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Journal Article
Stroke (1970), ISSN 0039-2499, 01/2015, Volume 46, Issue 1, pp. 182 - 189
Journal Article
Pediatrics (Evanston), ISSN 1098-4275, 2006, Volume 118, Issue 2, pp. 536 - 548
OBJECTIVES. The aim was to survey the range of cerebral injury and abnormalities of cerebral development in infants born between 23 and 30 weeks' gestation... 
Neurodevelopment | Brain imaging | Magnetic resonance imaging | Premature infants | premature infants | brain imaging | magnetic resonance imaging | NEONATAL BRAIN | CRANIAL ULTRASOUND | ABNORMALITIES | DISORDERS | DEVELOPMENTAL-DISABILITY | TERM | CHILDREN | FEATURES | neurodevelopment | INTRAVENTRICULAR HEMORRHAGE | PEDIATRICS | PREMATURE | Follow-Up Studies | Humans | Infant | Male | Leukomalacia, Periventricular - pathology | Developmental Disabilities - pathology | Dilatation, Pathologic - pathology | Intensive Care Units, Neonatal | Female | Brain Damage, Chronic - pathology | Dilatation, Pathologic - etiology | Infant, Newborn | Cerebral Hemorrhage - etiology | Cerebral Infarction - pathology | Cerebral Ventricles - pathology | Severity of Illness Index | Leukomalacia, Periventricular - etiology | Cerebral Palsy - epidemiology | Developmental Disabilities - etiology | Basal Ganglia - pathology | Gestational Age | Fetal Growth Retardation - pathology | Neuropsychological Tests | Magnetic Resonance Imaging | Brain Damage, Chronic - etiology | Cerebral Palsy - etiology | Infant, Premature | Brain - pathology | Cerebral Infarction - etiology | Infant, Small for Gestational Age | London - epidemiology | Cerebral Hemorrhage - pathology | Cohort Studies | Infant, Premature, Diseases - pathology | Brain | Medical examination | Infants (Premature) | Diagnosis | Health aspects | Injuries | Risk factors | Studies | Cerebral palsy | Nuclear magnetic resonance--NMR | Child development | Premature birth | Brain damage | Birth weight
Journal Article
Frontiers in cellular and infection microbiology, ISSN 2235-2988, 2014, Volume 4
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 8, pp. E881 - E890
Cerebral amyloid angiopathy (CAA) is characterized by deposition of amyloid β peptide (Aβ) within walls of cerebral arteries and is an important cause of... 
Alzheimer' s disease | Cerebral amyloid angiopathy | Reactive oxygen species | Vasomotor dysfunction | NADPH oxidase | OXIDATIVE STRESS | ALZHEIMERS-DISEASE | MEMORY DEFICITS | MULTIDISCIPLINARY SCIENCES | vasomotor dysfunction | BETA-PEPTIDES | cerebral amyloid angiopathy | reactive oxygen species | IN-VIVO | NITRIC-OXIDE | TRANSGENIC MOUSE MODEL | Alzheimer's disease | FUNCTIONAL HYPEREMIA | NADPH OXIDASES | CEREBROVASCULAR ENDOTHELIAL DYSFUNCTION | Microglia - metabolism | Reactive Oxygen Species - metabolism | Humans | Astrocytes - pathology | Myocytes, Smooth Muscle - pathology | Vasomotor System - drug effects | Spin Labels | Cerebral Hemorrhage - physiopathology | Apolipoproteins E - metabolism | Cerebral Amyloid Angiopathy - complications | Cyclic N-Oxides - pharmacology | Vasomotor System - physiopathology | Acetophenones - pharmacology | Microglia - pathology | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Astrocytes - drug effects | Cricetinae | Microglia - drug effects | Cerebral Amyloid Angiopathy - physiopathology | Brain - physiopathology | Mice, Inbred C57BL | Cerebral Arteries - physiopathology | Mice, Transgenic | Aging - pathology | Brain - drug effects | Cerebral Amyloid Angiopathy - pathology | Muscle, Smooth, Vascular - pathology | Animals | Vasomotor System - pathology | Cerebral Arteries - pathology | Brain - pathology | Oxidative Stress - drug effects | Cerebral Hemorrhage - complications | Cerebral Hemorrhage - pathology | Astrocytes - metabolism | Development and progression | Amyloidosis | Genetic aspects | Health aspects | Amyloid beta-protein | Biological Sciences | PNAS Plus
Journal Article
Journal Article
Nature (London), ISSN 1476-4687, 2018, Volume 564, Issue 7736, pp. 415 - 419
We previously reported(1) the presence of amyloid-beta protein (A beta) deposits in individuals with Creutzfeldt-Jakob disease (CJD) who had been treated... 
SEEDS | CEREBRAL-HEMORRHAGE | ALZHEIMERS-DISEASE | DURA-MATER | MULTIDISCIPLINARY SCIENCES | HYPOTHESIS | MUTATION | ANGIOPATHY | CREUTZFELDT-JAKOB-DISEASE | MODEL | Alzheimer Disease - etiology | Humans | tau Proteins - metabolism | Male | Drug Contamination - prevention & control | Case-Control Studies | Disease Transmission, Infectious - prevention & control | Disease Transmission, Infectious - statistics & numerical data | Amyloid beta-Peptides - metabolism | Female | Creutzfeldt-Jakob Syndrome - chemically induced | Iatrogenic Disease | Alzheimer Disease - chemically induced | Growth Hormone - administration & dosage | Disease Models, Animal | Prions - metabolism | Reproducibility of Results | Amyloid beta-Protein Precursor - adverse effects | Growth Hormone - pharmacology | Recombinant Proteins - pharmacology | Recombinant Proteins - administration & dosage | Creutzfeldt-Jakob Syndrome - etiology | Drug Contamination - statistics & numerical data | Animals | Models, Biological | Amyloid beta-Peptides - analysis | Amyloid beta-Protein Precursor - administration & dosage | Mice | tau Proteins - analysis | Cadaver | Brain | Seeds | Peptides | Creutzfeldt-Jakob disease | Proteins | Surgery | Vials | Children | Prion protein | Alzheimer's disease | Deposition | Public health | Plaques | Neurodegenerative diseases | Medical treatment | Patients | Disease control | Amyloid precursor protein | Laboratory tests | Pathology | Hypotheses | Tau protein | Prions | Pituitary | Cadavers | Growth hormones | Growth hormone | Laboratory animals | Alzheimers disease
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 01/2009, Volume 4, Issue 1, p. e4317
Journal Article