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Nature communications, ISSN 2041-1723, 2012, Volume 3, Issue 1, p. 1208
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 2009, Volume 296, Issue 2, pp. H404 - H412
.... In the present study, we hypothesize that Ac-SDKP prevents Gal-3-induced cardiac inflammation, remodeling, and dysfunction, and these effects are mediated by the transforming growth factor (TGF)-β... 
N-acetyl-seryl-aspartyl-lysyl-proline | Inflammation | Cardiac dysfunction | Fibrosis | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MYOCARDIAL-INFARCTION | HEART-FAILURE | MAST-CELLS | AC-SDKP | PRO | ANGIOTENSIN-CONVERTING ENZYME | MACROPHAGE ACTIVATION | inflammation | ANGIOGENESIS IN-VITRO | PERIPHERAL VASCULAR DISEASE | cardiac dysfunction | fibrosis | HYPERTENSIVE-RATS | Inflammation - chemically induced | Phosphorylation | Body Weight - drug effects | Myocardial Contraction - drug effects | Male | Smad3 Protein - metabolism | Heart Rate - drug effects | Inflammation - metabolism | Time Factors | Myocardium - metabolism | Echocardiography, Doppler | Anti-Inflammatory Agents - administration & dosage | Blood Pressure - drug effects | Disease Models, Animal | Macrophages - pathology | Oligopeptides - blood | Anti-Inflammatory Agents - pharmacology | Cardiac Output - drug effects | Ventricular Function, Left - drug effects | Cardiomegaly - physiopathology | Infusions, Parenteral | Rats | Stroke Volume - drug effects | Myocardium - pathology | Cardiotonic Agents - pharmacology | Mast Cells - drug effects | Rats, Sprague-Dawley | Anti-Inflammatory Agents - blood | Collagen - metabolism | Animals | Cardiomegaly - prevention & control | Cardiotonic Agents - administration & dosage | Signal Transduction - drug effects | Cardiotonic Agents - blood | Isoproterenol - pharmacology | Mast Cells - pathology | Cardiomegaly - chemically induced | Galectin 3 | Macrophages - drug effects | Hemodynamics - drug effects | Inflammation - prevention & control | Oligopeptides - administration & dosage | Oligopeptides - pharmacology | Transforming Growth Factor beta - metabolism | Ventricular Remodeling - drug effects | Cardiomegaly - metabolism | Inflammation - physiopathology | Index Medicus
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 08/2016, Volume 55, Issue 2, pp. 201 - 212
Human immunodeficiency virus (HIV)-related pulmonary arterial hypertension has been found to be more prevalent in intravenous drug users... 
Pulmonary hypertension; drug abuse; smooth muscle; bone morphogenetic protein receptor; human immunodeficiency virus-1 | bone morphogenetic protein receptor | OXIDATIVE STRESS | RISK-FACTORS | pulmonary hypertension | BIOCHEMISTRY & MOLECULAR BIOLOGY | smooth muscle | DOWN-REGULATION | MUSCLE-CELLS | CELL BIOLOGY | human immunodeficiency virus-1 | IN-VITRO | drug abuse | RESPIRATORY SYSTEM | ARTERIAL-HYPERTENSION | HIV-1 TAT | TYPE-1 TAT PROTEIN | EXPRESSION | MORPHOGENETIC PROTEIN-RECEPTOR | HIV - physiology | Apoptosis - drug effects | Humans | Rats, Inbred F344 | Myocytes, Smooth Muscle - pathology | Systole - drug effects | Male | Viral Proteins - metabolism | HIV - drug effects | Lung - virology | Blood Pressure - drug effects | Lung - metabolism | Vascular Remodeling - drug effects | Cocaine - adverse effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Rats, Transgenic | Cell Separation | Bone Morphogenetic Protein Receptors - metabolism | Lung - physiopathology | Pulmonary Artery - physiopathology | Pulmonary Artery - drug effects | Down-Regulation - drug effects | Rats, Sprague-Dawley | Muscle, Smooth, Vascular - pathology | Animals | Signal Transduction - drug effects | Lung - drug effects | Cell Proliferation - drug effects | Hemodynamics - drug effects | Hypertension | Heart | Pulmonary arteries | Laboratories | Diagnostic tests | Mortality | Smooth muscle | Infections | Proteins | Hepatitis | Rodents | Human immunodeficiency virus--HIV | Protein expression | Cocaine | Intubation | Apoptosis
Journal Article
Circulation research, ISSN 1524-4571, 2004, Volume 95, Issue 8, pp. 830 - 840
The pulmonary arteries (PA) in pulmonary arterial hypertension (PAH) are constricted and remodeled;. They have suppressed apoptosis, partly attributable to... 
Vascular remodeling | Smooth muscle | Proliferation | Apoptosis | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | proliferation | POTASSIUM CHANNELS | CARDIAC MYOCYTES | smooth muscle | RATS | apoptosis | RELAXATION | vascular remodeling | PERIPHERAL VASCULAR DISEASE | DIVERSITY | HEMATOLOGY | EXPRESSION | K+ CHANNELS | VASOCONSTRICTION | Apoptosis - drug effects | Myocytes, Smooth Muscle - pathology | Hypertrophy, Right Ventricular - etiology | Dichloroacetic Acid - therapeutic use | Hypertension, Pulmonary - chemically induced | Heart Failure - prevention & control | Oxidative Phosphorylation - drug effects | Potassium Channels, Voltage-Gated - metabolism | Vascular Resistance - drug effects | Hypertrophy, Right Ventricular - pathology | Hypertension, Pulmonary - drug therapy | Drug Evaluation, Preclinical | Myocytes, Smooth Muscle - drug effects | Heart Failure - etiology | Muscle, Smooth, Vascular - drug effects | Potassium Channels, Voltage-Gated - genetics | Cells, Cultured - drug effects | Rats | Monocrotaline - toxicity | Mitochondria - drug effects | Pulmonary Artery - drug effects | Organ Specificity | Shab Potassium Channels | Kv1.5 Potassium Channel | Cell Division - drug effects | Pulmonary Artery - cytology | Gene Expression Regulation - drug effects | Muscle, Smooth, Vascular - pathology | Animals | Dichloroacetic Acid - pharmacology | Hemodynamics - drug effects | Hypertension, Pulmonary - pathology | Hypertension, Pulmonary - complications
Journal Article
Journal of cerebral blood flow and metabolism, ISSN 1559-7016, 2015, Volume 35, Issue 11, pp. 1871 - 1881
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2016, Volume 311, Issue 6, pp. H1360 - H1366
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2010, Volume 56, Issue 25, pp. 2115 - 2125
Objectives In this study, we have investigated the effects of cannabidiol (CBD) on myocardial dysfunction, inflammation, oxidative/nitrative stress, cell death... 
Cardiovascular | Internal Medicine | diabetic complications | cannabinoids | inflammation | oxidative stress | CONSTITUENT | CARDIAC & CARDIOVASCULAR SYSTEMS | METALLOTHIONEIN | MITOCHONDRIAL | ENDOCANNABINOID SYSTEM | ANTAGONISM | PROTECTS | INHIBITION | DISEASE | NF-KAPPA-B | Diabetic Cardiomyopathies - metabolism | Reactive Oxygen Species - metabolism | Diabetic Cardiomyopathies - drug therapy | Apoptosis - drug effects | Humans | Body Weight - drug effects | Male | NF-kappa B - metabolism | Glucose | Cannabidiol - therapeutic use | Drug Evaluation, Preclinical | Disease Models, Animal | Mice, Inbred C57BL | Cells, Cultured | Pancreas - drug effects | Cannabidiol - pharmacology | Myocardium - pathology | Pancreas - metabolism | Blood Glucose - drug effects | Insulin - metabolism | Animals | MAP Kinase Signaling System - drug effects | Myocytes, Cardiac - drug effects | Fibrosis | Diabetic Cardiomyopathies - pathology | Hemodynamics - drug effects | Mice | Oxidative Stress - drug effects | Phosphates | Oxidative stress | Medical colleges | Niacinamide | Cardiomyopathy | Alcoholism | Radiation | Superoxide | Biochemistry | Dextrose | Purines | Cannabinoids | Cell death | Analysis | Nitric oxide | Protein kinases | Heart diseases | Adenosine triphosphatase | Jewish schools | Mortality | Cardiovascular disease | Cardiomyocytes | Kinases | Experiments | Variance analysis | Antioxidants | Proteins | Polymerase chain reaction | Rodents | Atherosclerosis | Diabetes | Pancreas | Apoptosis
Journal Article
Neuropsychopharmacology (New York, N.Y.), ISSN 1740-634X, 2004, Volume 29, Issue 8, pp. 1558 - 1572
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2003, Volume 108, Issue 17, pp. 2066 - 2069
...) inhibitor, is an effective and selective pulmonary vasodilator in PAH. However, the long-term effects of PD-5 inhibition and its mechanism of action in human pulmonary arteries (PAs) are unknown... 
Potassium | Hypertension, pulmonary | Sildenafil | CARDIAC & CARDIOVASCULAR SYSTEMS | potassium | THERAPY | hypertension, pulmonary | PERIPHERAL VASCULAR DISEASE | VASODILATOR | HEMATOLOGY | sildenafil | INHALED NITRIC-OXIDE | Large-Conductance Calcium-Activated Potassium Channels | Piperazines - administration & dosage | Phosphoric Diester Hydrolases - biosynthesis | Liver - enzymology | Vasodilator Agents - therapeutic use | Cyclic Nucleotide Phosphodiesterases, Type 5 | Humans | Middle Aged | Hypertension, Pulmonary - physiopathology | Piperazines - economics | Male | Time | Sildenafil Citrate | Purines | Liver - drug effects | Exercise Test - drug effects | Vascular Resistance - drug effects | Adult | Female | Hypertension, Pulmonary - drug therapy | Vasodilator Agents - economics | Sulfones | Muscle, Smooth, Vascular - drug effects | Administration, Oral | Cell Separation | Phosphoric Diester Hydrolases - drug effects | Stroke Volume - drug effects | Treatment Outcome | Piperazines - therapeutic use | Pulmonary Artery - physiopathology | Pulmonary Artery - drug effects | Piperazines - adverse effects | Patch-Clamp Techniques | Pilot Projects | Heart Ventricles - physiopathology | Potassium Channels, Calcium-Activated - drug effects | 3',5'-Cyclic-GMP Phosphodiesterases | Creatinine - analysis | Hemodynamics - drug effects | Vasodilator Agents - adverse effects | In Vitro Techniques | Muscle, Smooth, Vascular - enzymology | Vasodilator Agents - administration & dosage | Heart Ventricles - drug effects
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 11, p. e79543
... for Drug Evaluation and Research, U.S. Food and Drug Administration, Bethesda, Maryland, United States of America, Interagency Oncology Task Force (IOTF) Fellowship... 
BREAST-CANCER | INDUCED CARDIOTOXICITY | INDUCED APOPTOSIS | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | RECEPTOR | STRESS | BLOOD-PRESSURE | MODULATION | CIRCADIAN VARIATION | TROPONIN-I | Caspase 7 - metabolism | Oligonucleotide Array Sequence Analysis | Muscle Contraction - genetics | Heart - physiology | Caspase 3 - metabolism | Apoptosis - genetics | DNA Repair - genetics | Troponin I - blood | Mitochondria - genetics | Heart Ventricles - ultrastructure | Stress, Physiological - drug effects | Antibodies, Monoclonal, Humanized - adverse effects | DNA Repair - drug effects | Hemodynamics - genetics | Myocytes, Cardiac - cytology | Stress, Physiological - genetics | Transcriptome - drug effects | Mitochondria - drug effects | Animals | Myocytes, Cardiac - drug effects | Muscle Contraction - drug effects | Heart - drug effects | Myocytes, Cardiac - metabolism | Heart Ventricles - metabolism | Hemodynamics - drug effects | Mice | Myosin Light Chains - blood | Oxidative Stress - drug effects | Myocytes, Cardiac - ultrastructure | Trastuzumab | Heart Ventricles - drug effects | Heart | Genetic research | Genes | Heart cells | Myosin | Apoptosis | Oxidative stress | Biotechnology | Laboratories | Cardiomyopathy | Mitochondrial DNA | DNA repair | Cancer therapies | Epidermal growth factor | Rodents | Calcium-binding protein | Heart diseases | Pharmaceutical sciences | Deoxyribonucleic acid--DNA | Food | Heart failure | Immunoglobulins | Alcoholism | Cardiomyocytes | Breast cancer | Gene expression | Electron microscopy | ErbB-2 protein | Serum levels | Studies | Troponin | Womens health | Stem cells | Monoclonal antibodies | Myocardium | Biomarkers | Ventricle | Deoxyribonucleic acid | DNA
Journal Article