X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (71210) 71210
Newspaper Article (5202) 5202
Newsletter (735) 735
Book Chapter (518) 518
Magazine Article (224) 224
Book / eBook (71) 71
Reference (33) 33
Trade Publication Article (32) 32
Dissertation (27) 27
Conference Proceeding (17) 17
Transcript (12) 12
Streaming Video (7) 7
Government Document (5) 5
Publication (5) 5
Web Resource (5) 5
Book Review (2) 2
Report (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (33946) 33946
male (32829) 32829
animals (32233) 32233
female (22488) 22488
cardiac & cardiovascular systems (20471) 20471
heart (17977) 17977
heart failure (16770) 16770
rats (13539) 13539
middle aged (13411) 13411
aged (9806) 9806
adult (9419) 9419
cardiovascular disease (9360) 9360
cardiology (9142) 9142
heart diseases (7714) 7714
physiology (7635) 7635
heart ventricles - drug effects (7514) 7514
electrocardiography (7509) 7509
heart - drug effects (7078) 7078
myocardium - metabolism (6961) 6961
echocardiography (6946) 6946
mortality (6488) 6488
pharmacology & pharmacy (6290) 6290
heart rate - drug effects (5875) 5875
risk factors (5862) 5862
hypertension (5742) 5742
time factors (5724) 5724
ventricle (5681) 5681
care and treatment (5632) 5632
heart-failure (5582) 5582
research (5580) 5580
analysis (5419) 5419
treatment outcome (5367) 5367
blood pressure - drug effects (5347) 5347
medicine & public health (5325) 5325
heart attacks (5178) 5178
cardiomyopathy (5152) 5152
cardiovascular (5042) 5042
mice (4933) 4933
blood pressure (4912) 4912
dogs (4816) 4816
myocardial contraction - drug effects (4806) 4806
ischemia (4728) 4728
rats, sprague-dawley (4697) 4697
peripheral vascular disease (4565) 4565
heart ventricles - physiopathology (4528) 4528
rodents (4511) 4511
health aspects (4507) 4507
disease models, animal (4288) 4288
in vitro techniques (4277) 4277
cell biology (4256) 4256
patients (4256) 4256
heart rate (4179) 4179
myocardium - pathology (4066) 4066
medicine (4022) 4022
chronic illnesses (3978) 3978
surgery (3929) 3929
physiological aspects (3918) 3918
heart failure - physiopathology (3900) 3900
rats, wistar (3900) 3900
cardiovascular diseases (3740) 3740
cardiac arrhythmia (3707) 3707
research article (3624) 3624
cardiovascular system (3602) 3602
heart ventricles (3559) 3559
myocardial infarction (3559) 3559
hypertrophy (3474) 3474
cardiomyocytes (3448) 3448
cardiac patients (3445) 3445
dose-response relationship, drug (3439) 3439
studies (3422) 3422
heart - physiology (3379) 3379
hemodynamics - drug effects (3352) 3352
diagnosis (3338) 3338
oxidative stress (3318) 3318
expression (3299) 3299
disease (3266) 3266
proteins (3200) 3200
apoptosis (3159) 3159
prognosis (3090) 3090
cells, cultured (3036) 3036
abridged index medicus (3015) 3015
myocytes, cardiac - drug effects (3009) 3009
myocytes, cardiac - metabolism (2990) 2990
myocardial-infarction (2965) 2965
drug therapy (2955) 2955
hemodynamics (2882) 2882
medicine, research & experimental (2842) 2842
multidisciplinary sciences (2842) 2842
heart ventricles - metabolism (2836) 2836
therapy (2802) 2802
activation (2772) 2772
heart - physiopathology (2753) 2753
arrhythmia (2724) 2724
myocardium (2695) 2695
follow-up studies (2685) 2685
heart failure - drug therapy (2668) 2668
dysfunction (2651) 2651
prospective studies (2634) 2634
calcium - metabolism (2617) 2617
coronary vessels (2579) 2579
more...
Library Location Library Location
Library Location Library Location
X
Sort by Item Count (A-Z)
Filter by Count
Gerstein Science - Stacks (17) 17
Collection Dvlpm't (Acquisitions) - Vendor file (7) 7
Scarborough Hospital - General (3) 3
Sunnybrook Health Sciences Centre - Sunnybrook Stacks (3) 3
Online Resources - Online (2) 2
St. Michael's Hospital - Stacks (2) 2
Trillium Health Centre - Stacks (2) 2
Credit Valley Hospital - Stacks (1) 1
Scarborough Hospital - Online (1) 1
St. Michael's Hospital - Circulation Desk (1) 1
Women's College Hospital - Online (1) 1
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (76317) 76317
German (586) 586
Japanese (530) 530
Russian (482) 482
French (276) 276
Portuguese (189) 189
Chinese (177) 177
Italian (177) 177
Polish (125) 125
Spanish (113) 113
Czech (32) 32
Ukrainian (27) 27
Turkish (18) 18
Norwegian (17) 17
Bulgarian (13) 13
Dutch (10) 10
Hungarian (8) 8
Slovak (8) 8
Danish (6) 6
Romanian (4) 4
Serbian (4) 4
Hebrew (3) 3
Arabic (2) 2
Lithuanian (2) 2
Slovenian (2) 2
Afrikaans (1) 1
Finnish (1) 1
Swedish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


British journal of pharmacology, ISSN 0007-1188, 06/2013, Volume 169, Issue 3, pp. 528 - 538
Background and Purpose PDE3 and/or PDE4 control ventricular effects of catecholamines in several species but their relative effects in failing human ventricle are unknown... 
inotropism and lusitropism | human heart failure | phosphodiesterases 3 and 4 | noradrenaline and adrenaline | metoprolol | β1 and β2 adrenoceptors | and β | adrenoceptors | Adrenergic beta-1 Receptor Antagonists - adverse effects | Heart Failure - surgery | Cyclic Nucleotide Phosphodiesterases, Type 3 - chemistry | Norepinephrine - pharmacology | Adrenergic alpha-Agonists - chemistry | Phosphodiesterase 3 Inhibitors - pharmacology | Humans | Middle Aged | Adrenergic beta-1 Receptor Antagonists - pharmacology | Heart Failure - physiopathology | Myocardial Contraction - drug effects | Receptors, Adrenergic, beta-1 - chemistry | Adrenergic beta-1 Receptor Antagonists - therapeutic use | Adrenergic beta-Agonists - chemistry | Epinephrine - agonists | Receptors, Adrenergic, beta-2 - chemistry | Cardiotonic Agents - therapeutic use | Cyclic Nucleotide Phosphodiesterases, Type 3 - metabolism | Cyclic Nucleotide Phosphodiesterases, Type 4 - metabolism | Cyclic Nucleotide Phosphodiesterases, Type 4 - chemistry | Adrenergic beta-2 Receptor Antagonists - pharmacology | Adrenergic beta-Agonists - pharmacology | Heart Transplantation | Metoprolol - therapeutic use | Receptors, Adrenergic, beta-1 - metabolism | Drug Resistance - drug effects | Heart Failure - metabolism | Anti-Arrhythmia Agents - therapeutic use | Cardiotonic Agents - pharmacology | Metoprolol - adverse effects | Norepinephrine - agonists | Phosphodiesterase 4 Inhibitors - pharmacology | Receptors, Adrenergic, beta-2 - metabolism | Heart Failure - drug therapy | Heart Ventricles - physiopathology | Anti-Arrhythmia Agents - adverse effects | Adrenergic alpha-Agonists - pharmacology | Heart Ventricles - metabolism | In Vitro Techniques | Epinephrine - pharmacology | Heart Ventricles - drug effects | Research Paper with
Journal Article
Journal Article
Cell stem cell, ISSN 1934-5909, 2015, Volume 16, Issue 1, pp. 51 - 66
Mesenchymal stem cells (MSCs) reside in the perivascular niche of many organs, including kidney, lung, liver, and heart, although their roles in these tissues... 
REGULATOR | ORIGIN | SONIC HEDGEHOG | PATHWAY | BONE-MARROW NICHE | MYOFIBROBLASTS | NG2 PROTEOGLYCAN | EXPRESSION | MESENCHYMAL STEM-CELLS | GROWTH FACTOR-AA | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Organ Specificity - drug effects | Diphtheria Toxin - pharmacology | Neovascularization, Physiologic - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Fibrosis - metabolism | Cell Lineage - drug effects | Myofibroblasts - metabolism | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Kruppel-Like Transcription Factors - metabolism | Pericytes - pathology | Bone Marrow Cells - drug effects | Colony-Forming Units Assay | Aorta - physiopathology | Homeostasis - drug effects | Heart Ventricles - pathology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Mesenchymal Stromal Cells - drug effects | Endothelial Cells - metabolism | Aorta - drug effects | Pericytes - metabolism | Cells, Cultured | Proteoglycans - metabolism | Antigens - metabolism | Aorta - pathology | Myofibroblasts - cytology | Animals | Heart Ventricles - physiopathology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Blood Vessels - drug effects | Mice | Stem Cell Niche - drug effects | Zinc Finger Protein GLI1 | Fibrosis - pathology | Bone Marrow Cells - metabolism | Endothelial Cells - drug effects | Heart Ventricles - drug effects
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 11/2013, Volume 305, Issue 10, pp. H1440 - H1450
Xanthine oxidase (XO) is increased in human and rat left ventricular (LV) myocytes with volume overload (VO) of mitral regurgitation and aortocaval fistula... 
Heart failure | Xanthine oxidase | Mitochondria | Volume overload | volume overload | heart failure | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | INDUCED HEART-FAILURE | mitochondria | MITRAL REGURGITATION | SUBSARCOLEMMAL MITOCHONDRIA | ALLOPURINOL | CA2+ ATPASE | SARCOLIPIN | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | xanthine oxidase | RETICULUM CALCIUM-TRANSPORT | CREATINE-KINASE | Sarcoplasmic Reticulum - drug effects | Heart Failure - enzymology | Heart Failure - physiopathology | Systole - drug effects | Heart Ventricles - diagnostic imaging | Sarcoplasmic Reticulum - enzymology | Myocytes, Cardiac - enzymology | Time Factors | Adenosine Triphosphate - metabolism | Heart Ventricles - enzymology | Ultrasonography | Diastole - drug effects | Heart Failure - etiology | Xanthine Oxidase - antagonists & inhibitors | Disease Models, Animal | Xanthine Oxidase - metabolism | Ventricular Function, Left - drug effects | Enzyme Inhibitors - pharmacology | Ventricular Pressure - drug effects | Rats | Stroke Volume - drug effects | Cardiotonic Agents - pharmacology | Rats, Sprague-Dawley | Heart Failure - drug therapy | Animals | Myocytes, Cardiac - drug effects | Heart Ventricles - physiopathology | Calcium Signaling - drug effects | Oxygen Consumption - drug effects | Creatine Kinase - metabolism | Allopurinol - pharmacology | Hemodynamics - drug effects | Energy Metabolism - drug effects | Heart Ventricles - drug effects | Oxidases | Physiological aspects | Xanthine | Health aspects | Heart ventricle, Left | Call for Papers
Journal Article
Journal Article
European journal of heart failure, ISSN 1388-9842, 2016, Volume 18, Issue 4, pp. 386 - 393
Journal Article
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35
...) and its free acid form (RvD1) in the left ventricle (LV) and splenic remodeling post-MI. 8 to 12-week old male, C57BL/6J-mice were subjected to coronary artery ligation and Lipo-RvD1 or RvD1 (3 μg/kg/day... 
Cardiovascular | Myocardial infarction | Splenic remodeling | Resolution of inflammation | Metabololipidomics | Resolvin D1 | Neutrophils | SURVIVAL | CHRONIC HEART-FAILURE | PROTECTIVE ACTIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | RESOLUTION | TISSUE | RECEPTOR | LIPID MEDIATORS | CELL BIOLOGY | THERAPY | PATHWAY | DISEASE | Cell Count | Male | Myocardial Infarction - diagnostic imaging | Spleen - drug effects | Extracellular Matrix - genetics | Inflammation - complications | Pulmonary Edema - physiopathology | Inflammation - drug therapy | Pulmonary Edema - complications | Receptors, Formyl Peptide - metabolism | Ultrasonography | Myocardial Infarction - physiopathology | Cell Polarity - drug effects | Spleen - pathology | Neutrophil Infiltration - drug effects | Ventricular Function - drug effects | Docosahexaenoic Acids - therapeutic use | Arachidonate 5-Lipoxygenase - metabolism | Docosahexaenoic Acids - chemistry | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Cardiomegaly - physiopathology | Pulmonary Edema - drug therapy | Cardiomegaly - complications | Collagen - metabolism | Docosahexaenoic Acids - pharmacology | Myocardial Infarction - complications | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Prostaglandin-Endoperoxide Synthases - metabolism | Macrophages - drug effects | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Inflammation | Heart attack | metabololipidomics | liposomes | myocardial infarction | splenic remodeling | neutrophils | resolution of inflammation
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 50, pp. 20785 - 20798
A key feature of acute myocardial infarction (AMI) is an alteration in cardiac architecture. Signaling events that result in the inhibition of glycogen... 
CELLS | PROTECTION | angiogenesis | endothelial cell | BIOCHEMISTRY & MOLECULAR BIOLOGY | myocardial infarction | GSK-3-BETA | INJURY | ISCHEMIA | allosteric regulation | TIDEGLUSIB | glycogen synthase kinase 3 (GSK-3) | STRATEGIES | Wnt signaling | CARDIOPROTECTION | EXPRESSION | MODULATION | Neovascularization, Physiologic - drug effects | Apoptosis - drug effects | Atrial Remodeling - drug effects | Male | Aorta - metabolism | Angiogenesis Inducing Agents - therapeutic use | Ligation | Protein Processing, Post-Translational - drug effects | Thiadiazoles - therapeutic use | Myocardial Infarction - pathology | Female | Myocardial Infarction - physiopathology | Angiogenesis Inducing Agents - pharmacology | Phosphorylation - drug effects | Heart Ventricles - pathology | Disease Models, Animal | Thiadiazoles - pharmacology | Allosteric Regulation - drug effects | Coronary Vessels - drug effects | Coronary Vessels - pathology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Aorta - drug effects | Mice, Inbred C57BL | Myocardial Infarction - metabolism | Glycogen Synthase Kinase 3 - metabolism | Aorta - pathology | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Protein Kinase Inhibitors - therapeutic use | Aorta - surgery | Heart Ventricles - metabolism | Mice, Inbred BALB C | Protein Kinase Inhibitors - pharmacology | In Vitro Techniques | Heart Ventricles - drug effects | Gene Regulation
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2013, Volume 183, Issue 3, pp. 720 - 734
Hypoplastic left heart syndrome (HLHS) is a severe cardiac malformation characterized by left ventricle (LV... 
Pathology | FIBROSIS | PREMATURE SENESCENCE | EPITHELIAL-CELLS | CARDIOMYOCYTES | HIF-1-ALPHA | PATHOLOGY | HYPOXIA | EXPRESSION | CONTRIBUTES | FIBROBLASTS | PATHOPHYSIOLOGY | Mutagens - toxicity | Humans | Fetus - metabolism | Myocytes, Smooth Muscle - pathology | Cellular Senescence - drug effects | Fetus - pathology | Heart Ventricles - embryology | Cell Lineage - drug effects | Pluripotent Stem Cells - pathology | Hypoplastic Left Heart Syndrome - embryology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Heart Ventricles - pathology | Fibroblasts - metabolism | Cell Hypoxia - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Embryoid Bodies - pathology | Myocardium - pathology | Fibroblasts - pathology | Embryoid Bodies - drug effects | Cellular Reprogramming - drug effects | Fetus - drug effects | Pluripotent Stem Cells - metabolism | Up-Regulation - drug effects | Phenotype | Hypoplastic Left Heart Syndrome - pathology | Cell Cycle Checkpoints - drug effects | Cell Differentiation - drug effects | Fibroblasts - drug effects | Pluripotent Stem Cells - drug effects | Morphogenesis - drug effects | Human Umbilical Vein Endothelial Cells - pathology | DNA Damage | Transforming Growth Factor beta - metabolism | Cell Line, Transformed | Heart Ventricles - drug effects
Journal Article
PloS one, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, p. e0135988
.... However, their relative importance in mediating estrogen protective action is unknown. We found that, in the male mouse ventricle, Gper1 transcripts are three-and seventeen-fold more abundant than Esr1 and Esr2 mRNAs, respectively... 
ISCHEMIA-REPERFUSION INJURY | PERMEABILITY TRANSITION PORE | ACTIVATION | MOUSE HEART | MULTIDISCIPLINARY SCIENCES | IN-VIVO | KINASE | ALPHA | RAT-HEART | EXPRESSION | BETA | Receptors, Estrogen - metabolism | Estrogens - pharmacology | Mitochondria, Heart - metabolism | Receptors, G-Protein-Coupled - metabolism | Recovery of Function - drug effects | Calcium - metabolism | Glycogen Synthase Kinase 3 | Glycogen Synthase Kinase 3 beta | Male | Mitochondria, Heart - drug effects | RNA, Messenger - metabolism | Myocardial Reperfusion Injury - enzymology | Estrogens - therapeutic use | Cardiotonic Agents - therapeutic use | Estrogen Receptor beta - metabolism | Myocardial Reperfusion Injury - pathology | Estrogen Receptor beta - genetics | Time Factors | Protein Kinase C - metabolism | Heart Ventricles - enzymology | Estrogen Receptor alpha - metabolism | Myocardial Reperfusion Injury - drug therapy | Phosphorylation - drug effects | Heart Ventricles - pathology | Mitochondrial Membrane Transport Proteins - metabolism | Receptors, Estrogen - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Cardiotonic Agents - pharmacology | Myocardial Reperfusion Injury - physiopathology | Animals | Estrogen Receptor alpha - genetics | MAP Kinase Signaling System - drug effects | Heart Ventricles - physiopathology | Signal Transduction - drug effects | Models, Biological | Receptors, G-Protein-Coupled - genetics | Heart Ventricles - drug effects | Heart | Phosphorylation | Protein kinase C | Mitochondrial permeability transition pore | Estrogens | Estrogen | Membrane permeability | Science | Recovery of function | Estrogen receptors | AKT protein | Heart function | Kinases | Proteins | Receptors | Reperfusion | Salvage | Ischemia | Rodents | Physiology | Inhibition | Protocol | Translocation | Adenosine | Extracellular signal-regulated kinase | Permeability | Calcium permeability | Calcium (mitochondrial) | Medicine | Signaling | Perfusion | Coronary vessels | Infarction | MPTP | Ventricle | Cell and Molecular Biology | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Cell- och molekylärbiologi | Medicinska och farmaceutiska grundvetenskaper
Journal Article
European journal of heart failure, ISSN 1388-9842, 09/2012, Volume 14, Issue 9, pp. 1067 - 1074
Journal Article