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Journal of cellular and molecular medicine, ISSN 1582-1838, 01/2013, Volume 17, Issue 1, pp. 30 - 54
Accumulating lines of experimental evidence have revealed that hypoxia‐inducible factors, HIF‐1α and HIF‐2α... 
Hypoxia‐inducible factors | Targeted therapies | Cancer‐initiating cells | Hypoxia | Cancer progression | Metabolic pathways | Cancer stem/progenitor cells | Metastasis‐initiating cells | Metastases | Hypoxia-inducible factors | Cancer-initiating cells | Metastasis-initiating cells | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Neoplasms - metabolism | Hypoxia - drug therapy | Neoplastic Stem Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Molecular Targeted Therapy | Hypoxia - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Neoplasm Metastasis | Basic Helix-Loop-Helix Transcription Factors - metabolism | Neoplasms - genetics | Neoplastic Stem Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Intercellular Signaling Peptides and Proteins - genetics | Transcription Factors - genetics | Neoplasms - drug therapy | Transcription Factors - metabolism | Hypoxia - genetics | Hypoxia - pathology | Cell Transformation, Neoplastic - drug effects | Neoplasms - pathology | Apoptosis | Prevention | Glucose metabolism | Physiological aspects | Development and progression | Genetic transcription | Metastasis | Transforming growth factors | Vascular endothelial growth factor | Cancer | Energy metabolism | Transcription factors | Mesenchyme | AKT protein | Insulin-like growth factors | Kinases | Autophagy | Cancer therapies | Neoplasms | Proteins | Homing | Angiogenesis | Signal transduction | Receptors | Epidermal growth factor | Bone marrow | Tumor necrosis factor-TNF | miRNA | Growth factors | Deprivation | Cell survival | Cytokines | Epidermal growth factor receptors | Breast cancer | Rapamycin | Metabolism | Gene expression | Cell differentiation | Insulin | CXCR4 protein | 1-Phosphatidylinositol 3-kinase | Signaling | Pancreatic cancer | Stem cells | Glycolysis | Regulation | Prostate | Cell migration | Tumors | Index Medicus | Reviews | Cancer stem | progenitor cells
Journal Article
Nutrition (Burbank, Los Angeles County, Calif.), ISSN 0899-9007, 02/2016, Volume 32, Issue 2, pp. 174 - 178
Abstract Flavonoid resveratrol modulates the transcription factor NF-κB; inhibits the cytochrome P450 isoenzyme CYP1 A1... 
Gastroenterology and Hepatology | Obesity | Flavonoids | Microbiota | Type 2 diabetes mellitus | Alzheimer's disease | Resveratrol | Life Sciences & Biomedicine | Nutrition & Dietetics | Science & Technology | Autistic Disorder - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Brain-Derived Neurotrophic Factor - genetics | Apoptosis - drug effects | Humans | Tumor Necrosis Factor-alpha - genetics | Cytochrome P-450 CYP1A1 - antagonists & inhibitors | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Lipoxins - pharmacology | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Stilbenes - pharmacology | Metabolic Syndrome - chemically induced | Vascular Endothelial Growth Factor A - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Brain-Derived Neurotrophic Factor - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Diabetes Mellitus, Type 2 - prevention & control | Gastrointestinal Tract - microbiology | Interleukin-17 - genetics | Gastrointestinal Tract - drug effects | Antioxidants - pharmacology | Autistic Disorder - prevention & control | Transcription Factors - genetics | Cytochrome P-450 CYP1A1 - metabolism | Transcription Factors - metabolism | Interleukin-17 - metabolism | Gastrointestinal Microbiome - drug effects | Interleukin-17 - antagonists & inhibitors | NF-kappa B - genetics | Cell Differentiation - drug effects | Metabolic Syndrome - prevention & control | Cell Proliferation - drug effects | Diabetes Mellitus, Type 2 - chemically induced | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Type 2 diabetes | COX-2 inhibitors | Bisphenol-A | Endothelial growth factors | Microbiota (Symbiotic organisms) | Cytochrome P-450 | Schizophrenia | Bipolar disorder | T cells | Cell differentiation | Fatty acids | Autism | Stem cells | Tumor proteins | Pathogenesis | p53 Protein | Lipids | Proteins | Antioxidants | Signal transduction | Lymphocytes | Rodents | Oxidation | Growth factors | Cytochromes P450 | Diabetes mellitus | Rapamycin | Metabolism | Bisphenol A | Alloxan | Brain-derived neurotrophic factor | Insulin resistance | Hypoxia | Diabetes | Alzheimers disease | Autoimmune diseases | Metabolic disorders | Dementia | Apoptosis | Tumors | Index Medicus
Journal Article
Circulation research, ISSN 1524-4571, 06/2015, Volume 117, Issue 1, pp. 65 - 79
Hypoxia inducible factors (HIFs) are α/β heterodimeric transcription factors that direct multiple cellular and systemic responses in response to changes in oxygen availability... 
hypoxia | dioxygenases | prolyl hydroxylases | hypoxia inducible factor | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Adaptation, Physiological | Polycythemia - genetics | Heart Defects, Congenital - embryology | Cardiovascular Diseases - drug therapy | Heart - embryology | Humans | Hypoxia-Inducible Factor-Proline Dioxygenases - antagonists & inhibitors | Cardiovascular Diseases - enzymology | Oxygen - metabolism | Hypoxia - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - deficiency | Hypoxia-Inducible Factor-Proline Dioxygenases - deficiency | Cell Hypoxia | Repressor Proteins - physiology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Heart Defects, Congenital - enzymology | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Mixed Function Oxygenases - physiology | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Basic Helix-Loop-Helix Transcription Factors - deficiency | Basic Helix-Loop-Helix Transcription Factors - genetics | Hypoxia-Inducible Factor-Proline Dioxygenases - physiology | Cardiovascular Diseases - metabolism | Hydroxylation | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Hypoxia-Inducible Factor-Proline Dioxygenases - genetics | Hypertension, Pulmonary - metabolism | Cardiovascular System - enzymology | Animals | Iron - physiology | Polycythemia - enzymology | Cardiovascular System - metabolism | Ischemic Preconditioning, Myocardial | Protein Isoforms | Mice | Protein Processing, Post-Translational | Altitude | Index Medicus | Hypoxia | oxygen sensing | HIF | 2-oxoglutarate oxygenase | hydroxylase
Journal Article
Gene expression, ISSN 1052-2166, 1998, Volume 7, Issue 3, pp. 205 - 213
Hypoxia inducible factors (HIFs) are heterodimeric transcription factors that regulate a number of adaptive responses to low oxygen tension... 
Molecular characterization | Hypoxia inducible factor | HIF3α | Genetics & Heredity | Life Sciences & Biomedicine | Biotechnology & Applied Microbiology | Science & Technology
Journal Article