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Circulation (New York, N.Y.), ISSN 1524-4539, 2015, Volume 131, Issue 6, pp. 550 - 559
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 485, Issue 7397, pp. 251 - 255
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 19 - 26
Genetic mutations account for a significant percentage of cardiomyopathies, which are a leading cause of congestive heart failure... 
MEDICINE, RESEARCH & EXPERIMENTAL | SARCOMERE PROTEIN GENES | COMPLEX | HYPERTROPHIC CARDIOMYOPATHY | POLYMORPHISMS | TITIN | DREIFUSS MUSCULAR-DYSTROPHY | LAMIN-A/C DEFICIENCY | RISK | CONGESTIVE-HEART-FAILURE | MECHANOTRANSDUCTION | Cardiomyopathy, Dilated - pathology | Humans | Heart Failure - physiopathology | Myosin Heavy Chains - genetics | Myosin Heavy Chains - metabolism | Arrhythmias, Cardiac - physiopathology | Sarcomeres - pathology | Arrhythmias, Cardiac - pathology | Mitochondria - genetics | Sarcomeres - genetics | Myocardium - metabolism | Heart Failure - etiology | Arrhythmias, Cardiac - genetics | Cardiac Myosins - metabolism | Arrhythmias, Cardiac - metabolism | Calcium-Binding Proteins - metabolism | Cardiomyopathy, Dilated - genetics | Cytoskeleton - pathology | Cardiomyopathy, Dilated - complications | Sarcomeres - metabolism | Cardiac Myosins - genetics | Cytoskeleton - genetics | Heart Failure - genetics | Myocardium - pathology | Arrhythmias, Cardiac - etiology | Heart Failure - metabolism | Mitochondria - metabolism | Heart Failure - pathology | Mitochondria - pathology | Cardiomyopathy, Dilated - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cardiomyopathy, Dilated - physiopathology | Cytoskeleton - metabolism | Mutation | Calcium-Binding Proteins - genetics | Gene mutations | Cardiomyopathy | Genetic aspects | Congestive heart failure | Diagnosis | Heart diseases | Genetic screening | Methods | Review Series
Journal Article
Human Pathology, ISSN 0046-8177, 2015, Volume 49, pp. 27 - 32
Summary Complex I deficiency causes Leigh syndrome, fatal infant lactic acidosis, and neonatal cardiomyopathy. Mutations in more than 100 nuclear DNA and... 
Pathology | Metabolic disease | Lethal neonatal lactic acidosis | Whole-exome sequencing | Mitochondriopathy | Complex I deficiency | ACAD9 defect | Mitochondrial hyperplasia | Multiorgan failure | DIAGNOSIS | CARDIOMYOPATHY | PATHOLOGY | GENE | DNA | EXPRESSION | Immunohistochemistry | Fibroblasts - enzymology | Diaphragm - pathology | Mitochondria, Heart - pathology | Electron Transport Complex I - deficiency | Humans | Hyperplasia | Male | Cardiomyopathy, Hypertrophic - enzymology | Acyl-CoA Dehydrogenases - genetics | Leigh Disease - pathology | Muscle Weakness - genetics | Amino Acid Metabolism, Inborn Errors - genetics | Autopsy | DNA Mutational Analysis | Electron Transport Complex I - genetics | Fatal Outcome | Muscle Weakness - pathology | Acyl-CoA Dehydrogenases - deficiency | Amino Acid Metabolism, Inborn Errors - pathology | Kidney Tubules - pathology | Infant, Newborn | Multiple Organ Failure - pathology | Acyl-CoA Dehydrogenase - genetics | Cardiomyopathy, Hypertrophic - genetics | Genetic Predisposition to Disease | Mitochondria, Liver - pathology | Mitochondria, Heart - enzymology | Fibroblasts - pathology | Leigh Disease - genetics | Acidosis, Lactic - pathology | Cardiomyopathy, Hypertrophic - diagnosis | Mitochondrial Diseases - enzymology | Phenotype | Acyl-CoA Dehydrogenase - deficiency | Kidney Tubules - enzymology | Acidosis - diagnosis | Multiple Organ Failure - enzymology | Acidosis - genetics | Amino Acid Metabolism, Inborn Errors - enzymology | Mitochondrial Diseases - pathology | Muscle Weakness - diagnosis | Acidosis, Lactic - genetics | Cause of Death | Acidosis, Lactic - enzymology | Amino Acid Metabolism, Inborn Errors - diagnosis | DNA, Mitochondrial - genetics | Muscle Weakness - enzymology | Transfection | Mitochondrial Diseases - genetics | Mitochondria, Muscle - enzymology | Mitochondria, Muscle - pathology | Leigh Disease - enzymology | Cells, Cultured | Multiple Organ Failure - genetics | Codon, Nonsense | Mitochondria, Liver - enzymology | Multiple Organ Failure - diagnosis | Diaphragm - enzymology | Acidosis - pathology | Leigh Disease - diagnosis | Acidosis - enzymology | Mitochondrial Diseases - diagnosis | Acidosis, Lactic - diagnosis | Cardiomyopathy, Hypertrophic - pathology | Infants (Newborn) | Muscles | Genetic aspects | Mitochondrial DNA | Liver | Heart | Urine | Cytochrome | Antigens | Enzymes | Cytomegalovirus | Cardiomyopathy | Defects | Musculoskeletal system | Databases | Rodents | Fibroblasts | Oxidation | Mutation | Metabolic disorders | Deoxyribonucleic acid--DNA
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2013, Volume 128, Issue 13, pp. 1420 - 1432
Journal Article
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2017, Volume 127, Issue 5, pp. 1600 - 1612
.... Mechanical stress due to pressure and volume overload and metabolic dysfunction also induce profound changes in ECM composition that contribute to the pathogenesis of heart failure... 
ISCHEMIA-REPERFUSION INJURY | MEDICINE, RESEARCH & EXPERIMENTAL | VOLUME OVERLOAD | HEART-FAILURE | CHRONIC PRESSURE-OVERLOAD | LEFT-VENTRICULAR DILATION | MATRICELLULAR PROTEINS | SMOOTH-MUSCLE-CELLS | ANGIOTENSIN-II | CARDIAC FIBROSIS | MICE LACKING OSTEOPONTIN | Inflammation - pathology | Leukocytes - pathology | Myocardium - immunology | Myocytes, Cardiac - immunology | Humans | Extracellular Matrix - immunology | Extracellular Matrix - metabolism | Stress, Mechanical | Heart Failure, Diastolic - metabolism | Myocardial Infarction - immunology | Leukocytes - immunology | Intercellular Signaling Peptides and Proteins - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Myocardial Infarction - pathology | Myocardium - metabolism | Extracellular Matrix Proteins - metabolism | Myocardium - pathology | Inflammation - immunology | Myocardial Infarction - metabolism | Heart Failure, Diastolic - pathology | Myocytes, Cardiac - pathology | Animals | Extracellular Matrix Proteins - immunology | Heart Failure, Diastolic - immunology | Myocytes, Cardiac - metabolism | Extracellular Matrix - pathology | Leukocytes - metabolism | Intercellular Signaling Peptides and Proteins - immunology | Extracellular matrix | Care and treatment | Research | Regeneration (Biology) | Heart attack | Heart failure | Hypertension | Myocardial infarction | Heart attacks | Pathogenesis | Cardiomyocytes | Inflammation | Metabolism | Proteins | Ischemia | Collagen | Rodents | Fibroblasts | Plastics | Growth factors | Heart diseases | Binding sites
Journal Article