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STEM CELLS, ISSN 1066-5099, 06/2016, Volume 34, Issue 6, pp. 1664 - 1678
Journal Article
Blood, ISSN 0006-4971, 05/2014, Volume 123, Issue 19, pp. 3016 - 3026
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e98586
Journal Article
Blood, ISSN 0006-4971, 04/2005, Volume 105, Issue 7, pp. 2707 - 2716
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2018, Volume 13, Issue 8, p. e0201220
In recent years, immunogenic cell death (ICD) has emerged as a revolutionary concept in the development of novel anticancer therapies. This particular form of... 
CALRETICULIN EXPOSURE | KILLS CANCER-CELLS | THERAPY | MELANOMA | PROTEIN | MULTIDISCIPLINARY SCIENCES | LTX-315 | RELEASE | TARGETING AAC-11 | EXPRESSION | TUMORS | Apoptosis Regulatory Proteins - pharmacology | Recombinant Fusion Proteins - pharmacology | Humans | Cell Death - immunology | Nuclear Proteins - pharmacology | Peptide Fragments - pharmacology | Sarcoma, Experimental - immunology | Cell-Penetrating Peptides - pharmacology | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Cell-Penetrating Peptides - chemistry | Amino Acid Sequence | Jurkat Cells | Apoptosis Regulatory Proteins - chemistry | Mice, Inbred C57BL | Recombinant Fusion Proteins - chemistry | Antineoplastic Agents - chemistry | Nuclear Proteins - chemistry | Sarcoma, Experimental - pathology | Sarcoma, Experimental - drug therapy | Xenograft Model Antitumor Assays | Peptide Fragments - chemistry | Animals | Cell Line, Tumor | HL-60 Cells | Mice | Care and treatment | Peptides | Cell death | Analysis | Cancer cells | Research | Cancer | Dehydrogenases | Vaccination | Inflammatory response | Lymphocytes T | Leucine | Kinases | HMGB1 protein | Blood | Metastases | Anticancer properties | Proteins | Calreticulin | Immunology | Gangrene | Immune system | Medical research | Cell survival | Immune response | Mortality | Melanoma | Hazards | Inflammation | Mode of action | Immunogenicity | Antitumor activity | Infiltration | Chemokines | Apoptosis | Tumors
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 6, p. e21077
The slow-releasing hydrogen sulfide (H2S) donor, GYY4137, caused concentration-dependent killing of seven different human cancer cell lines (HeLa, HCT-116, Hep... 
CELLS | APOPTOSIS | OXIDATIVE STRESS | INHIBITION | P38 | CASPASE-3 | BIOLOGY | Organothiophosphorus Compounds - therapeutic use | Humans | Antineoplastic Agents - therapeutic use | Organothiophosphorus Compounds - chemistry | Organothiophosphorus Compounds - pharmacology | Female | Antineoplastic Agents - pharmacology | Morpholines - therapeutic use | Cell Line | Cell Survival - drug effects | HCT116 Cells | Hydrogen Sulfide - chemistry | Morpholines - pharmacology | Antineoplastic Agents - chemistry | Mice, SCID | Blotting, Western | Morpholines - chemistry | Hep G2 Cells | Xenograft Model Antitumor Assays | Animals | Cell Line, Tumor | HL-60 Cells | Cell Proliferation - drug effects | Mice | Cell Cycle - drug effects | Hydrogen sulfide | Exhibitions | Care and treatment | Cancer | Sulfide | Cell culture | Biotechnology | Oxidative stress | Nuclear magnetic resonance--NMR | Hydrogen | Leukemia | Colorectal cancer | Smooth muscle | Biochemistry | Kinases | Anticancer properties | Ischemia | Rodents | Xenografts | Cell cycle | Fibroblasts | Sulfur | Hydrogen ion concentration | Killing | Enzymes | Incubation | Poly(ADP-ribose) polymerase | Caspase | Tumor cell lines | Releasing | Chemotherapy | Mutagenesis | Sodium | Lungs | Coronary vessels | Cell lines | Caspase-9 | Scientific imaging | Apoptosis | Nuclear magnetic resonance | NMR
Journal Article
Journal Article
BMC Cell Biology, ISSN 1471-2121, 01/2014, Volume 15, Issue 1, pp. 4 - 4
Background: Epigenetic regulation is known to affect gene expression, and recent research shows that aberrant DNA methylation patterning and histone... 
Histones | KG1 cells | Gene methylation/demethylation | Neutrophils | CD34+ cells | CHRONIC MYELOGENOUS LEUKEMIA | CD34+cells | DNA METHYLATION | HL-60 CELLS | E-CADHERIN | P16(INK4A) METHYLATION STATUS | CELL BIOLOGY | BREAST-CANCER CELLS | IN-VITRO | RETINOIC ACID | HISTONE DEACETYLASE INHIBITORS | STROMAL CELLS | Myeloid Cells - cytology | Neutrophils - cytology | Granulocytes - cytology | Antigens, CD34 - metabolism | Cadherins - metabolism | Epigenesis, Genetic | Humans | Receptors, Retinoic Acid - genetics | DNA Methylation | Cell Cycle Proteins - genetics | Phenylbutyrates - pharmacology | Granulocytes - metabolism | Cadherins - genetics | Neutrophils - metabolism | Promoter Regions, Genetic | Cell Cycle Proteins - metabolism | Cells, Cultured | Receptors, Retinoic Acid - metabolism | Hematopoietic Stem Cells - metabolism | Histones - genetics | Cell Differentiation - drug effects | Hematopoietic Stem Cells - cytology | Myeloid Cells - metabolism | Histone Deacetylase Inhibitors - pharmacology | Histones - metabolism | Physiological aspects | Methylation | Gene expression | Analysis | Hematopoietic stem cells | Epigenetic inheritance | DNA binding proteins | Research | Comparative analysis | Cell differentiation | Pathogenesis | DNA methylation | Epigenetics | Mutation | Experiments | Deoxyribonucleic acid--DNA | Methods
Journal Article
by Yoo, HJ and Lee, JS and Kim, JE and Gu, J and Koh, Y and Kim, I and Kim, HK
PLOS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0163982
Journal Article