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Journal of Biological Chemistry, ISSN 0021-9258, 02/2014, Volume 289, Issue 6, pp. 3126 - 3137
Background: The chemokine receptor CXCR3-B initiates inhibitory signals. Results: CXCR3-B-mediated signal induced apoptosis and inhibited autophagy of breast... 
Signal Transduction | SPLICE VARIANT | BIOCHEMISTRY & MOLECULAR BIOLOGY | I-TAC | Heme Oxygenase | HEME OXYGENASE-1 GENE | CXC CHEMOKINES | HUMAN BREAST-CANCER | Signaling | MURINE MODEL | EPITHELIAL-CELLS | Nrf2 | Nuclear Transport | TRANSCRIPTION FACTOR | EXPRESSION | Chemokines | Apoptosis | Basic-Leucine Zipper Transcription Factors - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | bcl-X Protein - genetics | Apoptosis - genetics | Breast Neoplasms - metabolism | MAP Kinase Signaling System | Heme Oxygenase-1 - genetics | Fanconi Anemia Complementation Group Proteins - metabolism | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Cell Nucleus - pathology | Mitogen-Activated Protein Kinase 1 - genetics | Apoptosis Regulatory Proteins - genetics | Female | NF-E2-Related Factor 2 - genetics | Membrane Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Active Transport, Cell Nucleus - genetics | Beclin-1 | Receptors, CXCR3 - metabolism | Heme Oxygenase-1 - biosynthesis | Mitogen-Activated Protein Kinase 3 - genetics | Membrane Proteins - genetics | Alternative Splicing - genetics | Fanconi Anemia Complementation Group Proteins - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Basic-Leucine Zipper Transcription Factors - genetics | Apoptosis Regulatory Proteins - metabolism | Breast Neoplasms - genetics | Cell Nucleus - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Breast Neoplasms - pathology | NF-E2-Related Factor 2 - metabolism | Receptors, CXCR3 - genetics | Cell Line, Tumor | bcl-X Protein - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Protein Isoforms - genetics
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 04/2015, Volume 35, Issue 6, pp. 2333 - 2348
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2010, Volume 126, Issue 2, pp. 338 - 346.e10
Background Regulatory T–cell dysfunction is associated with development of the complex genetic conditions atopy and asthma. Therefore, we hypothesized that... 
Allergy and Immunology | T regulatory cells | birth cohort | IgE | atopy | interaction | asthma | MDR | polymorphism | Interaction | Birth cohort | Asthma | Atopy | Polymorphism | SERUM IGE | AIRWAY INFLAMMATION | IMMUNOLOGY | CHILDREN | MULTIFACTOR-DIMENSIONALITY REDUCTION | PROMOTER POLYMORPHISMS | ALLERGY | TRANSFORMING GROWTH-FACTOR-BETA-1 | ALLERGIC DISEASE | ASSOCIATION | IMMUNOGLOBULIN-E | Forkhead Transcription Factors - immunology | Allergens - adverse effects | Receptors, Transforming Growth Factor beta - genetics | Prospective Studies | Receptors, Transforming Growth Factor beta - immunology | Toll-Like Receptor 2 - genetics | Humans | Genetic Loci - genetics | Child, Preschool | Infant | Male | Genetic Loci - immunology | Heme Oxygenase-1 - genetics | Transforming Growth Factor beta1 - immunology | Immunoglobulin E - immunology | Interleukin-2 - immunology | Interleukin-2 Receptor alpha Subunit - genetics | Asthma - immunology | Female | Receptors, Interleukin-6 - immunology | T-Lymphocytes - pathology | Child | Interleukin-2 Receptor alpha Subunit - immunology | Genetic Predisposition to Disease | Interleukin-6 - genetics | Receptors, Interleukin-6 - genetics | Protein-Serine-Threonine Kinases - genetics | Logistic Models | Transforming Growth Factor beta1 - genetics | Forkhead Transcription Factors - genetics | Asthma - genetics | Polymorphism, Genetic | Interleukin-2 - genetics | Interleukin-10 - genetics | Epistasis, Genetic - immunology | Heme Oxygenase-1 - immunology | Interleukin-6 - immunology | Toll-Like Receptor 2 - immunology | Protein-Serine-Threonine Kinases - immunology | T-Lymphocytes - immunology | Models, Genetic | Asthma - pathology | Interleukin-10 - immunology | Allergy | Genetic disorders | Child development | Asthma in children | Immunoglobulin E | Family medicine | Children | Transforming growth factors | T cells | Health aspects | Allergic reaction | Public health | Studies | Food allergies | Genes | Statistical methods | Allergies | Deoxyribonucleic acid--DNA | Milk
Journal Article
Inflammatory Bowel Diseases, ISSN 1078-0998, 05/2016, Volume 22, Issue 8, pp. 1812 - 1834
Background:Clinical observations or animal studies implicate enteric glial cells in motility disorders, irritable bowel syndrome, inflammatory bowel disease,... 
adenosine | human enteric glial cells | purinergic signaling | signaling | mechanosensitivity | bacterial lipopolysaccharide | ATP | Ca 2 | NERVOUS-SYSTEM | OXIDATIVE STRESS | Ca2+ signaling | CROHNS-DISEASE | ULCERATIVE-COLITIS | PROTECT NEURONS | ASTROCYTES | INTESTINAL INFLAMMATION | INFLAMMATORY-BOWEL-DISEASE | MOUSE MODEL | MICE | GASTROENTEROLOGY & HEPATOLOGY | Enteric Nervous System - cytology | Superoxide Dismutase - genetics | Gene Expression - drug effects | Calcium - metabolism | Humans | Gastrointestinal Motility | Heme Oxygenase-1 - genetics | Inflammation - metabolism | Adenosine Triphosphate - metabolism | Caspase 3 - genetics | Jejunum - cytology | S100 Calcium Binding Protein beta Subunit - metabolism | Calcium Channels - genetics | Cytokines - genetics | Mechanotransduction, Cellular - genetics | Cytokines - metabolism | Vesicular Transport Proteins - genetics | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Signal Transduction - genetics | Transcription Factors - genetics | Receptors, Purinergic - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Receptors, Purinergic - genetics | Tryptophan Hydroxylase - genetics | Up-Regulation - drug effects | Carrier Proteins - genetics | Phenotype | Gastrointestinal Diseases - genetics | Gastrointestinal Diseases - metabolism | Nitric Oxide Synthase Type II - genetics | Lipopolysaccharides - pharmacology | Colon, Sigmoid - cytology | Inflammation - genetics | Neuroglia - metabolism | Interferon-gamma - pharmacology | Human Enteric Glial Cells
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2015, Volume 308, Issue 4, pp. L314 - L324
Sickle cell disease (SCD) is an autosomal recessive disorder in the gene encoding the beta -chain of hemoglobin. Deoxygenation causes the mutant hemoglobin S... 
Sickle cell disease | Nitric oxide | Pulmonary hypertension | Cell free hemoglobin | LACTATE-DEHYDROGENASE | nitric oxide | PHYSIOLOGY | pulmonary hypertension | NITRIC-OXIDE BIOAVAILABILITY | EXERCISE CAPACITY | KNOCKOUT MICE | sickle cell disease | cell free hemoglobin | ACUTE CHEST SYNDROME | RESPIRATORY SYSTEM | ARTERIAL-HYPERTENSION | FETAL-HEMOGLOBIN | TRANSGENIC MOUSE MODEL | PLATELET ACTIVATION | FREE HEMOGLOBIN | Humans | Heart Failure - physiopathology | Immunity, Innate - genetics | Oxidative Stress - immunology | Hypertension, Pulmonary - physiopathology | Acute Chest Syndrome - physiopathology | Hypertension, Pulmonary - therapy | Hemolysis - immunology | Renal Insufficiency, Chronic - therapy | Vascular Resistance - genetics | Heart Failure - immunology | Renal Insufficiency, Chronic - genetics | Reactive Oxygen Species - blood | Hemoglobin, Sickle - immunology | Toll-Like Receptor 4 - genetics | Vascular Diseases - genetics | Heart Failure - genetics | Toll-Like Receptor 4 - immunology | Renal Insufficiency, Chronic - physiopathology | Hemoglobin, Sickle - metabolism | Heme - immunology | Toll-Like Receptor 4 - blood | Nitric Oxide - genetics | Mutation | Vascular Diseases - blood | Practice Guidelines as Topic | Vascular Resistance - immunology | Heme - metabolism | Acute Chest Syndrome - therapy | Age Factors | Hypertension, Pulmonary - blood | Heart Failure - blood | Erythrocytes, Abnormal | Heart Failure - therapy | Heme - genetics | Reactive Oxygen Species - immunology | Acute Chest Syndrome - genetics | Acute Chest Syndrome - immunology | Hemolysis - genetics | Nitric Oxide - blood | Nitric Oxide - immunology | Vascular Diseases - immunology | Renal Insufficiency, Chronic - immunology | Vascular Diseases - therapy | Oxidative Stress - genetics | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - immunology | Hemoglobin, Sickle - genetics | Vascular Diseases - physiopathology | Renal Insufficiency, Chronic - blood | Acute Chest Syndrome - blood | Genetic aspects | Sickle cell anemia | Vasculitis | Diagnosis | Risk factors | Physiology in Medicine
Journal Article
The Plant Cell, ISSN 1040-4651, 12/2011, Volume 23, Issue 12, pp. 4476 - 4491
5-Aminolevulinic acid (ALA) is the universal precursor for tetrapyrrole biosynthesis and is synthesized in plants in three enzymatic steps: ligation of... 
Proteins | Enzymes | Yeasts | Chloroplasts | Tetrapyrroles | Thylakoids | Biosynthesis | Plants | Chlorophylls | Seedlings | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHLOROPHYLL BIOSYNTHESIS | MAGNESIUM CHELATASE | ESCHERICHIA-COLI | TETRAPYRROLE BIOSYNTHESIS | HORSERADISH-PEROXIDASE | THALIANA CHLOROPLASTS | PLANT SCIENCES | CELL BIOLOGY | TRANSFER-RNA REDUCTASE | CHLAMYDOMONAS-REINHARDTII | GENE-EXPRESSION | PHOTOSYSTEM-II | Heme - metabolism | Genes, Plant | Agrobacterium tumefaciens - genetics | Molecular Sequence Data | RNA, Plant - metabolism | Chlorophyll - genetics | Arabidopsis Proteins - metabolism | Thylakoid Membrane Proteins - metabolism | Heme - genetics | RNA Interference | Aminolevulinic Acid - metabolism | Escherichia coli - metabolism | Gene Expression Regulation, Plant | Plasmids - genetics | Transcription, Genetic | Recombinant Proteins - metabolism | Amino Acid Sequence | Arabidopsis Proteins - genetics | Chloroplasts - ultrastructure | Plants, Genetically Modified - genetics | Tobacco - metabolism | Recombinant Proteins - genetics | Aldehyde Oxidoreductases - genetics | RNA, Plant - genetics | Thylakoid Membrane Proteins - genetics | Plasmids - metabolism | Chloroplasts - metabolism | Aldehyde Oxidoreductases - metabolism | Arabidopsis - metabolism | Protein Interaction Mapping | Arabidopsis - genetics | Carrier Proteins - genetics | Two-Hybrid System Techniques | Carrier Proteins - metabolism | Plants, Genetically Modified - metabolism | Agrobacterium tumefaciens - metabolism | Escherichia coli - genetics | Tobacco - genetics | Chlorophyll - biosynthesis | Arabidopsis thaliana | Physiological aspects | Amino acids | Research | Binding proteins
Journal Article
Lymphatic Research and Biology, ISSN 1539-6851, 02/2018, Volume 16, Issue 1, pp. 43 - 55
Background: Lymphatic endothelial cells (LECs) derived from lymphatic malformations (LMs) bear activated PIK3CA alleles yet display an inflammatory gene... 
PIK3CA | endothelial cells | lymphatic malformations | MEDICINE, RESEARCH & EXPERIMENTAL | PHYSIOLOGY | PERCUTANEOUS SCLEROTHERAPY | PHOSPHORYLATION | PROTEIN-KINASE | CANCER | FAT STORAGE | PI3K | INSULIN-RESISTANCE | AMPK | MUTATIONS | LIPID DROPLETS | Interleukin-8 - genetics | Heme Oxygenase-1 - metabolism | Class I Phosphatidylinositol 3-Kinases - genetics | Vesicular Transport Proteins - metabolism | Humans | Aminoimidazole Carboxamide - pharmacology | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Lipid Metabolism - genetics | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Repressor Proteins - metabolism | Histone Deacetylases - genetics | Interleukin-6 - genetics | Signal Transduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Lymphoid Tissue - metabolism | Repressor Proteins - genetics | Lymphatic Abnormalities - pathology | Lymphoid Tissue - pathology | Aminoimidazole Carboxamide - analogs & derivatives | Vascular Endothelial Growth Factor C - metabolism | Angiopoietin-like 4 Protein - metabolism | Mutation | Endothelial Cells - pathology | Autophagy-Related Proteins - metabolism | AMP-Activated Protein Kinases - genetics | Angiopoietin-like 4 Protein - genetics | Lymphatic Abnormalities - genetics | AMP-Activated Protein Kinases - metabolism | Class I Phosphatidylinositol 3-Kinases - metabolism | Ribonucleotides - pharmacology | Heme Oxygenase-1 - genetics | Membrane Proteins - metabolism | Autophagy-Related Proteins - genetics | Cell Line | Vesicular Transport Proteins - genetics | Gene Expression Regulation | Lymphatic Abnormalities - metabolism | E-Selectin - metabolism | Histone Deacetylases - metabolism | Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Transcription Factors - metabolism | Ankyrins - genetics | Alleles | Ankyrins - metabolism | Cyclooxygenase 2 - metabolism | Primary Cell Culture | Endothelial Cells - drug effects
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e95004
Chinese medicine is a complex system guided by traditional Chinese medicine (TCM) theories, which has proven to be especially effective in treating chronic and... 
SIGNALING PATHWAYS | ACTIVATED PROTEIN-KINASE | MYOCARDIAL-INFARCTION | RECEPTOR-GAMMA | SYSTEMS PHARMACOLOGY | MULTIDISCIPLINARY SCIENCES | CELL-PROLIFERATION | PPAR-GAMMA AGONISTS | HEME OXYGENASE-1 | RAW264.7 MACROPHAGES | EXPRESSION | Myocardial Infarction - genetics | Heme Oxygenase-1 - metabolism | Oligonucleotide Array Sequence Analysis | Cell Survival - genetics | Drugs, Chinese Herbal - pharmacology | Male | Gene Regulatory Networks - drug effects | PPAR gamma - metabolism | Heme Oxygenase-1 - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Membrane Proteins - metabolism | Phytotherapy - methods | PPAR gamma - genetics | Cell Line | Cell Survival - drug effects | Gene Regulatory Networks - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Membrane Proteins - genetics | Cells, Cultured | Signal Transduction - genetics | Transcriptome - drug effects | Transcriptome - genetics | Macrophages - cytology | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Blotting, Western | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Sesquiterpenes - pharmacology | Drug Combinations | Mitogen-Activated Protein Kinase 1 - metabolism | Epoxy resins | Heart attack | Myocardial infarction | Phosphorylation | Heart attacks | Target recognition | Biology | Kinases | Proteins | Chinese medicine | Immunology | Nerolidol | Rodents | Heme | Informatics | Pharmaceutical sciences | Food | Traditional Chinese medicine | Extracellular signal-regulated kinase | Heme oxygenase (decyclizing) | Pharmacology | Inflammation | Gene expression | Mode of action | Panax notoginseng | Medicine | Oxygenase | Salvia miltiorrhiza | Multilevel | Nitric oxide | Infarction | Cardiovascular diseases
Journal Article
Journal Article
Eukaryotic Cell, ISSN 1535-9778, 03/2010, Volume 9, Issue 3, pp. 460 - 471
Journal Article
Nature, ISSN 0028-0836, 07/2011, Volume 475, Issue 7354, pp. 106 - 110
Reactive oxygen species (ROS) are mutagenic and may thereby promote cancer(1). Normally, ROS levels are tightly controlled by an inducible antioxidant program... 
TRANSFORMATION | OXIDATIVE STRESS | ACTIVATION | INHIBITION | K-RAS | PATHWAY | MULTIDISCIPLINARY SCIENCES | PANCREATIC-CANCER | HEME OXYGENASE-1 | MUTATIONS | EXPRESSION | NIH 3T3 Cells | Cell Proliferation | Pancreatic Neoplasms - metabolism | Reactive Oxygen Species - metabolism | Cytoskeletal Proteins - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Antioxidants - metabolism | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | MAP Kinase Signaling System | Mitogen-Activated Protein Kinase Kinases - metabolism | Cell Transformation, Neoplastic - genetics | Cytoskeletal Proteins - metabolism | NF-E2-Related Factor 2 - genetics | Intracellular Signaling Peptides and Proteins - genetics | Kelch-Like ECH-Associated Protein 1 | Proto-Oncogene Proteins B-raf - metabolism | Fibroblasts - metabolism | Proto-Oncogene Proteins p21(ras) - metabolism | Oncogenes - genetics | Oxidation-Reduction | Pancreatic Neoplasms - pathology | Cells, Cultured | Pancreatic Neoplasms - genetics | NF-E2-Related Factor 2 - deficiency | Cell Transformation, Neoplastic - metabolism | Animals | Proto-Oncogene Proteins B-raf - genetics | Genes, myc - genetics | NF-E2-Related Factor 2 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Alleles | Cell Line, Tumor | Mice | Adaptor Proteins, Signal Transducing - metabolism | Cell Transformation, Neoplastic - pathology | Polymerase chain reaction | Usage | Reactive oxygen species | Physiological aspects | Research | Gene expression | Oncogenes | Studies | Mass spectrometry | Rodents | Evacuations & rescues | Cancer
Journal Article
Journal Article