) 7
lung cancer (7) 7
proliferation (7) 7
renal cell carcinoma (7) 7
tyrosine (7) 7
vascular endothelial growth factor (7) 7
activation (6) 6
antineoplastic agents - pharmacology (6) 6
antineoplastic agents - therapeutic use (6) 6
cells (6) 6
everolimus (6) 6
hepatocyte growth factor - genetics (6) 6
kidney neoplasms - drug therapy (6) 6
liver neoplasms - genetics (6) 6
migration (6) 6
phosphorylation (6) 6
protein kinase inhibitors - therapeutic use (6) 6
research (6) 6
signaling pathway (6) 6
stem cells (6) 6
targeted therapy (6) 6
vegf (6) 6
anilides - therapeutic use (5) 5
carcinoma (5) 5
carcinoma, hepatocellular - genetics (5) 5
care and treatment (5) 5
cell cycle (5) 5
cell movement - drug effects (5) 5
cross-talk (5) 5
deoxyribonucleic acid--dna (5) 5
drug resistance (5) 5
gene amplification (5) 5
immunohistochemistry (5) 5
inhibitor (5) 5
kidney cancer (5) 5
ligands (5) 5
liver (5) 5
liver cancer (5) 5
lung neoplasms (5) 5
neoplasm invasiveness (5) 5
patients (5) 5
pi3k (5) 5
protein-tyrosine kinase (5) 5
pyridines - therapeutic use (5) 5
receptor (5) 5
sunitinib (5) 5
therapy (5) 5
tyrosine kinase (5) 5
xenograft model antitumor assays (5) 5
akt (4) 4
analysis (4) 4
antineoplastic agents - administration & dosage (4) 4
beta-catenin (4) 4
breast cancer (4) 4
carcinoma, non-small-cell lung - drug therapy (4) 4
carcinoma, renal cell (4) 4
carcinoma, renal cell - metabolism (4) 4
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1. Full Text Cabozantinib versus Everolimus in Advanced Renal-Cell Carcinoma
by Choueiri, Toni K and Escudier, Bernard and Powles, Thomas and Mainwaring, Paul N and Rini, Brian I and Donskov, Frede and Hammers, Hans and Hutson, Thomas E and Lee, Jae-Lyun and Peltola, Katriina and Roth, Bruce J and Bjarnason, Georg A and Géczi, Lajos and Keam, Bhumsuk and Maroto, Pablo and Heng, Daniel Y.C and Schmidinger, Manuela and Kantoff, Philip W and Borgman-Hagey, Anne and Hessel, Colin and Scheffold, Christian and Schwab, Gisela M and Tannir, Nizar M and Motzer, Robert J and METEOR Investigators
The New England Journal of Medicine, ISSN 0028-4793, 11/2015, Volume 373, Issue 19, pp. 1814 - 1823
In a phase 3 clinical trial involving previously treated patients with advanced renal-cell carcinoma, progression-free survival was significantly longer with... 
PHASE-III TRIAL | MEDICINE, GENERAL & INTERNAL | XL184 | METASTASIS | PROGNOSTIC-FACTORS | C-MET | SUNITINIB | HGF/C-MET | SORAFENIB | TUMORS | NEPHRECTOMY | Anilides - therapeutic use | Sirolimus - analogs & derivatives | Sirolimus - therapeutic use | Anilides - adverse effects | Humans | Middle Aged | Male | Antineoplastic Agents - therapeutic use | Kidney Neoplasms - mortality | Carcinoma, Renal Cell - mortality | Disease-Free Survival | Pyridines - adverse effects | Antineoplastic Agents - adverse effects | Survival Analysis | Aged, 80 and over | Quality of Life | Adult | Female | Aged | Carcinoma, Renal Cell - drug therapy | Kidney Neoplasms - drug therapy | Everolimus | Sirolimus - adverse effects | Pyridines - therapeutic use | Treatment outcome | Care and treatment | Carcinoma, Renal cell | Analysis | Tyrosine | Inhibitor drugs | Metastasis | Drug resistance | Drug development | Patients | Vascular endothelial growth factor receptors | Metastases | Axl protein | c-Met protein | Chemotherapy | Medical prognosis | Kidney cancer | Vascular endothelial growth factor | Protein-tyrosine kinase | Clear cell-type renal cell carcinoma | Tumors | Index Medicus | Abridged Index Medicus
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2. The Progress of Antibody-Based Anti-Tumor Drugs Targeting HGF/c-Met Signaling Pathway
by Guo, Bin-Sheng and Xu, Han-Mei and Qu, Yu
Chinese Journal of Pharmaceutical Biotechnology, ISSN 1005-8915, 12/2017, Volume 24, Issue 6, pp. 534 - 540
Antibody | Anti-cancer drugs | HGF/c-Met signaling pathway | Cancer
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3. Full Text Paracrine HGF/c-MET enhances the stem cell-like potential and glycolysis of pancreatic cancer cells via activation of YAP/HIF-1α
by Yan, Bin and Jiang, Zhengdong and Cheng, Liang and Chen, Ke and Zhou, Cancan and Sun, Liankang and Qian, Weikun and Li, Jie and Cao, Junyu and Xu, Qinhong and Ma, Qingyong and Lei, Jianjun
Experimental Cell Research, ISSN 0014-4827, 10/2018, Volume 371, Issue 1, pp. 63 - 71
Pancreatic stellate cells (PSCs), a pivotal component of the tumor microenvironment, contribute to tumor growth and metastasis. PSC-derived factors are... 
Glycolysis | Pancreatic stellate cells | HGF/c-MET | YAP | HIF-1α | Cancer stem cells | HYPOXIA-INDUCIBLE FACTOR | HIF-1 alpha | FIBROBLASTS | CELL BIOLOGY | INVASION | ONCOLOGY | PATHWAY | YAP/TAZ | INCREASE | C-MET | GROWTH | RESISTANCE | Index Medicus
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4. Research progress of HGF/c-Met-related targeted therapy of gastric cancer
by Zhu, Xue-Ru and Zheng, Lei-Zhen
Journal of Shanghai Jiaotong University (Medical Science), ISSN 1674-8115, 01/2016, Volume 36, Issue 1, pp. 133 - 137
Targeted therapy | HGF/c-Met | Gastric cancer
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5. Full Text ROLE OF NEUREGULINS AND HEPATOCYTE GROWTH FACTOR IN THE DEVELOPMENT OF CASTRATION-RESISTANT PROSTATE CANCER
by L. V. Spirina and E. A. Usynin and E. M. Slonimskaya and I. V. Kondakova and A. K. Gorbunov
Sibirskij Onkologičeskij Žurnal, ISSN 1814-4861, 02/2016, Volume 1, Issue 4, pp. 80 - 86
The development of castration-resistant prostate cancer is an important problem of modern oncology. The review is focused on the system of growth factors –... 
prostate cancer, neuregulins, HGF, c-Met, ErbB
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6. Full Text Adipose-derived mesenchymal stem cells (ASCs) may favour breast cancer recurrence via HGF/c-Met signaling
by Eterno, Vincenzo and Zambelli, Alberto and Pavesi, Lorenzo and Villani, Laura and Zanini, Vittorio and Petrolo, Gianfranco and Manera, Stefania and Tuscano, Antonella and Amato, Angela
Oncotarget, ISSN 1949-2553, 2014, Volume 5, Issue 3, pp. 613 - 633
Adipose tissue is a reservoir of Mesenchymal Stem Cells (Adipose-derived Mesenchymal Stem Cells, ASCs), endowed with regenerative properties. Fat graft was... 
Neoangiogenesis | Adipose-derived mesenchymal stem cells (ASCs) | Breast cancer | Microenvironment | HGF/c-met crosstalk | Index Medicus
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7. Full Text Differential expression of c-Met between primary and metastatic sites in clear-cell renal cell carcinoma and its association with PD-L1 expression
by Lalani, Aly-Khan A and Gray, Kathryn P and Albiges, Laurence and Callea, Marcella and Pignon, Jean-Christophe and Pal, Soumitro and Gupta, Mamta and Bhatt, Rupal S and McDermott, David F and Atkins, Michael B and Woude, G.F. Vande and Harshman, Lauren C and Choueiri, Toni K and Signoretti, Sabina
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 61, pp. 103428 - 103436
In preclinical models, c-Met promotes survival of renal cancer cells through the regulation of programmed death-ligand 1 (PD-L1). However, this relationship in... 
PD-L1 | Metastasis | Renal cell carcinoma | C-Met | Primary | CABOZANTINIB | EVEROLIMUS | VEGF | HEPATOCYTE GROWTH-FACTOR | HGF/C-MET | CANCER | CELL BIOLOGY | renal cell carcinoma | metastasis | SIGNALING PATHWAY | SUNITINIB | PROGNOSTIC MARKER | c-Met | primary
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8. HGF stimulates proliferation through the HGF/c-Metpathway in nasopharyngeal carcinoma cells
by Sun, Rui and Zhang, Qing and Guo, Ling and Chen, Ming-Yuan and Sun, Ying and Cao, Brian and Sun, Jian
Oncology Letters, ISSN 1792-1074, 05/2012, Volume 3, Issue 5, pp. 1124 - 1128
Nasopharyngeal carcinoma | HGF/c-Met | Proliferation | Migration
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9. Full Text Exosomes serve as nanoparticles to suppress tumor growth and angiogenesis in gastric cancer by delivering hepatocyte growth factor siRNA
by Zhang, Haiyang and Wang, Yi and Bai, Ming and Wang, Junyi and Zhu, Kegan and Liu, Rui and Ge, Shaohua and Li, JiaLu and Ning, Tao and Deng, Ting and Fan, Qian and Li, Hongli and Sun, Wu and Ying, Guoguang and Ba, Yi
Cancer Science, ISSN 1347-9032, 03/2018, Volume 109, Issue 3, pp. 629 - 641
Exosomes derived from cells have been found to mediate signal transduction between cells and to act as efficient carriers to deliver drugs and small RNA .... 
RNA | angiogenesis | exosomes | hepatocyte growth factor | tumor therapy | siRNA | ACTIVATION | DRUG-DELIVERY | VEGF | MICROVESICLES | HGF/C-MET | CELL MIGRATION | MULTIVESICULAR BODIES | ONCOLOGY | MEMBRANE-VESICLES | C-MET | CARCINOMA | Stomach Neoplasms - genetics | Coculture Techniques | Humans | RNA, Small Interfering - pharmacology | Exosomes - genetics | Stomach Neoplasms - drug therapy | Nanoparticles | Up-Regulation - drug effects | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Animals | Hepatocyte Growth Factor - antagonists & inhibitors | Cell Line, Tumor | Hepatocyte Growth Factor - genetics | Cell Proliferation - drug effects | Mice | Gene Expression Regulation, Neoplastic - drug effects | RNA, Small Interfering - administration & dosage | Cellular signal transduction | Neovascularization | Stomach cancer | Analysis | Cancer | Index Medicus | Original
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10. Full Text Mechanism of Chinese herbal formula QHF against breast cancer MCF-7 cells invasion and migration
by Hu, Xiang-yang and Hu, Wei and Liu, Dan and Jiao, Liang-bo and Zhang, Wen and Gan, Shen-tong and Wang, Xin-yi and Chen, Tao
Chinese Herbal Medicines, ISSN 1674-6384, 10/2018, Volume 10, Issue 4, pp. 405 - 410
To investigate the effects of Chinese herbal formula Qinghuofu (QHF) on the migration and invasion of breast cancer MCF-7 cells and its possible molecular... 
migration | breast cancer | invasion | formula QHF | HGF/c-Met signaling pathway
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11. Full Text Hepatocyte growth factor inhibition: A novel therapeutic approach in pancreatic cancer
by Pothula, Srinivasa P and Xu, Zhihong and Goldstein, David and Biankin, Andrew V and Pirola, Romano C and Wilson, Jeremy S and Apte, Minoti V
British Journal of Cancer, ISSN 0007-0920, 02/2016, Volume 114, Issue 3, pp. 269 - 280
Background: Pancreatic stellate cells (PSCs, which produce the stroma of pancreatic cancer (PC)) interact with cancer cells to facilitate PC growth. A... 
pancreatic stellate cells | stromal-tumour interactions | orthotopic model | pancreatic cancer | HGF inhibition | HGF/c-MET pathway | STEM-CELLS | CARCINOMA CELLS | DUCTAL ADENOCARCINOMA | PHOSPHATIDYLINOSITOL 3-KINASE | STELLATE CELLS | MONOCLONAL-ANTIBODY | PHASE-III TRIAL | EPITHELIAL-MESENCHYMAL TRANSITION | ONCOLOGY | SIGNALING PATHWAY | C-MET | Proto-Oncogene Proteins c-met - metabolism | Neoplasm Transplantation | Pancreatic Neoplasms - metabolism | Hepatocyte Growth Factor - secretion | Apoptosis - drug effects | Antibodies, Monoclonal - pharmacology | Humans | Pancreatic Stellate Cells - drug effects | Carcinoma, Pancreatic Ductal - metabolism | Deoxycytidine - pharmacology | Epithelial-Mesenchymal Transition - drug effects | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Animals | Hepatocyte Growth Factor - antagonists & inhibitors | Mice, Nude | Pancreatic Stellate Cells - secretion | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Mice | Neovascularization, Pathologic - metabolism | In Vitro Techniques | Deoxycytidine - analogs & derivatives | Index Medicus | Translational Therapeutics | c-MET pathway | HGF
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12. Full Text c‐Met Signaling Is Essential for Mouse Adult Liver Progenitor Cells Expansion After Transforming Growth Factor‐β‐Induced Epithelial–Mesenchymal Transition and Regulates Cell Phenotypic Switch
by Almalé, Laura and García‐Álvaro, María and Martínez‐Palacián, Adoración and García‐Bravo, María and Lazcanoiturburu, Nerea and Addante, Annalisa and Roncero, Cesáreo and Sanz, Julián and López, María and Bragado, Paloma and Mikulits, Wolfgang and Factor, Valentina M and Thorgeirsson, Snorri S and Casal, J. Ignacio and Segovia, José‐Carlos and Rial, Eduardo and Fabregat, Isabel and Herrera, Blanca and Sánchez, Aránzazu
STEM CELLS, ISSN 1066-5099, 08/2019, Volume 37, Issue 8, pp. 1108 - 1118
Adult hepatic progenitor cells (HPCs)/oval cells are bipotential progenitors that participate in liver repair responses upon chronic injury. Recent findings... 
TGF‐β | Oval cell | HGF/c‐Met | EMT | HGF/c-Met | TGF-β | FIBROSIS | APOPTOSIS | HGF | REGENERATION | FETAL HEPATOCYTES | DIFFERENTIATION FACTORS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | TGF-beta | IN-VITRO | HEPATIC STELLATE CELLS | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | c-Met | EXTRACELLULAR-MATRIX | OVAL CELLS | HEMATOLOGY | Oxidative stress | Profiling | Mesenchyme | Hepatocyte growth factor | Syngeneic grafts | Liver | Transforming growth factor | Carbon tetrachloride | Stem cell transplantation | Transplantation | Progenitor cells | c-Met protein | Regeneration | Signaling | Allografts | Cell fate | Counterbalances | Hepatocytes | Stem cells | Signal processing | Growth factors | Niches | Apoptosis | Index Medicus
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13. Full Text Targeting the HGF/c-MET pathway: Stromal remodelling in pancreatic cancer
by Pothula, Srinivasa P and Xu, Zhihong and Goldstein, David and Merrett, Neil and Pirola, Romano C and Wilson, Jeremy S and Apte, Minoti V
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 44, pp. 76722 - 76739
Stromal-tumor interactions in pancreatic cancer (PC) impact on treatment outcomes. Pancreatic stellate cells (PSCs) produce the collagenous stroma of PC and... 
Pancreatic stellate cells | Orthotopic model | Pancreatic cancer | HGF/c-MET pathway | Stromal-tumor interactions | pancreatic stellate cells | stromal-tumor interactions | STEM-CELLS | CARCINOMA CELLS | DUCTAL ADENOCARCINOMA | HEPATOCYTE GROWTH-FACTOR | STELLATE CELLS | SMALL-MOLECULE INHIBITOR | CELL BIOLOGY | RECEPTOR-MEDIATED MIGRATION | pancreatic cancer | orthotopic model | C-MET | IN-VIVO | TUMOR-GROWTH
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14. Full Text MicroRNA‐198 inhibition of HGF/c‐MET signaling pathway overcomes resistance to radiotherapy and induces apoptosis in human non–small‐cell lung cancer
by Zhu, Lin and Xue, Feng and Xu, Xiangying and Xu, Jianyu and Hu, Songliu and Liu, Shanshan and Cui, Ying and Gao, Chunzi
Journal of Cellular Biochemistry, ISSN 0730-2312, 09/2018, Volume 119, Issue 9, pp. 7873 - 7886
Non–small‐cell lung cancer (NSCLC) is the most common cause of death from cancer worldwide. MicroRNAs (miRNAs) are a group of important regulators in NSCLC,... 
HGF/c‐MET signaling pathway | microRNA‐198 | non–small‐cell lung cancer | radioresistance | lung adenocarcinoma | microRNA-198 | non–small-cell lung cancer | HGF/c-MET signaling pathway | HGF | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | HEPATOCELLULAR-CARCINOMA | INVASION | MIR-198 | GENE | c-MET signaling pathway | C-MET | GROWTH | non-small-cell lung cancer | MicroRNA | Radiotherapy | Lung cancer, Non-small cell | Apoptosis | Cell proliferation | Phosphorylation | Regulators | Hepatocyte growth factor | Lung cancer | Xenotransplantation | MiRNA | Irradiated | Radiation therapy | Kinases | Ribonucleic acid--RNA | c-Met protein | Signal transduction | Molecular modelling | Xenografts | Inhibition | Growth factors | Cell migration | Cancer | Tumors
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15. The Challenges of Modeling Drug Resistance to Antiangiogenic Therapy
by Mastri, M and Rosario, S and Tracz, A and Frink, RE and Brekken, RA and Ebos, JML
CURRENT DRUG TARGETS, ISSN 1389-4501, 2016, Volume 17, Issue 15, pp. 1747 - 1754
Drug resistance remains an ongoing challenge for the majority of patients treated with inhibitors of the vascular endothelial growth factor (VEGF) pathway, a... 
mouse models | CANCER CELLS | VEGF | syngeneic | ACQUIRED-RESISTANCE | HGF/C-MET | MESENCHYMAL TRANSITION | RENAL-CELL CARCINOMA | SPONTANEOUS METASTASIS | TUMOR RESISTANCE | metastasis | orthotopic | PHARMACOLOGY & PHARMACY | drug resistance | GEMMs | ANTI-VEGF THERAPY | MALIGNANT PROGRESSION
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16. Full Text Pancreatic stellate cells increase pancreatic cancer cells invasion through the hepatocyte growth factor /c-Met/survivin regulated by P53/P21
by Yang, Xiao-Peng and Liu, Shang-Long and Xu, Jian-Fei and Cao, Shou-Gen and Li, Yu and Zhou, Yan-Bing
Experimental Cell Research, ISSN 0014-4827, 08/2017, Volume 357, Issue 1, pp. 79 - 87
Pancreatic stellate cells (PSCs) are a key cellular component of the pancreatic tumor microenvironment and are considered to contribute to tumor invasion and... 
Pancreatic stellate cells | HGF/c-Met | Survivin | P21 | P53 | SURVIVIN EXPRESSION | METASTASIS | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | GENOMIC INSTABILITY | ONCOLOGY | MET SIGNALING PATHWAY | NEGATIVE REGULATION | CHEMORESISTANCE | MUTATIONS | TUMOR MICROENVIRONMENT | Proto-Oncogene Proteins c-met - metabolism | Pancreatic Neoplasms - metabolism | Neoplasm Invasiveness | Humans | Pancreatic Neoplasms - pathology | Tumor Suppressor Protein p53 - metabolism | Tumor Microenvironment | Cell Movement - physiology | Hepatocyte Growth Factor - metabolism | Cell Line, Tumor | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Inhibitor of Apoptosis Proteins - metabolism | Pancreatic Stellate Cells - metabolism | Medical colleges | Development and progression | Metastasis | Tumor proteins | Pancreatic cancer | Index Medicus
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17. Full Text Correlation between HGF/c-Met and Notch1 signaling pathways in human gastric cancer cells
by Huang, Kuo-Hung and Sung, I-Cheng and Fang, Wen-Liang and Chi, Chin-Wen and Yeh, Tien-Shun and Lee, Hsin-Chen and Yin, Pen-Hui and Li, Anna Fen-Yau and Wu, Chew-Wun and Shyr, Yi-Ming and Yang, Muh-Hwa
Oncology Reports, ISSN 1021-335X, 07/2018, Volume 40, Issue 1, pp. 294 - 302
In recent decades, research concerning gastric carcinogenesis has rapidly progressed. It is evident that hepatocyte growth factor (HGF) is clinically related... 
HGF/c-Met | Notch pathway | Jagged1 | Hepatocyte growth factor | Gastric cancer | gastric cancer | HGF | TYROSINE KINASE | HEPATOCYTE GROWTH-FACTOR | CYCLOOXYGENASE-2 PROTEIN | MAPK ACTIVATION | INVASIVE GROWTH | ONCOLOGY | hepatocyte growth factor | THERAPEUTIC TARGET | c-Met | EXPRESSION | CARCINOMA | PROGRESSION | Lymphatic system | Laboratories | Mortality | Metastasis | Kinases | Cancer therapies | Stomach cancer | Cell adhesion & migration | Proteins | Signal transduction | Angiogenesis | Surgery | Ligands | Growth factors | Tumors | Index Medicus
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18. Cabozantinib in patients with hepatocellular carcinoma failing previous treatment with sorafenib
by Personeni, N and Pressiani, T and Rimassa, L
FUTURE ONCOLOGY, ISSN 1479-6694, 07/2019, Volume 15, Issue 21, pp. 2449 - 2462
Over the past 10 years, sorafenib has been the only systemic agent approved for the treatment of patients with unresectable hepatocellular carcinoma. Recently,... 
VEGFR2 | PLACEBO | XL184 | second-line | metastatic | advanced | angiogenesis | TYROSINE KINASE | HGF/C-MET | immune checkpoint inhibitors | cabozantinib | ONCOLOGY | AXL | C-MET | GROWTH | DOUBLE-BLIND | 2ND-LINE TREATMENT | INHIBITOR | MET | third-line | hepatocellular carcinoma
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19. Full Text MACC1 Suppresses Cell Apoptosis in Hepatocellular Carcinoma by Targeting the HGF/c-MET/AKT Pathway
by Yao, Yingmin and Dou, Chanwei and Lu, Zhongtang and Zheng, Xin and Liu, Qingguang
Cellular Physiology and Biochemistry, ISSN 1015-8987, 02/2015, Volume 35, Issue 3, pp. 983 - 996
Background & Aims: To investigate the expression and prognostic value of MACC1 in patients with HCC and identify the mechanism by which MACC1 inhibits HCC cell... 
Original Paper | Hepatocellular carcinoma | AKT | HGF/c-MET signaling | MACC1 | Apoptosis | MIGRATION | PREDICTS | PHYSIOLOGY | METASTASIS | MARKER | PROLIFERATION | CANCER | INVASION | MET | Oncogene Protein v-akt - biosynthesis | Caspase 9 - metabolism | Humans | Gene Expression Regulation, Neoplastic | Heterografts | Forkhead Transcription Factors - metabolism | Carcinoma, Hepatocellular - genetics | Oncogene Protein v-akt - genetics | Hepatocyte Growth Factor - genetics | bcl-Associated Death Protein - metabolism | Liver Neoplasms - pathology | Hepatocyte Growth Factor - biosynthesis | Cell Proliferation - genetics | Liver Neoplasms - genetics | Signal Transduction | Transcription Factors - biosynthesis | Transcription Factors - genetics | Proto-Oncogene Proteins c-met - genetics | Phosphatidylinositol 3-Kinases - genetics | Animals | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Mice | Forkhead Box Protein O3 | RNA, Messenger | Index Medicus
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