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PLoS ONE, ISSN 1932-6203, 05/2009, Volume 4, Issue 5, pp. e5549 - e5549
Journal Article
The American Journal of Surgical Pathology, ISSN 0147-5185, 05/2016, Volume 40, Issue 5, pp. 645 - 655
Journal Article
Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 4, pp. 376 - 379
Journal Article
Journal Article
Journal of Medical Genetics, ISSN 0022-2593, 03/2010, Volume 47, Issue 3, pp. 176 - 181
Background Radiotherapy-induced DNA double-strand breaks (DSBs) are critical cytotoxic lesions. Inherited defects in DNA DSB repair pathways lead to... 
CATALYTIC SUBUNIT | REPAIR | PHOSPHORYLATION | GENETICS & HEREDITY | SENSITIVITY | DOUBLE-STRAND BREAKS | HISTONE H2AX | EXCISION | IONIZING-RADIATION | FIBROBLASTS | LINES | Skin Neoplasms - radiotherapy | Xeroderma Pigmentosum - genetics | Humans | Cell Survival - genetics | Molecular Sequence Data | Hemangiosarcoma - genetics | Xeroderma Pigmentosum - pathology | Radiation Injuries - genetics | DNA-Activated Protein Kinase - genetics | Nuclear Proteins - genetics | Isoenzymes - physiology | Skin Neoplasms - pathology | Amino Acid Sequence | Hemangiosarcoma - pathology | Isoenzymes - genetics | Cells, Cultured | Radiation Tolerance - genetics | Alternative Splicing - physiology | DNA-Activated Protein Kinase - physiology | Cell Survival - radiation effects | Protein Isoforms - physiology | Head and Neck Neoplasms - pathology | Sequence Homology, Amino Acid | Head and Neck Neoplasms - radiotherapy | Scalp | Skin Neoplasms - genetics | Hemangiosarcoma - radiotherapy | Head and Neck Neoplasms - genetics | Nuclear Proteins - physiology | Protein Isoforms - genetics | Complications and side effects | Genetic variation | Patient outcomes | Analysis | Xeroderma pigmentosum | Genetic aspects | Research | Radiotherapy | Protein kinases | DNA repair | Cytomegalovirus | Amino acids | Cytotoxicity | Kinases | Gene expression | Patients | Defects | Proteins | Plasmids | Fibroblasts | Mutation | Deoxyribonucleic acid--DNA | Cancer
Journal Article
Human Pathology, ISSN 0046-8177, 2015, Volume 46, Issue 9, pp. 1360 - 1366
Summary Alternative lengthening of telomeres (ALT) is a mechanism using homologous recombination to maintain telomere length and sustain limitless... 
Pathology | TERT | Angiosarcoma | Alternative lengthening of telomeres | Telomere | ATRX | Immunohistochemistry | Nuclear Proteins - analysis | Prognosis | Humans | Middle Aged | Male | Hemangiosarcoma - genetics | X-linked Nuclear Protein | Liver Neoplasms - mortality | Hemangioendothelioma, Epithelioid - enzymology | Skin Neoplasms - enzymology | Telomerase - genetics | DNA Mutational Analysis | Hemangiosarcoma - enzymology | Adult | Female | Liver Neoplasms - pathology | Liver Neoplasms - enzymology | Telomere - genetics | Skin Neoplasms - pathology | Hemangiosarcoma - pathology | Promoter Regions, Genetic | Liver Neoplasms - genetics | Biomarkers, Tumor - analysis | Down-Regulation | In Situ Hybridization, Fluorescence | Hemangiosarcoma - mortality | Telomere Homeostasis | Hemangioendothelioma, Epithelioid - pathology | Sarcoma, Kaposi - pathology | Sarcoma, Kaposi - enzymology | Hemangioendothelioma, Epithelioid - genetics | Skin Neoplasms - genetics | DNA Helicases - analysis | Aged | Biomarkers, Tumor - genetics | Mutation | Sarcoma, Kaposi - genetics | Cellular proteins | Sarcoma | Analysis | Genetic research | Genetic aspects | Chromosomes | Telomerase | Pathogenesis | Radiation therapy | Metastasis | Kinases | Cancer therapies | Studies | Genotype & phenotype | Chemotherapy | Gene amplification | Surgery | Gangrene | Tumors | Cancer
Journal Article
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 05/2017, Volume 16, Issue 5, pp. 956 - 965
Sarcomas differ from carcinomas in their mesenchymal origin. Therapeutic advancements have come slowly, so alternative drugs and models are urgently needed.... 
CANINE HEMANGIOSARCOMA | SYNOVIAL SARCOMA | RECOMBINANT IMMUNOTOXIN | SOFT-TISSUE SARCOMAS | ONCOLOGY | UROKINASE PLASMINOGEN-ACTIVATOR | EPIDERMAL-GROWTH-FACTOR | SPLENIC HEMANGIOSARCOMA | PHASE-I | PSEUDOMONAS EXOTOXIN | LIGAND-DIRECTED TOXIN | Virulence Factors - genetics | Humans | ErbB Receptors - genetics | Hemangiosarcoma - genetics | Molecular Targeted Therapy | ADP Ribose Transferases - administration & dosage | Exotoxins - administration & dosage | Epidermal Growth Factor - chemistry | Bacterial Toxins - genetics | Urokinase-Type Plasminogen Activator - chemistry | ADP Ribose Transferases - chemistry | Disease Models, Animal | Doxorubicin - administration & dosage | Hemangiosarcoma - pathology | ErbB Receptors - antagonists & inhibitors | Exotoxins - chemistry | Epidermal Growth Factor - genetics | Hemangiosarcoma - drug therapy | Virulence Factors - chemistry | Urokinase-Type Plasminogen Activator - genetics | Bacterial Toxins - chemistry | Receptors, Urokinase Plasminogen Activator - antagonists & inhibitors | ADP Ribose Transferases - genetics | Animals | Dogs | Exotoxins - genetics | Cell Line, Tumor | Bacterial Toxins - administration & dosage | Mice | Receptors, Urokinase Plasminogen Activator - genetics | Neoplasm Staging | Virulence Factors - administration & dosage | Spleen | Drugs | Animal models | Carcinoma | Sarcoma | Epidermal growth factor receptors | Mesenchyme | Toxicity | Data acquisition | Minimal residual disease | Doxorubicin | Biological activity | U-Plasminogen activator | Urokinase | Epidermal growth factor | Life span | Data collection | Biocompatibility | In vivo methods and tests | Drug dosages | Cancer | targeted toxin | canine | sarcoma | epidermal growth factor receptor | adaptive clinical trial | urokinase plasminogen activator receptor | Clinical Medicine | Medical and Health Sciences | Cancer and Oncology | Klinisk medicin | Medicin och hälsovetenskap | Cancer och onkologi
Journal Article
PLoS Genetics, ISSN 1553-7390, 2015, Volume 11, Issue 2, pp. 1 - 24
Journal Article
Journal Article