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Gastroenterology, ISSN 0016-5085, 2017, Volume 153, Issue 1, pp. 166 - 177
Background & Aims Esophageal squamous cell carcinoma (ESCC) is the most common subtype of esophageal cancer. Little is known about the genetic changes that... 
Gastroenterology and Hepatology | Driver Mutation | Sequencing | Carcinogenesis | Esophagus | MUTATIONAL PROCESSES | POPULATION | PRECANCEROUS LESIONS | ADENOCARCINOMA | BARRETTS-ESOPHAGUS | CELL CARCINOMA | SOMATIC MUTATIONS | CANCER DEVELOPMENT | HELICOBACTER-PYLORI INFECTION | DNA | GASTROENTEROLOGY & HEPATOLOGY | Carcinoma, Squamous Cell - genetics | Humans | Apoptosis - genetics | DNA Repair - genetics | Phylogeny | Esophagitis - metabolism | Esophagitis - pathology | Tumor Suppressor Protein p53 - genetics | DNA Copy Number Variations | Chromosomes, Human, Pair 2 - genetics | Chromosomes, Human, Pair 9 - genetics | Carcinoma in Situ - genetics | Hyperplasia - genetics | SOXB1 Transcription Factors - genetics | DNA Mutational Analysis | NF-E2-Related Factor 2 - genetics | APOBEC Deaminases - genetics | Cell Adhesion - genetics | Cell Proliferation - genetics | Esophagus - metabolism | Chromosomes, Human, Pair 11 - genetics | Esophagus - pathology | Cyclin-Dependent Kinase Inhibitor p18 - genetics | Esophageal Neoplasms - genetics | High-Throughput Nucleotide Sequencing | Chromosomes, Human, Pair 3 - genetics | Medical colleges | Medical research | Dysplasia | Squamous cell carcinoma | Analysis | Business parks | Medicine, Experimental | Genetic research | Development and progression | Genomics | Index Medicus | Abridged Index Medicus
Journal Article
PLoS Genetics, ISSN 1553-7390, 2009, Volume 5, Issue 12, p. e1000785
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, p. e28083
Background: Potential functional allele A/T single nucleotide polymorphism (SNP) of Interleukin 8 (IL-8) promoter -251has been implicated in gastric cancer... 
DISEASES | GENETIC POLYMORPHISMS | ANGIOGENESIS | HELICOBACTER-PYLORI INFECTION | ADENOCARCINOMA | CARDIA | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | SQUAMOUS-CELL CARCINOMA | CHINESE POPULATION | STOMACH | Stomach Neoplasms - genetics | Interleukin-8 - genetics | Stomach Neoplasms - ethnology | Genetic Predisposition to Disease - genetics | Gene Frequency | Humans | Risk Factors | Asian Continental Ancestry Group - genetics | Bacterial Proteins - genetics | Helicobacter pylori - genetics | Genotype | Helicobacter Infections - genetics | Antigens, Bacterial - genetics | Promoter Regions, Genetic - genetics | Host-Pathogen Interactions - genetics | China | Helicobacter pylori - physiology | Polymorphism, Single Nucleotide | Odds Ratio | Helicobacter Infections - microbiology | Interleukins | Development and progression | Helicobacter infections | Genetic aspects | Disease susceptibility | Single nucleotide polymorphisms | Health aspects | Stomach cancer | Risk factors | Cancer | Nuclear medicine | Genes | Interleukin | Risk | Single-nucleotide polymorphism | Gene polymorphism | Data bases | Impact analysis | Proteins | Helicobacter pylori | Intestine | Quality | Hepatology | Gastroenterology | Gastric cancer | Interleukin 8 | Sensitivity analysis | Statistical analysis | Cytokines | Health risks | Esophagus | Meta-analysis | Studies | Biomarkers | Protein expression | Polymorphism
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e60327
Background: In addition to Helicobacter pylori infection, host genetic factors contribute to gastric cancer (GC). Recognition of H. pylori is known to involve... 
PROMOTER POLYMORPHISMS | TOLL-LIKE RECEPTOR-4 | EPITHELIAL-CELLS | HELICOBACTER-PYLORI INFECTION | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER RISK | SINGLE-NUCLEOTIDE POLYMORPHISM | BORDETELLA-PERTUSSIS | NF-KAPPA-B | THR399ILE POLYMORPHISMS | CHINESE POPULATION | Stomach Neoplasms - genetics | Toll-Like Receptor 2 - genetics | Genetic Association Studies | Humans | Middle Aged | Asian Continental Ancestry Group - genetics | Toll-Like Receptor 4 - genetics | Male | Case-Control Studies | Polymorphism, Genetic | Stomach Neoplasms - microbiology | Helicobacter pylori | Cell Transformation, Neoplastic - genetics | Lipopolysaccharide Receptors - genetics | Publication Bias | Adult | Female | Aged | Odds Ratio | Helicobacter Infections - microbiology | Toll-like receptors | Genetic aspects | Research | Carcinogenesis | Health aspects | Genetic polymorphisms | Biotechnology | Transcription factors | Control methods | Dyspepsia | Genomics | Genes | Systematic review | Infections | Kinases | CD14 antigen | Population genetics | Stomach cancer | Risk factors | Proteins | Confidence intervals | Signal transduction | Receptors | Carcinogens | Clonal deletion | Gastroenterology | Deletion | Genetic factors | Gastric cancer | Immune system | NF-κB protein | Antigens | Mortality | Mass spectroscopy | Inflammation | TLR4 protein | Minority & ethnic groups | Polymerase chain reaction | Signaling | Genotyping | TLR2 protein | Alleles | Mutation | Mass spectrometry | Prostate cancer | Binding sites
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2011, Volume 108, Issue 36, pp. 14944 - 14949
The bacterial pathogen Helicobacter pylori chronically infects the human gastric mucosa and is the leading risk factor for the development of gastric cancer.... 
Helicobacter pylori | Epithelial cells | DNA damage | DNA | Cell nucleus | Cell lines | Bacteria | Cultured cells | Infections | Mice | Chromosome breaks | DNA damage signaling | Gastric tumorigenesis | Genomic instability | CYTOLETHAL DISTENDING TOXIN | MULTIDISCIPLINARY SCIENCES | RISK | MISMATCH REPAIR | COLONIZATION | GENETIC INSTABILITY | EPITHELIAL-CELLS | chromosome breaks | GASTRIC-CANCER | INFLAMMATION | INFECTION | MICE | genomic instability | gastric tumorigenesis | Phosphorylation | Epithelial Cells - metabolism | Humans | Stomach Neoplasms - metabolism | Helicobacter Infections - complications | Stomach Neoplasms - pathology | Intracellular Signaling Peptides and Proteins - metabolism | Antigens, Bacterial - genetics | DNA Breaks, Double-Stranded | Helicobacter Infections - pathology | DNA-Binding Proteins - metabolism | Tumor Suppressor Proteins - genetics | Trans-Activators - genetics | Cell Cycle Proteins - genetics | Helicobacter Infections - metabolism | Nuclear Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Stomach Neoplasms - genetics | Tumor Suppressor Proteins - metabolism | Chromosomal Proteins, Non-Histone - metabolism | Bacterial Proteins - genetics | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Genomic Islands | Epithelial Cells - pathology | Nuclear Proteins - metabolism | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Chromosomal Proteins, Non-Histone - genetics | Bacterial Adhesion | Stomach Neoplasms - microbiology | Animals | Histones - genetics | Helicobacter pylori - metabolism | Epithelial Cells - microbiology | Chromosome Aberrations | Cell Line, Tumor | Bacterial Proteins - metabolism | Trans-Activators - metabolism | Histones - metabolism | Tumor Suppressor p53-Binding Protein 1 | Antigens, Bacterial - metabolism | Carcinogens | Cocarcinogens | Physiological aspects | Genetic aspects | Research | Health aspects | Cells | Biological Sciences
Journal Article
Cytokine, ISSN 1043-4666, 2005, Volume 29, Issue 4, pp. 141 - 152
The aim of this study was to evaluate whether there was any correlation between -associated diseases and (1) virulence genes or (2) , , , , genetic... 
Interleukin-1 alpha | Tumor necrosis factor alpha | Helicobacter pylori | Interleukin-10 | Gastric cancer | gastric cancer | BIOCHEMISTRY & MOLECULAR BIOLOGY | GAMMA-INTERFERON | INTERLEUKIN-1 POLYMORPHISMS | MUCOSAL INTERLEUKIN-1-BETA | IMMUNOLOGY | interleukin-10 | CELL BIOLOGY | tumor necrosis factor alpha | interleukin-1 alpha | INCREASED RISK | ATROPHIC GASTRITIS | GASTRIC-ACID SECRETION | TNF-ALPHA | NF-KAPPA-B | PEPTIC-ULCER | ALPHA PROMOTER POLYMORPHISM | Interleukin-1 - genetics | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Gastritis - genetics | Helicobacter pylori - genetics | Helicobacter pylori - pathogenicity | Metaplasia - etiology | Helicobacter Infections - complications | Male | Stomach Ulcer - etiology | Duodenal Ulcer - genetics | Gastritis - etiology | Virulence - genetics | Aged, 80 and over | Adult | Female | Stomach Ulcer - genetics | Helicobacter Infections - diagnosis | Metaplasia - genetics | Intestinal Diseases - etiology | Stomach Neoplasms - etiology | Cytokines - genetics | Stomach Neoplasms - genetics | Gene Frequency | Intestinal Diseases - genetics | Genotype | Duodenal Ulcer - etiology | Helicobacter Infections - genetics | Interleukin-10 - genetics | Adolescent | Italy | Polymorphism, Genetic - genetics | Aged | Anti-inflammatory drugs | Cytokines | Nurses | Genetic research | Genetic aspects | Bacterial genetics | Health aspects | Musicians
Journal Article