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PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, p. e0159442
Millions of mild traumatic brain injuries (TBIs) occur every year in the United States, with many people subject to multiple head injuries that can lead to... 
MILD | ASTROCYTE ACTIVATION | OPTIC-NERVE | TRAUMATIC BRAIN-INJURY | BEHAVIORAL IMPAIRMENTS | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | COGNITIVE IMPAIRMENT | SPINAL-CORD-INJURY | CONCUSSION | AXONAL INJURY | Optic Tract - physiopathology | Memory Disorders - physiopathology | Neurons - pathology | Optic Nerve - pathology | Corpus Callosum - metabolism | Brain Concussion - pathology | Brain Concussion - physiopathology | Corpus Callosum - physiopathology | Neuroglia - pathology | Optic Tract - pathology | Cerebral Cortex - pathology | Memory Disorders - metabolism | Head Injuries, Closed - metabolism | Male | Gliosis - physiopathology | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Head Injuries, Closed - physiopathology | Gliosis - pathology | Neurofilament Proteins - genetics | Pyramidal Tracts - physiopathology | Pyramidal Tracts - metabolism | Neurons - metabolism | Optic Nerve - physiopathology | Pyramidal Tracts - pathology | Memory Disorders - pathology | Disease Models, Animal | Psychomotor Performance | Gene Expression | Head Injuries, Closed - pathology | Gliosis - metabolism | Mice, Inbred C57BL | Hippocampus - pathology | Optic Tract - metabolism | Hippocampus - metabolism | Animals | Corpus Callosum - pathology | Neuroglia - metabolism | Mice | Neurofilament Proteins - metabolism | Hippocampus - physiopathology | Brain Concussion - metabolism | Optic Nerve - metabolism | Complications and side effects | Care and treatment | Head injuries | Research | Memory, Disorders of | Risk factors | Intervals | Brain | Traumatic brain injury | Gait | Neuronal-glial interactions | Visual system | Histopathology | Neurodegeneration | Neurofilament protein | Physiology | Acute effects | Stains | Injuries | Head | Optic nerve | Motor ability | Abnormalities | Cortex (visual) | Inflammation | Trauma | Corpus callosum | Optic tract | Medicine | Axons | Injury prevention | Brain research | Gliosis | Chronic traumatic encephalopathy | Cell death | Spinal cord injuries | Laboratory animals | Human behavior | Apoptosis | Animal cognition
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 12/2009, Volume 26, Issue 12, pp. 2233 - 2243
Frontal impact, closed head trauma is a frequent cause of traumatic brain injury (TBI) in motor vehicle and sports accidents. Diffuse axonal injury (DAI) is... 
β-amyloid precursor protein | Traumatic brain injury | Diffuse axonal injury | Frontal impact | Impact acceleration | Activated caspase-3 | activated caspase-3 | diffuse axonal injury | traumatic brain injury | AMYLOID PRECURSOR PROTEIN | frontal impact | BIOMECHANICS | NEUROSCIENCES | PEPTIDE | DAMAGE | CLINICAL NEUROLOGY | ACCELERATION | MILD | TRAUMATIC BRAIN-INJURY | FLUID PERCUSSION INJURY | beta-amyloid precursor protein | impact acceleration | DEFICITS | CRITICAL CARE MEDICINE | Neurons - pathology | Diffuse Axonal Injury - physiopathology | Rats, Long-Evans | Caspase 3 - metabolism | Head Injuries, Closed - metabolism | Male | Brain Injuries - metabolism | Brain Injuries - physiopathology | Head Injuries, Closed - physiopathology | Amyloid beta-Protein Precursor - metabolism | Diffuse Axonal Injury - metabolism | Neurons - metabolism | Rotation - adverse effects | Disease Models, Animal | Biomarkers - metabolism | Acceleration - adverse effects | Head Injuries, Closed - pathology | Purkinje Cells - metabolism | Brain - physiopathology | Biomarkers - analysis | Axons - metabolism | Rats | Hippocampus - pathology | Frontal Bone - injuries | Physics | Hippocampus - metabolism | Biomechanical Phenomena | Animals | Axons - pathology | Brain - pathology | Brain Injuries - pathology | Purkinje Cells - pathology | Diffuse Axonal Injury - pathology | Complications and side effects | Brain | Usage | Head injuries | Animal models in research | Models | Research | Injuries | Risk factors | Traffic accidents & safety | Rodents | Original
Journal Article
by Gu, Y and Zhao, Y and Qian, K and Sun, M
Neuroscience, ISSN 0306-4522, 2014, Volume 291, pp. 331 - 340
Highlights • Taurine attenuates hippocampal injury after closed head injury in rats. • Taurine reduced white matter damage after closed head injury in rats. •... 
Neurology | calpain | taurine | closed head injury | corpus callosum | hippocampus | Taurine | Calpain | Closed head injury | Corpus callosum | Hippocampus | WHITE-MATTER | ALPHA-II-SPECTRIN | PROTEOLYSIS | NEUROSCIENCES | PATHOPHYSIOLOGY | MYELIN BASIC-PROTEIN | DOWN-REGULATING CALPAIN | TRAUMATIC BRAIN-INJURY | BEHAVIORAL DEFICITS | DEGRADATION | CALPASTATIN | Neurons - pathology | Calpain - metabolism | Corpus Callosum - metabolism | White Matter - metabolism | Head Injuries, Closed - metabolism | Male | Gray Matter - drug effects | CA1 Region, Hippocampal - drug effects | CA1 Region, Hippocampal - pathology | CA3 Region, Hippocampal - drug effects | Neuroprotective Agents - pharmacology | Gray Matter - metabolism | CA3 Region, Hippocampal - metabolism | CA1 Region, Hippocampal - metabolism | Neurons - metabolism | Cell Death - drug effects | Neurons - drug effects | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Head Injuries, Closed - pathology | Myelin Basic Protein - metabolism | White Matter - drug effects | Corpus Callosum - drug effects | Random Allocation | Rats, Sprague-Dawley | White Matter - pathology | Animals | Corpus Callosum - pathology | Gray Matter - pathology | Taurine - pharmacology | CA3 Region, Hippocampal - pathology | Neurofilament Proteins - metabolism | Head Injuries, Closed - drug therapy | Head injuries
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 07/2005, Volume 22, Issue 7, pp. 750 - 762
Journal Article
Journal of Biomedical Optics, ISSN 1083-3668, 06/2009, Volume 14, Issue 3, pp. 034015 - 0340111
Journal Article
Journal Article