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PLoS pathogens, ISSN 1553-7374, 2013, Volume 9, Issue 11, p. e1003773
Interferons (IFNs) are a group of cytokines with a well-established antiviral function. They can be induced by viral infection, are secreted and bind to... 
CELLS | DEFENSE | VIRUS-INFECTION | DISTINCT | RIG-I | MICROBIOLOGY | IRF-7 | ANTIVIRAL RESPONSE | VIROLOGY | GENE | SIGNALING PATHWAY | ADAPTER PROTEIN | PARASITOLOGY | Epithelial Cells - metabolism | Influenza A virus - genetics | Respiratory Mucosa - virology | Interferon Regulatory Factor-7 - genetics | Interferon Type I - immunology | Interferon Regulatory Factor-3 - genetics | Interleukins - metabolism | Orthomyxoviridae Infections - genetics | Respiratory Mucosa - pathology | Interleukins - genetics | Interleukins - immunology | Respiratory Mucosa - immunology | Adaptor Proteins, Signal Transducing - immunology | Interferon Type I - metabolism | Influenza A virus - immunology | Membrane Proteins - metabolism | Interferon Regulatory Factor-3 - immunology | Nerve Tissue Proteins - immunology | Membrane Proteins - genetics | Orthomyxoviridae Infections - metabolism | Epithelial Cells - pathology | Membrane Proteins - immunology | Interferon Regulatory Factor-7 - immunology | Interferon Regulatory Factor-7 - metabolism | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Influenza A virus - metabolism | Epithelial Cells - immunology | Epithelial Cells - virology | Adaptor Proteins, Signal Transducing - genetics | Interferon Regulatory Factor-3 - metabolism | Interferon Type I - genetics | Mice | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Orthomyxoviridae Infections - immunology | Epithelial cells | Influenza | Physiological aspects | Host-parasite relationships | Interferon | Genetic aspects | Genetic transcription | Research | Health aspects | Airway (Medicine) | Interleukins/metabolism | Nerve Tissue Proteins/immunology | Orthomyxoviridae Infections/genetics | Orthomyxoviridae Infections/metabolism | Membrane Proteins/genetics | Adaptor Proteins, Signal Transducing/genetics | Interferon Regulatory Factor-7/genetics | Interleukins/immunology | Membrane Proteins/immunology | Life Sciences | Orthomyxoviridae Infections/immunology | Influenza A virus/genetics | Nerve Tissue Proteins/metabolism | Respiratory Mucosa/immunology | Immunology | Interferon Type I/immunology | Epithelial Cells/immunology | Epithelial Cells/virology | Interferon Regulatory Factor-7/immunology | Epithelial Cells/metabolism | Interferon Regulatory Factor-3/immunology | Respiratory Mucosa/pathology | Influenza A virus/immunology | Interleukins/genetics | Interferon Regulatory Factor-3/genetics | Influenza A virus/metabolism | Adaptor Proteins, Signal Transducing/metabolism | Epithelial Cells/pathology | Adaptor Proteins, Signal Transducing/immunology | Interferon Type I/genetics | Interferon Regulatory Factor-3/metabolism | Membrane Proteins/metabolism | Nerve Tissue Proteins/genetics | Interferon Regulatory Factor-7/metabolism | Interferon Type I/metabolism | Respiratory Mucosa/metabolism | Respiratory Mucosa/virology | Cytokines | Genes | Rodents | Genomics | Genomes | Kinases | Experiments | Viral infections
Journal Article
Molecular plant-microbe interactions, ISSN 0894-0282, 1987
Journal
PLoS genetics, ISSN 1553-7404, 2010, Volume 6, Issue 2, p. e1000827
Journal Article
PLoS Pathogens, ISSN 1553-7366, 05/2011, Volume 7, Issue 5, p. e1002027
During infection Neisseria meningitidis (Nm) encounters multiple environments within the host, which makes rapid adaptation a crucial factor for meningococcal... 
FACTOR-H-BINDING | MICROBIOLOGY | BACTERIAL | GENOME | PATHOGENIC NEISSERIAE | VACCINE CANDIDATES | VIROLOGY | OUTER-MEMBRANE PROTEIN | GENE-EXPRESSION | STAPHYLOCOCCUS-AUREUS | SERINE-PROTEASE | GROUP-B | PARASITOLOGY | Adaptation, Physiological | Sequence Deletion | Virulence Factors - genetics | Genes, Bacterial - genetics | Humans | Neisseria meningitidis, Serogroup B - physiology | Transcriptome | Male | Neisseria meningitidis, Serogroup B - genetics | Meningococcal Infections - microbiology | Antigens, Bacterial - genetics | Bacteremia - microbiology | Bacteremia - blood | RNA, Bacterial - genetics | Adult | Female | Neisseria meningitidis, Serogroup B - pathogenicity | Bacterial Proteins - genetics | Up-Regulation - genetics | Down-Regulation - genetics | Neisseria meningitidis, Serogroup B - growth & development | Genome, Bacterial - genetics | Gene Expression Regulation, Bacterial - genetics | Host-Pathogen Interactions - genetics | Models, Biological | Meningococcal Infections - blood | Cluster Analysis | Causes of | Sepsis | Neisseria meningitidis | Genetic aspects | Research | Health aspects | Microbiology | Pathogenesis | Mortality | Genomes | Gene expression | Blood | Proteins | Studies | Evolutionary biology | Mutagenesis | DNA methylation | Meningitis | Deoxyribonucleic acid--DNA | Deoxyribonucleic acid | DNA
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2014, Volume 111, Issue 31, pp. 11491 - 11496
.... Genetic transfer of these mutations into the human TB agent, Mycobacterium tuberculosis, resulted in downregulation of the PhoP regulon, with loss of biologically active lipids, reduced secretion... 
Mycobacterium | Tuberculosis | Mycobacterium tuberculosis | Mycobacterium bovis | Secretion | Humans | Virulence | Alleles | Lipids | Genetic mutation | Evolution | Phylogeny | Zoonosis | Adaptation | MYCOBACTERIUM-TUBERCULOSIS | COMPLEX | UNITED-KINGDOM | MULTIDISCIPLINARY SCIENCES | CATTLE | adaptation | evolution | IDENTIFICATION | zoonosis | LIPID BIOSYNTHESIS | GENE-CLUSTER | BOVIS | phylogeny | INFECTION | SECRETION | Conserved Sequence - genetics | Bacterial Proteins - genetics | Tuberculosis - microbiology | Antigens, Bacterial | Bacterial Proteins - secretion | Mutation - genetics | Mycobacterium - genetics | Biological Evolution | Host-Pathogen Interactions | Mycobacterium tuberculosis - pathogenicity | Animals | Tuberculosis - genetics | Virulence - genetics | Cattle | Gene Deletion | Polymorphism, Single Nucleotide - genetics | Mutagenesis, Insertional | Mycobacterium bovis - pathogenicity | Mycobacterium bovis - genetics | Mycobacterium tuberculosis - genetics | Tuberculosis in cattle | Microbiological research | Research | Natural history | Microbial mutation | Mycobacterium tuberculosis / pathogenicity | Tuberculosis / microbiology | Life Sciences | Bacterial Proteins / genetics | Conserved Sequence / genetics | Bacterial Proteins / metabolism | Virulence / genetics | Mycobacterium bovis / pathogenicity | Tuberculosis / genetics | Mycobacterium tuberculosis / genetics | Bacteriology | Mycobacterium bovis / genetics | Microbiology and Parasitology | Polymorphism, Single Nucleotide / genetics | Mutation / genetics | Mycobacterium / genetics | Biological Sciences
Journal Article
PLoS genetics, ISSN 1553-7404, 2010, Volume 6, Issue 10, pp. e1001165 - 10
Conjugation is one mechanism for intra- and inter-species horizontal gene transfer among bacteria. Conjugative elements have been instrumental in many... 
REPAIR SYSTEMS | RECOMBINATION | PSIB GENES | EVOLUTION | ESCHERICHIA-COLI | GENETICS & HEREDITY | TRANSCRIPTION INITIATION | GENE-TRANSFER | ENTRY EXCLUSION | PLASMID R64 | VIBRIO-CHOLERAE | Conjugation, Genetic | Green Fluorescent Proteins - metabolism | Integrons - genetics | Bacteria - metabolism | Vibrio cholerae - genetics | Lac Operon - genetics | Gene Transfer, Horizontal | Green Fluorescent Proteins - genetics | Drug Resistance, Microbial - genetics | Bacteria - genetics | Recombinant Fusion Proteins - metabolism | SOS Response (Genetics) - genetics | Vibrio cholerae - metabolism | DNA, Single-Stranded - genetics | Time Factors | Adaptation, Physiological - genetics | Escherichia coli - genetics | Escherichia coli - metabolism | Plasmids - genetics | Recombinant Fusion Proteins - genetics | Integrases - metabolism | Gene Expression Regulation, Bacterial | Integrases - genetics | Adaptations | Integrase | Pathogens | Recombination | Gene transfer | Transcription | DNA damage | Data processing | Genomes | SOS response | Infection | Antibiotics | Mutagenesis | Bioreactors | Plasmids | Antibiotic resistance | Development | Host range | Conjugation | gene rearrangement | Adaptation, Physiological | Integrons | Green Fluorescent Proteins | Vibrio cholerae | Escherichia coli | Recombinant Fusion Proteins | Integrases | SOS Response, Genetics | Life Sciences | Drug Resistance, Microbial | Bacteria | Genetics | DNA, Single-Stranded | Lac Operon | Cholera | Microbiology | Gene expression | Bacteriology
Journal Article
PLoS genetics, ISSN 1553-7404, 2017, Volume 13, Issue 3, p. e1006705
... and integrative and conjugative elements (ICEs) code for T4SS to promote their own dissemination by conjugation. These self-transmissible mobile genetic elements often bear... 
PAIR STABILIZATION | INCA/C PLASMIDS | GENE | TYPHIMURIUM DT104 | ESCHERICHIA-COLI | GENETICS & HEREDITY | ANTIBIOTIC-RESISTANCE | IV SECRETION | SYSTEMS | F-PLASMID | EXCISION | Genomic Islands - genetics | Salmonella enterica - pathogenicity | Salmonella enterica - genetics | Salmonella enterica - growth & development | Salmonella enterica - drug effects | Interspersed Repetitive Sequences - genetics | DNA, Bacterial - genetics | Chromosomes - genetics | Escherichia coli - genetics | Gene Expression Regulation, Developmental | Plasmids - genetics | Drug Resistance, Multiple, Bacterial - genetics | Sequence Deletion - genetics | Salmonella | Drug resistance in microorganisms | Promoters (Genetics) | Analysis | Genetic aspects | Dosage and administration | Research | Genetic transcription | Carbapenems | Transformation | Colleges & universities | Drug resistance | Assaying | Business competition | Proteins | T-cell receptor | Genetics | Complementation | Deoxyribonucleic acid--DNA | Data acquisition | Redundancy | Nucleic acids | Gene expression | Membrane proteins | Polymerase chain reaction | Collection | Conjugation | Mutation | Cargo | Populations | Transcription | Propagation | Microbiology | Genes | Clinical isolates | Homology | Genomes | R factors | Defects | Deletion mutant | Arabinose | E coli | Clonal deletion | Modulation | Antibiotic resistance | Bacteria | Islands | Expression vectors | Cassettes | Nucleotide sequence | Secretion | Multidrug resistance | Electroporation | Data processing | Grants | Antibiotics | Plasmids | Host systems | Gene mapping | Deoxyribonucleic acid | DNA
Journal Article
Nature immunology, ISSN 1529-2916, 2010, Volume 11, Issue 5, pp. 411 - 418
Sensors of pathogens, such as Toll-like receptors (TLRs), detect microbes to activate transcriptional programs that orchestrate adaptive responses to specific... 
UNFOLDED PROTEIN RESPONSE | PATHWAY | ER STRESS | HOST-DEFENSE | ENDOPLASMIC-RETICULUM STRESS | FRANCISELLA-TULARENSIS INFECTION | IMMUNOLOGY | LIVE VACCINE STRAIN | INFLAMMATORY RESPONSE | NEGATIVE REGULATOR | SYSTEMS BIOLOGY | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis
Journal Article
PLoS genetics, ISSN 1553-7404, 2012, Volume 8, Issue 7, p. e1002784
Journal Article