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by Pereyra, Florencia and Jia, Xiaoming and McLaren, Paul J and Telenti, Amalio and de Bakker, Paul I. W and Walker, Bruce D and Ripke, Stephan and Brumme, Chanson J and Pulit, Sara L and Carrington, Mary and Kadie, Carl M and Carlson, Jonathan M and Heckerman, David and Graham, Robert R and Plenge, Robert M and Deeks, Steven G and Gianniny, Lauren and Crawford, Gabriel and Sullivan, Jordan and Gonzalez, Elena and Davies, Leela and Camargo, Amy and Moore, Jamie M and Beattie, Nicole and Gupta, Supriya and Crenshaw, Anew and Burtt, Noël P and Guiducci, Candace and Gupta, Namrata and Gao, Xiaojiang and Qi, Ying and Yuki, Yuko and Piechocka-Trocha, Alicja and Cutrell, Emily and Rosenberg, Rachel and Moss, Kristin L and Lemay, Paul and O'Leary, Jessica and Schaefer, Todd and Verma, Pranshu and Toth, Ildiko and Block, Brian and Baker, Brett and Rothchild, Alissa and Lian, Jeffrey and Proudfoot, Jacqueline and Alvino, Donna Marie L and Vine, Seanna and Addo, Marylyn M and Allen, Todd M and Altfeld, Marcus and Henn, Matthew R and Le Gall, Sylvie and Streeck, Henik and Haas, David W and Kuritzkes, Daniel R and Robbins, Gregory K and Shafer, Robert W and Gulick, Roy M and Shikuma, Cecilia M and Haubrich, Richard and Riddler, Sharon and Sax, Paul E and Daar, Eric S and Ribaudo, Heather J and Agan, Brian and Agarwal, Shanu and Ahern, Richard L and Allen, Brady L and Altidor, Sherly and Altschuler, Eric L and Ambardar, Sujata and Anastos, Kathryn and Anderson, Ben and Anderson, Val and Anady, Ushan and Antoniskis, Diana and Bangsberg, David and Barbaro, Daniel and Barrie, William and Bartczak, J and Barton, Simon and Basden, Patricia and Basgoz, Nesli and Bazner, Suzane and Bellos, Nicholaos C and Benson, Anne M and Berger, Judith and Bernard, Nicole F and Bernard, Annette M and Birch, Christopher and Bodner, Stanley J and Bolan, Robert K and Boueaux, Emilie T and Bradley, Meg and Braun, James F and Brndjar, Jon E and Brown, Stephen J and Brown, Katherine and Brown, Sheldon T and ... and Int HIV Controllers Study and International HIV Controllers Study and The International HIV Controllers Study
Science, ISSN 0036-8075, 12/2010, Volume 330, Issue 6010, pp. 1551 - 1557
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2015, Volume 125, Issue 10, pp. 3981 - 3991
Adoptively transferred tumor-infiltrating T lymphocytes (TILs) that mediate complete regression of metastatic melanoma have been shown to recognize mutated... 
CD4+T CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | METASTATIC MELANOMA | TCR | THERAPY | RECOGNITION | ANTI-CTLA-4 | MUTATIONAL LANDSCAPE | CD4(+) | ANTIGENS | BLOCKADE | Humans | Middle Aged | Neoplasm Proteins - immunology | Molecular Sequence Data | Male | Antigen-Antibody Reactions | Epitopes - immunology | Exome | Melanoma - genetics | Peptide Fragments - immunology | Adult | Female | Receptor, ErbB-2 - immunology | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | Transcription Factors - immunology | Amino Acid Sequence | Antigens, Neoplasm - genetics | DNA-Binding Proteins - immunology | HLA-A2 Antigen - immunology | Antigens, Neoplasm - immunology | Interferon-gamma Release Tests | Cells, Cultured | HLA-A1 Antigen - chemistry | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Epitopes - genetics | Nuclear Proteins - immunology | Melanoma - secondary | HLA-A2 Antigen - chemistry | Algorithms | Genes, erbB-2 | Melanoma - immunology | Adolescent | RNA, Neoplasm - genetics | T-Cell Antigen Receptor Specificity | T-Lymphocytes - immunology | HLA-A1 Antigen - immunology | DNA, Neoplasm - genetics | Mutation | Antigens, Neoplasm - classification | Lymphocytes, Tumor-Infiltrating - immunology | Physiological aspects | T cells | Lymphocytes | Identification and classification | Health aspects | Tumors | Antigens | Peptides | Cloning | Melanoma | T cell receptors | Metastasis | Cancer therapies | Patients | Studies | Immunotherapy | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
Science, ISSN 0036-8075, 10/2011, Volume 334, Issue 6052, pp. 89 - 94
Whole genome comparisons identified introgression from archaic to modern humans. Our analysis of highly polymorphic human leukocyte antigen (HLA) class I,... 
HLA antigens | Haplotypes | Human populations | HLA A antigens | REPORTS | Alleles | HLA B antigens | Admixtures | Ligands | Genomes | HLA C antigens | MIGRATION | POPULATION | EVOLUTION | HAPLOTYPE MAP | MULTIDISCIPLINARY SCIENCES | HLA-B ALLELES | MHC | DIVERSITY | POLYMORPHISM | SELECTION | HISTORY | Humans | Asian Continental Ancestry Group - genetics | Adaptation, Biological | Molecular Sequence Data | Hominidae - genetics | HLA-A Antigens - metabolism | Genetic Variation | HLA-C Antigens - immunology | Hybridization, Genetic | Receptors, KIR - metabolism | Killer Cells, Natural - immunology | African Continental Ancestry Group - genetics | Oceanic Ancestry Group - genetics | HLA-B Antigens - genetics | European Continental Ancestry Group - genetics | HLA-B Antigens - immunology | Selection, Genetic | Continental Population Groups - genetics | Hominidae - immunology | Linkage Disequilibrium | HLA-B Antigens - metabolism | HLA-A Antigens - immunology | Animals | HLA-A Antigens - genetics | Genes, MHC Class I | HLA-C Antigens - genetics | Receptors, KIR - immunology | Evolution, Molecular | Histocompatibility antigens | Genomics | HLA histocompatibility antigens | Physiological aspects | Genetic aspects | Research | Immune system | Leucocytes | Antigens | Polymorphism | Index Medicus | Biological Sciences | Evolutionsbiologi | Naturvetenskap | Natural Sciences | Evolutionary Biology | Biologiska vetenskaper
Journal Article
Brain, ISSN 0006-8950, 2012, Volume 135, Issue 4, pp. 1042 - 1054
Peptides presented at the cell surface reflect the protein content of the cell; those on HLA class I molecules comprise the critical peptidome elements... 
peptidome | glioblastoma | tumour-infiltrating lymphocytes | tumour antigen | immunotherapy | AVIDITY | IDENTIFICATION | NEUROSCIENCES | CLINICAL NEUROLOGY | GLIOMA-CELL | GROWTH | CHONDROITIN SULFATE PROTEOGLYCAN | SELECTION | EXPRESSION | T-CELLS | PROGRESSION | TENASCIN-C | Oligonucleotide Array Sequence Analysis | Humans | Brain Neoplasms - pathology | Gene Expression Profiling | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Antigens, Neoplasm - chemistry | HLA-A Antigens - analysis | Antigens, CD - metabolism | Sequence Analysis, Protein | Brain Neoplasms - immunology | Flow Cytometry | Antigens, Neoplasm - therapeutic use | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Mass Spectrometry | Chromatography, Liquid | Cytokines - metabolism | Antigens, Neoplasm - immunology | Peptides - immunology | Glioblastoma - therapy | HLA-A Antigens - immunology | Glioblastoma - immunology | Glioblastoma - pathology | Brain Neoplasms - therapy | HLA-A Antigens - chemistry | CD8-Positive T-Lymphocytes - immunology | Antigen Presentation - physiology | Peptides - analysis | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - metabolism | Index Medicus | Abridged Index Medicus | Immunogenicity | CD8 antigen | Immunotherapy | Glioblastoma | Data processing | Histocompatibility antigen HLA | Lymphocytes T | Vaccines | Cell surface | Immunological tolerance
Journal Article
Cancer Research, ISSN 0008-5472, 06/2010, Volume 70, Issue 11, pp. 4335 - 4345
Journal Article
Blood, ISSN 0006-4971, 08/2010, Volume 116, Issue 6, pp. 935 - 944
Type 1 T regulatory (Tr1) cells suppress immune responses in vivo and in vitro and play a key role in maintaining tolerance to self- and non-self-antigens.... 
G MOLECULE | IN-VITRO | CD16(+) MONOCYTES | HUMAN DENDRITIC CELLS | HLA-G EXPRESSION | CD4(+) | PERIPHERAL-BLOOD | HEMATOLOGY | MACROPHAGE ACTIVATION | IL-10 | MYELOMONOCYTIC CELLS | Cell Communication - immunology | T-Lymphocytes, Regulatory - metabolism | Membrane Glycoproteins - metabolism | Monocytes - cytology | Dendritic Cells - immunology | Humans | Monocytes - metabolism | Monocytes - immunology | HLA Antigens - genetics | T-Lymphocytes, Regulatory - immunology | Histocompatibility Antigens Class I - metabolism | Signal Transduction - immunology | Flow Cytometry | Immune Tolerance - immunology | Gene Expression - immunology | T-Lymphocytes, Regulatory - cytology | Interleukin-10 - metabolism | Interleukin-10 - secretion | Membrane Glycoproteins - immunology | Receptors, Immunologic - immunology | Interleukin-12 - secretion | Dendritic Cells - metabolism | Histocompatibility Antigens Class I - immunology | HLA Antigens - immunology | Immunophenotyping | Histocompatibility Antigens Class I - genetics | Interleukin-12 - metabolism | HLA-G Antigens | HLA Antigens - metabolism | Membrane Glycoproteins - genetics | Cell Differentiation - immunology | Dendritic Cells - cytology | Receptors, Immunologic - genetics | Interleukin-10 - immunology | Receptors, Immunologic - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Nature, ISSN 0028-0836, 03/2011, Volume 471, Issue 7337, pp. 220 - 224
Under physiological conditions the gut-associated lymphoid tissues not only prevent the induction of a local inflammatory immune response, but also induce... 
TGF-BETA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | CLASS-I | ORAL TOLERANCE | MAJOR HISTOCOMPATIBILITY COMPLEX | DIFFERENTIATION | GLUTEN SENSITIVITY | CELIAC-DISEASE | BOWEL-DISEASE | TRANSGENIC MICE | Glutens - immunology | Coculture Techniques | Dendritic Cells - immunology | Humans | Middle Aged | Child, Preschool | T-Lymphocytes, Regulatory - immunology | Young Adult | Forkhead Transcription Factors - metabolism | Intestinal Mucosa - immunology | Phosphorylation - drug effects | Child | Interleukin-12 - secretion | Interleukin-12 - biosynthesis | Tretinoin - pharmacology | Celiac Disease - chemically induced | Administration, Oral | Mitogen-Activated Protein Kinase 8 - metabolism | Mice, Transgenic | Interleukin-23 - immunology | T-Lymphocytes, Regulatory - drug effects | Diet | Dendritic Cells - enzymology | Glutens - administration & dosage | Adolescent | Interleukin-12 - immunology | Mice | Receptors, Interleukin-12 - deficiency | T-Lymphocytes, Regulatory - metabolism | Adjuvants, Immunologic - pharmacology | Interleukin-15 - immunology | HLA-DQ Antigens - immunology | Celiac Disease - etiology | Intestinal Mucosa - cytology | Celiac Disease - immunology | T-Lymphocytes, Regulatory - cytology | Adult | Dendritic Cells - drug effects | Interleukin-23 - secretion | Dendritic Cells - metabolism | Immune Tolerance - drug effects | Gliadin - immunology | Mice, Inbred C57BL | Cells, Cultured | Tretinoin - immunology | Inflammation - immunology | Animals | Gliadin - administration & dosage | Interleukin-15 - genetics | HLA-DQ Antigens - genetics | Physiological aspects | Antigens | Immune response | Research | Health aspects | Tretinoin | Proteins | Celiac disease | Immunology | Rodents | Index Medicus
Journal Article
PLoS Biology, ISSN 1544-9173, 06/2010, Volume 8, Issue 6, pp. e1000407 - e1000407
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, pp. e64683 - e64683
DNA sequence variation within human leukocyte antigen (HLA) genes mediate susceptibility to a wide range of human diseases. The complex genetic structure of... 
CLASSICAL HLA ALLELES | EXTENDED HUMAN MHC | DISEASE ASSOCIATION | GENOTYPE IMPUTATION | MULTIDISCIPLINARY SCIENCES | COMMON DISEASES | SUSCEPTIBILITY | MAJOR HISTOCOMPATIBILITY COMPLEX | HAPLOTYPE | INFERENCE | GENOME-WIDE ASSOCIATION | Haplotypes | Genetic Predisposition to Disease | Genome-Wide Association Study | HapMap Project | Humans | Amino Acids - immunology | Histocompatibility Antigens Class I - immunology | Diabetes Mellitus, Type 1 - genetics | HLA Antigens - immunology | European Continental Ancestry Group | Chromosome Mapping | Histocompatibility Antigens Class I - genetics | HLA Antigens - genetics | Amino Acids - genetics | Linkage Disequilibrium | Histocompatibility Antigens Class II - immunology | Alleles | Polymorphism, Single Nucleotide | Diabetes Mellitus, Type 1 - immunology | Histocompatibility Antigens Class II - genetics | Diabetes Mellitus, Type 1 - ethnology | Histocompatibility antigens | Type 1 diabetes | HLA histocompatibility antigens | Amino acids | Disease susceptibility | Genetic aspects | Nucleotide sequencing | Genetic polymorphisms | DNA sequencing | DQA1 protein | Genes | Linkage disequilibrium | Genomes | Single-nucleotide polymorphism | Density | Population genetics | Medical schools | Consortia | Proteins | Drb1 protein | Genetics | Chromosomes | Deoxyribonucleic acid--DNA | Antigens | Nucleotide sequence | Genetic structure | Diabetes mellitus | Rheumatology | Amino acid sequence | Loci | Medicine | Studies | White blood cells | Hospitals | DNA microarrays | Major histocompatibility complex | Acids | Genotyping | Womens health | Computer applications | Histocompatibility antigen HLA | Diabetes | Gene mapping | Histocompatibility | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article