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Cell Stem Cell, ISSN 1934-5909, 01/2015, Volume 16, Issue 1, pp. 51 - 66
Mesenchymal stem cells (MSCs) reside in the perivascular niche of many organs, including kidney, lung, liver, and heart, although their roles in these tissues... 
REGULATOR | ORIGIN | SONIC HEDGEHOG | PATHWAY | BONE-MARROW NICHE | MYOFIBROBLASTS | NG2 PROTEOGLYCAN | EXPRESSION | MESENCHYMAL STEM-CELLS | GROWTH FACTOR-AA | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Organ Specificity - drug effects | Diphtheria Toxin - pharmacology | Neovascularization, Physiologic - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Fibrosis - metabolism | Cell Lineage - drug effects | Myofibroblasts - metabolism | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Kruppel-Like Transcription Factors - metabolism | Pericytes - pathology | Bone Marrow Cells - drug effects | Colony-Forming Units Assay | Aorta - physiopathology | Homeostasis - drug effects | Heart Ventricles - pathology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Mesenchymal Stromal Cells - drug effects | Endothelial Cells - metabolism | Aorta - drug effects | Pericytes - metabolism | Cells, Cultured | Proteoglycans - metabolism | Antigens - metabolism | Aorta - pathology | Myofibroblasts - cytology | Animals | Heart Ventricles - physiopathology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Blood Vessels - drug effects | Mice | Stem Cell Niche - drug effects | Zinc Finger Protein GLI1 | Fibrosis - pathology | Bone Marrow Cells - metabolism | Endothelial Cells - drug effects | Heart Ventricles - drug effects | Index Medicus
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2015, Volume 21, Issue 7, pp. 777 - 785
Journal Article
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35
Abstract Unresolved inflammation is a major contributor to the development of heart failure following myocardial infarction (MI). Pro-resolving lipid... 
Cardiovascular | Myocardial infarction | Splenic remodeling | Resolution of inflammation | Metabololipidomics | Resolvin D1 | Neutrophils | SURVIVAL | CHRONIC HEART-FAILURE | PROTECTIVE ACTIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | RESOLUTION | TISSUE | RECEPTOR | LIPID MEDIATORS | CELL BIOLOGY | THERAPY | PATHWAY | DISEASE | Cell Count | Male | Myocardial Infarction - diagnostic imaging | Spleen - drug effects | Extracellular Matrix - genetics | Inflammation - complications | Pulmonary Edema - physiopathology | Inflammation - drug therapy | Pulmonary Edema - complications | Receptors, Formyl Peptide - metabolism | Ultrasonography | Myocardial Infarction - physiopathology | Cell Polarity - drug effects | Spleen - pathology | Neutrophil Infiltration - drug effects | Ventricular Function - drug effects | Docosahexaenoic Acids - therapeutic use | Arachidonate 5-Lipoxygenase - metabolism | Docosahexaenoic Acids - chemistry | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Cardiomegaly - physiopathology | Pulmonary Edema - drug therapy | Cardiomegaly - complications | Collagen - metabolism | Docosahexaenoic Acids - pharmacology | Myocardial Infarction - complications | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Prostaglandin-Endoperoxide Synthases - metabolism | Macrophages - drug effects | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Inflammation | Heart attack | Index Medicus | metabololipidomics | liposomes | myocardial infarction | splenic remodeling | neutrophils | resolution of inflammation
Journal Article
SCIENCE, ISSN 0036-8075, 02/2016, Volume 351, Issue 6273, pp. 617 - 621
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 03/2016, Volume 67, Issue 12, pp. 1444 - 1455
Journal Article
Cardiovascular Research, ISSN 0008-6363, 07/2013, Volume 99, Issue 1, pp. 164 - 174
Nucleotide-binding oligomerization domain-Like Receptor with a Pyrin domain 3 (NLRP3) is considered necessary for initiating a profound sterile inflammatory... 
Heart | Myocardial infarction | Inflammasome | Ischaemia-reperfusion | IL-1β | Potassium - metabolism | Up-Regulation | Inflammasomes - metabolism | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Caspase 1 - metabolism | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Myocardial Reperfusion Injury - pathology | Time Factors | Interleukin-1beta - metabolism | Adenosine Triphosphate - metabolism | Myocardial Infarction - physiopathology | Toll-Like Receptors - metabolism | Myocardial Reperfusion Injury - genetics | Disease Models, Animal | Fibroblasts - metabolism | Myocardial Reperfusion Injury - immunology | Rats | Receptors, Cytoplasmic and Nuclear - genetics | Myocardial Infarction - metabolism | Myocardial Reperfusion Injury - physiopathology | Mice, Knockout | Fibroblasts - drug effects | Lipopolysaccharides - pharmacology | Fibroblasts - immunology | Heart Ventricles - metabolism | Mice | Interleukin-18 - genetics | Interleukin-18 - metabolism | Myocardial Reperfusion Injury - prevention & control | Heart Ventricles - drug effects | Receptors, Cytoplasmic and Nuclear - metabolism | Myocardial Infarction - genetics | Rats, Wistar | Ventricular Function, Left | Heart Ventricles - immunology | Myocardial Infarction - immunology | Cytoskeletal Proteins - deficiency | Myocardial Infarction - pathology | Heart Ventricles - pathology | Myocardial Contraction | Mice, Inbred C57BL | Cells, Cultured | Inflammasomes - genetics | Carrier Proteins - genetics | Myocardial Reperfusion Injury - metabolism | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Heart Ventricles - physiopathology | CARD Signaling Adaptor Proteins | Apoptosis | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 05/2013, Volume 62, Issue 5, pp. 1697 - 1708
Impaired cardiac microvascular function contributes to cardiovascular complications in diabetes. Glucagon-like peptide-1 (GLP-1) exhibits potential... 
OXIDATIVE STRESS | IN-VITRO | GLP-1 | RECEPTOR AGONISTS | GLUCOSE | ENDOTHELIAL-CELLS | DISEASE | KINASE | ENDOCRINOLOGY & METABOLISM | ANGIOTENSIN-II | DYSFUNCTION | Endothelium, Vascular - cytology | Microvessels - physiopathology | AMP-Activated Protein Kinases - metabolism | Diabetic Cardiomyopathies - metabolism | Microvessels - metabolism | Microvessels - pathology | Endothelium, Vascular - drug effects | Male | Diabetic Cardiomyopathies - physiopathology | Cardiotonic Agents - therapeutic use | Diabetic Angiopathies - physiopathology | Diabetic Angiopathies - pathology | Diabetic Cardiomyopathies - prevention & control | Diabetic Angiopathies - prevention & control | Hyperglycemia - physiopathology | Cyclic AMP - metabolism | Heart Ventricles - pathology | Disease Models, Animal | Hypoglycemic Agents - therapeutic use | Diabetic Angiopathies - metabolism | Second Messenger Systems - drug effects | Glucagon-Like Peptide 1 - metabolism | Glucagon-Like Peptide 1 - analogs & derivatives | Cells, Cultured | Microvessels - drug effects | Rats | Random Allocation | Rats, Sprague-Dawley | Animals | Heart Ventricles - physiopathology | Hyperglycemia - blood | Endothelium, Vascular - metabolism | rho GTP-Binding Proteins - metabolism | Venoms - therapeutic use | Diabetic Cardiomyopathies - pathology | Endothelium, Vascular - pathology | Heart Ventricles - metabolism | Oxidative Stress - drug effects | Peptides - therapeutic use | Glucagon-Like Peptide 1 - therapeutic use | Heart Ventricles - drug effects | Prevention | Type 2 diabetes | Complications and side effects | Glucagon | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Cardiovascular diseases | Risk factors | Index Medicus | Abridged Index Medicus | Original Research
Journal Article