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The Journal of Immunology, ISSN 0022-1767, 11/2008, Volume 181, Issue 9, pp. 6644 - 6653
Journal Article
细胞研究:英文版, ISSN 1001-0602, 2012, Volume 22, Issue 3, pp. 457 - 472
Journal Article
The Journal of Pathology, ISSN 0022-3417, 12/2013, Volume 231, Issue 4, pp. 424 - 432
Journal Article
Journal Article
Cellular and Molecular Life Sciences, ISSN 1420-682X, 08/2017, Volume 74, Issue 15, pp. 2761 - 2771
Although surgical excision, chemo-, and radiotherapy are clearly advanced, tumors may relapse due to cells of the so-called "minimal residual disease". Indeed,... 
Dormancy | Tumor microenvironment | Acidosis | CANCER-CELLS | LACTIC-ACID | STEM-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RADIATION-INDUCED APOPTOSIS | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | HUMAN GLIOBLASTOMA CELLS | IN-VIVO | HUMAN-MELANOMA CELLS | PUMP INHIBITOR PANTOPRAZOLE | EXTRACELLULAR PH | Immunologic Surveillance | Neoplasm, Residual - pathology | Acidosis - immunology | Cell Proliferation | Prognosis | Humans | Neoplastic Stem Cells - immunology | Tumor Microenvironment | Killer Cells, Natural - pathology | Neoplasm Recurrence, Local - etiology | Neovascularization, Pathologic - etiology | Neovascularization, Pathologic - pathology | T-Lymphocytes, Cytotoxic - pathology | Neoplasm Recurrence, Local - immunology | Neoplasm Recurrence, Local - pathology | Acidosis - complications | Neoplasms - therapy | Killer Cells, Natural - immunology | Neoplastic Stem Cells - pathology | Neoplasm, Residual - immunology | Neovascularization, Pathologic - immunology | T-Lymphocytes, Cytotoxic - immunology | Animals | Neoplasms - immunology | Acidosis - pathology | Neoplasm, Residual - complications | Neoplasms - pathology | Apoptosis | Hydrogen-Ion Concentration | Chemotherapy | Killer cells | Cancer | Therapy | Target recognition | Tumor cells | Minimal residual disease | Cytotoxicity | Radio | Acidity | Radiation therapy | Tissues | Patients | Cells | Immunology | Acids | Lymphocytes | Surgery | Clusters | Remission | Natural killer cells | Tumors
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2013, Volume 15, Issue 7, pp. 818 - 828
Dysfunctional telomeres suppress tumour progression by activating cell-intrinsic programs that lead to growth arrest. Increased levels of TRF2, a key factor in... 
DAMAGE RESPONSE | PROTEIN | TELOMERE DYSFUNCTION | IN-VIVO | HUMAN-MELANOMA CELLS | ATM | SENESCENCE | HEPARAN-SULFATE PROTEOGLYCANS | UP-REGULATION | EXPRESSION | CELL BIOLOGY | RNA, Small Interfering - genetics | Cell Proliferation | Breast Neoplasms - immunology | Colonic Neoplasms - prevention & control | Humans | Melanoma, Experimental - metabolism | Killer Cells, Natural - pathology | DNA Primers - chemistry | Colonic Neoplasms - metabolism | Breast Neoplasms - metabolism | Melanoma, Experimental - prevention & control | Flow Cytometry | Melanoma, Experimental - immunology | Colonic Neoplasms - immunology | Killer Cells, Natural - immunology | Female | Sulfotransferases - metabolism | Tumor Cells, Cultured | Real-Time Polymerase Chain Reaction | Sulfotransferases - genetics | Telomeric Repeat Binding Protein 2 - antagonists & inhibitors | RNA, Messenger - genetics | Discoidin Domain Receptor 1 | Receptor Protein-Tyrosine Kinases - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion | Blotting, Western | Breast Neoplasms - prevention & control | Animals | Lymphocytes, Tumor-Infiltrating - pathology | Receptor Protein-Tyrosine Kinases - genetics | Mice, Nude | Telomeric Repeat Binding Protein 2 - metabolism | Mice | HeLa Cells | Killer Cells, Natural - metabolism | Telomeric Repeat Binding Protein 2 - genetics | Apoptosis | Lymphocytes, Tumor-Infiltrating - immunology | Killer cells | Colorectal cancer | Cancer cells | Development and progression | Genetic aspects | Research | Health aspects
Journal Article