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Journal Article
by Zhao, Wei and Rasheed, Asif and Tikkanen, Emmi and Lee, Jung-Jin and Butterworth, Adam S and Howson, Joanna M. M and Assimes, Themistocles L and Chowdhury, Rajiv and Orho-Melander, Marju and Damrauer, Scott and Small, Aeron and Asma, Senay and Imamura, Minako and Yamauch, Toshimasa and Chambers, John C and Chen, Peng and Sapkota, Bishwa R and Shah, Nabi and Jabeen, Sehrish and Surendran, Praveen and Lu, Yingchang and Zhang, Weihua and Imran, Atif and Abbas, Shahid and Majeed, Faisal and Trindade, Kevin and Qamar, Nadeem and Mallick, Nadeem Hayyat and Yaqoob, Zia and Saghir, Tahir and Hasan Rizvi, Syed Nadeem and Memon, Anis and Rasheed, Syed Zahed and Memon, Fazal-Ur-Rehman and Mehmood, Khalid and Ahmed, Naveeduddin and Hussain Qureshi, Irshad and Tanveer-Us-Salam, Tanveer-Us-Salam and Iqbal, Wasim and Malik, Uzma and Mehra, Narinder and Kuo, Jane Z and Sheu, Wayne H.-H and Guo, Xiuqing and Hsiung, Chao A and Juang, Jyh-Ming J and Taylor, Kent D and Hung, Yi-Jen and Lee, Wen-Jane and Quertermous, Thomas and Lee, I-Te and Hsu, Chih-Cheng and Bottinger, Erwin P and Ralhan, Sarju and Teo, Yik Ying and Wang, Tzung-Dau and Alam, Dewan S and Di Angelantonio, Emanuele and Epstein, Steve and Nielsen, Sune F and Nordestgaard, Borge G and Tybjaerg-Hansen, Anne and Young, Robin and Benn, Marianne and Frikke-Schmidt, Ruth and Kamstrup, Pia R and Jukema, J. Wouter and Sattar, Naveed and Smit, Roelof and Chung, Ren-Hua and Liang, Kae-Woei and Anand, Sonia and Sanghera, Dharambir K and Ripatti, Samuli and Loos, Ruth J. F and Kooner, Jaspal S and Tai, E. Shyong and Rotter, Jerome I and Ida Chen, Yii-Der and Frossard, Philippe and Maeda, Shiro and Kadowaki, Takashi and Reilly, Muredach and Pare, Guillaume and Melander, Olle and Salomaa, Veikko and Rader, Daniel J and Danesh, John and Voight, Benjamin F and Saleheen, Danish and EPIC-CVD Consortium and CHD Exome Consortium and EPIC-Interact Consortium and Michigan Biobank and CHD Exome+ Consortium and Diabetes - Cardiovascular Disease and Diabetes - kardiovaskulär sjukdom and Kardiovaskulär forskning - hypertoni and Cardiovascular Research - Hypertension and Lund University and EpiHealth: Epidemiology for Health and Lunds universitet and EXODIAB: Excellence in Diabetes Research in Sweden
Nature Genetics, ISSN 1061-4036, 10/2017, Volume 49, Issue 10, pp. 1450 - 1457
To evaluate the shared genetic etiology of type 2 diabetes (T2D) and coronary heart disease (CHD), we conducted a genome-wide, multi-ancestry study of genetic... 
METAANALYSIS | AP2 | VARIANTS | ACID-BINDING PROTEIN | GENETICS & HEREDITY | ATHEROSCLEROSIS | RISK | GENETIC ARCHITECTURE | MENDELIAN RANDOMIZATION | GENOME-WIDE ASSOCIATION | ARTERY-DISEASE | Diabetes Mellitus, Type 2 - genetics | Humans | Asian Continental Ancestry Group - genetics | Genetic Loci - genetics | Coronary Disease - epidemiology | Mutation, Missense | Molecular Targeted Therapy | Diabetes Mellitus, Type 2 - epidemiology | Diabetes Mellitus, Type 2 - etiology | HLA-DRB5 Chains - genetics | Metabolic Syndrome - epidemiology | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Comorbidity | Risk Factors | Coronary Disease - etiology | Europe - epidemiology | Asia - epidemiology | Metabolic Networks and Pathways - genetics | Coronary Disease - genetics | Metabolic Syndrome - genetics | Biomarkers | Polymorphism, Single Nucleotide | Diabetes Mellitus, Type 2 - drug therapy | Quantitative trait loci | Type 2 diabetes | Genetic variation | Genetic aspects | Health aspects | Coronary heart disease | Risk factors | Drugs | Diabetes mellitus | Cardiovascular disease | Triglycerides | Genomes | Genetic diversity | Adipocytes | Risk analysis | Fatty acids | Loci | Coronary artery disease | Meta-analysis | Proteins | Pathways | Etiology | Atherosclerosis | Gene loci | Histocompatibility antigen HLA | Diabetes | Cardiovascular diseases | Bioinformatics | Heart diseases | Medical and Health Sciences | single nucleotide polymorphism | major clinical study | ischemic heart disease | Genetic Loci | HLA DRB5 gene | genetic risk | genetic predisposition | East Asian | Diabetes Mellitus, Type 2 | genetic susceptibility | metabolic syndrome X | Basic Medicine | gene | Europe | Caucasian | genetic variation | European Continental Ancestry Group | South Asian | European | non insulin dependent diabetes mellitus | Metabolic Networks and Pathways | HLA-DRB5 Chains | metabolism | molecularly targeted therapy | comparative study | HLA DRB5 antigen | CCDC92 gene | Medicin och hälsovetenskap | Article | fatty acid binding protein 4 | missense mutation | Medicinsk genetik | Medical Genetics | genetics | Coronary Disease | icosapentaenoic acid ethyl ester | priority journal | human | risk factor | genetic variability | gene locus | comorbidity | biological marker | controlled study | Asian continental ancestry group | Asia | genome-wide association study | Medicinska och farmaceutiska grundvetenskaper
Journal Article
JAMA Neurology, ISSN 2168-6149, 06/2016, Volume 73, Issue 6, pp. 691 - 697
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, p. e29819
The human leukocyte antigen (HLA) DRB1*1501 has been consistently associated with multiple sclerosis (MS) in nearly all populations tested. This points to a... 
ALLELES | DISEASE | BIOLOGY | SUSCEPTIBILITY LOCI | COMMON | MHC | MECHANISMS | MAJOR HISTOCOMPATIBILITY COMPLEX | HLA | EPISTASIS | GENOME-WIDE ASSOCIATION | Genetics, Population | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Genetic Association Studies | HLA-DRB1 Chains - genetics | Humans | Risk Factors | Gene Expression Regulation | Linkage Disequilibrium - genetics | Logistic Models | Male | Multiple Sclerosis - genetics | HLA-DQ beta-Chains - genetics | Haplotypes - genetics | Alleles | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Polymorphism, Single Nucleotide - genetics | Adult | Female | HLA-DRB5 Chains - genetics | Quantitative Trait Loci - genetics | Quantitative genetics | Multiple sclerosis | Histocompatibility antigens | Type 1 diabetes | Gastrointestinal diseases | Genes | Genomics | HLA histocompatibility antigens | Genetic aspects | Disease susceptibility | Genomes | Gene expression | Haplotypes | Inflammatory bowel diseases | Populations | Disease | Risk | Diabetes mellitus (insulin dependent) | Single-nucleotide polymorphism | Immunoglobulin A | Population genetics | Human populations | Consortia | Intestine | Drb1 protein | Population | Genetics | Genotypes | Lupus | Antigen presentation | Antigens | Diabetes mellitus | Loci | Asthma | Carriers | Quantitative trait loci | Inflammatory bowel disease | White blood cells | Studies | Rheumatoid arthritis | Histocompatibility antigen HLA | Ulcerative colitis | Data processing
Journal Article
Diabetes, ISSN 0012-1797, 03/2016, Volume 65, Issue 3, pp. 710 - 718
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, pp. e0150283 - e0150283
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 5, p. e36870
Journal Article
Journal Article
Neurotoxicology, ISSN 0161-813X, 07/2017, Volume 61, pp. 243 - 264
Journal Article