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P L o S Genetics (Print), ISSN 1553-7390, 01/2010, Volume 6, Issue 1, pp. e1000798 - e1000798
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2006, Volume 103, Issue 11, pp. 4168 - 4173
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2009, Volume 4, Issue 9, pp. e7230 - e7230
Journal Article
Journal Article
PLoS Neglected Tropical Diseases, ISSN 1935-2727, 10/2017, Volume 11, Issue 10, pp. e0005979 - e0005979
Background Human African Trypanosomiasis (HAT) is a neglected disease targeted for elimination as a public health problem by 2020. Elimination requires a... 
BRAZILIAN POPULATION | HUMAN AFRICAN TRYPANOSOMIASIS | SUSCEPTIBILITY | DISEASE | FOCUS | ALPHA | INTERLEUKIN-4 RECEPTOR | GENETIC-VARIANTS | TRYPANOLYTIC FACTOR | HUMAN SERUM | PARASITOLOGY | TROPICAL MEDICINE | Humans | Middle Aged | Haptoglobins - genetics | Male | Case-Control Studies | Young Adult | Trypanosomiasis, African - epidemiology | Aged, 80 and over | Neglected Diseases - epidemiology | Adult | Female | Trypanosoma brucei gambiense - isolation & purification | Trypanosomiasis, African - genetics | Child | Neglected Diseases - ethnology | Antigens, Neoplasm - genetics | Genetic Predisposition to Disease | Genetic Association Studies | Gene Frequency | Risk Factors | Neglected Diseases - genetics | Asymptomatic Diseases - epidemiology | Genotype | Trypanosomiasis, African - ethnology | Cameroon - epidemiology | Trypanosomiasis, African - blood | Adolescent | Alleles | Aged | Polymorphism, Single Nucleotide | Neglected Diseases - parasitology | Haptoglobin | Genetic aspects | Research | Management | Trypanosomiasis | Health aspects | Genetic polymorphisms | Public health | Risk factors | Genes | Biology | Infections | Biochemistry | Single-nucleotide polymorphism | Epidemiology | Gene polymorphism | Blood | Tropical diseases | Interleukin 6 | Proteins | Consortia | Risks | Interleukin 4 | Control | Interleukin 1 | Evolution | Interleukin 4 receptors | Protection | Interleukin 8 | African trypanosomiasis | Deoxyribonucleic acid--DNA | Vector-borne diseases | Parasitology | Cytokines | Tumor necrosis factor-α | Loci | Genotyping | Samples | DNA | Quality control | Interleukin 10 | Histocompatibility antigen HLA | Polymorphism | Index Medicus | Deoxyribonucleic acid
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2012, Volume 287, Issue 3, pp. 1847 - 1860
Obesity and insulin resistance are associated with chronic, low grade inflammation. Moreover, regulation of energy metabolism and immunity are highly... 
DIABETES-MELLITUS | SKELETAL-MUSCLE | HEPATIC GLUCONEOGENESIS | METABOLIC SYNDROME | INSULIN-RESISTANCE | NUCLEAR RECEPTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CALORIC RESTRICTION | HUMAN OBESITY | NECROSIS-FACTOR-ALPHA | ENDOPLASMIC-RETICULUM STRESS | Enzyme Activators - pharmacology | RNA-Binding Proteins - genetics | AMP-Activated Protein Kinases - metabolism | Liver - pathology | Receptors, Interleukin-15 - biosynthesis | Haptoglobins - biosynthesis | Caloric Restriction | Haptoglobins - genetics | Hepatocytes - pathology | Interleukin 1 Receptor Antagonist Protein - genetics | Male | Hepatocytes - metabolism | Obesity - genetics | Gene Knockdown Techniques | Receptors, Interleukin-15 - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Inflammation Mediators - metabolism | Trans-Activators - genetics | Mice, Inbred DBA | Fasting - metabolism | Interleukin 1 Receptor Antagonist Protein - biosynthesis | C-Reactive Protein - biosynthesis | Physical Conditioning, Animal | Gene Expression Regulation - genetics | Liver - metabolism | Metformin - pharmacology | Cells, Cultured | Rats | PPAR alpha - genetics | C-Reactive Protein - genetics | Transcription Factors - genetics | Hypoglycemic Agents - pharmacology | Obesity - metabolism | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Animals | Energy Metabolism | Insulin Resistance - genetics | Obesity - therapy | Trans-Activators - metabolism | Mice | PPAR alpha - metabolism | AMP-Activated Protein Kinases - genetics | RNA-Binding Proteins - metabolism | Index Medicus | IL15Rα | IL1Rn | Coactivator Transcription | Liver | Inflammation | Cellular Immune Response | Diabetes | Tweak | Metformin | Metabolism | Nuclear Receptors | necrosis-factor-alpha | skeletal-muscle | metabolic syndrome | nuclear receptors | hepatic gluconeogenesis | insulin-resistance | caloric restriction | diabetes-mellitus | endoplasmic-reticulum stress | human obesity
Journal Article
Circulation Research, ISSN 0009-7330, 07/2007, Volume 101, Issue 1, pp. 106 - 110
In individuals with diabetes mellitus (DM), the haptoglobin (Hp) genotype is a major determinant of susceptibility to myocardial infarction. We have proposed... 
Haptoglobin polymorphism | Hemoglobin | Diabetes mellitus | Intraplaque hemorrhage | Macrophage | haptoglobin polymorphism | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | MECHANISM | MACROPHAGES | HAPTOGLOBIN GENOTYPE | CD163 EXPRESSION | macrophage | diabetes mellitus | HEME UPTAKE | RELEASE | HUMAN MONOCYTES | hemoglobin | IN-VITRO | intraplaque hemorrhage | PERIPHERAL VASCULAR DISEASE | NEOVASCULARIZATION | HEMATOLOGY | Diabetes Mellitus - blood | Diabetes Mellitus - pathology | Myocardial Infarction - blood | Myocardial Infarction - genetics | Myocardial Infarction - epidemiology | Antigens, CD - biosynthesis | Diabetes Mellitus - genetics | Humans | Haptoglobins - genetics | Haptoglobins - metabolism | Receptors, Scavenger - antagonists & inhibitors | Antigens, CD - genetics | Incidence | Receptors, Cell Surface - biosynthesis | Genetic Predisposition to Disease - epidemiology | Antigens, Differentiation, Myelomonocytic - biosynthesis | Hemorrhage - genetics | Hemorrhage - epidemiology | Hemoglobins - metabolism | Genotype | Hemoglobins - genetics | Receptors, Cell Surface - blood | Receptors, Scavenger - blood | Hemorrhage - blood | Antigens, Differentiation, Myelomonocytic - genetics | Down-Regulation - genetics | Antigens, CD - blood | Macrophages - metabolism | Antigens, Differentiation, Myelomonocytic - blood | Receptors, Scavenger - genetics | Receptors, Cell Surface - genetics | Index Medicus
Journal Article
by Howson, Joanna M M and Zhao, Wei and Barnes, Daniel R and Ho, Weang-Kee and Young, Robin and Paul, Dirk S and Waite, Lindsay L and Freitag, Daniel F and Fauman, Eric B and Salfati, Elias L and Sun, Benjamin B and Eicher, John D and Johnson, Andrew D and Sheu, Wayne H H and Nielsen, Sune F and Lin, Wei-Yu and Surendran, Praveen and Malarstig, Anders and Wilk, Jemma B and Tybjærg-Hansen, Anne and Rasmussen, Katrine L and Kamstrup, Pia R and Deloukas, Panos and Erdmann, Jeanette and Kathiresan, Sekar and Samani, Nilesh J and Schunkert, Heribert and Watkins, Hugh and Do, Ron and Rader, Daniel J and Johnson, Julie A and Hazen, Stanley L and Quyyumi, Arshed A and Spertus, John A and Pepine, Carl J and Franceschini, Nora and Justice, Anne and Reiner, Alex P and Buyske, Steven and Hindorff, Lucia A and Carty, Cara L and North, Kari E and Kooperberg, Charles and Boerwinkle, Eric and Young, Kristin and Graff, Mariaelisa and Peters, Ulrike and Absher, Devin and Hsiung, Chao A and Lee, Wen-Jane and Taylor, Kent D and Chen, Ying-Hsiang and Lee, I-Te and Guo, Xiuqing and Chung, Ren-Hua and Hung, Yi-Jen and Rotter, Jerome I and Juang, Jyh-Ming J and Quertermous, Thomas and Wang, Tzung-Dau and Rasheed, Asif and Frossard, Philippe and Alam, Dewan S and Majumder, Abdulla Al Shafi and Di Angelantonio, Emanuele and Chowdhury, Rajiv and Chen, Yii-Der Ida and Nordestgaard, Børge G and Assimes, Themistocles L and Danesh, John and Butterworth, Adam S and Saleheen, Danish and EPIC-CVD and CARDIoGRAMplusC4D
Nature Genetics, ISSN 1061-4036, 07/2017, Volume 49, Issue 7, pp. 1113 - 1119
Journal Article
Journal Article