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The Journal of infectious diseases, ISSN 1537-6613, 2011, Volume 204, Issue 8, pp. 1227 - 1236
Background.  Pro-inflammatory monocytes/macrophages may contribute to increased atherosclerosis in human immunodeficiency virus (HIV)-infected patients. We... 
HEMOGLOBIN | INFECTIOUS DISEASES | ATHEROSCLEROSIS | MICROBIOLOGY | ANTIRETROVIRAL THERAPY | IMMUNOLOGY | CYTOMEGALOVIRUS | SCAVENGER RECEPTOR CD163 | COMPUTED-TOMOGRAPHY | RISK-FACTOR | DISEASE | IMMUNE ACTIVATION | FLOW-CYTOMETRY | Osteopontin - blood | Antigens, CD - immunology | Prospective Studies | Chemokine CCL2 - immunology | Plaque, Atherosclerotic - virology | Humans | Middle Aged | Male | HIV - immunology | Young Adult | HIV Infections - immunology | Flow Cytometry | Interleukin-6 - blood | Statistics, Nonparametric | Adult | Anti-HIV Agents - therapeutic use | Antigens, Differentiation, Myelomonocytic - immunology | Macrophages - immunology | Macrophage Activation - immunology | Anti-HIV Agents - adverse effects | HIV Infections - virology | Lipopolysaccharide Receptors - immunology | Receptors, Cell Surface - blood | Plaque, Atherosclerotic - immunology | Chemokine CCL2 - blood | Receptors, Cell Surface - immunology | Lipopolysaccharide Receptors - blood | Osteopontin - immunology | Coronary Angiography | Antigens, CD - blood | Antigens, Differentiation, Myelomonocytic - blood | Adolescent | Interleukin-6 - immunology | HIV Infections - drug therapy | Macrophages - drug effects | Macrophage Activation - drug effects | Highly active antiretroviral therapy | Patient outcomes | Atherosclerosis | Causes of | Macrophage colony stimulating factor | Research | Drug therapy | HIV infection | Risk factors | Major and Brief Reports
Journal Article
The Journal of Immunology, ISSN 0022-1767, 10/2008, Volume 181, Issue 8, pp. 5627 - 5636
S100A8 and S100A9, highly expressed by neutrophils, activated macrophages, and microvascular endothelial cells, are secreted during inflammatory processes. Our... 
CALCIUM-BINDING PROTEINS | MAST-CELL ACTIVATION | HUMAN NEUTROPHILS | TRANSENDOTHELIAL MIGRATION | ENDOTHELIAL-CELLS | NITRIC-OXIDE | NADPH OXIDASE ACTIVATION | MACROPHAGE EXPRESSION | ARACHIDONIC-ACID | IMMUNOLOGY | CHEMOTACTIC PROTEIN | Free Radical Scavengers - pharmacology | Free Radical Scavengers - chemistry | Humans | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Leukocytes - immunology | Calgranulin A - chemistry | Free Radical Scavengers - immunology | Nitric Oxide Synthase Type II - immunology | Protein Processing, Post-Translational - drug effects | Calgranulin B - immunology | Biological Transport - drug effects | Microcirculation - immunology | Calgranulin A - immunology | Protein Processing, Post-Translational - immunology | Calgranulin B - chemistry | Mast Cells - immunology | Nitric Oxide - immunology | Hemoglobins - immunology | Rats | Anti-Inflammatory Agents, Non-Steroidal - immunology | Inflammation - immunology | p-Methoxy-N-methylphenethylamine - pharmacology | Cysteine - chemistry | Biological Transport - immunology | Endothelial Cells - immunology | Oxidants - immunology | Animals | Lipopolysaccharides - pharmacology | Mice | Mice, Inbred BALB C | Interleukin-10 - immunology | Interferon-gamma - pharmacology | Cysteine - immunology
Journal Article
European Journal of Immunology, ISSN 0014-2980, 02/2011, Volume 41, Issue 2, pp. 537 - 548
The release of hemoglobin (Hb) occurs in some infectious and autoimmune diseases characterized by inflammation. As levels of haptoglobin (Hp) fall, free Hb can... 
Autoimmunity | Anti‐hemoglobin autoantibodies | Systemic lupus erythematosus | Anti-hemoglobin autoantibodies | SYSTEMIC-LUPUS-ERYTHEMATOSUS | DNA ANTIBODIES | HAPTOGLOBIN | IMMUNOLOGY | HEMOLYTIC-ANEMIA | ENDOTHELIAL-CELLS | E-SELECTIN | DISEASE | HYDROGEN-PEROXIDE | BINDING-SITES | AUTOANTIBODY PRODUCTION | Autoantibodies - metabolism | Antigen-Antibody Complex - pharmacology | Autoantibodies - blood | Humans | Immunoglobulin Isotypes - metabolism | Lupus Erythematosus, Systemic - blood | Monocytes - immunology | Haptoglobins - metabolism | snRNP Core Proteins - immunology | Cell Movement - immunology | Aging - immunology | Antibody Specificity - immunology | Autoimmunity - immunology | Base Sequence | Binding Sites - physiology | Hemoglobins - pharmacology | Antibodies, Monoclonal - immunology | Leishmaniasis - immunology | Binding Sites, Antibody - immunology | Endothelial Cells - metabolism | Leishmaniasis - blood | Malaria - immunology | Hemoglobins - immunology | Oxyhemoglobins - immunology | Monocytes - drug effects | Cell Movement - drug effects | Autoantigens - immunology | Immunoglobulin Isotypes - immunology | Mice | Mice, Inbred BALB C | Methemoglobin - pharmacology | Malaria - blood | Animal Structures - metabolism | Animal Structures - immunology | Vaccination | Molecular Sequence Data | Culture Media, Conditioned - pharmacology | Monocytes - metabolism | Lupus Erythematosus, Systemic - immunology | Cytokines - metabolism | Inflammation - immunology | Mice, Inbred NZB | Aging - blood | Endothelial Cells - immunology | Antibodies, Monoclonal - genetics | Autoantibodies - immunology | Immunoglobulin Isotypes - blood | Animals | Methemoglobin - immunology | Immunoglobulin Variable Region - genetics | Antigen-Antibody Complex - immunology | Endothelial Cells - drug effects | Lupus | Hemoglobin | Autoimmune diseases | Immune system
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2015, Volume 308, Issue 4, pp. L314 - L324
Sickle cell disease (SCD) is an autosomal recessive disorder in the gene encoding the beta -chain of hemoglobin. Deoxygenation causes the mutant hemoglobin S... 
Sickle cell disease | Nitric oxide | Pulmonary hypertension | Cell free hemoglobin | LACTATE-DEHYDROGENASE | nitric oxide | PHYSIOLOGY | pulmonary hypertension | NITRIC-OXIDE BIOAVAILABILITY | EXERCISE CAPACITY | KNOCKOUT MICE | sickle cell disease | cell free hemoglobin | ACUTE CHEST SYNDROME | RESPIRATORY SYSTEM | ARTERIAL-HYPERTENSION | FETAL-HEMOGLOBIN | TRANSGENIC MOUSE MODEL | PLATELET ACTIVATION | FREE HEMOGLOBIN | Humans | Heart Failure - physiopathology | Immunity, Innate - genetics | Oxidative Stress - immunology | Hypertension, Pulmonary - physiopathology | Acute Chest Syndrome - physiopathology | Hypertension, Pulmonary - therapy | Hemolysis - immunology | Renal Insufficiency, Chronic - therapy | Vascular Resistance - genetics | Heart Failure - immunology | Renal Insufficiency, Chronic - genetics | Reactive Oxygen Species - blood | Hemoglobin, Sickle - immunology | Toll-Like Receptor 4 - genetics | Vascular Diseases - genetics | Heart Failure - genetics | Toll-Like Receptor 4 - immunology | Renal Insufficiency, Chronic - physiopathology | Hemoglobin, Sickle - metabolism | Heme - immunology | Toll-Like Receptor 4 - blood | Nitric Oxide - genetics | Mutation | Vascular Diseases - blood | Practice Guidelines as Topic | Vascular Resistance - immunology | Heme - metabolism | Acute Chest Syndrome - therapy | Age Factors | Hypertension, Pulmonary - blood | Heart Failure - blood | Erythrocytes, Abnormal | Heart Failure - therapy | Heme - genetics | Reactive Oxygen Species - immunology | Acute Chest Syndrome - genetics | Acute Chest Syndrome - immunology | Hemolysis - genetics | Nitric Oxide - blood | Nitric Oxide - immunology | Vascular Diseases - immunology | Renal Insufficiency, Chronic - immunology | Vascular Diseases - therapy | Oxidative Stress - genetics | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - immunology | Hemoglobin, Sickle - genetics | Vascular Diseases - physiopathology | Renal Insufficiency, Chronic - blood | Acute Chest Syndrome - blood | Genetic aspects | Sickle cell anemia | Vasculitis | Diagnosis | Risk factors | Physiology in Medicine
Journal Article
Brain, behavior, and immunity, ISSN 0889-1591, 2012, Volume 26, Issue 4, pp. 623 - 634
Journal Article
The journal of histochemistry and cytochemistry, ISSN 1551-5044, 2006, Volume 54, Issue 7, pp. 763 - 771
CD163 is a member of the scavenger receptor cysteine-rich superfamily restricted to the monocyte/macrophage lineage and is thought to be a useful marker for... 
Macrophage heterogeneity | Macrophage markers | CD163 | Atherosclerotic lesions | Granulomatous diseases | NITRIC-OXIDE SYNTHASE | GIANT-CELL TUMOR | atherosclerotic lesions | MONOCLONAL-ANTIBODY | GLUCOCORTICOID-INDUCIBLE MEMBER | HUMAN MONOCYTES | CYSTEINE-RICH FAMILY | CELL BIOLOGY | macrophage heterogeneity | TUMOR-NECROSIS-FACTOR | DIFFERENTIATION ANTIGEN | MEMBRANE-PROTEIN RM3/1 | macrophage markers | granulomatous diseases | HEMOGLOBIN SCAVENGER RECEPTOR | Immunohistochemistry | Antigens, CD - immunology | Tuberculosis, Pulmonary - metabolism | Histiocytosis, Non-Langerhans-Cell - metabolism | Giant Cell Tumors - immunology | Antigens, CD - biosynthesis | Humans | Synovial Membrane - pathology | Histiocytosis, Sinus - metabolism | Giant Cell Tumors - metabolism | Antigens, CD - metabolism | Bone Neoplasms - metabolism | Paraffin Embedding | Soft Tissue Neoplasms - immunology | Histiocytosis, Sinus - immunology | Foreign-Body Reaction - immunology | Mass Spectrometry | Sarcoidosis - immunology | Cell Differentiation | Receptors, Cell Surface - biosynthesis | Antigens, Differentiation, Myelomonocytic - immunology | Histiocytosis, Non-Langerhans-Cell - immunology | Macrophages - immunology | Antigens, Differentiation, Myelomonocytic - biosynthesis | Atherosclerosis - immunology | Gaucher Disease - metabolism | Electrophoresis, Polyacrylamide Gel | Soft Tissue Neoplasms - metabolism | Foreign-Body Reaction - metabolism | Gaucher Disease - immunology | Receptors, Cell Surface - metabolism | Bone Neoplasms - immunology | Macrophages - cytology | Receptors, Cell Surface - immunology | Atherosclerosis - metabolism | Macrophage Activation | Macrophages - metabolism | Phenotype | Tuberculosis, Pulmonary - immunology | Antigens, Differentiation, Myelomonocytic - metabolism | Sarcoidosis - metabolism
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