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Nature Medicine, ISSN 1078-8956, 12/2010, Volume 16, Issue 12, pp. 1400 - 1406
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2011, Volume 108, Issue 11, pp. 4274 - 4280
Glioblastoma (GBM) is the most malignant brain tumor and is highly resistant to intensive combination therapies and anti-VEGF therapies. To assess the... 
Angiogenesis | Myeloid cells | Cell lines | Blood vessels | Antibodies | Cultured cells | Mice | Endothelial cells | Tumors | Cancer | Hypoxia-inducible factor 1 | Mouse model | Glioma | GLIOMAS | VASCULOGENIC MIMICRY | mouse model | BEVACIZUMAB | angiogenesis | MULTIDISCIPLINARY SCIENCES | MOUSE | HYDROXYLATION | TUMORS | STEM-LIKE CELLS | THERAPY | hypoxia-inducible factor 1 | glioma | DIFFERENTIATION | HIF-ALPHA | Endothelial Cells - metabolism | Humans | Spheroids, Cellular - metabolism | Spheroids, Cellular - pathology | Vascular Endothelial Growth Factor A - metabolism | Cell Hypoxia | Xenograft Model Antitumor Assays | Cell Fusion | Receptor, Epidermal Growth Factor - metabolism | Animals | Receptors, Vascular Endothelial Growth Factor - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glioblastoma - pathology | Biomarkers, Tumor - metabolism | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Cell Transdifferentiation | Glioblastoma - metabolism | Neovascularization, Pathologic - metabolism | Tumor Cells, Cultured | Endothelial Cells - pathology | Disease Models, Animal | Transdifferentiation | Cancer cells | Diagnosis | Neovascularization | Research | Properties | Glioblastoma multiforme | Brain | Signal transduction | Transplants & implants | Rodents | Hypoxia | Vascular endothelial growth factor | Cancer therapies | Cells | Index Medicus | Biological Sciences
Journal Article
American Journal of Hematology, ISSN 0361-8609, 06/2013, Volume 88, Issue 6, pp. 517 - 517
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 511, Issue 7509, pp. 312 - 318
Journal Article
Nature Cell Biology, ISSN 1465-7392, 2012, Volume 14, Issue 3, pp. 249 - 256
The shear-responsive transcription factor Kruppel-like factor 2 (KLF2) is a critical regulator of endothelial gene expression patterns induced by... 
MECHANISM | ANGIOGENESIS | IN-VIVO | DISEASE | MICRORNAS | MICROPARTICLES | TRANSCRIPTION FACTOR | EXPRESSION | KLF2 | FLOW | CELL BIOLOGY | Exosomes - metabolism | Apolipoproteins E - deficiency | Human Umbilical Vein Endothelial Cells - metabolism | Lovastatin - pharmacology | Atherosclerosis - genetics | Coculture Techniques | Humans | Endothelial Cells - transplantation | Stress, Mechanical | MicroRNAs - metabolism | Lovastatin - analogs & derivatives | Aorta - metabolism | Transfection | Kruppel-Like Transcription Factors - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Exosomes - ultrastructure | Atherosclerosis - pathology | Exosomes - transplantation | Endothelial Cells - metabolism | Cells, Cultured | Signal Transduction - genetics | Microscopy, Electron | Reverse Transcriptase Polymerase Chain Reaction | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Gene Expression Regulation - drug effects | Microscopy, Confocal | Animals | Endothelial Cells - cytology | Apolipoproteins E - genetics | Mice | MicroRNAs - genetics | Kruppel-Like Transcription Factors - genetics | Transcription factors | MicroRNA | Atherosclerosis | Physiological aspects | Genetic aspects | Research | Gene expression | Risk factors | Index Medicus
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 11/2012, Volume 303, Issue 9, pp. C991 - C999
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 11/2009, Volume 297, Issue 5, pp. 1876 - 1881
The production of hyperglycemia-induced mitochondrial reactive oxygen species (mtROS) is a key event in the development of diabetic complications. Because... 
Histone deacetylase | Vasoprotection | Sirtuin 1 | sirtuin 1 | LIFE-SPAN | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | SUPEROXIDE-PRODUCTION | vasoprotection | histone deacetylase | LIVE CELLS | SIRT1 | SKELETAL-MUSCLE | CALORIC RESTRICTION | IN-VIVO | PERIPHERAL VASCULAR DISEASE | HYDROGEN-PEROXIDE | ENERGY-METABOLISM | SMALL-MOLECULE ACTIVATORS | Reactive Oxygen Species - metabolism | Glutathione - metabolism | Humans | Chromatography, High Pressure Liquid | Stilbenes - pharmacology | Hyperglycemia - drug therapy | Coronary Vessels - metabolism | Dose-Response Relationship, Drug | Transfection | RNA Interference | Sirtuins - genetics | Superoxide Dismutase - metabolism | Coronary Vessels - drug effects | Endothelial Cells - metabolism | Cells, Cultured | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Hydrogen Peroxide - metabolism | Hyperglycemia - metabolism | Biosensing Techniques | Glucose - metabolism | Oxidative Stress - drug effects | Sirtuins - metabolism | Microscopy, Fluorescence | Endothelial Cells - drug effects | Oxidative stress | Resveratrol | Physiological aspects | Histones | Vascular endothelium | Dosage and administration | Mitochondrial DNA | Research | Proteins | Cell culture | Coronary vessels | Fluorescence | Physiology | Diabetes | Glucose | Gene expression | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 04/2015, Volume 520, Issue 7546, pp. 192 - 197
The metabolism of endothelial cells during vessel sprouting remains poorly studied. Here we report that endothelial loss of CFT1A, a rate-limiting enzyme of... 
OXIDATION | DISTRIBUTIONS | GLYCOLYSIS | REDUCTIVE GLUTAMINE-METABOLISM | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | TUMOR | PROLIFERATION | GROWTH-FACTOR | CANCER | CULTURE | Carnitine O-Palmitoyltransferase - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Neovascularization, Pathologic - pathology | Nucleotides - chemistry | Carnitine O-Palmitoyltransferase - antagonists & inhibitors | Adenosine Triphosphate - metabolism | Carnitine O-Palmitoyltransferase - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Nucleotides - biosynthesis | Nucleotides - pharmacology | Oxidation-Reduction - drug effects | DNA - biosynthesis | Carnitine O-Palmitoyltransferase - deficiency | Fatty Acids - metabolism | Disease Models, Animal | Carbon - metabolism | Fatty Acids - chemistry | Human Umbilical Vein Endothelial Cells - drug effects | Endothelial Cells - metabolism | Gene Silencing | Acetic Acid - pharmacology | Blood Vessels - cytology | Citric Acid Cycle | Animals | Retinopathy of Prematurity - drug therapy | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Blood Vessels - drug effects | Cell Line, Tumor | Glucose - metabolism | Human Umbilical Vein Endothelial Cells - pathology | Cell Proliferation - drug effects | Mice | Neovascularization, Pathologic - metabolism | Retinopathy of Prematurity - metabolism | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Retinopathy of Prematurity - pathology | Physiological aspects | Deoxyribonucleotides | Fatty acids | Carbon | Endothelium | Proteins | Angiogenesis | Protein synthesis | Glucose | Kinases | Deoxyribonucleic acid--DNA | Cells | Index Medicus
Journal Article