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Cell Metabolism, ISSN 1550-4131, 11/2012, Volume 16, Issue 5, pp. 625 - 633
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 09/2008, Volume 205, Issue 10, pp. 2409 - 2417
The current goal of diabetes therapy is to reduce time-averaged mean levels of glycemia, measured as HbA1c, to prevent diabetic complications. However, HbA1c... 
DIABETES-MELLITUS | MEDICINE, RESEARCH & EXPERIMENTAL | HISTONE LYSINE METHYLATION | ACTIVATION | ENDOTHELIAL-CELLS | TRANSCRIPTION | ATHEROSCLEROSIS | CHROMATIN MODIFICATIONS | COMPLICATIONS | IMMUNOLOGY | CORONARY HEART-DISEASE | NF-KAPPA-B | Protein Methyltransferases | Superoxide Dismutase - genetics | Diabetes Complications | Reactive Oxygen Species - metabolism | Epigenesis, Genetic | Glycated Hemoglobin A - metabolism | Humans | Ion Channels - genetics | Male | Mitochondrial Proteins - genetics | Transcription Factor RelA - genetics | Cattle | Mitochondrial Proteins - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Chemokine CCL2 - metabolism | Endothelial Cells - physiology | Superoxide Dismutase - metabolism | Promoter Regions, Genetic | Histone-Lysine N-Methyltransferase - genetics | Mice, Inbred C57BL | Risk Factors | Cells, Cultured | Gene Expression Regulation | Diabetes Mellitus - metabolism | Chemokine CCL2 - genetics | Mitochondria - metabolism | Mice, Knockout | Hyperglycemia - metabolism | Animals | Histone-Lysine N-Methyltransferase - metabolism | Ion Channels - metabolism | Transcription Factor RelA - metabolism | Endothelial Cells - cytology | Glucose - metabolism | Mice | Uncoupling Protein 1 | Blood Glucose - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Glycated Hemoglobin A - genetics | Index Medicus
Journal Article
Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 7, pp. 678 - 684
Recovery from blood loss requires a greatly enhanced supply of iron to support expanded erythropoiesis. After hemorrhage, suppression of the iron-regulatory... 
OVERLOAD | HOMEOSTASIS | DIFFERENTIATION FACTOR 15 | BETA-THALASSEMIA-INTERMEDIA | ERYTHROPOIESIS | GENETICS & HEREDITY | MOUSE-LIVER | MICE | HEPCIDIN EXPRESSION | ANEMIA | HYPOXIA | Hepcidins - metabolism | beta-Thalassemia - pathology | Liver - pathology | Iron Overload - drug therapy | Molecular Sequence Data | Erythropoiesis - drug effects | Male | Gene Expression Profiling | Iron Overload - pathology | Iron Overload - metabolism | Epoetin Alfa | Erythropoietin - metabolism | Real-Time Polymerase Chain Reaction | Anemia - etiology | Disease Models, Animal | Recombinant Proteins - metabolism | Hormones - pharmacology | Enzyme-Linked Immunosorbent Assay | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Hemorrhage - complications | Hemoglobins - metabolism | Hemorrhage - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Iron - metabolism | Blotting, Western | Erythropoiesis - physiology | Mice, Knockout | Anemia - metabolism | Animals | Mice | beta-Thalassemia - metabolism | Physiological aspects | Genetic research | Genetic aspects | Iron in the body | Hormones | Research | Genetic regulation | Identification and classification | Flow cytometry | Plasma | Phosphorylation | Bone marrow | Homeostasis | Iron | Grants | Metabolism | Gene expression | Experiments | Hemorrhage | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e62068 - e62068
The NF-kappa B pathway plays an important role in chronic inflammatory and autoimmune diseases. Recently, NF-kappa B has also been suggested as an important... 
METABOLIC SYNDROME | OXIDATIVE STRESS | ACTIVATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | MESANGIAL CELLS | INDUCED INFLAMMATION | DIABETIC-NEPHROPATHY | BINDING-ACTIVITY | TRANSCRIPTION FACTOR | ADIPOSE-TISSUE | Albuminuria - complications | Diabetes Mellitus, Experimental - drug therapy | Liver - pathology | Triterpenes - pharmacology | Kidney - pathology | Glycated Hemoglobin A - metabolism | Triterpenes - therapeutic use | Kidney Glomerulus - physiopathology | Male | NF-kappa B - metabolism | Kidney Function Tests | Diabetes Mellitus, Experimental - blood | Adipose Tissue - metabolism | Albuminuria - physiopathology | Kidney - metabolism | Liver - drug effects | Lipids - blood | Inflammation Mediators - metabolism | Lipid Peroxidation - drug effects | Kidney Glomerulus - metabolism | Albuminuria - metabolism | Diabetes Mellitus, Experimental - metabolism | Fatty Acids - metabolism | Kidney - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Albuminuria - pathology | NF-kappa B - antagonists & inhibitors | Kidney - drug effects | Podocytes - metabolism | Cytokines - metabolism | Kidney Glomerulus - drug effects | Liver - metabolism | Adipose Tissue - pathology | Cells, Cultured | Insulin Resistance | Kidney Glomerulus - pathology | Podocytes - pathology | Animals | Podocytes - drug effects | Mice | Oxidative Stress - drug effects | Adipose Tissue - drug effects | Transcription factors | Nephrology | Adipose tissue | Disease | Syngeneic grafts | Oxidative metabolism | Liver | Body weight | Neuropeptides | Adipocytes | Glucose | Animal tissues | Lipid metabolism | Stresses | Obesity | Medical research | NF-κB protein | Cytokines | Internal medicine | Mortality | Abnormalities | Polyamide-imides | Metabolism | Insulin | Fatty acids | Inhibitors | Nephropathy | Weight reduction | Metabolic disorders | Structure-function relationships | Oxidative stress | Animal models | Homeostasis | Adiponectin | Oxidation resistance | Rodents | Collagen (type IV) | Creatinine | Urine | Hypertension | Excretion | Fasting | Kidneys | Diabetes mellitus | Organs | Inflammation | Stress | Medicine | Insulin resistance | Diabetes | Autoimmune diseases | Index Medicus
Journal Article
Diabetes/Metabolism Research and Reviews, ISSN 1520-7552, 10/2016, Volume 32, Issue 7, pp. 754 - 761
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2012, Volume 59, Issue 2, pp. 166 - 177
Objectives The purpose of this study was to examine selective macrophage differentiation occurring in areas of intraplaque hemorrhage in human atherosclerosis.... 
Cardiovascular | Internal Medicine | atherosclerosis | macrophages | ABC transporters | inflammation | hemoglobin | reactive oxygen species | ACTIVATION | CD163 | CARDIAC & CARDIOVASCULAR SYSTEMS | IRON | HAPTOGLOBIN | RESPONSES | SCAVENGER RECEPTOR | CHOLESTEROL EFFLUX | SUDDEN CORONARY DEATH | INTRAPLAQUE HEMORRHAGE | BINDING | Mannose-Binding Lectins - metabolism | Reactive Oxygen Species - metabolism | Monocytes - cytology | Humans | Neovascularization, Pathologic | Orphan Nuclear Receptors - metabolism | Antigens, CD - metabolism | Lectins, C-Type - metabolism | Liver X Receptors | Hemoglobins - physiology | ATP-Binding Cassette Transporters - metabolism | Interleukin-10 - metabolism | Cell Differentiation | Monocytes - physiology | Plaque, Atherosclerotic - pathology | Receptors, Scavenger - metabolism | Macrophages - physiology | Rabbits | Atherosclerosis - pathology | Atherosclerosis - immunology | Down-Regulation | Hemorrhage - immunology | Interleukin-4 - metabolism | Receptors, Cell Surface - metabolism | Macrophages - cytology | Plaque, Atherosclerotic - immunology | Iron - metabolism | Animals | Antigens, Differentiation, Myelomonocytic - metabolism | Haptoglobin | Low density lipoproteins | Atherosclerosis | Glycosylated hemoglobin | Fluorescence | Superoxide | Apolipoproteins | Macrophages | Mitogens | Sugars | Monosaccharides | Hemoglobin | Medical colleges | Cell differentiation | Polymerase chain reaction | Angiogenesis | Flow cytometry | Cell culture | Genotype & phenotype | Rodents | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 09/2012, Volume 97, Issue 9, pp. 3333 - 3341
Context: Sitagliptin is an inhibitor of the enzyme dipeptidyl peptidase-IV (DPP-IV), which degrades the incretins, glucagon-like peptide-1 and... 
IMMUNITY | DIPEPTIDYL-PEPTIDASE-IV | HEALTHY-SUBJECTS | INSULIN-RESISTANCE | INFLAMMATION | ENDOCRINOLOGY & METABOLISM | RECEPTOR | MICE | CD26 | INHIBITOR SITAGLIPTIN | Glycated Hemoglobin A - analysis | Tumor Necrosis Factor-alpha - metabolism | Interleukin-6 - analysis | Dipeptidyl Peptidase 4 - metabolism | Prospective Studies | Humans | Middle Aged | Male | Monocytes - metabolism | I-kappa B Kinase - analysis | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | C-Reactive Protein - metabolism | MAP Kinase Kinase 4 - analysis | MAP Kinase Kinase 4 - metabolism | I-kappa B Kinase - metabolism | Glucagon-Like Peptide 1 - analysis | Adult | Female | Sitagliptin Phosphate | C-Reactive Protein - analysis | Interleukin-6 - metabolism | Toll-Like Receptor 4 - analysis | Dipeptidyl Peptidase 4 - analysis | Anti-Inflammatory Agents, Non-Steroidal | Blood Glucose - analysis | Double-Blind Method | Glucagon-Like Peptide 1 - metabolism | Cell Separation | Toll-Like Receptor 2 - metabolism | Receptors, CCR2 - analysis | Toll-Like Receptor 4 - metabolism | Blotting, Western | Tumor Necrosis Factor-alpha - analysis | Monocytes - drug effects | Triazoles - pharmacology | Receptors, CCR2 - metabolism | Diabetes Mellitus, Type 2 - physiopathology | Toll-Like Receptor 2 - analysis | Aged | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Pyrazines - pharmacology | Index Medicus | Abridged Index Medicus | Endocrine Research
Journal Article
Science, ISSN 0036-8075, 12/2008, Volume 322, Issue 5909, pp. 1839 - 1842
Differences in the amount of fetal hemoglobin (HbF) that persists into adulthood affect the severity of sickle cell disease and the ?-thalassemia syndromes.... 
Protein isoforms | K562 cells | Erythroblasts | Genes | Cell lines | Small interfering RNA | Genetic loci | Reports | Erythroid cells | Gene expression | Hemoglobins | LOCUS-CONTROL REGION | COMPLEX | VARIANTS | MULTIDISCIPLINARY SCIENCES | DISEASE | ZINC-FINGER PROTEIN | DIFFERENTIATION | RECRUITS | BETA-THALASSEMIA | LINEAGE | GENOME-WIDE ASSOCIATION | Erythropoiesis | Fetal Hemoglobin - biosynthesis | Multigene Family | Mi-2 Nucleosome Remodeling and Deacetylase Complex | Humans | Erythroblasts - metabolism | gamma-Globins - metabolism | Protein Isoforms - metabolism | RNA Interference | Transcription, Genetic | gamma-Globins - genetics | Nuclear Proteins - genetics | Down-Regulation | Cells, Cultured | Erythroid Cells - metabolism | Gene Expression Regulation | Histone Deacetylases - metabolism | Nuclear Proteins - metabolism | GATA1 Transcription Factor - metabolism | beta-Globins - genetics | Hemoglobinopathies - therapy | Transcription Factors - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | beta-Globins - metabolism | Fetal Hemoglobin - genetics | K562 Cells | Cell Line, Tumor | Erythroid Precursor Cells - metabolism | Mice | Polymorphism, Single Nucleotide | Protein Isoforms - genetics | Sickle cell anemia | Physiological aspects | Thalassemia | Genetic aspects | Risk factors | Fetal hemoglobin | Medical research | Genotype & phenotype | Hematology | Sickle cell disease | Index Medicus
Journal Article