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Thorax, ISSN 0040-6376, 12/2017, Volume 72, Issue Suppl 3, p. A160
IntroductionHypoxic challenge test consisting of breathing 15% FiO2 for 20 min with blood gas measurements is recommended by BTS guidelines for the assessment... 
Hypoxia
Journal Article
Heart, ISSN 1355-6037, 02/2018, Volume 104, Issue Suppl 2, p. A10
PurposeCurrently there is no UK consensus on fitness to fly in congenital heart patients. Evidence for the need for supplemental oxygen during flights for... 
Hypoxia
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 01/2017, Volume 195
Introduction: High altitude exposure results in acute hypoxia and an increase in the ventilatory drive. The aim of the current study was to examine the changes... 
Hypoxia
Journal Article
Journal of Science and Medicine in Sport, ISSN 1440-2440, 11/2017, Volume 20, pp. S94 - S95
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 01/2013, Volume 17, Issue 1, pp. 30 - 54
Accumulating lines of experimental evidence have revealed that hypoxia‐inducible factors, HIF ‐1α and HIF ‐2α, are key regulators of the adaptation of cancer‐... 
Hypoxia‐inducible factors | Targeted therapies | Cancer‐initiating cells | Hypoxia | Cancer progression | Metabolic pathways | Cancer stem/progenitor cells | Metastasis‐initiating cells | Metastases | Hypoxia-inducible factors | Cancer-initiating cells | Metastasis-initiating cells | MEDICINE, RESEARCH & EXPERIMENTAL | CLINICALLY RELEVANT SUBTYPES | PANCREATIC-CANCER | Cancer stem | progenitor cells | CHRONIC MYELOID-LEUKEMIA | INHIBITS TUMOR-GROWTH | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | PROSTATE-CANCER | GENE-EXPRESSION | FATTY-ACID SYNTHASE | ENDOTHELIAL GROWTH-FACTOR | Neoplasms - metabolism | Hypoxia - drug therapy | Neoplastic Stem Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Molecular Targeted Therapy | Hypoxia - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Neoplasm Metastasis | Basic Helix-Loop-Helix Transcription Factors - metabolism | Neoplasms - genetics | Neoplastic Stem Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Intercellular Signaling Peptides and Proteins - genetics | Transcription Factors - genetics | Neoplasms - drug therapy | Transcription Factors - metabolism | Hypoxia - genetics | Hypoxia - pathology | Cell Transformation, Neoplastic - drug effects | Neoplasms - pathology | Apoptosis | Prevention | Glucose metabolism | Physiological aspects | Development and progression | Genetic transcription | Metastasis | Transforming growth factors | Vascular endothelial growth factor | Cancer | Gene expression | Cells | Tumors | Index Medicus | Reviews
Journal Article
Neurochemical Research, ISSN 0364-3190, 6/2019, Volume 44, Issue 6, pp. 1425 - 1436
Post-conditioning is exposure of an injured organism to the same harmful factors but of milder intensity which mobilizes endogenous protective mechanisms.... 
Neurochemistry | Biochemistry, general | Neurology | Oxidative stress | Neuroprotection | Neurosciences | Biomedicine | Severe hypoxia | Pentose phosphate pathway | Hypoxic post-conditioning | HIF1 | Cell Biology | SEVERE HYPOBARIC HYPOXIA | BIOCHEMISTRY & MOLECULAR BIOLOGY | REPERFUSION INJURY | NEUROSCIENCES | ISCHEMIC POSTCONDITIONING PROTECTS | CORTICAL-NEURONS | GLUCOSE-METABOLISM | SIGNALING PATHWAY | HIF-1-ALPHA | EXPRESSION | CEREBRAL ISCHEMIA/REPERFUSION INJURY | Neocortex - pathology | Pentose Phosphate Pathway - physiology | Rats, Wistar | Glutathione - metabolism | Oxidative Stress - physiology | Neuroprotection - physiology | Male | Hippocampus - pathology | Hypoxia - metabolism | Hypoxia, Brain - metabolism | Hypoxia, Brain - pathology | Brain - metabolism | Hypoxia, Brain - prevention & control | Hippocampus - metabolism | Neocortex - metabolism | Animals | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Hypoxia, Brain - therapy | Brain - pathology | Glucosephosphate Dehydrogenase - metabolism | NADP - metabolism | Phosphates | Brain | Hypoxia-inducible factor 1 | Stabilization | Lipid peroxidation | Lipids | Neuroendocrine system | Antioxidants | Reduction | Glucose dehydrogenase | Cascades | Pentose | NADP | Conditioning | Glucosephosphate dehydrogenase | Redox properties | Glutathione | Peroxidation | Hypoxia-inducible factors | Pentose phosphate | Pathological effects | Cortex | Cell death | Hypoxia | Hippocampus
Journal Article
Acta Ophthalmologica, ISSN 1755-375X, 09/2014, Volume 92, Issue s253
Purpose Retinal hypoxia with consequent changes in blood perfusion is a central feature in common vision threatening diseases. The aim of this study was to... 
Hypoxia
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 517 - 12
Journal Article
Cell Death and Disease, ISSN 2041-4889, 03/2013, Volume 4, Issue 3, pp. e544 - e544
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2013, Volume 110, Issue 43, pp. 17462 - 17467
Both preclinical and clinical studies suggest that brief cycles of ischemia and reperfusion in the arm or leg may protect the heart against injury following... 
Heart | Femoral artery | Phosphorylation | Messenger RNA | Reperfusion | Ischemia | Genes | Antibodies | Hypoxia | Ischemic preconditioning | Myocardial infarction | Cardioprotection | Cardiac surgery | Coronary heart disease | ARTERY-BYPASS SURGERY | COACTIVATOR | PROTECTION | LIMB ISCHEMIA | MULTIDISCIPLINARY SCIENCES | myocardial infarction | REPERFUSION INJURY | KINASE | FACTOR-I | HIF-1 | INTERMITTENT HYPOXIA | cardioprotection | cardiac surgery | coronary heart disease | Myocardial Infarction - genetics | Interleukin-10 - blood | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Immunoblotting | Aryl Hydrocarbon Receptor Nuclear Translocator - metabolism | Hypoxia-Inducible Factor 1 - metabolism | Hypoxia-Inducible Factor 1 - genetics | Cell Hypoxia | Myoblasts, Skeletal - cytology | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Myocardium - metabolism | Interleukin-10 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Myoblasts, Skeletal - metabolism | Acriflavine - pharmacology | Cell Line | Aryl Hydrocarbon Receptor Nuclear Translocator - antagonists & inhibitors | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Myocardium - pathology | Myocardial Infarction - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Animals | Interleukin-10 - genetics | Protein Binding | Ischemic Preconditioning, Myocardial - methods | Aryl Hydrocarbon Receptor Nuclear Translocator - genetics | Mice | Myocardial Infarction - prevention & control | Myoblasts, Skeletal - drug effects | Hypoxia-Inducible Factor 1 - antagonists & inhibitors | Physiological aspects | Heart attack | Plasma | Chromatin | Heart attacks | Cytokines | Coronary vessels | Rodents | Index Medicus | Biological Sciences
Journal Article