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The New England Journal of Medicine, ISSN 0028-4793, 09/2010, Volume 363, Issue 14, pp. 1324 - 1334
Journal Article
Trends in Immunology, ISSN 1471-4906, 2015, Volume 36, Issue 10, pp. 651 - 662
Multiple sclerosis (MS) is the prototypical inflammatory disease of the central nervous system (CNS). MS lesions harbor different immune cells, but the... 
Allergy and Immunology | monocytes | CNS | tissue damage | GM-CSF | neuroinflammation | Neuroinflammation | Monocytes | Tissue damage | RHEUMATOID-ARTHRITIS | CONTROLLED PHASE-3 | REMITTING MULTIPLE-SCLEROSIS | COLONY-STIMULATING FACTOR | DENDRITIC CELLS | BONE-MARROW | IN-VIVO | INTERFERON-BETA | IMMUNOLOGY | T-CELLS | INFLAMMATORY CYTOKINE | Myeloid Cells - cytology | Central Nervous System Diseases - genetics | Central Nervous System Diseases - pathology | Inflammation - pathology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Central Nervous System Diseases - metabolism | Inflammation - metabolism | Myeloid Cells - immunology | T-Lymphocytes, Helper-Inducer - immunology | Cell Differentiation | Encephalomyelitis, Autoimmune, Experimental - genetics | Multiple Sclerosis - metabolism | Encephalomyelitis, Autoimmune, Experimental - pathology | T-Lymphocytes, Helper-Inducer - metabolism | Gene Expression Regulation | Cell Communication | Central Nervous System Diseases - immunology | Multiple Sclerosis - genetics | Inflammation - immunology | Multiple Sclerosis - therapy | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Animals | Inflammation - genetics | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Myeloid Cells - metabolism | Neuroimmunomodulation | Studies | Antigens | Transcription factors | Target recognition | Disease | Cytokines | Lymphocytes | Inflammation | Gene expression
Journal Article
FRONTIERS IN IMMUNOLOGY, ISSN 1664-3224, 09/2019, Volume 10
Pre-clinical models and clinical trials demonstrate that targeting the action of the cytokine, granulocyte macrophage-colony stimulating factor (GM-CSF), can... 
pain | ACTIVATION | cell survival | COLONY-STIMULATING FACTOR | DENDRITIC CELLS | CYTOKINE | IMMUNOLOGY | inflammation | IRF4 | B-CELLS PROTECT | polarization | FETAL MONOCYTES | GROWTH-FACTOR | DIFFERENTIATION | CCL17 | MACROPHAGE POLARIZATION | HUMAN-MONOCYTES
Journal Article
Cancer Cell, ISSN 1535-6108, 06/2012, Volume 21, Issue 6, pp. 836 - 847
Stromal responses elicited by early stage neoplastic lesions can promote tumor growth. However, the molecular mechanisms that underlie the early recruitment of... 
COLONY-STIMULATING FACTORS | T-CELL RESPONSES | TUMOR PROGRESSION | P21 RAS | ONCOLOGY | DUCTAL ADENOCARCINOMA | INFLAMMATION | MOUSE | MICE | SUPPRESSOR-CELLS | CANCER | CELL BIOLOGY | Immunohistochemistry | Adenocarcinoma - pathology | Epithelial Cells - metabolism | Pancreatic Neoplasms - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Immunoblotting | Green Fluorescent Proteins - genetics | Pancreatic Ducts - metabolism | Carcinoma, Pancreatic Ductal - genetics | Adenocarcinoma - metabolism | RNA Interference | HEK293 Cells | CD8-Positive T-Lymphocytes - metabolism | Adenocarcinoma - genetics | Proto-Oncogene Proteins p21(ras) - metabolism | Green Fluorescent Proteins - metabolism | Pancreatic Ducts - cytology | Mice, Inbred C57BL | Pancreatic Neoplasms - pathology | Receptors, Chemokine - metabolism | Cells, Cultured | Mice, Transgenic | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Animals | Cell Transformation, Neoplastic | Myeloid Cells - metabolism | Mice | CD11b Antigen - metabolism | Microscopy, Fluorescence | T cells | Tumors | granulocyte-macrophage colony stimulating factor | immunosuppression | Kras | cancer | inflammation | pancreas
Journal Article