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Acta Neuropathologica, ISSN 0001-6322, 9/2011, Volume 122, Issue 3, pp. 293 - 311
Cerebrovascular lesions related to congophilic amyloid angiopathy (CAA) often accompany deposition of β-amyloid (Aβ) in Alzheimer’s disease (AD), leading to... 
Pathology | Neurosciences | Congophilic amyloid angiopathy | Medicine & Public Health | Alzheimer’s disease | Astrocytes | Cerebral glucose metabolism | Alzheimer's disease | MICROVASCULAR PATHOLOGY | ALZHEIMERS-DISEASE | NEUROVASCULAR MECHANISMS | GLUCOSE-UTILIZATION | PATHOLOGY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | MOUSE MODEL | ENDOTHELIAL-CELLS | A-BETA | SMOOTH-MUSCLE-CELLS | RAT-BRAIN | Microdialysis - methods | Humans | Astrocytes - pathology | Dystroglycans - metabolism | Glucose Transporter Type 1 - metabolism | Muscle, Smooth - ultrastructure | Glial Fibrillary Acidic Protein - metabolism | Microscopy, Electron, Scanning - methods | Amyloid beta-Peptides - genetics | Amyloid beta-Peptides - metabolism | Cell Culture Techniques | Disease Models, Animal | Endothelium - pathology | Mice, Transgenic | Cerebral Arteries - ultrastructure | Basement Membrane - metabolism | Disease Progression | Symporters - metabolism | Astrocytes - ultrastructure | Blood-Brain Barrier - pathology | Cerebral Arteries - metabolism | Brain - pathology | Glucose - metabolism | Plaque, Amyloid - metabolism | Mice | Astrocytes - metabolism | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Cerebral Amyloid Angiopathy - complications | Monocarboxylic Acid Transporters - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Hemorrhage - etiology | Amyloid beta-Protein Precursor - metabolism | Lactase - metabolism | Cerebral Amyloid Angiopathy - genetics | Astrocytes - drug effects | Plaque, Amyloid - pathology | Gene Expression Regulation - genetics | Hemorrhage - metabolism | Muscle, Smooth - metabolism | Cerebrovascular Disorders - etiology | Cerebral Amyloid Angiopathy - pathology | Animals | Endothelium - metabolism | Glucose Transporter Type 1 - genetics | Symporters - genetics | Cerebral Arteries - pathology | Laminin - metabolism | Monocarboxylic Acid Transporters - genetics | Cerebrovascular Disorders - pathology | Hemorrhage - pathology | Muscle, Smooth - pathology | Index Medicus | Glucose transporter | Leakage | Brain | Neurodegenerative diseases | Cognitive ability | Transgenic mice | Blood vessels | Data processing | Smooth muscle | beta -Amyloid | Glucose transport | Metabolism | Amyloid precursor protein | Blood-brain barrier | Cerebral blood flow | Lactic acid | Mutation | Original Paper
Journal Article
Journal of Forensic Sciences, ISSN 0022-1198, 09/2013, Volume 58, Issue 5, pp. 1183 - 1188
If in clinical practice definitive diagnostic criteria had been established, after death sepsis is often difficult to diagnose, especially if a site of origin... 
forensic science | Gram‐negative sepsis | Acinetobacter | sepsis diagnosed or confirmed at the autopsy | forensic microbiology | etiology of sepsis in legal medicine | Forensic microbiology | Forensic science | Etiology of sepsis in legal medicine | Gram-negative sepsis | Sepsis diagnosed or confirmed at the autopsy | INFECTIONS | MEDICINE, LEGAL | BLOOD CULTURES | Adrenal Glands - pathology | Inflammation - pathology | Liver - pathology | Kidney - pathology | Humans | Middle Aged | Child, Preschool | Infant | Male | Meningitis, Bacterial - pathology | Gram-Negative Bacteria - isolation & purification | Necrosis - pathology | Cause of Death | Young Adult | Drug Resistance, Multiple, Bacterial | Disseminated Intravascular Coagulation - pathology | Adult | Female | Forensic Pathology | Spleen - pathology | Child | Heart Ventricles - pathology | Skin - pathology | Neutrophils - pathology | Lung - pathology | Intestines - pathology | Encephalitis - pathology | Myocardium - pathology | Purpura - pathology | Empyema - pathology | Thrombosis - pathology | Abscess - pathology | Adolescent | Brain - pathology | Gram-Positive Bacteria - isolation & purification | Muscle, Skeletal - pathology | Edema - pathology | Hemorrhage - pathology | Sepsis - microbiology | Causes and theories of causation | Diseases | Autopsies | Digestive system | Sepsis | Hospitalization | Clinical medicine | Gram-negative bacteria | Medical diagnosis | Index Medicus | Autopsy
Journal Article
Journal Article
Journal of Forensic Sciences, ISSN 0022-1198, 07/2012, Volume 57, Issue 4, pp. 976 - 978
Journal Article
Journal Article
Pediatrics, ISSN 0031-4005, 08/2006, Volume 118, Issue 2, pp. 536 - 548
OBJECTIVES. The aim was to survey the range of cerebral injury and abnormalities of cerebral development in infants born between 23 and 30 weeks' gestation... 
Neurodevelopment | Brain imaging | Magnetic resonance imaging | Premature infants | premature infants | brain imaging | magnetic resonance imaging | NEONATAL BRAIN | CRANIAL ULTRASOUND | ABNORMALITIES | INJURY | DEVELOPMENTAL-DISABILITY | TERM | CHILDREN | neurodevelopment | INTRAVENTRICULAR HEMORRHAGE | PEDIATRICS | PREMATURE | WEIGHT | Follow-Up Studies | Humans | Infant | Male | Leukomalacia, Periventricular - pathology | Developmental Disabilities - pathology | Dilatation, Pathologic - pathology | Intensive Care Units, Neonatal | Female | Brain Damage, Chronic - pathology | Dilatation, Pathologic - etiology | Infant, Newborn | Cerebral Hemorrhage - etiology | Cerebral Infarction - pathology | Cerebral Ventricles - pathology | Severity of Illness Index | Leukomalacia, Periventricular - etiology | Cerebral Palsy - epidemiology | Developmental Disabilities - etiology | Basal Ganglia - pathology | Gestational Age | Fetal Growth Retardation - pathology | Neuropsychological Tests | Magnetic Resonance Imaging | Brain Damage, Chronic - etiology | Cerebral Palsy - etiology | Infant, Premature | Brain - pathology | Cerebral Infarction - etiology | Infant, Small for Gestational Age | London - epidemiology | Cerebral Hemorrhage - pathology | Cohort Studies | Infant, Premature, Diseases - pathology | Brain | Medical examination | Infants (Premature) | Diagnosis | Health aspects | Injuries | Risk factors | Studies | Cerebral palsy | Nuclear magnetic resonance--NMR | Child development | Premature birth | Brain damage | Birth weight | Index Medicus | Abridged Index Medicus
Journal Article
Stroke, ISSN 0039-2499, 01/2015, Volume 46, Issue 1, pp. 182 - 189
Journal Article
Journal Article
Frontiers in Cellular and Infection Microbiology, ISSN 2235-2988, 2014, Volume 4, pp. 104 - 104
Journal Article
Journal of Forensic Sciences, ISSN 0022-1198, 05/2008, Volume 53, Issue 3, pp. 720 - 723
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2015, Volume 112, Issue 8, pp. E881 - E890
Cerebral amyloid angiopathy (CAA) is characterized by deposition of amyloid β peptide (Aβ) within walls of cerebral arteries and is an important cause of... 
Alzheimer' s disease | Cerebral amyloid angiopathy | Reactive oxygen species | Vasomotor dysfunction | NADPH oxidase | OXIDATIVE STRESS | ALZHEIMERS-DISEASE | MEMORY DEFICITS | MULTIDISCIPLINARY SCIENCES | vasomotor dysfunction | BETA-PEPTIDES | cerebral amyloid angiopathy | reactive oxygen species | IN-VIVO | NITRIC-OXIDE | TRANSGENIC MOUSE MODEL | Alzheimer's disease | FUNCTIONAL HYPEREMIA | NADPH OXIDASES | CEREBROVASCULAR ENDOTHELIAL DYSFUNCTION | Microglia - metabolism | Reactive Oxygen Species - metabolism | Humans | Astrocytes - pathology | Myocytes, Smooth Muscle - pathology | Vasomotor System - drug effects | Spin Labels | Cerebral Hemorrhage - physiopathology | Apolipoproteins E - metabolism | Cerebral Amyloid Angiopathy - complications | Cyclic N-Oxides - pharmacology | Vasomotor System - physiopathology | Acetophenones - pharmacology | Microglia - pathology | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Astrocytes - drug effects | Cricetinae | Microglia - drug effects | Cerebral Amyloid Angiopathy - physiopathology | Brain - physiopathology | Mice, Inbred C57BL | Cerebral Arteries - physiopathology | Mice, Transgenic | Aging - pathology | Brain - drug effects | Cerebral Amyloid Angiopathy - pathology | Muscle, Smooth, Vascular - pathology | Animals | Vasomotor System - pathology | Cerebral Arteries - pathology | Brain - pathology | Oxidative Stress - drug effects | Cerebral Hemorrhage - complications | Cerebral Hemorrhage - pathology | Astrocytes - metabolism | Development and progression | Amyloidosis | Genetic aspects | Health aspects | Amyloid beta-protein | Brain | Oxidative stress | Oxygen | Pathogenesis | Rodents | Aging | Hemorrhage | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2009, Volume 4, Issue 1, pp. e4317 - e4317
Journal Article