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Journal Article
CARDIOVASCULAR RESEARCH, ISSN 0008-6363, 01/2017, Volume 113, Issue 1, pp. 40 - 51
Aims Myxomatous valve disease (MVD) is the most common aetiology of primary mitral regurgitation. Recent studies suggest that defects in heart valve... 
CARDIAC & CARDIOVASCULAR SYSTEMS | MOUSE | ECM remodelling | BETA-CATENIN | INTERSTITIAL-CELLS | Wnt/beta-catenin signalling | PATHOGENESIS | ORGANIZATION | Myxomatous valve disease | MARFAN-SYNDROME | Heart valve development | Axin2 | EXTRACELLULAR-MATRIX | DIFFERENTIATION | MITRAL-VALVE | UP-REGULATION | Axin Protein - deficiency | Cell Proliferation | Axin Protein - genetics | Aortic Valve - abnormalities | Extracellular Matrix - metabolism | Mitral Valve - metabolism | Mitral Valve - physiopathology | Mitral Valve Insufficiency - genetics | Morphogenesis | Heart Defects, Congenital - genetics | Bone Morphogenetic Proteins - metabolism | Gene Expression Regulation, Developmental | Fibrillin-1 - metabolism | Fibrillin-1 - genetics | Wnt Signaling Pathway | SOX9 Transcription Factor - metabolism | Aortic Valve Insufficiency - metabolism | Genetic Predisposition to Disease | Heart Defects, Congenital - pathology | Aortic Valve - physiopathology | Aortic Valve Insufficiency - physiopathology | Aortic Valve Insufficiency - pathology | Aggrecans - metabolism | Aortic Valve - metabolism | Marfan Syndrome - genetics | Mice, Knockout | Collagen - metabolism | Mitral Valve Insufficiency - pathology | Phenotype | Animals | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Heart Defects, Congenital - metabolism | Heart Defects, Congenital - physiopathology | Mutation | Extracellular Matrix - pathology | Mitral Valve Insufficiency - physiopathology | SOX9 Transcription Factor - genetics | Mitral Valve - abnormalities | Mitral Valve Insufficiency - metabolism | β-catenin signalling | Original | Axin2 • Heart valve development • Myxomatous valve disease • ECM remodelling • Wnt
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 07/2013, Volume 99, Issue 1, pp. 175 - 184
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2013, Volume 33, Issue 1, pp. 121 - 130
OBJECTIVE—Inflammatory activation of valve endothelium is an early phase of aortic valve disease pathogenesis, but subsequent mechanisms are poorly understood.... 
calcification | invasion | transforming growth factor-β | smooth muscle actin | tumor necrosis factor-α | PROGENITOR CELLS | ENDOCARDIAL CUSHION TISSUE | ATHEROSCLEROSIS | FACTOR-KAPPA-B | INTERSTITIAL-CELLS | transforming growth factor-beta | tumor necrosis factor-alpha | HEART | ARTERIAL | GROWTH | RECEPTOR ACTIVATOR | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Heart Valve Diseases - pathology | Humans | Transforming Growth Factor beta1 - metabolism | NF-kappa B - metabolism | Epithelial-Mesenchymal Transition - genetics | Heart Valve Diseases - immunology | Aortic Valve - pathology | Inflammation - metabolism | Transfection | Time Factors | Swine | Inflammation Mediators - metabolism | Calcinosis - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Protein-Serine-Threonine Kinases - metabolism | Aortic Valve - immunology | Recombinant Proteins - metabolism | Signal Transduction | Endothelial Cells - metabolism | Aortic Valve - embryology | Cells, Cultured | Gene Expression Regulation | Activin Receptors, Type I - metabolism | Inflammation - immunology | Quail | Aortic Valve - metabolism | Chick Embryo | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | Receptors, Transforming Growth Factor beta - metabolism | Heart Valve Diseases - metabolism | Heart Valve Diseases - genetics | Inflammation - genetics | Calcinosis - immunology | Primary Cell Culture | Calcinosis - pathology | Endothelial Cells - pathology | Cell Movement
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 03/2010, Volume 106, Issue 4, pp. 712 - 719
RATIONALE:Calcification of heart valve structures is the most common form of valvular disease and is characterized by the appearance of bone-like phenotypes... 
Calcification | Extracellular matrix | Mouse model | Sox9 | Heart valves | CARDIAC & CARDIOVASCULAR SYSTEMS | mouse model | MOUSE EMBRYOS | AORTIC-VALVE | INTERSTITIAL-CELLS | CELL LINEAGE | MATRIX METALLOPROTEINASES | RETINOIC ACID | heart valves | BONE-FORMATION | TOLL-LIKE RECEPTORS | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | calcification | HEMATOLOGY | extracellular matrix | CARDIAC VALVES | Calcinosis - genetics | Inflammation - pathology | Age Factors | Heart Valve Diseases - pathology | Calcium - metabolism | Extracellular Matrix - metabolism | Male | Mitral Valve - metabolism | Gene Knockdown Techniques | Tricuspid Valve - embryology | Tricuspid Valve - drug effects | Inflammation - metabolism | Transfection | Mitral Valve - drug effects | Aging | Female | Calcinosis - metabolism | Osteogenesis - genetics | Disease Models, Animal | Tretinoin - pharmacology | Animals, Newborn | SOX9 Transcription Factor - metabolism | Tissue Culture Techniques | Tricuspid Valve - metabolism | Down-Regulation | Genotype | Mice, Transgenic | Chick Embryo | Collagen Type II - genetics | Phenotype | Animals | Tricuspid Valve - pathology | Heart Valve Diseases - metabolism | Heart Valve Diseases - genetics | Mitral Valve - embryology | Mice | Mitral Valve - pathology | Calcinosis - pathology | Extracellular Matrix - pathology | Integrases - genetics | SOX9 Transcription Factor - genetics
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e48843
The bicuspid aortic valve (BAV) is the most common congenital cardiac anomaly and is frequently associated with calcific aortic valve disease (CAVD). The most... 
MATRIX METALLOPROTEINASES | OSTEOPONTIN | STENOSIS | MULTIDISCIPLINARY SCIENCES | FREQUENCY | MATRIX-METALLOPROTEINASE-9 | PHENOTYPES | BIOLOGICAL-PROPERTIES | CYCLIC PRESSURE | EXPRESSION | INTERSTITIAL-CELLS | Aorta - abnormalities | Immunohistochemistry | Heart Valve Diseases - pathology | Aortic Valve - abnormalities | Transforming Growth Factor beta1 - metabolism | Stress, Mechanical | Aorta - metabolism | Bone Morphogenetic Protein 4 - metabolism | Aortic Valve - pathology | Tissue Inhibitor of Metalloproteinase-2 - metabolism | Matrix Metalloproteinase 9 - metabolism | Swine | Aorta - physiopathology | Calcinosis - metabolism | Heart Valve Diseases - physiopathology | Matrix Metalloproteinase 2 - metabolism | Models, Cardiovascular | Tricuspid Valve - metabolism | Aortic Valve - physiopathology | Tricuspid Valve - physiopathology | Aortic Valve - metabolism | Blotting, Western | Algorithms | Animals | Tricuspid Valve - pathology | Heart Valve Diseases - metabolism | Blood Flow Velocity | Hemodynamics | Calcinosis - pathology | In Vitro Techniques | Apoptosis | Calcinosis - physiopathology | Transforming growth factors | Heart valve diseases | Bone morphogenetic proteins | Calcification (ectopic) | Heart | Tissue inhibitor of metalloproteinases | Laboratories | Pathogenesis | Homeostasis | Immunoblotting | Activation | Shear stresses | Mechanical engineering | Genotype & phenotype | Cell activation | Biomedical materials | Biocompatibility | Aorta | Extracellular matrix | Rheumatic heart disease | Bone matrix | Acute effects | Growth factors | Heart diseases | Cbfa-1 protein | Osteocalcin | Bone morphogenetic protein 4 | Cathepsin L | Congenital diseases | Cytokines | Secretion | Abnormalities | Inflammation | Cathepsin S | Gene expression | Gelatinase B | Blood flow | Gelatinase A | Signaling | Valve leaflets | Calcification | Shear stress | Mechanical stimuli | Aortic valve | Osteogenesis
Journal Article