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1. Full Text Recurrent mutations in multiple components of the cohesin complex in myeloid neoplasms
by Kon, Ayana and Shih, Lee-Yung and Minamino, Masashi and Sanada, Masashi and Shiraishi, Yuichi and Nagata, Yasunobu and Yoshida, Kenichi and Okuno, Yusuke and Bando, Masashige and Nakato, Ryuichiro and Ishikawa, Shumpei and Sato-Otsubo, Aiko and Nagae, Genta and Nishimoto, Aiko and Haferlach, Claudia and Nowak, Daniel and Sato, Yusuke and Alpermann, Tamara and Nagasaki, Masao and Shimamura, Teppei and Tanaka, Hiroko and Chiba, Kenichi and Yamamoto, Ryo and Yamaguchi, Tomoyuki and Otsu, Makoto and Obara, Naoshi and Sakata-Yanagimoto, Mamiko and Nakamaki, Tsuyoshi and Ishiyama, Ken and Nolte, Florian and Hofmann, Wolf-Karsten and Miyawaki, Shuichi and Chiba, Shigeru and Mori, Hiraku and Nakauchi, Hiromitsu and Koeffler, H Phillip and Aburatani, Hiroyuki and Haferlach, Torsten and Shirahige, Katsuhiko and Miyano, Satoru and Ogawa, Seishi
Nature Genetics, ISSN 1061-4036, 10/2013, Volume 45, Issue 10, pp. 1232 - 1237
Cohesin is a multimeric protein complex that is involved in the cohesion of sister chromatids, post-replicative DNA repair and transcriptional regulation. Here... 
GENE-EXPRESSION | RESISTANT | EVOLUTION | LEUKEMIA | DNA | GENETICS & HEREDITY | Chromosomal Proteins, Non-Histone - metabolism | Humans | Cell Cycle Proteins - metabolism | Cell Cycle Proteins - genetics | Cell Line, Tumor | Male | Hematologic Neoplasms - genetics | Hematologic Neoplasms - pathology | Mutation | Hematologic Neoplasms - metabolism | Chromosomal Proteins, Non-Histone - genetics | Gene mutations | Myelocytic leukemia | Genetic aspects | Nonlymphoid leukemia | Research | Health aspects | Risk factors | Studies | Cell growth | Expected values | DNA methylation | Gene expression | Experiments | Index Medicus
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2. Full Text Thrombopoietin receptor activation by myeloproliferative neoplasm associated calreticulin mutants
by Chachoua, Ilyas and Pecquet, Christian and El-Khoury, Mira and Nivarthi, Harini and Albu, Roxana-Irina and Marty, Caroline and Gryshkova, Vitalina and Defour, Jean-Philippe and Vertenoeil, Gäelle and Ngo, Anna and Koay, Ann and Raslova, Hana and Courtoy, Pierre J and Choong, Meng Ling and Plo, Isabelle and Vainchenker, William and Kralovics, Robert and Constantinescu, Stefan N
Blood, ISSN 0006-4971, 03/2016, Volume 127, Issue 10, pp. 1325 - 1335
Mutations in the calreticulin gene (CALR) represented by deletions and insertions in exon 9 inducing a -1/+2 frameshift are associated with a significant... 
MYELOFIBROSIS | PATHOGENESIS | ERYTHROPOIETIN RECEPTOR | TRANSMEMBRANE | SURFACE EXPRESSION | DIMERIZATION | MUTATIONS | ESSENTIAL THROMBOCYTHEMIA | JAK2 | TYPE-1 | HEMATOLOGY | STAT Transcription Factors - metabolism | Hematologic Neoplasms - pathology | Myeloproliferative Disorders - pathology | Phosphatidylinositol 3-Kinases - metabolism | LIM Domain Proteins - metabolism | Neoplasm Proteins - metabolism | Calreticulin - genetics | Myeloproliferative Disorders - genetics | Phosphorylation - genetics | Janus Kinase 2 - metabolism | Myeloproliferative Disorders - metabolism | Muscle Proteins - metabolism | Calreticulin - metabolism | Neoplasm Proteins - genetics | Hematologic Neoplasms - metabolism | Janus Kinase 2 - genetics | Glycosylation | Signal Transduction - genetics | Receptors, Thrombopoietin - metabolism | Protein Transport - genetics | Muscle Proteins - genetics | Phosphatidylinositol 3-Kinases - genetics | Receptors, Thrombopoietin - genetics | STAT Transcription Factors - genetics | Animals | Cell Line, Tumor | LIM Domain Proteins - genetics | Hematologic Neoplasms - genetics | Mice | Mutation | Index Medicus | Abridged Index Medicus
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3. Full Text Deletion of Tet2 in mice leads to dysregulated hematopoietic stem cells and subsequent development of myeloid malignancies
by Li, Zhe and Cai, Xiaoqiang and Cai, Chen-Leng and Wang, Jiapeng and Zhang, Wenyong and Petersen, Bruce E and Yang, Feng-Chun and Xu, Mingjiang
Blood, ISSN 0006-4971, 10/2011, Volume 118, Issue 17, pp. 4509 - 4518
TET2 is mutated/deleted with high frequencies in multiple forms of myeloid malignancies including MDS, CMML, MPN, and AML. However, little is known regarding... 
MUTANT TET2 | 5-METHYLCYTOSINE | MOUSE | MYELOPROLIFERATIVE NEOPLASMS | 5-HYDROXYMETHYLCYTOSINE | MUTATIONS | SPECIFICATION | LEUKEMIA | MLL | HEMATOLOGY | T(10/11)(Q22,Q23) | Neoplasm Transplantation | Gene Expression Regulation, Neoplastic | Hematopoietic Stem Cells - pathology | Male | Green Fluorescent Proteins - genetics | Hematologic Neoplasms - pathology | Bone Marrow Neoplasms - genetics | DNA-Binding Proteins - metabolism | Gene Deletion | Genes, Tumor Suppressor - physiology | Female | Hematologic Neoplasms - metabolism | Disease Models, Animal | Proto-Oncogene Proteins - metabolism | DNA-Binding Proteins - physiology | Mice, Inbred C57BL | Proto-Oncogene Proteins - genetics | Hematopoietic Stem Cells - metabolism | DNA-Binding Proteins - genetics | Mice, Knockout | Animals | Bone Marrow Neoplasms - pathology | Proto-Oncogene Proteins - physiology | Hematologic Neoplasms - genetics | Mice | Index Medicus | Abridged Index Medicus | Plenary Paper | Myeloid Neoplasia
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4. Full Text Activation of the thrombopoietin receptor by mutant calreticulin in CALR-mutant myeloproliferative neoplasms
by Araki, Marito and Yang, Yinjie and Masubuchi, Nami and Hironaka, Yumi and Takei, Hiraku and Morishita, Soji and Mizukami, Yoshihisa and Kan, Shin and Shirane, Shuichi and Edahiro, Yoko and Sunami, Yoshitaka and Ohsaka, Akimichi and Komatsu, Norio
Blood, ISSN 0006-4971, 03/2016, Volume 127, Issue 10, pp. 1307 - 1316
Recurrent somatic mutations of calreticulin (CALR) have been identified in patients harboring myeloproliferative neoplasms; however, their role in... 
PLURIPOTENT STEM-CELLS | ERYTHROPOIETIN | LINE | PHENOTYPE | DIMERIZATION | ENDOPLASMIC-RETICULUM | MUTATIONS | ESSENTIAL THROMBOCYTHEMIA | JAK2 | ESTABLISHMENT | HEMATOLOGY | Induced Pluripotent Stem Cells - pathology | Protein Structure, Tertiary | Phosphorylation | Thrombopoietin - metabolism | Hematologic Neoplasms - mortality | Humans | Myeloproliferative Disorders - pathology | Neoplasm Proteins - metabolism | Calreticulin - genetics | Myeloproliferative Disorders - genetics | Receptors, Thrombopoietin - metabolism | Thrombopoiesis - genetics | Receptors, Thrombopoietin - genetics | Janus Kinase 2 - metabolism | Myeloproliferative Disorders - metabolism | HEK293 Cells | Calreticulin - metabolism | Cell Line, Tumor | Hematologic Neoplasms - genetics | Neoplasm Proteins - genetics | Hematologic Neoplasms - metabolism | Induced Pluripotent Stem Cells - metabolism | Index Medicus | Abridged Index Medicus
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5. Full Text Novel markers of normal and neoplastic human plasmacytoid dendritic cells
by Marafioti, Teresa and Paterson, Jennifer C and Ballabio, Erica and Reichard, Kaaren K and Tedoldi, Sara and Hollowood, Kevin and Dictor, Michael and Hansmann, Martin-Leo and Pileri, Stefano A and Dyer, Martin J and Sozzani, Silvano and Dikic, Ivan and Shaw, Andrey S and Petrella, Tony and Stein, Harald and Isaacson, Peter G and Facchetti, Fabio and Mason, David Y and Oncology and Pathology, Kamprad Lab and Lund University and Lunds universitet and Onkologi och Patologi, Kampradlab
Blood, ISSN 0006-4971, 04/2008, Volume 111, Issue 7, pp. 3778 - 3792
Plasmacytoid dendritic cells (pDCs) are involved in innate immunity (eg, by secreting interferons) and also give rise to CD4(+)CD56(+) hematodermic neoplasms.... 
LINEAGE-NEGATIVE MALIGNANCIES | TOLL-LIKE RECEPTORS | INTERFERON-PRODUCING CELLS | INTRACELLULAR SIGNALING MOLECULES | MYELOMONOCYTIC LEUKEMIA | GTPASE-ACTIVATING PROTEIN | HEMATOLOGY | IFN-ALPHA PRODUCTION | T-CELLS | VIRAL-INFECTION | I INTERFERON | Neoplasms, Plasma Cell - diagnosis | Humans | Leukemia, Myeloid, Acute - metabolism | Middle Aged | Interferon Regulatory Factors - metabolism | Dendritic Cells - pathology | Male | Hematologic Neoplasms - pathology | Plasma Cells - pathology | Neoplasms, Plasma Cell - pathology | Skin Neoplasms - diagnosis | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Cytoskeletal Proteins - metabolism | Female | Plasma Cells - metabolism | Dendritic Cells - metabolism | Hematologic Neoplasms - metabolism | Proto-Oncogene Proteins - metabolism | Skin Neoplasms - pathology | Hematologic Neoplasms - diagnosis | Diagnosis, Differential | Toll-Like Receptor 7 - metabolism | Leukemia, Myeloid, Acute - pathology | Neoplasms, Plasma Cell - metabolism | Nuclear Proteins - metabolism | Skin Neoplasms - metabolism | Carrier Proteins - metabolism | Leukemia, Myeloid, Acute - diagnosis | Biopsy | Trans-Activators - metabolism | Aged | Adaptor Proteins, Signal Transducing - metabolism | Toll-Like Receptor 9 - metabolism | Index Medicus | Abridged Index Medicus | Clinical Medicine | Hematologi | Medical and Health Sciences | Hematology | Klinisk medicin | Medicin och hälsovetenskap
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6. Full Text Transgenic Expression of Helicobacter pylori CagA Induces Gastrointestinal and Hematopoietic Neoplasms in Mouse
by Naomi Ohnishi and Hitomi Yuasa and Shinya Tanaka and Hirofumi Sawa and Motohiro Miura and Atsushi Matsui and Hideaki Higashi and Manabu Musashi and Kazuya Iwabuchi and Misao Suzuki and Gen Yamada and Takeshi Azuma and Masanori Hatakeyama
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2008, Volume 105, Issue 3, pp. 1003 - 1008
Infection with cagA-positive Helicobacter pylori is associated with gastric adenocarcinoma and gastric mucosa-associated lymphoid tissue (MALT) lymphoma of B... 
Adenocarcinoma | Stomach | Phosphorylation | Helicobacter pylori | Epithelial cells | Transgenic animals | Mice | Infections | Bone marrow cells | Tumors | Transgenic mouse | Bacterial oncoprotein | SHP-2 TYROSINE PHOSPHATASE | PROTEIN | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | transgenic mouse | CELL DEVELOPMENT | CANCER | bacterial oncoprotein | BIOLOGICAL-ACTIVITY | ATROPHIC GASTRITIS | PTPN11 MUTATIONS | INFECTION | MONGOLIAN GERBILS | Gastrointestinal Neoplasms - genetics | Bacterial Proteins - genetics | Gene Expression Regulation, Neoplastic | Helicobacter pylori - genetics | Helicobacter pylori - pathogenicity | Mice, Transgenic | Hematologic Neoplasms - pathology | Antigens, Bacterial - genetics | Cell Transformation, Neoplastic - metabolism | Phosphotyrosine - metabolism | Gastrointestinal Neoplasms - pathology | Animals | Helicobacter pylori - metabolism | Cell Transformation, Neoplastic - genetics | Bacterial Proteins - metabolism | Hematologic Neoplasms - genetics | Cell Transformation, Neoplastic - pathology | Antigens, Bacterial - metabolism | Hematologic Neoplasms - metabolism | Gastrointestinal Neoplasms - metabolism | Gastrointestinal tumors | Causes of | Influence | Genetic aspects | Hematopoietic system | Gene expression | Leucocytes | Bacterial proteins | Rodents | Bacteria | Models | Mutation | Kinases | Index Medicus | Biological Sciences
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7. Full Text Blastic Plasmacytoid Dendritic Cell Neoplasms: Clinico-immunohistochemical Correlations in a Series of 91 Patients
by Julia, Fanny and Dalle, Stephane and Duru, Gerard and Balme, Brigitte and Vergier, Béatrice and Ortonne, Nicolas and Vignon-Pennamen, Marie D and Costes-Martineau, Valérie and Lamant, Laurence and Dalac, Sophie and Delattre, Claire and Déchelotte, Pierre and Courville, Philippe and Carlotti, Agnès and De Muret, Anne and Fraitag, Sylvie and Levy, Annie and Mitchell, Andrew and Petrella, Tony
The American Journal of Surgical Pathology, ISSN 0147-5185, 05/2014, Volume 38, Issue 5, pp. 673 - 680
Blastic plasmacytoid dendritic cell neoplasm is a rare clinicopathologic entity, characterized by strong skin tropism and a poor prognosis. The diagnosis is... 
Ki-67 | CD303 | immunohistochemistry | prognostic factor | BPDCN | SURGERY | CD4+CD56+HEMATODERMIC NEOPLASMS | PATHOLOGY | FEATURES | LYMPHOMAS | ENTITY | BDCA-2 | LEUKEMIA | EXPRESSION | Immunohistochemistry | Skin Neoplasms - pathology | Biomarkers, Tumor - analysis | Hematologic Neoplasms - mortality | Humans | Middle Aged | Kaplan-Meier Estimate | Dendritic Cells - pathology | Male | Hematologic Neoplasms - pathology | Skin Neoplasms - metabolism | Young Adult | Skin Neoplasms - mortality | Adolescent | Aged, 80 and over | Adult | Female | Aged | Child | Dendritic Cells - metabolism | Hematologic Neoplasms - metabolism | Index Medicus
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8. Full Text Two routes to leukemic transformation after a JAK2 mutation-positive myeloproliferative neoplasm
by Beer, Philip A and Delhommeau, François and LeCouédic, Jean-Pierre and Dawson, Mark A and Chen, Edwin and Bareford, David and Kušec, Rajko and McMullin, Mary Frances and Harrison, Claire N and Vannucchi, Alessandro M and Vainchenker, William and Green, Anthony R
Blood, ISSN 0006-4971, 04/2010, Volume 115, Issue 14, pp. 2891 - 2900
Acute myeloid leukemia (AML) may follow a JAK2-positive myeloproliferative neoplasm (MPN), although the mechanisms of disease evolution, often involving loss... 
V617F MUTATION | MYELOFIBROSIS | POLYCYTHEMIA-VERA | 17P DELETION | PROGNOSTIC-FACTORS | TYROSINE KINASE | CLONAL HEMATOPOIESIS | DISORDERS | ESSENTIAL THROMBOCYTHEMIA | HEMATOLOGY | CHRONIC MYELOID-LEUKEMIA | Proto-Oncogene Proteins c-cbl - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Middle Aged | Male | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Janus Kinase 2 - metabolism | Female | Neoplasms, Second Primary - genetics | Hematologic Neoplasms - metabolism | Primary Myelofibrosis - metabolism | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins c-cbl - genetics | Janus Kinase 2 - genetics | Tumor Suppressor Protein p53 - metabolism | Proto-Oncogene Proteins - genetics | DNA-Binding Proteins - genetics | Neoplasms, Second Primary - metabolism | Primary Myelofibrosis - genetics | Aged | Blast Crisis - genetics | Hematologic Neoplasms - genetics | Blast Crisis - metabolism | Mutation | Leukemia, Myeloid, Acute - genetics | Index Medicus | Abridged Index Medicus
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9. Full Text Prognostically relevant breakdown of 123 patients with systemic mastocytosis associated with other myeloid malignancies
by Pardanani, Animesh and Lim, Ken-Hong and Lasho, Terra L and Finke, Christy and McClure, Rebecca F and Li, Chin-Yang and Tefferi, Ayalew
Blood, ISSN 0006-4971, 10/2009, Volume 114, Issue 18, pp. 3769 - 3772
The prognostic heterogeneity of the World Health Organization category of "systemic mastocytosis with associated clonal hematologic nonmast cell lineage... 
C-KIT | DISORDERS | HEMATOLOGY | CELL | MUTATION | Eosinophilia - metabolism | Oncogene Proteins, Fusion - metabolism | Prognosis | mRNA Cleavage and Polyadenylation Factors - metabolism | Humans | Middle Aged | Male | Eosinophilia - epidemiology | Hematologic Neoplasms - epidemiology | Mastocytosis, Systemic - diagnosis | Mastocytosis, Systemic - genetics | Eosinophilia - genetics | Mastocytosis, Systemic - metabolism | Aged, 80 and over | Adult | Female | Hematologic Neoplasms - complications | Mastocytosis, Systemic - epidemiology | Neoplasms, Second Primary - genetics | mRNA Cleavage and Polyadenylation Factors - genetics | Eosinophilia - diagnosis | Hematologic Neoplasms - metabolism | Hematologic Neoplasms - diagnosis | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Neoplasms, Second Primary - diagnosis | Neoplasms, Second Primary - epidemiology | Neoplasms, Second Primary - metabolism | Oncogene Proteins, Fusion - genetics | Receptor, Platelet-Derived Growth Factor alpha - genetics | Aged | Eosinophilia - complications | Hematologic Neoplasms - genetics | Mastocytosis, Systemic - complications | Index Medicus | Abridged Index Medicus
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10. Hematologic malignancies
by Tomblyn, Michael and Winkfield, Karen Marie, 1970 and Dabaja, Bouthaina
2012, Radiation medicine rounds, ISBN 9781936287772, Volume 3, issue 3
Hematologic Neoplasms | Lymphoma, Non-Hodgkin | Hodgkin Disease
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11. Full Text Randomized Phase II, Double-Blind, Placebo-Controlled Study of Exemestane With or Without Entinostat in Postmenopausal Women With Locally Recurrent or Metastatic Estrogen Receptor-Positive Breast Cancer Progressing on Treatment With a Nonsteroidal Aromatase Inhibitor
by Denise A. Yardley and Roohi R. Ismail-Khan and Bohuslav Melichar and Mikhail Lichinitser and Pamela N. Munster and Pamela M. Klein and Scott Cruickshank and Kathy D. Miller and Min J. Lee and Jane B Trepel
Journal of Clinical Oncology, ISSN 0732-183X, 06/2013, Volume 31, Issue 17, pp. 2128 - 2135
Purpose Entinostat is an oral isoform selective histone deacetylase inhibitor that targets resistance to hormonal therapies in estrogen receptor-positive (ER+)... 
TRIAL | DEPSIPEPTIDE | HISTONE DEACETYLASE INHIBITOR | MULTICENTER | MS-275 | ONCOLOGY | ANTICANCER AGENTS | T-CELL LYMPHOMA | REFRACTORY SOLID TUMORS | HEMATOLOGIC MALIGNANCIES | Receptors, Estrogen - metabolism | Humans | Middle Aged | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Breast Neoplasms - metabolism | Neoplasm Recurrence, Local - pathology | Aromatase Inhibitors - adverse effects | Benzamides - administration & dosage | Neoplasm Metastasis | Pyridines - adverse effects | Aged, 80 and over | Biomarkers, Tumor - metabolism | Postmenopause | Adult | Female | Benzamides - adverse effects | Aromatase Inhibitors - administration & dosage | Pyridines - administration & dosage | Double-Blind Method | Breast Neoplasms - drug therapy | Disease-Free Survival | Androstadienes - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Breast Neoplasms - pathology | Aged | Androstadienes - therapeutic use | Androstadienes - adverse effects | Index Medicus | Original Reports | Breast Cancer | Bc7
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12. Full Text SPIB, a novel immunohistochemical marker for human blastic plasmacytoid dendritic cell neoplasms: Characterization of its expression in major hematolymphoid neoplasms
by Montes-Moreno, Santiago and Ramos-Medina, Rocio and Martínez-López, Azahara and Cornejo, Carlos Barrionuevo and Cubillos, Alejandro Parra and Quintana-Truyenque, Shirley and Rodriguez Pinilla, Socorro María and Pajares, Raquel and Sanchez-Verde, Lydia and Martinez-Torrecuadrada, Jorge and Roncador, Giovanna and Piris, Miguel Angel
Blood, ISSN 0006-4971, 01/2013, Volume 121, Issue 4, pp. 643 - 647
SPIB is an Ets transcription factor that is expressed exclusively in mature B cells, T-cell progenitors, and plasmacytoid dendritic cells. In the present... 
LYMPHOMA | DIFFERENTIATION | LEUKEMIA | B-CELL | ETS PROTEIN | HEMATOLOGY | Hematologic Neoplasms - diagnosis | Lymphoma - diagnosis | Humans | Dendritic Cells - pathology | Lymphoid Tissue - metabolism | Lymphoma - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | DNA-Binding Proteins - metabolism | Lymphoma - metabolism | Transcription Factors - metabolism | Lymphoid Tissue - pathology | Biomarkers, Tumor - metabolism | Cell Line, Tumor | Biomarkers, Tumor - genetics | Hematologic Neoplasms - genetics | Dendritic Cells - metabolism | Hematologic Neoplasms - metabolism | Index Medicus | Abridged Index Medicus
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13. Full Text BET protein inhibition shows efficacy against JAK2V617F-driven neoplasms
by Wyspiańska, B.S and Bannister, A.J and Barbieri, I and Nangalia, J and Godfrey, A and Calero-Nieto, F.J and Robson, S and Rioja, I and Li, J and Wiese, M and Cannizzaro, E and Dawson, M.A and Huntly, B and Prinjha, R.K and Green, A.R and Gottgens, B and Kouzarides, T
Leukemia, ISSN 0887-6924, 2014, Volume 28, Issue 1, pp. 88 - 97
Small molecule inhibition of the BET family of proteins, which bind acetylated lysines within histones, has been shown to have a marked therapeutic benefit in... 
myeloproliferative neoplasms | BET proteins | JAK2V617F | LMO2 | RECRUITMENT | POLYCYTHEMIA-VERA | ESSENTIAL THROMBOCYTHEMIA | RUXOLITINIB | ONCOLOGY | MUTATION | TYROSINE KINASE JAK2 | LEUKEMIA | HEMATOLOGY | CELL | HEMATOPOIETIC STEM | Myeloproliferative Disorders - enzymology | Humans | Hematologic Neoplasms - enzymology | Hematologic Neoplasms - pathology | Myeloproliferative Disorders - pathology | Reverse Transcriptase Polymerase Chain Reaction | Oncogene Proteins - antagonists & inhibitors | Chromatin Immunoprecipitation | Janus Kinase 2 - metabolism | Myeloproliferative Disorders - metabolism | Cell Line, Tumor | Real-Time Polymerase Chain Reaction | Hematologic Neoplasms - metabolism | Proteins | Care and treatment | Oncology, Experimental | Research | Identification and classification | Tumors | Cancer | Index Medicus
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