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Journal of Clinical Investigation, ISSN 0021-9738, 12/2006, Volume 116, Issue 12, pp. 3150 - 3159
Genomic actions induced by 1 alpha 25-dihydroxyvitamin D-3 [1,25(OH)(2)D-3] are crucial for normal bone metabolism, mainly because they regulate active... 
PHOSPHATE | MEDICINE, RESEARCH & EXPERIMENTAL | IN-VITRO | BONE-FORMATION | GENE-EXPRESSION | TARGETED ABLATION | HYPERTROPHIC CHONDROCYTES | ANIMAL-MODEL | FIBROBLAST GROWTH FACTOR-23 | SKELETAL | KNOCKOUT MICE | Immunohistochemistry | Chondrocytes - cytology | Osteogenesis - physiology | Gene Expression - genetics | Bone Development - physiology | Receptors, Calcitriol - physiology | Fibroblast Growth Factors - genetics | Osteoclasts - cytology | RNA, Messenger - metabolism | Receptors, Calcitriol - genetics | Cell Differentiation - genetics | Vitamin D - genetics | Growth Plate - cytology | Time Factors | RANK Ligand - physiology | Osteoblasts - cytology | Cell Differentiation - physiology | Chondrocytes - metabolism | Osteogenesis - genetics | Animals, Newborn | Signal Transduction | RNA, Messenger - genetics | Cells, Cultured | Platelet Endothelial Cell Adhesion Molecule-1 - analysis | Growth Plate - metabolism | Mutation - genetics | Reverse Transcriptase Polymerase Chain Reaction | Osteoclasts - metabolism | RANK Ligand - genetics | Phosphates - metabolism | Animals | Homeostasis - physiology | Vitamin D - analogs & derivatives | Mice | Bone Development - genetics | Vitamin D - physiology | Osteoblasts - metabolism | Prevention | Calcifediol | Alfacalcidol | Vitamin D | Osteoblasts | Health aspects | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal of Cell Science, ISSN 0021-9533, 05/2003, Volume 116, Issue 9, pp. 1819 - 1826
The transcription factor HIF-1alpha plays a crucial role in modifying gene expression during low oxygen tension. In a previous study, we demonstrated that... 
Hypoxia | HIF-1α | Extacellular matrix | Chondrocytes | FACTOR VEGF | hypoxia | chondrocytes | HIF-1 alpha | OXYGEN-TENSION | INDUCIBLE FACTOR 1-ALPHA | ENDOCHONDRAL BONE-FORMATION | X COLLAGEN | PLATE CARTILAGE | CELL BIOLOGY | ARTICULAR-CARTILAGE | MONOLAYER-CULTURE | HYPERTROPHIC CHONDROCYTES | extracellular matrix | ENDOTHELIAL GROWTH-FACTOR
Journal Article
Journal Article
Journal Article
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2016, Volume 6, Issue 1, pp. 38616 - 38616
Members of the TGF-beta superfamily are important regulators of chondrocyte function. Sox9, a key transcriptional regulator of chondrogenesis, is required for... 
HYPERTROPHIC DIFFERENTIATION | ACTIVATION | INHIBITION | PATHWAY | MULTIDISCIPLINARY SCIENCES | ARTICULAR CHONDROCYTES | RECEPTOR | OSTEOARTHRITIS | TRANSCRIPTION FACTOR | EXPRESSION | CARTILAGE | Index Medicus
Journal Article
International Journal of Biological Sciences, ISSN 1449-2288, 01/2018, Volume 14, Issue 1, pp. 1 - 9
A recent breakthrough showing that direct trans-differentiation of chondrocytes into bone cells commonly occurs during endochondral bone formation in the... 
Osteoblast | Cartilage | Growth/development | Chondrocyte | Cell signaling | MANDIBULAR CONDYLE GROWTH | OSTEOCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | cartilage | CRE RECOMBINASE | osteoblast | MATURATION | chondrocyte | growth/development | TEMPOROMANDIBULAR-JOINT | cell signaling | OSTEOBLASTS | HYPERTROPHIC CHONDROCYTES | GENERATION | EXPRESSION | Cell Proliferation - genetics | Chondrocytes - cytology | Microscopy, Electron, Scanning | Osteogenesis - physiology | Chondrogenesis - genetics | Cell Proliferation - physiology | Mice, Transgenic | Signal Transduction - genetics | beta Catenin - metabolism | beta Catenin - genetics | Mice, Knockout | Cell Differentiation - genetics | Chondrogenesis - physiology | Microscopy, Confocal | Animals | Signal Transduction - physiology | Mice | Cell Differentiation - physiology | Chondrocytes - metabolism | Osteogenesis - genetics | Cell proliferation | Transformation | Gene regulation | Differentiation (biology) | Growth plate | β-catenin | Bone growth | Biomedical materials | Clonal deletion | Cell fate | Deletion | Biocompatibility | Chondrogenesis | Bone (endochondral) | Mandible | Cell lineage | Cartilage (articular) | Bone (subchondral) | Gene expression | Biological activity | Signaling | Transformations (mathematics) | Chondrocytes | Bone | Osteogenesis | Index Medicus
Journal Article