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2011, ISBN 0309164192, xvii, 96
"With the widespread use of highly active anti-retroviral treatment (HAART), HIV has become a chronic, rather than a fatal, disease. But for their treatment to... 
HIV infections | Treatment | Health Services Accessibility | therapy | AIDS (Disease) | HIV-positive persons | diagnosis | Health Policy | Government policy | Patients | Care | Counseling of
Book
Clinical Infectious Diseases, ISSN 1058-4838, 10/2018, Volume 67, Issue 10, pp. 1588 - 1594
Our trial of a single-tablet regimen containing elvitegravir, cobicistat, emtricitabine, and tenofovir disoproxil fumarate in antiretroviral therapy-naive... 
HIV-1
Journal Article
2000, Health, risk and society, ISBN 1857289099, xii, 234
To date, the majority of HIV/AIDS research has concentrated on education and prevention for those with a seronegative status, while studies of HIV positive... 
Psychological aspects | Social aspects | AIDS (Disease) | HIV-positive persons | HIV (Viruses)
Book
International Journal of Pure and Applied Mathematics, ISSN 1311-8080, 2018, Volume 119, Issue 12, pp. 7237 - 7248
Journal Article
Cell Host & Microbe, ISSN 1931-3128, 04/2017, Volume 21, Issue 4, pp. 494 - 506.e4
Journal Article
BMC Infectious Diseases, ISSN 1471-2334, 09/2017, Volume 17, Issue 1, pp. 605 - 605
Background: We describe the accumulation of HIV-1 drug resistance and its effect on the activity of next-line components in patients with virological failure... 
HIV-1 | Antiretroviral drug resistance | Virological failure | Second-line ART | HIV-1 RNA | INFECTIOUS DISEASES | SUB-SAHARAN AFRICA | LOAD | ADULTS | 1ST-LINE ART | STRATEGIES | CHILDREN | MUTATIONS | HIV-1 - pathogenicity | Tenofovir - therapeutic use | HIV-1 - drug effects | HIV Infections - virology | Humans | Male | Nevirapine - therapeutic use | CD4 Lymphocyte Count | HIV-1 - genetics | Benzoxazines - therapeutic use | Drug Resistance, Viral - genetics | Mozambique | Treatment Failure | Lamivudine - therapeutic use | Adult | Drug Resistance, Viral - drug effects | Female | HIV Infections - drug therapy | Zidovudine - therapeutic use | Mutation | Reverse Transcriptase Inhibitors - therapeutic use | Antiretroviral Therapy, Highly Active - methods | Viral Load - drug effects | Longitudinal Studies | Highly active antiretroviral therapy | Complications and side effects | Drug resistance in microorganisms | Usage | Patient outcomes | Development and progression | Research | Drug therapy | HIV infection | Risk factors | Drugs | Therapy | Nevirapine | Thymidine | Drug resistance | Efavirenz | Accumulation | Tenofovir | Rams | Acquired immune deficiency syndrome--AIDS | Antiretroviral agents | Human immunodeficiency virus--HIV | Emtricitabine | Antiretroviral drugs | Analogue | Lamivudine | Ribonucleic acid--RNA | Patients | Forecasting | Antiretroviral therapy | Switching | CD4 antigen | Protease inhibitors | Correlation analysis | Zidovudine | Index Medicus
Journal Article
Journal Article
PLoS Pathogens, ISSN 1553-7366, 2015, Volume 11, Issue 9, pp. e1005142 - e1005142
Pharmacologically-induced activation of replication competent proviruses from latency in the presence of antiretroviral treatment (ART) has been proposed as a... 
ACTIVATION | RNA | PERSISTENCE | CD4(+) T-CELLS | MICROBIOLOGY | VIROLOGY | HISTONE DEACETYLASES | ACTIVE ANTIRETROVIRAL THERAPY | IMMUNODEFICIENCY-VIRUS TYPE-1 | STEM-CELL | INFECTION | PARASITOLOGY | VIRAL RESERVOIR | Follow-Up Studies | Humans | Middle Aged | Antiretroviral Therapy, Highly Active - adverse effects | Male | RNA, Viral - blood | AIDS Vaccines - therapeutic use | Virus Latency - drug effects | AIDS Vaccines - adverse effects | Anti-HIV Agents - administration & dosage | HIV Infections - immunology | Drug Interactions | HIV-1 - physiology | Lymphocytes - immunology | Protein Processing, Post-Translational - drug effects | HIV-1 - isolation & purification | Adult | Anti-HIV Agents - therapeutic use | Female | Depsipeptides - therapeutic use | RNA, Viral - metabolism | Depsipeptides - adverse effects | Biomarkers - metabolism | Lymphocytes - metabolism | HIV-1 - drug effects | Anti-HIV Agents - adverse effects | HIV Infections - virology | Biomarkers - blood | Histones - blood | Acetylation - drug effects | HIV-1 - immunology | Virus Activation - drug effects | Lymphocytes - drug effects | HIV Infections - drug therapy | Histones - metabolism | Infusions, Intravenous | Viral Load - drug effects | Cohort Studies | Depsipeptides - administration & dosage | HIV Infections - metabolism | Index Medicus | Studies | Plasma | HIV | Acquired immune deficiency syndrome | Cytokines | Lymphocytes | Curing | AIDS | Infections | Human immunodeficiency virus | Research | Drug therapy
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 505, Issue 7484, pp. 509 - 514
The pathway causing CD4 T-cell death in HIV-infected hosts remains poorly understood although apoptosis has been proposed as a key mechanism. We now show that... 
APOPTOSIS | ACTIVATION | HUMAN LYMPHOID-TISSUE | DENDRITIC CELLS | MEMORY | REVERSE TRANSCRIPTION | MULTIDISCIPLINARY SCIENCES | IMMUNODEFICIENCY-VIRUS TYPE-1 | X4 HIV-1 | EX-VIVO | CXCR4-TROPIC HIV-1 | Anti-HIV Agents - pharmacology | Spleen - virology | Inflammation - pathology | Caspase Inhibitors - pharmacology | HIV-1 - pathogenicity | Palatine Tonsil - drug effects | Inflammasomes - metabolism | Humans | Caspase 3 - metabolism | Caspase 1 - metabolism | Male | HIV Infections - enzymology | Spleen - drug effects | CD4-Positive T-Lymphocytes - pathology | Lymph Nodes - enzymology | HIV Infections - immunology | Inflammation - complications | HIV-1 - growth & development | HIV Infections - pathology | Adult | Interleukin-1beta - biosynthesis | Cell Death - drug effects | Interleukin-1beta - secretion | Caspase Inhibitors - administration & dosage | Administration, Oral | HIV-1 - drug effects | CD4-Positive T-Lymphocytes - cytology | Inflammation - virology | Inflammation - immunology | CD4-Positive T-Lymphocytes - secretion | Virus Replication | Protein Precursors - biosynthesis | Inflammasomes - immunology | Palatine Tonsil - virology | HIV Infections - drug therapy | In Vitro Techniques | CD4-Positive T-Lymphocytes - drug effects | Medical research | Cell death | Physiological aspects | Host-parasite relationships | Medicine, Experimental | Research | T cells | Health aspects | HIV infection | Infections | Kinases | Human immunodeficiency virus--HIV | Apoptosis | Immune system | Index Medicus
Journal Article
Molecular epidemiology of HIV in Brazil: Polymorphism of the antigenically distinct HIV-1 B subtype strains, 12/1998
HIV is one of the most genetically polymorphic human emergent pathogens of this century. Two related viruses, HIV-1 and HIV-2 were identified as ethiological... 
HIV-1 polymorphism - molecular epidemiology - HIV-1 B subtype
Journal
by Frentz, Dineke and van de Vijver, David A. M. C and Abecasis, Ana B and Albert, Jan and Hamouda, Osamah and Jørgensen, Louise B and Kücherer, Claudia and Struck, Daniel and Schmit, Jean-Claude and Vercauteren, Jurgen and Åsjö, Birgitta and Balotta, Claudia and Beshkov, Danail and Camacho, Ricardo J and Clotet, Bonaventura and Coughlan, Suzie and Griskevicius, Algirdas and Grossman, Zehava and Horban, Anzej and Kolupajeva, Tatjana and Korn, Klaus and Kostrikis, Leondios G and Liitsola, Kirsi and Linka, Marek and Nielsen, Claus and Otelea, Dan and Paraskevis, Dimitrios and Paredes, Roger and Poljak, Mario and Puchhammer-Stöckl, Elisabeth and Sönnerborg, Anders and Stanekova, Danica and Stanojevic, Maja and van Wijngaerden, Eric and Wensing, Annemarie M. J and Boucher, Charles A. B and Puchhammer-Stockl, E and Sarcletti, M and Schmied, B and Geit, M and Balluch, G and Vandamme, A.-M and Vercauteren, J and Derdelinckx, I and Sasse, A and Bogaert, M and Ceunen, H and de Roo, A and de Wit, S and Echahidi, F and Fransen, K and Goffard, J.-C and Goubau, P and Goudeseune, E and Yombi, J.-C and Lacor, P and Liesnard, C and Moutschen, M and Pierard, D and Rens, R and Schrooten, Y and Vaira, D and Vandekerckhove, L. P. R and van den Heuvel, A and van der Gucht, B and van Ranst, M and van Wijngaerden, E and Vandercam, B and Vekemans, M and Verhofstede, C and Clumeck, N and van Laethem, K and Kostrikis, L and Demetriades, I and Kousiappa, I and Demetriou, V and Hezka, J and Bruckova, M and Linka, M and Machala, L and Nielsen, C and Jørgensen, L. B and Gerstoft, J and Mathiesen, L and Pedersen, C and Nielsen, H and Laursen, A and Kvinesdal, B and Salminen, M and Ristola, M and Liitsola, K and Suni, J and Sutinen, J and Korn, K and Kücherer, C and Berg, T and Braun, P and Poggensee, G and Däumer, M and Eberle, J and ... and SPREAD Programme
BMC infectious diseases, ISSN 1471-2334, 2014, Volume 14, Issue 1, pp. 407 - 407
Journal Article