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Cellular Microbiology, ISSN 1462-5814, 07/2017, Volume 19, Issue 7, pp. e12728 - n/a
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e70783
The mouse pathobiont Helicobacter hepaticus can induce typhlocolitis in interleukin-10-deficient mice, and H. hepaticus infection of immunodeficient mice is... 
ALTERED SCHAEDLER FLORA | INFLAMMATORY-BOWEL-DISEASE | SWISS WEBSTER MICE | BILOPHILA-WADSWORTHIA | EPITHELIAL-CELLS | CYTOLETHAL DISTENDING TOXIN | IMMUNE-RESPONSES | MULTIDISCIPLINARY SCIENCES | GUT MICROBIOTA | BACTERIAL-INFECTION | MULTIPLE SEQUENCE ALIGNMENT | DNA, Complementary - genetics | Colitis - pathology | Colon - immunology | RNA, Ribosomal, 16S - classification | Cecum - microbiology | Helicobacter Infections - pathology | DNA, Complementary - classification | Disease Susceptibility - immunology | Helicobacter hepaticus - pathogenicity | Colitis - immunology | Interleukin-10 - deficiency | Microbiota - immunology | Colon - pathology | Mice, Inbred C57BL | Helicobacter hepaticus - physiology | Helicobacter Infections - immunology | Disease Susceptibility - microbiology | Sequence Analysis, DNA | Mice, Knockout | Cecum - pathology | Animals | Colitis - microbiology | RNA, Ribosomal, 16S - genetics | Colon - microbiology | Mice | Interleukin-10 - immunology | Cecum - immunology | Helicobacter Infections - microbiology | Infection | Interleukins | Microbiota (Symbiotic organisms) | Gastrointestinal diseases | Analysis | Colitis | Health aspects | Animal models | Fourier transforms | Inflammatory bowel diseases | rRNA 16S | Pathogenesis | Interleukin | Opportunist infection | Infections | Epidemiology | Institutions | Medical schools | Hepatitis | Microbiota | Intestine | Rodents | Cell cycle | Interleukin 1 | Bacteria | Colon | Intestinal microflora | Bioinformatics | Pathogens | Bacterial infections | Cecum | Immunodeficiency | Inflammation | Inflammatory bowel disease | Pathology | Hospitals | Acids | Interleukin 10 | Laboratory animals
Journal Article
Journal Article
Nature, ISSN 0028-0836, 02/2018, Volume 554, Issue 7692, pp. 373 - 377
Both microbial and host genetic factors contribute to the pathogenesis of autoimmune diseases(1-4). There is accumulating evidence that microbial species that... 
INTESTINAL INFLAMMATION | RESPONSES | IMMUNITY | MULTIDISCIPLINARY SCIENCES | IN-VIVO | DISEASE | HUMAN MICROBIOTA | MICE | DIFFERENTIATION | EXPRESSION | ANTIGEN | Bioengineering | T-Lymphocytes, Regulatory - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - antagonists & inhibitors | Homeostasis | Colitis - pathology | Male | Intestines - immunology | T-Lymphocytes, Regulatory - immunology | Forkhead Transcription Factors - metabolism | Helicobacter hepaticus - pathogenicity | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | T-Lymphocytes, Regulatory - cytology | Female | Th17 Cells - cytology | Colitis - immunology | Helicobacter hepaticus - genetics | Interleukin-10 - deficiency | Helicobacter hepaticus - immunology | Immune Tolerance | Host-Pathogen Interactions | Animals | Colitis - microbiology | Intestines - microbiology | Proto-Oncogene Proteins c-maf - deficiency | Th17 Cells - immunology | Interleukin-10 - biosynthesis | Proto-Oncogene Proteins c-maf - metabolism | Mice | Interleukin-10 - immunology | Host-bacteria relationships | Inflammatory bowel diseases | Helicobacter infections | Physiological research | Research | T cells | Immunological tolerance | Pathogenesis | Helper cells | Lymphocytes T | Inactivation | Small intestine | Accumulation | Microbiota | Microorganisms | Immunology | Lymphocytes | Rodents | Bacteria | Foxp3 protein | Genetic factors | Deactivation | Immunoregulation | T cell receptors | c-Maf protein | Inflammatory bowel disease | Large intestine | Interleukin 10 | Colitis | Autoimmune diseases | Differentiation | Colonization
Journal Article
Nature, ISSN 0028-0836, 05/2008, Volume 453, Issue 7195, pp. 620 - 625
Humans are colonized by multitudes of commensal organisms representing members of five of the six kingdoms of life; however, our gastrointestinal tract... 
HOST IMMUNE-SYSTEM | SUPPRESS | BACTERIA | CROHNS-DISEASE | GUT MICROBIOME | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | REGULATORY T-CELLS | HELICOBACTER-HEPATICUS | INTERLEUKIN-10-DEFICIENT MICE | BOWEL-DISEASE | Inflammation - chemically induced | Intestines - drug effects | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Inflammatory Bowel Diseases - immunology | Symbiosis - physiology | Intestines - immunology | CD4-Positive T-Lymphocytes - immunology | Polysaccharides, Bacterial - genetics | Colitis - chemically induced | Helicobacter hepaticus - pathogenicity | Inflammatory Bowel Diseases - pathology | Interleukin-10 - metabolism | Colitis - immunology | Disease Models, Animal | Polysaccharides, Bacterial - pharmacology | Bacteroides fragilis - immunology | Inflammation - microbiology | Inflammatory Bowel Diseases - prevention & control | Intestines - pathology | Bacteroides fragilis - physiology | CD4-Positive T-Lymphocytes - metabolism | Polysaccharides, Bacterial - metabolism | Polysaccharides, Bacterial - immunology | Interleukin-17 - metabolism | Animals | Colitis - microbiology | Intestines - microbiology | Interleukin-10 - genetics | Germ-Free Life | Bacteroides fragilis - genetics | Inflammatory Bowel Diseases - microbiology | Interleukin-10 - biosynthesis | Inflammation - prevention & control | Mice | Colitis - prevention & control | Tumor Necrosis Factor-alpha - biosynthesis | CD4-Positive T-Lymphocytes - drug effects | Prevention | Physiological aspects | Development and progression | Inflammatory bowel diseases | Pathogenic microorganisms | Research | Inflammatory bowel disease | Molecules | Cytokines | Clinical trials | Bacteria | Small intestine | Cells | Crohns disease | Index Medicus
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