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Blood, ISSN 0006-4971, 09/2011, Volume 118, Issue 12, pp. e50 - e61
Monocytes are a heterogeneous cell population with subset-specific functions and phenotypes. The differential expression of CD14 and CD16 distinguishes... 
CD16(+) MONOCYTES | SUBPOPULATIONS | HUMAN PERIPHERAL-BLOOD | DIALYSIS PATIENTS | DISEASE | GENE-EXPRESSION PROFILES | INFECTION | ANGIOPOIETIN-2 | IDENTIFICATION | HEMATOLOGY | PREFERENTIALLY HARBORS HIV-1 | Cell Proliferation | Monocytes - cytology | Atherosclerosis - genetics | Humans | RNA, Messenger - analysis | Monocytes - metabolism | Monocytes - immunology | Lipopolysaccharide Receptors - metabolism | HIV Infections - immunology | RNA, Messenger - biosynthesis | Inflammation - metabolism | Flow Cytometry | HIV-1 - physiology | Neovascularization, Pathologic - immunology | Monocytes - classification | Expressed Sequence Tags | Atherosclerosis - immunology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Neovascularization, Physiologic - genetics | HIV Infections - genetics | HIV Infections - virology | Antigen Presentation - genetics | Gene Expression Profiling - methods | Lipopolysaccharide Receptors - immunology | Inflammation - immunology | Antigen Presentation - immunology | Neovascularization, Physiologic - immunology | Atherosclerosis - metabolism | HIV-1 - immunology | Inflammation - genetics | Lipopolysaccharide Receptors - genetics | Neovascularization, Pathologic - genetics | High-Throughput Nucleotide Sequencing | Immunity - genetics | HIV Infections - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
EMBO reports, ISSN 1469-221X, 09/2015, Volume 16, Issue 9, pp. 1203 - 1218
S treptococcus pneumoniae coinfection is a major cause of influenza‐associated mortality; however, the mechanisms underlying pathogenesis or protection remain... 
influenza | related apoptosis‐inducing ligand | treptococcus pneumoniae | 2 | CCR | S | TRAIL | C‐C‐chemokine receptor type | ) | neutrophil | TNF | TNF-related apoptosis-inducing ligand (TRAIL) | C-C-chemokine receptor type (CCR) 2 | Streptococcus pneumoniae | VIRUS-INFECTION | DENDRITIC CELLS | PANDEMIC INFLUENZA | BIOCHEMISTRY & MOLECULAR BIOLOGY | LETHAL SYNERGISM | NECROSIS-FACTOR-ALPHA | CELL BIOLOGY | BACTERIAL PNEUMONIA | NEGATIVE REGULATION | MOUSE MODEL | PNEUMOCOCCAL PNEUMONIA | TNF-ALPHA | Receptors, CCR2 - genetics | Lung - microbiology | Streptococcus pneumoniae - growth & development | Influenza A Virus, H3N2 Subtype | Humans | Orthomyxoviridae Infections - complications | Monocytes - immunology | Coinfection - physiopathology | Streptococcus pneumoniae - immunology | Lung - virology | Pneumococcal Infections - immunology | Streptococcus pneumoniae - pathogenicity | Tumor Necrosis Factor-alpha - immunology | Cytokines - immunology | Disease Models, Animal | Pneumococcal Infections - complications | Disease Susceptibility | Coinfection - immunology | Mice, Inbred C57BL | Neutrophils - immunology | Lung - physiopathology | TNF-Related Apoptosis-Inducing Ligand - metabolism | Animals | Mice | Receptors, CCR2 - deficiency | Lung - immunology | Orthomyxoviridae Infections - immunology | Ligands | Lungs | Influenza | Streptococcus infections | Apoptosis | Index Medicus
Journal Article
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 01/2017, Volume 101, Issue 1, pp. 339 - 350
Journal Article
Journal Article