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Arthritis & Rheumatology, ISSN 2326-5191, 2016, Volume 68, Issue 11, pp. 2795 - 2805
Objective Mevalonate kinase deficiency (MKD) is a rare metabolic disease characterized by recurrent inflammatory episodes. This study was undertaken to... 
MVK | HYPER-IGD | PERIODIC FEVER SYNDROME | ACIDURIA | HYPERIMMUNOGLOBULINEMIA-D | AUTOINFLAMMATORY DISEASES | MUTATIONS | SPECTRUM | RHEUMATOLOGY | Arthralgia - etiology | Uveitis - etiology | Humans | Child, Preschool | Lymphadenopathy - etiology | Male | Arthralgia - physiopathology | Arthritis - genetics | Stomatitis, Aphthous - genetics | Cerebellar Diseases - physiopathology | Amyloidosis - genetics | Skin Diseases - physiopathology | Amyloidosis - etiology | Child | Stomatitis, Aphthous - etiology | Infant, Newborn | Myalgia - physiopathology | Amyloidosis - physiopathology | Genotype | Headache - physiopathology | Vomiting - genetics | Phenotype | Diarrhea - genetics | Abdominal Pain - genetics | Adolescent | Age of Onset | Intellectual Disability - etiology | Uveitis - genetics | Vomiting - physiopathology | Conjunctivitis - etiology | Conjunctivitis - genetics | Arthritis - physiopathology | Headache - etiology | Skin Diseases - genetics | Mevalonate Kinase Deficiency - physiopathology | Myalgia - genetics | Cerebellar Diseases - etiology | Diarrhea - physiopathology | Infant | Mevalonate Kinase Deficiency - genetics | Intellectual Disability - genetics | Pharyngitis - genetics | Arthritis - etiology | Female | Registries | Retrospective Studies | Arthralgia - genetics | Pharyngitis - physiopathology | Abdominal Pain - etiology | Pharyngitis - etiology | Lymphadenopathy - genetics | Abdominal Pain - physiopathology | Myalgia - etiology | Phosphotransferases (Alcohol Group Acceptor) - genetics | Headache - genetics | Intellectual Disability - physiopathology | Skin Diseases - etiology | Cerebellar Diseases - genetics | Conjunctivitis - physiopathology | Mevalonate Kinase Deficiency - complications | Stomatitis, Aphthous - physiopathology | Vomiting - etiology | Diarrhea - etiology | Lymphadenopathy - physiopathology | Uveitis - physiopathology | Kinases | Genotype & phenotype | Index Medicus | Abridged Index Medicus
Journal Article
Sleep Medicine Reviews, ISSN 1087-0792, 2005, Volume 9, Issue 4, pp. 269 - 310
Human narcolepsy is a chronic sleep disorder affecting 1:2000 individuals. The disease is characterized by excessive daytime sleepiness, cataplexy and other... 
Niemann-Pick disease | Cataplexy encephalitis | Prader-Willi syndrome | Encephalitis lethargica | Paraneoplastic syndromes | Hypocretin | Brain tumor | Cerebrospinal fluid (CSF) | Multiple sclerosis (MS) | Narcolepsy | Head trauma | Acute disseminated encephalomyelitis (ADEM) | Orexin | Guillain-Barré syndrome (GBS) | narcolepsy | GUILLAIN-BARRE-SYNDROME | CEREBROSPINAL-FLUID | acute disseminated encephalomyelitis (ADEM) | CLINICAL NEUROLOGY | NIEMANN-PICK-DISEASE | MULTIPLE-SCLEROSIS | COFFIN-LOWRY-SYNDROME | ACUTE DISSEMINATED ENCEPHALOMYELITIS | paraneoplastic syndromes | OREXIN-A LEVELS | CENTRAL-NERVOUS-SYSTEM | orexin | cataplexy encephalitis | Guillain-Barre syndrome (GBS) | multiple sclerosis (MS) | cerebrospinal fluid (CSF) | brain tumor | NEUROSCIENCES | head trauma | hypocretin | encephalitis lethargica | PRADER-WILLI-SYNDROME | EXCESSIVE DAYTIME SLEEPINESS | Disorders of Excessive Somnolence - physiopathology | Humans | Craniocerebral Trauma - physiopathology | Receptors, Neuropeptide - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | HLA-DQ Antigens - immunology | Disorders of Excessive Somnolence - metabolism | Myotonic Dystrophy - immunology | Narcolepsy - physiopathology | Multiple Sclerosis - physiopathology | Craniocerebral Trauma - cerebrospinal fluid | Prader-Willi Syndrome - physiopathology | Prader-Willi Syndrome - immunology | Niemann-Pick Diseases - cerebrospinal fluid | Child | Vascular Diseases - cerebrospinal fluid | Niemann-Pick Diseases - immunology | Orexins | Parkinson Disease, Postencephalitic - physiopathology | Multiple Sclerosis - cerebrospinal fluid | Parkinson Disease, Postencephalitic - cerebrospinal fluid | Coffin-Lowry Syndrome - physiopathology | Receptors, G-Protein-Coupled | Neuropeptides - metabolism | Disorders of Excessive Somnolence - immunology | Vascular Diseases - physiopathology | Orexin Receptors | Neurodegenerative Diseases - cerebrospinal fluid | Niemann-Pick Diseases - physiopathology | Hypothalamus - physiopathology | Narcolepsy - immunology | Myotonic Dystrophy - physiopathology | Hypothalamus - metabolism | Neurodegenerative Diseases - physiopathology | Narcolepsy - cerebrospinal fluid | Multiple Sclerosis - immunology | Myotonic Dystrophy - cerebrospinal fluid | HLA-DR Antigens - immunology | Coffin-Lowry Syndrome - cerebrospinal fluid | Chronic Disease | Prader-Willi Syndrome - cerebrospinal fluid | Brain | Multiple sclerosis | Parkinson's disease | Genetic disorders | Histocompatibility antigens | HLA histocompatibility antigens | Development and progression | Hypothalamic hormones | Tumors
Journal Article
Europace, ISSN 1099-5129, 11/2004, Volume 6, Issue 6, pp. 467 - 537
Journal Article
Nature medicine, ISSN 1078-8956, 01/2017, Volume 23, Issue 1, pp. 28 - 38
Journal Article
Journal Article
Pain, ISSN 0304-3959, 05/2010, Volume 149, Issue 2, pp. 235 - 242
Bright light can cause ocular discomfort and/or pain; however, the mechanism linking luminance to trigeminal nerve activity is not known. In this study we... 
Trigeminal subnucleus caudalis | Ocular pain | Light | Photophobia | NERVE-FIBERS | GENE-RELATED PEPTIDE | RAT | SUPERIOR SALIVATORY NUCLEUS | CHOROIDAL BLOOD-FLOW | MIGRAINE | NEUROSCIENCES | PERIPHERAL GANGLIA | CLINICAL NEUROLOGY | SENSORY INNERVATION | ANTERIOR EYE SEGMENT | NEURONS | ANESTHESIOLOGY | Headache - etiology | Photophobia - etiology | Visual Pathways - physiopathology | Afferent Pathways - physiopathology | Male | Rhombencephalon - drug effects | Anesthetics, Local - pharmacology | Regional Blood Flow - drug effects | Afferent Pathways - radiation effects | Pain - etiology | Parasympathetic Nervous System - physiology | Trigeminal Caudal Nucleus - radiation effects | Trigeminal Nerve - physiopathology | Nociceptors - radiation effects | Vasodilation - physiology | Vasoconstrictor Agents - pharmacology | Ophthalmic Nerve - radiation effects | Trigeminal Ganglion - radiation effects | Ophthalmic Nerve - physiopathology | Photophobia - physiopathology | Light - adverse effects | Ophthalmic Artery - innervation | Rats | Trigeminal Nerve - radiation effects | Headache - physiopathology | Rats, Sprague-Dawley | Rhombencephalon - physiology | Trigeminal Caudal Nucleus - physiopathology | Animals | Visual Pathways - drug effects | Pain - physiopathology | Parasympathetic Nervous System - drug effects | Trigeminal Ganglion - physiopathology | Vasodilation - drug effects | Regional Blood Flow - physiology | Ophthalmic Artery - physiopathology | ocular pain | trigeminal subnucleus caudalis | light | photophobia
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, p. e0159442
Millions of mild traumatic brain injuries (TBIs) occur every year in the United States, with many people subject to multiple head injuries that can lead to... 
MILD | ASTROCYTE ACTIVATION | OPTIC-NERVE | TRAUMATIC BRAIN-INJURY | BEHAVIORAL IMPAIRMENTS | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | COGNITIVE IMPAIRMENT | SPINAL-CORD-INJURY | CONCUSSION | AXONAL INJURY | Optic Tract - physiopathology | Memory Disorders - physiopathology | Neurons - pathology | Optic Nerve - pathology | Corpus Callosum - metabolism | Brain Concussion - pathology | Brain Concussion - physiopathology | Corpus Callosum - physiopathology | Neuroglia - pathology | Optic Tract - pathology | Cerebral Cortex - pathology | Memory Disorders - metabolism | Head Injuries, Closed - metabolism | Male | Gliosis - physiopathology | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Head Injuries, Closed - physiopathology | Gliosis - pathology | Neurofilament Proteins - genetics | Pyramidal Tracts - physiopathology | Pyramidal Tracts - metabolism | Neurons - metabolism | Optic Nerve - physiopathology | Pyramidal Tracts - pathology | Memory Disorders - pathology | Disease Models, Animal | Psychomotor Performance | Gene Expression | Head Injuries, Closed - pathology | Gliosis - metabolism | Mice, Inbred C57BL | Hippocampus - pathology | Optic Tract - metabolism | Hippocampus - metabolism | Animals | Corpus Callosum - pathology | Neuroglia - metabolism | Mice | Neurofilament Proteins - metabolism | Hippocampus - physiopathology | Brain Concussion - metabolism | Optic Nerve - metabolism | Complications and side effects | Care and treatment | Head injuries | Research | Memory, Disorders of | Risk factors | Intervals | Brain | Traumatic brain injury | Gait | Neuronal-glial interactions | Visual system | Histopathology | Neurodegeneration | Neurofilament protein | Physiology | Acute effects | Stains | Injuries | Head | Optic nerve | Motor ability | Abnormalities | Cortex (visual) | Inflammation | Trauma | Corpus callosum | Optic tract | Medicine | Axons | Injury prevention | Brain research | Gliosis | Chronic traumatic encephalopathy | Cell death | Spinal cord injuries | Laboratory animals | Human behavior | Apoptosis | Animal cognition
Journal Article
Journal Article