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Molecular Cell, ISSN 1097-2765, 2006, Volume 24, Issue 1, pp. 77 - 89
Journal Article
Experimental Cell Research, ISSN 0014-4827, 04/2011, Volume 317, Issue 7, pp. 1060 - 1070
Homeodomain-interacting protein kinase 2 (HIPK2) is a key regulator of various transcription factors including p53 and CtBP in the DNA damage signaling... 
SUMO-interaction motif | PML | p53 phosphorylation | HIPK2 | P53 phosphorylation | TRANSCRIPTION FACTORS | HOMEODOMAIN | PROTEIN KINASE-2 | HOMEODOMAIN-INTERACTING-PROTEIN-KINASE-2 HIPK2 | PHOSPHORYLATION | DNA-DAMAGE | RAR-ALPHA | CELL BIOLOGY | PROTEIN-KINASE-2 | ONCOLOGY | COVALENT MODIFICATION | ACUTE PROMYELOCYTIC LEUKEMIA | Phosphorylation | Humans | Molecular Sequence Data | Recombinant Fusion Proteins - metabolism | Tumor Suppressor Protein p53 - genetics | Cell Nucleus - metabolism | Protein Isoforms - metabolism | SUMO-1 Protein - genetics | Tumor Suppressor Proteins - genetics | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Amino Acid Sequence | Cell Line | Tumor Suppressor Proteins - metabolism | Mutagenesis, Site-Directed | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Transcription Factors - metabolism | Carrier Proteins - genetics | Sequence Alignment | Carrier Proteins - metabolism | Recombinant Fusion Proteins - genetics | Signal Transduction - physiology | SUMO-1 Protein - metabolism | Apoptosis - physiology | Promyelocytic Leukemia Protein | Protein Isoforms - genetics | Tumor proteins | Analysis | Proteins | Cellular biology | Kinases | DNA damage | Apoptosis | APOPTOSIS | MUTANTS | COLONY FORMATION | CELL PROLIFERATION | MASS SPECTROSCOPY | 60 APPLIED LIFE SCIENCES | DNA DAMAGES
Journal Article
Oncogene, ISSN 0950-9232, 08/2014, Volume 34, Issue 26, pp. 3463 - 3473
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2014, Volume 111, Issue 43, pp. 15532 - 15537
X-linked inhibitor of apoptosis (XIAP)-associated factor 1 (XAF1) is a tumor suppressor that is frequently inactivated in many human cancers. However, the... 
Phosphorylation | HCT116 cells | Ubiquitins | Control loops | Cell lines | Gene expression regulation | Mathematical functions | Tumors | Apoptosis | Cancer | XAF1 | ZNF313 | Siah2 | HIPK2 | P53 | CANCER-CELLS | DOMAIN | HYPERMETHYLATION | X-LINKED INHIBITOR | MULTIDISCIPLINARY SCIENCES | INTERACTING PROTEIN KINASE-2 | IDENTIFICATION | p53 | GENE | XIAP-ASSOCIATED FACTOR-1 | PROSTATE-CANCER | EXPRESSION | Neoplasms - metabolism | Apoptosis - drug effects | Humans | Antineoplastic Agents - therapeutic use | Intracellular Signaling Peptides and Proteins - metabolism | Neoplasm Proteins - metabolism | Proteolysis - drug effects | Protein Isoforms - metabolism | Ubiquitination - drug effects | Protein Binding - drug effects | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Antineoplastic Agents - pharmacology | Phosphorylation - drug effects | Proto-Oncogene Proteins c-mdm2 - metabolism | Protein-Serine-Threonine Kinases - metabolism | Protein Structure, Tertiary | HCT116 Cells | Tumor Suppressor Protein p53 - metabolism | Ubiquitin-Protein Ligases - metabolism | Neoplasm Proteins - chemistry | Nuclear Proteins - metabolism | Enzyme Activation - drug effects | Phosphoserine - metabolism | Remission Induction | Neoplasms - drug therapy | Carrier Proteins - metabolism | Signal Transduction - drug effects | Intracellular Signaling Peptides and Proteins - chemistry | Models, Biological | Protein Stability - drug effects | Feedback, Physiological - drug effects | Tumor Suppressor Protein p53 - chemistry | Neoplasms - pathology | Physiological aspects | Physiological research | Research | Zinc finger proteins | Tumor proteins | Protein kinases | Biological Sciences
Journal Article
Journal of Virology, ISSN 0022-538X, 03/2004, Volume 78, Issue 6, pp. 2984 - 2993
Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
NUCLEAR-BODIES | VIROLOGY | PROTEIN KINASE-2 HIPK2 | PHOSPHORYLATION | INFECTED-CELLS | VIRAL-DNA REPLICATION | BINDING PROTEIN | P53 ACTIVITY | EXPRESSION | SERINE/THREONINE KINASE | GENE-PRODUCT | Green Fluorescent Proteins | Phosphorylation | Humans | Molecular Sequence Data | Recombinant Fusion Proteins - metabolism | Viral Proteins - metabolism | RNA-Binding Proteins - pharmacology | Carrier Proteins - chemistry | Viral Proteins - pharmacology | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Herpesvirus 1, Human - metabolism | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Nuclear Proteins - chemistry | Sequence Analysis, DNA | Cell Division - drug effects | Carrier Proteins - genetics | Two-Hybrid System Techniques | Adaptor Proteins, Signal Transducing | Carrier Proteins - metabolism | Recombinant Fusion Proteins - genetics | Luminescent Proteins - genetics | Protein-Serine-Threonine Kinases - chemistry | HeLa Cells - cytology | Herpesvirus 1, Human - pathogenicity | RNA-Binding Proteins - metabolism | Luminescent Proteins - metabolism | Protein-Serine-Threonine Kinases | Viral Proteins | RNA-Binding Proteins | Cellular Biology | Recombinant Fusion Proteins | Herpesvirus 1, Human | Nuclear Proteins | Life Sciences | Cell Division | Hela Cells | Luminescent Proteins | Carrier Proteins | Virus-Cell Interactions
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 4, p. e10171
Background: Homeodomain interacting protein kinase 2 (HIPK2) is an evolutionary conserved serine/threonine whose activity is fundamental in maintaining... 
DISEASE PATIENTS | APOPTOSIS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | DOXORUBICIN | HYPOXIC RESPONSE | PRECURSOR PROTEIN | CONFORMATION | SECRETION | FIBROBLASTS | HIPK2 | Carrier Proteins - physiology | Promoter Regions, Genetic | Cell Survival | Protein-Serine-Threonine Kinases - physiology | Humans | Cells, Cultured | DNA - metabolism | Protein Folding | Alzheimer Disease - pathology | Carrier Proteins - metabolism | Protein Binding | Tumor Suppressor Protein p53 - chemistry | Amyloid beta-Protein Precursor | Protein-Serine-Threonine Kinases - metabolism | Advertising executives | Chromatin | Analysis | Amyloid beta-protein | Research | Tumor proteins | DNA-ligand interactions | Protein kinases | Alzheimer's disease | Metallothionein | Neurosciences | Phosphorylation | Immunoprecipitation | Peptides | Laboratories | Pathogenesis | p53 Protein | DNA damage | Oncology | Biology | Kinases | Tissues | HIPK2 protein | Recruitment | Homeobox | Proteins | Protein folding | Evolutionary conservation | Rodents | Cell cycle | Fibroblasts | Physiology | Deoxyribonucleic acid--DNA | Binding | Dopamine | Cell survival | Threonine | Neurodegenerative diseases | Neurons | Pharmacology | Gene expression | Patients | Survival | Promoters | Zinc | Amyloid precursor protein | Hypotheses | Molecular modelling | β-Amyloid | Hypoxia | Protein-serine/threonine kinase | Mutation | Alzheimers disease | Cancer | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Protein Science, ISSN 0961-8368, 03/2018, Volume 27, Issue 3, pp. 725 - 737
Journal Article
Insect Molecular Biology, ISSN 0962-1075, 04/2018, Volume 27, Issue 2, pp. 198 - 211
Journal Article
Journal Article
The FEBS Journal, ISSN 1742-464X, 11/2009, Volume 276, Issue 21, pp. 6074 - 6083
Journal Article
Oncogene, ISSN 0950-9232, 01/2016, Volume 35, Issue 2, pp. 228 - 240
Journal Article
Journal Article