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PLoS ONE, ISSN 1932-6203, 10/2017, Volume 12, Issue 10, p. e0186227
Therapeutic agents to the central nervous system (CNS) need to be efficiently delivered to the target site of action at appropriate therapeutic levels.... 
CLEARANCE | OXIDATIVE STRESS | PROTEIN-SYNTHESIS SYSTEM | SUBSTRATE | MULTIDISCIPLINARY SCIENCES | MODEL | EXPRESSION | BRAIN | PEPTIDE | PROGRESSION | Neurons - pathology | Neuroprotective Agents - chemistry | Small Molecule Libraries - administration & dosage | Humans | Central Nervous System - pathology | Imidazoles - administration & dosage | Small Molecule Libraries - chemistry | Animals | alpha-2-HS-Glycoprotein - chemistry | Cattle | Hemopexin - metabolism | Central Nervous System - drug effects | alpha-2-HS-Glycoprotein - metabolism | Mice | Cell Death - drug effects | Hemopexin - chemistry | Oxidative Stress - drug effects | Culture Media - chemistry | Neurons - drug effects | Neuroprotective Agents - administration & dosage | Acetamides - administration & dosage | Care and treatment | Nervous system diseases | Cell death | Amyotrophic lateral sclerosis | Glycoproteins | Research | Diagnosis | Health aspects | Drugs | Neuroprotection | Cell culture | Oxidative stress | Animal models | Biomedical research | Laboratories | Central nervous system | Hemopexin | Neuroblastoma | Serum proteins | Neuroprotective agents | Proteins | Blood-brain barrier | Rodents | Drug dosages | Injuries | Effectiveness | Glycoprotein | Pharmacology | Chemical compounds | Glycan | Neurological diseases | Medicine | Chemistry | Brain research | Apoptosis
Journal Article
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1279 - 1285
Toll-like receptor 4 (TLR4) has a key role in innate immunity by activating an inflammatory signaling pathway. Free fatty acids (FFAs) stimulate adipose tissue... 
PATHWAYS | MEDICINE, RESEARCH & EXPERIMENTAL | ADIPOCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR-KAPPA-B | SATURATED FATTY-ACIDS | DIET-INDUCED OBESITY | CELL BIOLOGY | TOLL-LIKE RECEPTOR-4 | CHRONIC INFLAMMATION | EXPRESSION | ADIPOSE-TISSUE | INNATE IMMUNITY | Inflammation - chemically induced | Humans | Molecular Sequence Data | NF-kappa B - metabolism | Adipocytes - drug effects | Fatty Acids, Nonesterified - pharmacology | Adipocytes - physiology | Obesity - blood | Insulin Resistance - physiology | Base Sequence | alpha-2-HS-Glycoprotein - antagonists & inhibitors | Toll-Like Receptor 4 - antagonists & inhibitors | Binding Sites | Cytokines - genetics | Toll-Like Receptor 4 - analysis | Blood Glucose - analysis | Obesity - physiopathology | Toll-Like Receptor 4 - chemistry | 3T3-L1 Cells | alpha-2-HS-Glycoprotein - physiology | Mice, Knockout | Gene Expression Regulation - drug effects | Toll-Like Receptor 4 - physiology | Receptors, Leptin - deficiency | Diabetes Mellitus, Type 2 - blood | alpha-2-HS-Glycoprotein - toxicity | Animals | Diabetes Mellitus, Type 2 - physiopathology | alpha-2-HS-Glycoprotein - chemistry | Signal Transduction - drug effects | Galactosides - metabolism | Ligands | Mice, Obese | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | Chronic Disease | Dietary Fats - toxicity | Cytokines - biosynthesis | Adipocytes - chemistry | Inflammation - physiopathology | Insulin resistance | Alpha fetoproteins | Care and treatment | Diagnosis | Health aspects | Ligands (Biochemistry) | Signal transduction | Lipids | Cytokines | Gene expression | Immune system
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, p. e16945
Background: A liver-derived protein, fetuin-A, was first purified from calf fetal serum in 1944, but its potential role in lethal systemic inflammation was... 
EXPERIMENTAL SEPSIS | ACUTE-PHASE PROTEIN | IFN-GAMMA | BOVINE FETUIN | TYROSINE KINASE | APOPTOTIC CELLS | BIOLOGY | HMGB1 RELEASE | MOBILITY GROUP BOX-1 | HUMAN ALPHA-2-HS GLYCOPROTEIN | BACTERIAL-DNA | alpha-2-HS-Glycoprotein - deficiency | Endotoxemia - prevention & control | Liver | Cytoplasm - metabolism | Male | Endotoxemia - pathology | HMGB1 Protein - secretion | Time Factors | HMGB1 Protein - metabolism | Systemic Inflammatory Response Syndrome - prevention & control | alpha-2-HS-Glycoprotein - pharmacology | Cell Line | Systemic Inflammatory Response Syndrome - metabolism | Disease Susceptibility | Cytokines - metabolism | Acute-Phase Proteins - metabolism | Gene Expression Regulation | Gene Knockout Techniques | Macrophages - metabolism | Systemic Inflammatory Response Syndrome - pathology | Animals | alpha-2-HS-Glycoprotein - metabolism | Macrophages - drug effects | Mice | Endotoxemia - metabolism | Cytoplasm - drug effects | alpha-2-HS-Glycoprotein - genetics | Inflammation | Chromosomal proteins | Comparative analysis | Pathogenesis | Critical care | Emergency medical care | Macrophages | Dosage | HMGB1 protein | Accumulation | Lipopolysaccharides | Proteins | Rodents | Extracellular matrix | Tumor necrosis factor-TNF | Physiology | Inhibition | Supplementation | Endotoxemia | Medical research | Cell survival | Cytokines | Cecum | Survival | Medicine | Pathology | Hospitals | Infectious diseases | Molecular modelling | γ-Interferon | Sepsis | Interferon | Disruption | Peritoneum | Apoptosis
Journal Article
Atherosclerosis, ISSN 0021-9150, 2015, Volume 241, Issue 1, pp. 130 - 137
Abstract Objective Adipose Tissue (AT) dysregulation contributes to the pro-inflammatory state and insulin resistance of Metabolic Syndrome (MetS). We examined... 
Cardiovascular | Adipose tissue | TLR | Metabolic syndrome | Lipopolysaccharide binding protein | Fetuin A | CARDIAC & CARDIOVASCULAR SYSTEMS | MARKER | ENDOTOXIN | MONOCYTES | HUMANS | OBESITY | PATTERN | INSULIN-RESISTANCE | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | RECEPTOR-ACTIVITY | EXPRESSION | Up-Regulation | alpha-2-HS-Glycoprotein - analysis | Carrier Proteins - secretion | Humans | Middle Aged | Male | Acute-Phase Proteins - genetics | RNA, Messenger - metabolism | Case-Control Studies | HMGB1 Protein - genetics | HMGB1 Protein - secretion | Young Adult | Metabolic Syndrome - blood | Subcutaneous Fat - metabolism | Adiposity | Subcutaneous Fat - secretion | HMGB1 Protein - metabolism | Metabolic Syndrome - physiopathology | Adult | Female | Adipogenesis | Disease Models, Animal | Carrier Proteins - blood | Acute-Phase Proteins - secretion | Mice, Inbred C57BL | alpha-2-HS-Glycoprotein - secretion | Membrane Glycoproteins - blood | Obesity - physiopathology | Biomarkers - blood | 3T3-L1 Cells | Lipopolysaccharide Receptors - blood | Membrane Glycoproteins - genetics | Obesity - metabolism | Carrier Proteins - genetics | Membrane Glycoproteins - secretion | Animals | Toll-Like Receptor 4 - blood | Metabolic Syndrome - diagnosis | Metabolic Syndrome - genetics | Adipocytes - metabolism | Toll-Like Receptor 2 - blood | alpha-2-HS-Glycoprotein - metabolism | Mice, Obese | Aged | Mice | alpha-2-HS-Glycoprotein - genetics | HMGB1 Protein - blood | Adipose tissues | Medical colleges | Insulin resistance | Mitogens | Atherosclerosis | Protein binding
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e60904
The formation of fetuin-A-containing calciprotein particles (CPP) may facilitate the clearance of calcium phosphate nanocrystals from the extracellular fluid.... 
MOLECULAR-MECHANISM | TUMOR-NECROSIS-FACTOR | PROTEIN-KINASE-C | IN-VITRO | CALCIUM-PHOSPHATE CRYSTALS | MULTIDISCIPLINARY SCIENCES | VASCULAR CALCIFICATION | HUMAN FIBROBLASTS | SMOOTH-MUSCLE-CELLS | SCAVENGER RECEPTOR EXPRESSION | GLYCOPROTEIN/FETUIN-A | Cell Line | Cell Survival - drug effects | Cytokines - metabolism | Apoptosis - drug effects | Humans | Cytokines - secretion | Durapatite - chemistry | Protein Transport - drug effects | Macrophages - cytology | Gene Expression Regulation - drug effects | Macrophages - metabolism | Macrophages - secretion | Animals | alpha-2-HS-Glycoprotein - chemistry | Uremia - blood | Durapatite - pharmacology | alpha-2-HS-Glycoprotein - metabolism | Macrophages - drug effects | Scavenger Receptors, Class A - genetics | Mice | Minerals - metabolism | Oxidative Stress - drug effects | Phosphorylation - drug effects | alpha-2-HS-Glycoprotein - pharmacology | Stress management | Calcium phosphate | Macrophages | Stress (Psychology) | Calcification (ectopic) | Phosphates | Crystal growth | Phosphorylation | Calcium | Pathogenesis | Interleukin | Smooth muscle | Hydroxyapatite | Kinases | Peritoneal dialysis | Proteins | Mineralization | Rodents | Cascades | Tumor necrosis factor-TNF | Reticuloendothelial system | Secretion | Glycoprotein | Crystals | Particulates | Glycoproteins | Inflammation | Tumor necrosis factor-α | IL-1β | Chromatography | Calcium phosphates | Studies | Nanocrystals | Calcification | TNF inhibitors | Kidney diseases | Viability | Tumors | Apoptosis
Journal Article
Nature Reviews Endocrinology, ISSN 1759-5029, 03/2013, Volume 9, Issue 3, pp. 144 - 152
The liver is known to be involved in the natural history of the ongoing epidemics of type 2 diabetes mellitus and cardiovascular disease. In particular, the... 
INCIDENT DIABETES-MELLITUS | LIFE-STYLE INTERVENTION | FETUIN-A LEVELS | GLYCOPROTEIN GENE | FATTY LIVER-DISEASE | TYROSINE KINASE |