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Nature, ISSN 0028-0836, 05/2017, Volume 545, Issue 7655, pp. 491 - 494
Journal Article
Plant Physiology, ISSN 0032-0889, 11/2011, Volume 157, Issue 3, pp. 1243 - 1254
Many eukaryotes have from one to three heat shock factors (Hsfs), but plants have more than 20 Hsfs, designated class A, B, and C Class A Hsfs are activators... 
Heat stress disorders | Transcriptional regulatory elements | Transcription factors | Genes | Plant roots | Heat tolerance | Plants | Repression | ENVIRONMENTAL STRESS AND ADAPTATION TO STRESS | Seedlings | Plant cells | SHOCK FACTORS | NUCLEAR IMPORT | PROTEIN | THALIANA | HIGH-TEMPERATURE | ENVIRONMENTAL-STRESS | IDENTIFICATION | FACTOR DREB2A | STRESS TRANSCRIPTION FACTORS | TOMATO | PLANT SCIENCES | Arabidopsis - physiology | Transcription Factors - chemistry | Hypocotyl - genetics | Molecular Sequence Data | Promoter Regions, Genetic - genetics | Heat Shock Transcription Factors | Arabidopsis Proteins - metabolism | DNA-Binding Proteins - metabolism | Heat-Shock Proteins - genetics | Adaptation, Physiological - genetics | Plant Proteins - chemistry | Gene Expression Regulation, Plant | Transcription, Genetic | Plant Proteins - metabolism | Repressor Proteins - metabolism | Protein Structure, Tertiary | Amino Acid Sequence | Arabidopsis Proteins - genetics | Repressor Proteins - chemistry | Heat-Shock Proteins - metabolism | Hypocotyl - growth & development | Repressor Proteins - genetics | Up-Regulation - genetics | Hot Temperature | Transcription Factors - genetics | DNA-Binding Proteins - genetics | DNA-Binding Proteins - chemistry | Heat-Shock Response - genetics | Arabidopsis - genetics | Transcription Factors - metabolism | Plant Proteins - genetics | Phenotype | Arabidopsis Proteins - chemistry | Genes, Plant - genetics | Models, Biological | Heat-Shock Proteins - chemistry | Arabidopsis thaliana | Heat shock proteins | Genetic aspects | Research | Phytochemistry | Plant genetics | Data processing | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 04/2013, Volume 78, Issue 1, pp. 65 - 80
Mutations in VCP cause multisystem degeneration impacting the nervous system, muscle, and/or bone. Patients may present with ALS, Parkinsonism, frontotemporal... 
PATHOGENESIS | PARKIN | VALOSIN-CONTAINING-PROTEIN | INCLUSION-BODY MYOPATHY | DROSOPHILA MODEL | FRONTOTEMPORAL DEMENTIA | PAGETS-DISEASE | BONE | NEUROSCIENCES | P97 | DEGENERATION | Embryo, Mammalian | Humans | Ganglia, Spinal - cytology | Drosophila Proteins - metabolism | HSP72 Heat-Shock Proteins - genetics | Protein Tyrosine Phosphatases - genetics | Neurons - ultrastructure | Time Factors | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Neuromuscular Junction - genetics | Neurons - metabolism | Protein-Serine-Threonine Kinases - metabolism | Mitochondrial Membrane Transport Proteins - metabolism | Cell Cycle Proteins - metabolism | Enzyme Inhibitors - pharmacology | Ubiquitin-Protein Ligases - metabolism | Adenosine Triphosphatases - metabolism | Mutation - genetics | Leupeptins - pharmacology | GTP Phosphohydrolases - metabolism | Luminescent Proteins - genetics | Adenosine Triphosphatases - genetics | Ubiquitin-Protein Ligases - genetics | RNA, Small Interfering - metabolism | Immunoprecipitation | Neuromuscular Junction - metabolism | Valosin Containing Protein | Mitochondria - ultrastructure | Transfection | Mitochondria - genetics | Proton Ionophores - pharmacology | Cell Cycle Proteins - genetics | Microscopy, Electron, Transmission | Animals, Genetically Modified | Gene Expression Regulation - genetics | Drosophila | RNA, Small Interfering - pharmacology | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | Proteins - metabolism | Drosophila Proteins - genetics | In Vitro Techniques | Ubiquitination - genetics | Luminescent Proteins - metabolism | Ubiquitin | Analysis | Genomics | Quality control | Amyotrophic lateral sclerosis | Genetic aspects | Mitochondrial DNA | Dementia | Neurons | Parkinsons disease | Biosynthesis | Digital cameras | Kinases | DNA repair | Proteins | Mitochondria | Brain research | Insects | Microscopy | Morphology | Mutation | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 10/2012, Volume 490, Issue 7418, pp. 49 - 54
Journal Article
Science, ISSN 0036-8075, 12/2010, Volume 330, Issue 6012, pp. 1820 - 1824
How can species remain unaltered for long periods yet also undergo rapid diversification? By linking genetic variation to phenotypic variation via... 
Quantitative trait loci | Oxidative stress | Phenotypes | Genetic variation | Capacitors | REPORTS | Evolutionary genetics | Evolution | Genomes | Genotypes | Phenotypic traits | MORPHOLOGICAL EVOLUTION | YEAST | PHENOTYPIC VARIATION | PROTEIN | MULTIDISCIPLINARY SCIENCES | CAPACITOR | DRUG-RESISTANCE | CHAPERONE | SACCHAROMYCES-CEREVISIAE | ACTS | Adaptation, Physiological | Hydroxyurea - pharmacology | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Saccharomyces cerevisiae - genetics | Open Reading Frames | Stress, Physiological | Intracellular Signaling Peptides and Proteins - metabolism | Mitochondrial Proteins - genetics | Genetic Variation | Mitochondrial Proteins - metabolism | HSP90 Heat-Shock Proteins - genetics | Drug Resistance, Fungal | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Saccharomyces cerevisiae - physiology | Protein-Serine-Threonine Kinases - genetics | Sulfurtransferases - genetics | Saccharomyces cerevisiae Proteins - genetics | Sirolimus - pharmacology | Polymorphism, Genetic | Biological Evolution | Deoxycholic Acid - pharmacology | Saccharomyces cerevisiae Proteins - metabolism | HSP90 Heat-Shock Proteins - metabolism | Quantitative Trait Loci | Saccharomyces cerevisiae - growth & development | Sulfurtransferases - metabolism | Crosses, Genetic | Phenotype | Heat shock proteins | Genotype | Research | Properties | Genotype & phenotype | Genetic diversity | Yeast | Cellular biology | Molecular biology | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 03/2010, Volume 12, Issue 3, pp. 213 - 223
Impaired selective turnover of p62 by autophagy causes severe liver injury accompanied by the formation of p62-positive inclusions and upregulation of... 
OXIDATIVE STRESS | PROTEIN | MECHANISM | CUL3-BASED E3 LIGASE | STRUCTURAL BASIS | DLG MOTIFS | DEGRADATION | MICE | BETA-CELL MASS | INDUCTION | CELL BIOLOGY | Adaptor Proteins, Signal Transducing - chemistry | Liver - pathology | Microtubule-Associated Proteins - genetics | Cytoskeletal Proteins - genetics | Gene Expression - genetics | Protein Interaction Domains and Motifs - physiology | Sequestosome-1 Protein | Humans | Oxidative Stress - physiology | Crystallography, X-Ray | Hepatocytes - pathology | Autophagy - physiology | Intracellular Signaling Peptides and Proteins - metabolism | Hepatocytes - metabolism | Liver - physiopathology | Heat-Shock Proteins - genetics | Mutation - physiology | Transfection | Organ Size - genetics | Cytoskeletal Proteins - metabolism | NF-E2-Related Factor 2 - genetics | Inclusion Bodies - metabolism | Kelch-Like ECH-Associated Protein 1 | Cell Line | Binding, Competitive - physiology | Heat-Shock Proteins - metabolism | Liver - metabolism | Models, Molecular | Mice, Transgenic | Cytoskeletal Proteins - chemistry | Mice, Knockout | Protein Interaction Mapping | Autophagy-Related Protein 7 | Animals | Models, Biological | NF-E2-Related Factor 2 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Calorimetry | Signal Transduction - physiology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Heat-Shock Proteins - chemistry | Protein Binding - physiology | Autophagy (Cytology) | Care and treatment | Transcription factors | Liver diseases | Physiological aspects | Genetic aspects | Research | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 12/2011, Volume 480, Issue 7377, pp. 391 - 395
Journal Article
PLoS Biology, ISSN 1544-9173, 02/2007, Volume 5, Issue 2, pp. 0179 - 0189
The mammalian circadian timing system consists of a master pacemaker in neurons of the suprachiasmatic nucleus (SCN) and clocks of a similar molecular makeup... 
RAT-LIVER | REVEALS PERSISTENT | SUPRACHIASMATIC NUCLEUS | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEAT-SHOCK RESPONSE | MOUSE | BIOLOGY | GENE-EXPRESSION | TIME | PERIPHERAL-TISSUES | BINDING | INDIVIDUAL FIBROBLASTS | Male | Biological Clocks - drug effects | DNA-Binding Proteins - metabolism | Hepatocytes - cytology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Liver - drug effects | Cell Cycle Proteins - genetics | Nuclear Proteins - genetics | Hepatocytes - drug effects | Hepatocytes - physiology | Nuclear Receptor Subfamily 1, Group D, Member 1 | ARNTL Transcription Factors | Basic Helix-Loop-Helix Transcription Factors - genetics | Cell Cycle Proteins - metabolism | Circadian Rhythm - genetics | Mice, Transgenic | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Period Circadian Proteins | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Animals | Biological Clocks - genetics | Chronobiology Phenomena | Liver - physiology | Protein Array Analysis | Doxycycline - pharmacology | Anti-Bacterial Agents - pharmacology | Liver - cytology | Mice | Circadian Rhythm - drug effects | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus | Neuroscience | Molecular Biology | Computational Biology | Genetics and Genomics | Mus (Mouse) | Biochemistry | Physiology | Mammals | Cell Biology | Circadian rhythm | Kinases | Experiments | Liver | Binding sites
Journal Article
Molecular Cell, ISSN 1097-2765, 05/2012, Volume 46, Issue 4, pp. 518 - 529
Translation arrest leads to an endonucleolytic cleavage of mRNA that is termed no-go decay (NGD). It has been reported that the Dom34:Hbs1 complex stimulates... 
YEAST | DEPENDENT TRANSLATION ARREST | TERMINATION CODON | LACKING | BIOCHEMISTRY & MOLECULAR BIOLOGY | NO-GO DECAY | EXOSOME | SURVEILLANCE | DEGRADATION | CONTROL PATHWAYS | POLY(A) TAIL | CELL BIOLOGY | Adaptor Proteins, Signal Transducing - chemistry | Endoribonucleases - chemistry | Endoribonucleases - genetics | Peptide Elongation Factors - metabolism | Peptide Chain Elongation, Translational | Saccharomyces cerevisiae - genetics | Ribosomes - metabolism | Multiprotein Complexes - genetics | RNA, Messenger - metabolism | GTP-Binding Proteins - genetics | Cell Cycle Proteins - chemistry | Saccharomyces cerevisiae - metabolism | Multiprotein Complexes - metabolism | Base Sequence | Cell Cycle Proteins - genetics | HSP70 Heat-Shock Proteins - chemistry | Protein Interaction Domains and Motifs | RNA, Fungal - genetics | Peptide Elongation Factors - genetics | Endoribonucleases - metabolism | GTP-Binding Proteins - chemistry | RNA, Messenger - genetics | Cell Cycle Proteins - metabolism | HSP70 Heat-Shock Proteins - genetics | Saccharomyces cerevisiae Proteins - genetics | RNA Stability | HSP70 Heat-Shock Proteins - metabolism | RNA, Fungal - metabolism | Nonsense Mediated mRNA Decay | Multiprotein Complexes - chemistry | Peptide Elongation Factors - chemistry | Adaptor Proteins, Signal Transducing - genetics | Saccharomyces cerevisiae Proteins - metabolism | Adaptor Proteins, Signal Transducing - metabolism | GTP-Binding Proteins - metabolism | Saccharomyces cerevisiae Proteins - chemistry | RNA | Genetic translation | Messenger RNA | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 09/2010, Volume 12, Issue 9, pp. 863 - 875
Accumulation of unwanted/misfolded proteins in aggregates has been observed in airways of patients with cystic fibrosis (CF), a life-threatening genetic... 
APOPTOSIS | UNFOLDED PROTEIN RESPONSE | BECLIN 1 | DOWN-REGULATION | ENDOPLASMIC-RETICULUM | MICE | GENE-TRANSFER | TISSUE TRANSGLUTAMINASE | NEURODEGENERATIVE DISEASES | CELL-DEATH | CELL BIOLOGY | Reactive Oxygen Species - metabolism | Respiratory Mucosa - drug effects | Sequestosome-1 Protein | Humans | Transglutaminases - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Autophagy - drug effects | Young Adult | Cystamine - therapeutic use | Inflammation - metabolism | Autophagy - genetics | Nasal Polyps - metabolism | Mice, Inbred CFTR | Respiratory Mucosa - cytology | Cystic Fibrosis - metabolism | Membrane Proteins - genetics | Mice, Transgenic | Protein Transport - genetics | Apoptosis Regulatory Proteins - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Models, Biological | Cystic Fibrosis - genetics | Transglutaminases - metabolism | Adolescent | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Mice | GTP-Binding Proteins | Cystic Fibrosis - drug therapy | Protein Binding - physiology | Microtubule-Associated Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Salicylates - pharmacology | Epithelial Sodium Channels - genetics | Apoptosis Regulatory Proteins - genetics | Adult | Cystamine - pharmacology | Membrane Proteins - metabolism | Acetylcysteine - therapeutic use | Beclin-1 | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Cell Line | Small Ubiquitin-Related Modifier Proteins - metabolism | Heat-Shock Proteins - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Antioxidants - pharmacology | Mice, Inbred Strains | Nasal Polyps - drug therapy | Antioxidants - therapeutic use | Animals | Acetylcysteine - pharmacology | Adaptor Proteins, Signal Transducing - genetics | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Organometallic Compounds - pharmacology | Complications and side effects | Active oxygen | Gene mutations | Lung diseases | Physiological aspects | Cystic fibrosis | Genetic aspects | Research | Health aspects | Risk factors | Index Medicus
Journal Article