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Nature, ISSN 0028-0836, 11/2014, Volume 515, Issue 7527, pp. 431 - 435
Journal Article
Journal Article
Nature, ISSN 0028-0836, 05/2016, Volume 534, Issue 7605, pp. 119 - 123
Myocardial infarction results in compromised myocardial function and heart failure owing to insufficient cardiomyocyte self-renewal(1). Unlike many... 
RIEGER-SYNDROME | ELEMENTS | OXIDATIVE STRESS | GENE | REGENERATION | MULTIDISCIPLINARY SCIENCES | CARDIOMYOCYTE PROLIFERATION | GROWTH | ENHANCERS | GENOME | FAMILY | Heart Injuries - pathology | Myocardial Infarction - genetics | Protein-Serine-Threonine Kinases - deficiency | Electron Transport - drug effects | Reactive Oxygen Species - metabolism | Antioxidants - metabolism | Electron Transport - genetics | Heart Injuries - metabolism | Homeodomain Proteins - metabolism | Transcription Factors - deficiency | Male | Phosphoproteins - metabolism | Regeneration - genetics | Myocardial Infarction - pathology | Myocardium - metabolism | Female | Wound Healing - genetics | Wound Healing - drug effects | Disease Models, Animal | Animals, Newborn | Heart Injuries - genetics | Free Radical Scavengers - metabolism | Antioxidants - pharmacology | Myocardial Infarction - metabolism | Transcription Factors - genetics | Regeneration - physiology | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Myocytes, Cardiac - pathology | Regeneration - drug effects | Animals | Myocytes, Cardiac - drug effects | NF-E2-Related Factor 2 - metabolism | Myocytes, Cardiac - metabolism | Heart Ventricles - metabolism | Mice | Wound Healing - physiology | Adaptor Proteins, Signal Transducing - metabolism | Heart Ventricles - drug effects | Heart | Physiological aspects | Genetic aspects | Regeneration (Biology) | Homeobox genes | Homeotic genes | Antioxidants | Transcription factors | Genetic regulation | Health aspects | Heart attack | Proteins | Heart attacks | Genes | DNA methylation | Cardiomyocytes | Kinases | Deoxyribonucleic acid--DNA | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 07/2013, Volume 99, Issue 1, pp. 164 - 174
Nucleotide-binding oligomerization domain-Like Receptor with a Pyrin domain 3 (NLRP3) is considered necessary for initiating a profound sterile inflammatory... 
Heart | Myocardial infarction | Inflammasome | Ischaemia-reperfusion | IL-1β | Potassium - metabolism | Up-Regulation | Inflammasomes - metabolism | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Caspase 1 - metabolism | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Myocardial Reperfusion Injury - pathology | Time Factors | Interleukin-1beta - metabolism | Adenosine Triphosphate - metabolism | Myocardial Infarction - physiopathology | Toll-Like Receptors - metabolism | Myocardial Reperfusion Injury - genetics | Disease Models, Animal | Fibroblasts - metabolism | Myocardial Reperfusion Injury - immunology | Rats | Receptors, Cytoplasmic and Nuclear - genetics | Myocardial Infarction - metabolism | Myocardial Reperfusion Injury - physiopathology | Mice, Knockout | Fibroblasts - drug effects | Lipopolysaccharides - pharmacology | Fibroblasts - immunology | Heart Ventricles - metabolism | Mice | Interleukin-18 - genetics | Interleukin-18 - metabolism | Myocardial Reperfusion Injury - prevention & control | Heart Ventricles - drug effects | Receptors, Cytoplasmic and Nuclear - metabolism | Myocardial Infarction - genetics | Rats, Wistar | Ventricular Function, Left | Heart Ventricles - immunology | Myocardial Infarction - immunology | Cytoskeletal Proteins - deficiency | Myocardial Infarction - pathology | Heart Ventricles - pathology | Myocardial Contraction | Mice, Inbred C57BL | Cells, Cultured | Inflammasomes - genetics | Carrier Proteins - genetics | Myocardial Reperfusion Injury - metabolism | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Heart Ventricles - physiopathology | CARD Signaling Adaptor Proteins | Apoptosis | Index Medicus
Journal Article
Journal Article
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 07/2015, Volume 117, Issue 4, pp. 352 - 363
RATIONALE:Necrosis is one of the main forms of cardiomyocyte death in heart disease. Recent studies have demonstrated that certain types of necrosis are... 
H19 long noncoding RNA | Myocardial infarction | Oxidative stress | Ischemia/reperfusion injury | MiRNA | Fas-associated death domain protein | Necrosis | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | NECROPTOSIS | ischemia/reperfusion injury | myocardial infarction | ISCHEMIA | CELL-DEATH | PROTECTS | HEART-DISEASE | necrosis | miRNA | PERIPHERAL VASCULAR DISEASE | RIP3 | HEMATOLOGY | EXPRESSION | oxidative stress | CYCLE | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Myocardial Infarction - genetics | Humans | Male | MicroRNAs - metabolism | Dose-Response Relationship, Drug | Myocardial Reperfusion Injury - pathology | Transfection | RNA Interference | Myocardial Infarction - pathology | HEK293 Cells | Myocardial Reperfusion Injury - genetics | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Fas-Associated Death Domain Protein - genetics | Hydrogen Peroxide - toxicity | Signal Transduction | Mice, Inbred C57BL | Fas-Associated Death Domain Protein - metabolism | Rats | RNA, Long Noncoding - genetics | Myocardial Infarction - metabolism | Tumor Necrosis Factor-alpha - toxicity | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | MicroRNAs - genetics | Myocardial Infarction - prevention & control | Oligonucleotides - administration & dosage | RNA, Long Noncoding - metabolism | Myocardial Reperfusion Injury - prevention & control | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 2010, Volume 87, Issue 3, pp. 535 - 544
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2013, Volume 57, Issue 1, pp. 1 - 12
Journal Article