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The New England Journal of Medicine, ISSN 0028-4793, 03/2012, Volume 366, Issue 10, pp. 914 - 924
Journal Article
European journal of endocrinology / European Federation of Endocrine Societies, ISSN 0804-4643, 2013, Volume 169, Issue 6, pp. 811 - 819
Journal Article
European Journal of Endocrinology, ISSN 0804-4643, 10/2014, Volume 171, Issue 4, pp. 519 - 526
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2011, Volume 121, Issue 12, pp. 4712 - 4721
Cushing disease is a condition in which the pituitary gland releases excessive adrenocorticotropic hormone (ACTH) as a result of an adenoma arising from the... 
NUCLEAR-LOCALIZATION | MEDICINE, RESEARCH & EXPERIMENTAL | DEPENDENT CUSHINGS-SYNDROME | GEFITINIB INDUCES APOPTOSIS | CYCLE INHIBITOR P27(KIP1) | EPIDERMAL-GROWTH-FACTOR | PROOPIOMELANOCORTIN GENE | CORTICOTROPH TUMORS | T-BOX FACTOR | FACTOR RECEPTOR | PROGNOSTIC VALUE | ACTH-Secreting Pituitary Adenoma - drug therapy | Neoplasm Transplantation | Humans | Cell Line, Tumor - drug effects | Molecular Targeted Therapy | Pituitary ACTH Hypersecretion - drug therapy | ACTH-Secreting Pituitary Adenoma - complications | Adenoma - enzymology | Adenoma - complications | Dog Diseases - drug therapy | Dog Diseases - etiology | Secretory Rate - drug effects | Adenoma - drug therapy | Corticosterone - blood | Gene Expression Regulation, Neoplastic - drug effects | Pro-Opiomelanocortin - genetics | Adenoma - veterinary | Promoter Regions, Genetic | Pituitary ACTH Hypersecretion - etiology | ACTH-Secreting Pituitary Adenoma - veterinary | Cell Line, Tumor - enzymology | Tumor Burden | Adrenocorticotropic Hormone - secretion | Pituitary ACTH Hypersecretion - blood | Adenoma - blood | Dog Diseases - blood | ACTH-Secreting Pituitary Adenoma - blood | Animals | Mice, Nude | Protein Kinase Inhibitors - therapeutic use | Quinazolines - therapeutic use | Dogs | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Mice | Protein Kinase Inhibitors - pharmacology | ACTH-Secreting Pituitary Adenoma - enzymology | Pituitary ACTH Hypersecretion - veterinary | Quinazolines - pharmacology | Drug Screening Assays, Antitumor | Care and treatment | Pituitary gland tumors | ACTH | Drug targeting | Adenoma | Diagnosis | Research | Properties | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 05/2013, Volume 110, Issue 21, pp. 8555 - 8560
Cushing disease (CD) is a life-threatening disorder attributed to excess pituitary tumor-derived adrenocorticotrophic hormone (ACTH) and adrenal steroid... 
Cell growth | Phosphorylation | Pituitary neoplasms | Secretion | Plasmids | Cell lines | Pituitary ACTH hypersecretion | Mice | Orphan nuclear receptors | Tumors | MANAGEMENT | ACID | PHOSPHORYLATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | MODULATOR | EXPRESSION | Transcriptional Activation - genetics | Cell Proliferation | Receptors, Steroid - metabolism | Humans | Neoplasm Proteins - metabolism | ACTH-Secreting Pituitary Adenoma - therapy | Adrenocorticotropic Hormone - genetics | Pituitary ACTH Hypersecretion - genetics | ACTH-Secreting Pituitary Adenoma - genetics | Receptors, Thyroid Hormone - genetics | Response Elements - genetics | Corticosterone - secretion | Neoplasm Proteins - genetics | Pro-Opiomelanocortin - genetics | Pituitary ACTH Hypersecretion - therapy | Gene Expression Regulation, Neoplastic - genetics | Pituitary ACTH Hypersecretion - pathology | ACTH-Secreting Pituitary Adenoma - metabolism | Corticosterone - genetics | Adrenocorticotropic Hormone - secretion | Receptors, Thyroid Hormone - metabolism | Animals | Pituitary ACTH Hypersecretion - metabolism | Receptors, Steroid - genetics | Pro-Opiomelanocortin - biosynthesis | Cell Line, Tumor | ACTH-Secreting Pituitary Adenoma - pathology | Physiological aspects | Pituitary gland | Research | Hydrocortisone | Health aspects | Causes and theories of causation | Diseases | Rodents | Genetics | T cell receptors | Binding sites | Index Medicus | Biological Sciences
Journal Article
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 08/2017, Volume 102, Issue 8, pp. 2836 - 2843
Context: Somatic mutations in the ubiquitin-specific protease 8 (USP8) gene have been recently identified as the most common genetic alteration in patients... 
TARGET | UBPY | ADOLESCENTS | ENDOCRINOLOGY & METABOLISM | RESISTANCE | CANCER | EGFR | DEUBIQUITINATING ENZYMES | PITUITARY-ADENOMAS | CHILDREN | Adenoma - surgery | Prognosis | Adenoma - genetics | Humans | Child, Preschool | Endosomal Sorting Complexes Required for Transport - genetics | Hydrocortisone - metabolism | Male | Adenoma - metabolism | Pituitary ACTH Hypersecretion - genetics | ACTH-Secreting Pituitary Adenoma - genetics | ACTH-Secreting Pituitary Adenoma - surgery | Pituitary ACTH Hypersecretion - surgery | Female | Retrospective Studies | Neuroendoscopy | Child | Adrenocorticotropic Hormone - metabolism | Cavernous Sinus - pathology | Pituitary ACTH Hypersecretion - pathology | ACTH-Secreting Pituitary Adenoma - metabolism | Neoplasm Invasiveness | Tumor Burden | Ubiquitin Thiolesterase - genetics | Endopeptidases - genetics | Pituitary ACTH Hypersecretion - metabolism | Adolescent | Age of Onset | Adenoma - pathology | Neoplasm Recurrence, Local - genetics | Mutation | ACTH-Secreting Pituitary Adenoma - pathology | Ubiquitin | Pediatrics | Nervous system diseases | Ubiquitin-specific proteinase | Nucleotide sequence | Hydrocortisone | Gene deletion | Patients | 14-3-3 protein | Adrenocorticotropic hormone | Clonal deletion | Sinus | Pituitary | Deletion | Deoxyribonucleic acid--DNA | Index Medicus | Abridged Index Medicus
Journal Article
Osteoporosis International, ISSN 0937-941X, 1/2018, Volume 29, Issue 1, pp. 211 - 221
Hypercortisolism in humans suppresses osteoblastogenesis and osteoblast function through the upregulation of Wnt-signaling antagonists (sclerostin, Dkk1) and... 
Glucocorticoid-induced osteoporosis | Medicine & Public Health | Cushing’s disease | Wnt-signaling | Orthopedics | Rheumatology | microRNA | Sclerostin | Endocrinology | OSTEOBLAST | STEM-CELLS | DOWN-REGULATION | PROLIFERATION | OSTEOGENESIS | OSTEOPOROSIS | OSTEOCLASTOGENESIS | Cushing's disease | ENDOCRINOLOGY & METABOLISM | SUPPRESSES | DIFFERENTIATION | ANTAGONISTS | Bone Density - physiology | Humans | Middle Aged | Wnt Signaling Pathway - physiology | Male | Bone Density - genetics | Bone Remodeling - physiology | Osteoporosis - etiology | Pituitary ACTH Hypersecretion - genetics | Cell Differentiation - genetics | Pituitary ACTH Hypersecretion - complications | Sphenoid Bone - pathology | Adult | Female | Osteoporosis - genetics | Osteoclasts - physiology | Osteoporosis - physiopathology | Pituitary ACTH Hypersecretion - pathology | RNA, Messenger - genetics | Gene Expression Regulation - physiology | Osteoblasts - pathology | Pituitary ACTH Hypersecretion - metabolism | Osteoporosis - pathology | Wnt Signaling Pathway - genetics | Sphenoid Bone - metabolism | MicroRNAs - genetics | Bone Remodeling - genetics | Osteoporosis | Corticosteroids | Messenger RNA | MicroRNA | Analysis | Stem cells | Bone morphogenetic proteins | Cell culture | Collagen (type I) | Mesenchyme | Glucocorticoids | Antagonists | mRNA | Adipocytes | Cartilage | Body mass index | Bone growth | LRP5 protein | Cbfa-1 protein | Nervous system diseases | Bone morphogenetic protein 2 | Bone remodelling | MiRNA | Chromosome 5 | Gene expression | Osteoclastogenesis | Adrenocorticotropic hormone | Osteoblastogenesis | Pituitary | MicroRNAs | Dkk1 protein | Osteogenesis | Index Medicus
Journal Article
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