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Journal Article
Journal of molecular endocrinology, ISSN 0952-5041, 07/2017, Volume 59, Issue 1, pp. 13 - 27
Parathyroid hormone-related peptide (PTHrP) is associated with several human cancers such as colon carcinoma. This disease is a complex multistep process that... 
RSK | colon cancer cells | ERK1/2-p38MAPK | PTHrP | cell cycle migration | ENHANCES MIGRATION | P38 MAPK | FAMILY | INVASION | HORMONE-RELATED PROTEIN | PATHWAY | GROWTH | ENDOCRINOLOGY & METABOLISM | RIBOSOMAL S6 KINASE | FOCAL ADHESION KINASE | UP-REGULATION | Cyclin D1 - metabolism | Focal Adhesion Kinase 1 - genetics | Neoplasm Transplantation | Colonic Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Male | Phosphatidylinositol 3-Kinases - metabolism | Colonic Neoplasms - metabolism | Tumor Suppressor Protein p53 - genetics | MAP Kinase Signaling System - genetics | Injections, Intralesional | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Focal Adhesion Kinase 1 - metabolism | Protein-Serine-Threonine Kinases - metabolism | Caco-2 Cells | Cell Survival - drug effects | Mitogen-Activated Protein Kinase 3 - genetics | HCT116 Cells | Cyclin-Dependent Kinase 6 - genetics | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | p38 Mitogen-Activated Protein Kinases - genetics | Cyclin-Dependent Kinase 6 - metabolism | Parathyroid Hormone-Related Protein - metabolism | Parathyroid Hormone-Related Protein - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Cell Movement - drug effects | Animals | Cyclin D1 - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Mice, Nude | Colonic Neoplasms - pathology | Mice | Mitogen-Activated Protein Kinase 1 - metabolism | Cell proliferation | Adenocarcinoma | Phosphorylation | p53 Protein | Cyclin D1 | Kinases | Cell adhesion & migration | Parathyroid hormone-related protein | Signal transduction | Colon cancer | Intestine | Cell cycle | Xenografts | Colon | Localization | Parathyroid | Wound healing | Threonine | Therapeutic applications | Extracellular signal-regulated kinase | MAP kinase | 1-Phosphatidylinositol 3-kinase | Parathyroid hormone | Protein expression | Protein-serine/threonine kinase | Focal adhesion kinase | Cell migration
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2011, Volume 121, Issue 12, pp. 4655 - 4669
Parathyroid hormone-related protein (PTHrP) is a secreted factor expressed in almost all normal fetal and adult tissues. It is involved in a wide range of... 
MEDICINE, RESEARCH & EXPERIMENTAL | LUNG-CANCER | HORMONE-RELATED PROTEIN | MAMMARY-GLAND DEVELOPMENT | CANCER METASTASIS | PROSTATE-CANCER | MOUSE MODEL | PARATHYROID-HORMONE | BONE METASTASIS | MONOCLONAL-ANTIBODY | TRANSGENIC MICE | Angiogenic Proteins - genetics | Cell Adhesion Molecules - genetics | Epithelial Cells - metabolism | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Neoplasm Proteins - immunology | Molecular Targeted Therapy | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - therapy | Mammary Glands, Animal - growth & development | Antibodies, Neutralizing - immunology | Antibodies, Neutralizing - therapeutic use | Mammary Neoplasms, Experimental - pathology | Apoptosis Regulatory Proteins - genetics | Cell Cycle Proteins - genetics | Female | Receptors, CXCR4 - genetics | Parathyroid Hormone-Related Protein - deficiency | Neoplasm Proteins - genetics | Angiogenic Proteins - biosynthesis | Apoptosis Regulatory Proteins - biosynthesis | Cell Adhesion Molecules - biosynthesis | Neoplasm Proteins - biosynthesis | Parathyroid Hormone-Related Protein - genetics | Cell Cycle Proteins - biosynthesis | Disease Progression | Mice, Knockout | Parathyroid Hormone-Related Protein - immunology | Xenograft Model Antitumor Assays | Neoplasm Metastasis - genetics | Receptors, CXCR4 - physiology | Animals | Mammary Glands, Animal - metabolism | Receptors, CXCR4 - biosynthesis | Breast Neoplasms - pathology | Parathyroid Hormone-Related Protein - physiology | Mice | Cellular proteins | Breast tumors | Parathyroid hormone | Development and progression | Metastasis | Research | Properties | Index Medicus | Abridged Index Medicus
Journal Article
Development, ISSN 0950-1991, 06/2008, Volume 135, Issue 11, pp. 1947 - 1956
Chondrocyte hypertrophy is an essential process required for endochondral bone formation. Proper regulation of chondrocyte hypertrophy is also required in... 
Ihh | Patched 1 | Cartilage | Chondrocyte hypertrophy | PTHrP | PATHWAYS | OSTEOBLAST | cartilage | chondrocyte hypertrophy | PROLIFERATION | DEVELOPMENTAL BIOLOGY | CARTILAGE DEVELOPMENT | ENDOCHONDRAL BONE | SKELETON | patched 1 | HORMONE-RELATED-PROTEIN | GROWTH-PLATE | DIFFERENTIATION | Immunohistochemistry | Hedgehog Proteins - pharmacology | Bone Morphogenetic Proteins - physiology | Hedgehog Proteins - metabolism | Hedgehog Proteins - physiology | Wnt Proteins - metabolism | Cell Differentiation - genetics | Bone Morphogenetic Proteins - metabolism | In Situ Hybridization | Wnt Proteins - genetics | Hedgehog Proteins - genetics | Receptors, Cell Surface - physiology | beta Catenin - physiology | Patched Receptors | Bone Morphogenetic Proteins - genetics | Cell Differentiation - physiology | Chondrocytes - metabolism | Organ Culture Techniques | Recombinant Proteins - metabolism | Chondrocytes - pathology | Parathyroid Hormone-Related Protein - genetics | Cells, Cultured | Receptors, Cell Surface - metabolism | Signal Transduction - genetics | Cell Enlargement | Recombinant Proteins - pharmacology | Forelimb - metabolism | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | beta Catenin - genetics | Animals | Signal Transduction - drug effects | Parathyroid Hormone-Related Protein - physiology | Signal Transduction - physiology | Forelimb - cytology | Mice | Wnt Proteins - physiology | Mutation | Patched-1 Receptor | Receptors, Cell Surface - genetics | Index Medicus
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 3/2004, Volume 164, Issue 5, pp. 747 - 758
Sox5 and Sox6 encode Sry-related transcription factors that redundantly promote early chondroblast differentiation. Using mouse embryos with three or four null... 
Development | Mouse | Chondrogenesis | Differentiation | Transcription factor | transcription factor | development | differentiation | OSTEOBLAST DIFFERENTIATION | INDIAN HEDGEHOG | FACTOR RECEPTOR-3 | MOUSE LIMB | CELL BIOLOGY | mouse | TARGETED DISRUPTION | CBFA1-DEFICIENT MICE | II COLLAGEN | BONE-FORMATION | COLLAGEN GENE | chondrogenesis | SKELETAL DEVELOPMENT | Growth Plate - pathology | Receptor, Fibroblast Growth Factor, Type 3 | Cartilage - pathology | Protein-Tyrosine Kinases - metabolism | Bone Development - physiology | Growth Plate - physiology | SOXD Transcription Factors | Hypertrophy - metabolism | Hedgehog Proteins | Bone Morphogenetic Proteins - metabolism | Patched Receptors | Neoplasm Proteins - genetics | Animals, Newborn | Gestational Age | Parathyroid Hormone-Related Protein - metabolism | Mice, Knockout | Cell Division - physiology | Membrane Proteins | Mice | Bone and Bones - embryology | Chondrocytes - cytology | Receptors, Cell Surface | Neoplasm Proteins - metabolism | DNA-Binding Proteins - metabolism | Bone and Bones - anatomy & histology | Protein-Tyrosine Kinases - genetics | Embryo, Mammalian - anatomy & histology | Growth Plate - cytology | Cartilage - cytology | Receptors, Fibroblast Growth Factor - genetics | Trans-Activators - genetics | Female | Core Binding Factor Alpha 1 Subunit | Nuclear Proteins - genetics | Bone Morphogenetic Proteins - genetics | Cell Differentiation - physiology | Chondrocytes - metabolism | Bone Morphogenetic Protein 6 | Cartilage - embryology | High Mobility Group Proteins - metabolism | Mice, Inbred C57BL | Parathyroid Hormone-Related Protein - genetics | Intracellular Signaling Peptides and Proteins | Nuclear Proteins - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Pregnancy | Proteins - genetics | Transcription Factors - metabolism | Embryo, Mammalian - physiology | Animals | Proteins - metabolism | Receptors, Fibroblast Growth Factor - metabolism | Trans-Activators - metabolism | Bone and Bones - abnormalities | High Mobility Group Proteins - genetics | Cytology | Research | Cartilage | Cellular biology | Embryos | Deoxyribonucleic acid | DNA | Index Medicus | chondrogenesis; development; differentiation; mouse; transcription factor
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e54261 - e54261
Cellular functions are regulated by complex networks of many different signaling pathways. The TGF beta and cAMP pathways are of particular importance in tumor... 
EPITHELIAL-MESENCHYMAL TRANSITION | GROWTH-FACTOR-BETA | STEM-CELLS | INHIBITION | HORMONE-RELATED PROTEIN | MESSENGER-RNA | BONE METASTASES | PATHWAY | MULTIDISCIPLINARY SCIENCES | BINDING PROTEIN | CAMP | Receptors, Transforming Growth Factor beta - genetics | Coculture Techniques | Humans | Smad3 Protein - metabolism | Phosphoproteins - metabolism | Breast Neoplasms - metabolism | Smad3 Protein - genetics | Mesenchymal Stromal Cells - cytology | Cyclin-Dependent Kinase Inhibitor p21 - genetics | RNA Interference | Mitogen-Activated Protein Kinase 1 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Female | Cell Culture Techniques | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Cyclic AMP - metabolism | Protein-Serine-Threonine Kinases - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Colforsin - pharmacology | Protein-Serine-Threonine Kinases - genetics | Mesenchymal Stromal Cells - metabolism | Phosphoproteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Transforming Growth Factor beta - pharmacology | Breast Neoplasms - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Receptors, Transforming Growth Factor beta - metabolism | Breast Neoplasms - pathology | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Cell Proliferation - drug effects | Adaptor Proteins, Signal Transducing - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Cell proliferation | Phosphorylation | Mesenchyme | Smad3 protein | Metastasis | Kinases | Proteins | Signal transduction | Pathways | Cellular communication | Acetylation | Growth factors | Immunoglobulins | AMP | Cyclic AMP | Cultures | Breast cancer | Cyclic AMP response element-binding protein | Gene expression | Yes-associated protein | Signaling | Stem cells | Reagents | Biomarkers | Breast | Cancer | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2005, Volume 7, Issue 7, pp. 698 - 705
WSB-1 is a SOCS-box-containing WD-40 protein of unknown function that is induced by Hedgehog signalling in embryonic structures during chicken development.... 
COMPLEX | TYPE-2 IODOTHYRONINE DEIODINASE | PROTEIN | BONE-FORMATION | RETICULUM-ASSOCIATED DEGRADATION | ENDOPLASMIC-RETICULUM | PROLIFERATION | SOCS | INDIAN HEDGEHOG | DIFFERENTIATION | CELL BIOLOGY | Gene Expression - genetics | Growth Plate - embryology | Humans | Ubiquitin - metabolism | Ubiquitin-Protein Ligases - physiology | Elongin | Trans-Activators - physiology | Hedgehog Proteins | RNA Interference | Iodide Peroxidase - metabolism | Trans-Activators - pharmacology | Protein Subunits - genetics | Amino Acid Sequence | Parathyroid Hormone-Related Protein - secretion | Ubiquitin-Protein Ligases - metabolism | Models, Molecular | Chick Embryo | Models, Biological | Mice | Iodide Peroxidase - genetics | Mutation | Ubiquitin-Protein Ligases - genetics | Immunoprecipitation | Thyroid Hormones - pharmacology | Gene Expression - drug effects | Molecular Sequence Data | Protein Subunits - metabolism | Chondrocytes - drug effects | In Situ Hybridization | Transfection | Trans-Activators - genetics | Tibia - metabolism | Protein Subunits - physiology | Cell Differentiation - physiology | Chondrocytes - metabolism | beta-Transducin Repeat-Containing Proteins - genetics | Proteins - physiology | Binding Sites - genetics | Growth Plate - metabolism | Tibia - drug effects | Proteins - genetics | Sequence Homology, Amino Acid | Tibia - cytology | Transcription Factors - metabolism | Two-Hybrid System Techniques | Animals | Proteins - metabolism | Cell Differentiation - drug effects | Thyroid Hormones - metabolism | Protein Binding | Physiological aspects | Research | Thyroid hormones | Gene expression | Ligases | Index Medicus | Life Sciences | Biochemistry, Molecular Biology
Journal Article
Developmental Cell, ISSN 1534-5807, 2004, Volume 6, Issue 2, pp. 303 - 309
The atypical PKCs (aPKCs) have been implicated genetically in at least two independent signaling cascades that control NF-κB and cell polarity, through the... 
INTERLEUKIN-1 | TARGETED DISRUPTION | TRAF6 | NF-KAPPA-B2 | KINASE-C ISOFORMS | DEVELOPMENTAL BIOLOGY | PAGETS-DISEASE | ZETA-PKC | BONE | OSTEOPETROSIS | NF-KAPPA-B | CELL BIOLOGY | Blotting, Southern | Protein Kinase C - genetics | Osteogenesis - physiology | Embryo, Mammalian | Cell Count | Immunoblotting | NF-kappa B - metabolism | Bone Remodeling - physiology | Osteoprotegerin | Immediate-Early Proteins - metabolism | Time Factors | Interleukin-6 - blood | Protein Kinase C - metabolism | Gene Expression - physiology | Macrophage Colony-Stimulating Factor - pharmacology | Electrophoretic Mobility Shift Assay | Bone Marrow | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Glycoproteins - genetics | Bone Resorption | Enzyme-Linked Immunosorbent Assay | Cells, Cultured | Receptors, Cytoplasmic and Nuclear - physiology | Receptors, Cytoplasmic and Nuclear - genetics | I-kappa B Kinase | Glycoproteins - physiology | Osteoclasts - metabolism | Parathyroid Hormone-Related Protein - metabolism | Precipitin Tests | Mice, Knockout | Carrier Proteins - genetics | Macrophages - metabolism | TNF Receptor-Associated Factor 6 | Animals | Carrier Proteins - metabolism | Immediate-Early Proteins - genetics | Proteins - metabolism | Receptors, Tumor Necrosis Factor | Fibroblasts - drug effects | Hematoxylin - metabolism | Mice | Transcription Factors | Protein Kinase C-epsilon | RANK-L protein | TRAF6 protein | p62 protein | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2014, Volume 124, Issue 4, pp. 1646 - 1659
Bone metastasis is a frequent complication of breast cancer that is often accelerated by TGF-beta signaling; however, little is known about how the TGF-beta... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | TGF-BETA | PROTEIN | PHOSPHORYLATION | GROWTH | GENES | KINASE | TUMOR-SUPPRESSOR | RHO-GTPASES | EXPRESSION | Tumor Suppressor Proteins - antagonists & inhibitors | Humans | Bone Neoplasms - secondary | Tumor Microenvironment | Smad3 Protein - metabolism | GTPase-Activating Proteins - metabolism | Mammary Neoplasms, Experimental - metabolism | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Gene Knockdown Techniques | Tumor Suppressor Proteins - genetics | Parathyroid Hormone-Related Protein - antagonists & inhibitors | Female | Osteoclasts - pathology | Tumor Suppressor Proteins - metabolism | Signal Transduction | GTPase-Activating Proteins - antagonists & inhibitors | Parathyroid Hormone-Related Protein - genetics | Osteoclasts - metabolism | Parathyroid Hormone-Related Protein - metabolism | Animals | Bone Neoplasms - prevention & control | rho GTP-Binding Proteins - metabolism | Cell Line, Tumor | Mice | Mice, Inbred BALB C | GTPase-Activating Proteins - genetics | Transforming Growth Factor beta - metabolism | Hormone antagonists | Complications and side effects | Care and treatment | Parathyroid hormone | Physiological aspects | Genetic aspects | Breast cancer | Research | Metastasis | Transforming growth factors | Immunization | Biomedical research | Kinases | Gene expression | Patients | Proteins | Survival analysis | Lungs | Plasmids | Cytoskeleton | Laboratory animals | Tumors | Index Medicus | Abridged Index Medicus
Journal Article
ONCOGENE, ISSN 0950-9232, 09/2012, Volume 31, Issue 37, pp. 4150 - 4163
MicroRNAs (miRNAs) as modulators of gene expression have been described to display both tumor-promoting and tumor-suppressive functions. Although their role... 
ACTIVATION | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | miR-520c | breast cancer | NF-kappaB | PROLIFERATION | MICRORNAS | ER-negative | CELL BIOLOGY | TGF-beta | INVASION | miR-373 | ONCOLOGY | EMBRYONIC STEM-CELLS | GENETICS & HEREDITY | BONE METASTASIS | GROWTH-FACTOR | UP-REGULATION | EXPRESSION | Receptors, Estrogen - metabolism | Receptors, Transforming Growth Factor beta - genetics | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | NF-kappa B - metabolism | Gene Expression Profiling | Receptors, Estrogen - deficiency | Transcription Factor RelA - genetics | Breast Neoplasms - metabolism | Parathyroid Hormone-Related Protein - biosynthesis | Angiopoietin-like 4 Protein | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Female | Protein-Serine-Threonine Kinases - metabolism | Angiopoietins - biosynthesis | Signal Transduction | Interleukin-8 - biosynthesis | Protein-Serine-Threonine Kinases - genetics | Parathyroid Hormone-Related Protein - metabolism | Neoplasm Metastasis - genetics | Plasminogen Activator Inhibitor 1 - biosynthesis | Breast Neoplasms - genetics | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - metabolism | Transcription Factor RelA - metabolism | Cell Line, Tumor | Interleukin-6 - biosynthesis | Transforming Growth Factor beta - metabolism | Index Medicus | Tumor cells | Estrogen receptors | Breast cancer | Genomes | Inflammation | Transforming growth factor- beta | Gene expression | Lymph nodes | Metastases | Parathyroid hormone-related protein | plasminogen | Signal transduction | NF- Kappa B protein | Tumor suppressor genes | miRNA | RelA protein | Interleukin 8 | Tumors
Journal Article