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Publication DateThe New England Journal of Medicine, ISSN 0028-4793, 01/2018, Volume 378, Issue 4, pp. 370 - 379
Understanding the pathophysiology of the body’s response to hemorrhage has led to improvements in prehospital care, more rapid hemostasis, avoidance of massive...
MORTALITY | PLATELET DYSFUNCTION | MEDICINE, GENERAL & INTERNAL | DAMAGE CONTROL RESUSCITATION | BRAIN-INJURY | ENDOVASCULAR BALLOON OCCLUSION | OXYGEN DEBT | MASSIVE TRANSFUSION | HYPOTENSIVE PATIENTS | FLUID RESUSCITATION | TRAUMA | Blood Transfusion | Resuscitation - methods | Humans | Shock, Hemorrhagic - physiopathology | Shock, Hemorrhagic - therapy | Shock, Hemorrhagic - diagnosis | Emergency Medical Services | Care and treatment | Usage | Hemorrhagic shock | Young adults | Patient outcomes | Diagnosis | Research | Health aspects | Risk factors | Resuscitation | Blood platelets | Hemostasis | Surgery | Mortality | Critical care | Mitochondrial DNA | Metabolism | Patients | Trauma | Hemorrhage | Endothelium
MORTALITY | PLATELET DYSFUNCTION | MEDICINE, GENERAL & INTERNAL | DAMAGE CONTROL RESUSCITATION | BRAIN-INJURY | ENDOVASCULAR BALLOON OCCLUSION | OXYGEN DEBT | MASSIVE TRANSFUSION | HYPOTENSIVE PATIENTS | FLUID RESUSCITATION | TRAUMA | Blood Transfusion | Resuscitation - methods | Humans | Shock, Hemorrhagic - physiopathology | Shock, Hemorrhagic - therapy | Shock, Hemorrhagic - diagnosis | Emergency Medical Services | Care and treatment | Usage | Hemorrhagic shock | Young adults | Patient outcomes | Diagnosis | Research | Health aspects | Risk factors | Resuscitation | Blood platelets | Hemostasis | Surgery | Mortality | Critical care | Mitochondrial DNA | Metabolism | Patients | Trauma | Hemorrhage | Endothelium
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Loading RightsCirculatory shock, ISSN 0092-6213, 1974
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The New England Journal of Medicine, ISSN 0028-4793, 05/2018, Volume 378, Issue 19, pp. 1850 - 1853
To the Editor: In his review article, Cannon (Jan. 25 issue) 1 reports on hemorrhagic shock. A meta-analysis has shown that the administration of tranexamic...
MEDICINE, GENERAL & INTERNAL | FIBRINOLYSIS SHUTDOWN | Blood Pressure | Humans | Shock, Hemorrhagic
MEDICINE, GENERAL & INTERNAL | FIBRINOLYSIS SHUTDOWN | Blood Pressure | Humans | Shock, Hemorrhagic
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Loading RightsNEW ENGLAND JOURNAL OF MEDICINE, ISSN 0028-4793, 05/2018, Volume 378, Issue 19, pp. 1851 - 1852
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Loading RightsNEW ENGLAND JOURNAL OF MEDICINE, ISSN 0028-4793, 05/2018, Volume 378, Issue 19, pp. 1851 - 1851
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Loading RightsNEW ENGLAND JOURNAL OF MEDICINE, ISSN 0028-4793, 05/2018, Volume 378, Issue 19, pp. 1850 - 1851
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Loading RightsScandinavian Journal of Trauma, Resuscitation and Emergency Medicine, ISSN 1757-7241, 12/2016, Volume 24, Issue 1
Background: A new classification of hypovolemic shock based on the shock index (SI) was proposed in 2013. This classification contains four classes of shock...
Shock index | Multiple trauma | Haemorrhagic shock | Traumatic brain injury | MORTALITY | EUROPEAN GUIDELINE | MANAGEMENT | MASSIVE TRANSFUSION | COAGULOPATHY | RESPONSES | EMERGENCY MEDICINE | MAJOR TRAUMA | UTILITY | REGISTRY | Shock | Evaluation | Brain | Complications and side effects | Care and treatment | Practice guidelines (Medicine) | Development and progression | Injuries
Shock index | Multiple trauma | Haemorrhagic shock | Traumatic brain injury | MORTALITY | EUROPEAN GUIDELINE | MANAGEMENT | MASSIVE TRANSFUSION | COAGULOPATHY | RESPONSES | EMERGENCY MEDICINE | MAJOR TRAUMA | UTILITY | REGISTRY | Shock | Evaluation | Brain | Complications and side effects | Care and treatment | Practice guidelines (Medicine) | Development and progression | Injuries
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Loading RightsJournal of Trauma and Acute Care Surgery, ISSN 2163-0755, 11/2013, Volume 75, Issue 5, pp. 888 - 895
SURGERY | HEMOGLOBIN SOLUTION | OXYGEN-CONSUMPTION | DAMAGE CONTROL RESUSCITATION | MASSIVE TRANSFUSION | DEPOLARIZES CELLS | BLOOD-VOLUME | FRESH-FROZEN PLASMA | BACTERIAL TRANSLOCATION | HYPERTONIC SALINE 7.5-PERCENT | FLUID RESUSCITATION | CRITICAL CARE MEDICINE | Animals | Resuscitation - methods | Shock, Hemorrhagic - etiology | Wounds and Injuries - therapy | Humans | Wounds and Injuries - complications | Wounds and Injuries - diagnosis | Shock, Hemorrhagic - therapy | Shock, Hemorrhagic - diagnosis | Trauma Severity Indices
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Loading Rights1994, ISBN 0340560215, xii, 187
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Annals of Surgery, ISSN 0003-4932, 07/2011, Volume 254, Issue 1, pp. 10 - 19
A trauma patient's survival depends on the ability to control 2 opposing conditions, bleeding at the early phase and thrombosis at a late phase of trauma. The...
HEMORRHAGIC-SHOCK | SURGERY | INFLAMMATORY RESPONSE SYNDROME | TUMOR-NECROSIS-FACTOR | MULTIPLE ORGAN DYSFUNCTION | ACUTE COAGULOPATHY | PLASMINOGEN-ACTIVATOR | BLOOD-COAGULATION | FIBRIN FORMATION | TISSUE FACTOR | PROCOAGULANT ACTIVITY | Disseminated Intravascular Coagulation - etiology | Disseminated Intravascular Coagulation - prevention & control | Humans | Shock - physiopathology | Wounds and Injuries - complications | Wounds and Injuries - diagnosis | Blood Coagulation - physiology | Wounds and Injuries - physiopathology | Shock - complications | Shock - diagnosis
HEMORRHAGIC-SHOCK | SURGERY | INFLAMMATORY RESPONSE SYNDROME | TUMOR-NECROSIS-FACTOR | MULTIPLE ORGAN DYSFUNCTION | ACUTE COAGULOPATHY | PLASMINOGEN-ACTIVATOR | BLOOD-COAGULATION | FIBRIN FORMATION | TISSUE FACTOR | PROCOAGULANT ACTIVITY | Disseminated Intravascular Coagulation - etiology | Disseminated Intravascular Coagulation - prevention & control | Humans | Shock - physiopathology | Wounds and Injuries - complications | Wounds and Injuries - diagnosis | Blood Coagulation - physiology | Wounds and Injuries - physiopathology | Shock - complications | Shock - diagnosis
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Loading RightsAnnals of Surgery, ISSN 0003-4932, 03/2011, Volume 253, Issue 3, pp. 431 - 441
Objective: To determine whether out-of-hospital administration of hypertonic fluids would improve survival after severe injury with hemorrhagic shock....
SURGERY | MULTICENTER TRIAL | NEUTROPHIL ACTIVATION | CONSENSUS CONFERENCE | INJURED PATIENTS | 7.5-PERCENT SODIUM-CHLORIDE | SALINE-DEXTRAN | OUTCOMES CONSORTIUM | IMPACT MANAGEMENT ERRORS | UNCONTROLLED HEMORRHAGIC-SHOCK | FLUID RESUSCITATION | Dextrans - adverse effects | Double-Blind Method | Hospital Mortality | Shock, Traumatic - therapy | Humans | Middle Aged | Male | Survival Rate | Combined Modality Therapy | Shock - mortality | Young Adult | Early Termination of Clinical Trials | Blood Transfusion | Saline Solution, Hypertonic - adverse effects | Adult | Female | Shock - therapy | Shock, Traumatic - mortality | Hypertonic Solutions - administration & dosage | Hypertonic Solutions - adverse effects | Saline Solution, Hypertonic - administration & dosage | Dextrans - administration & dosage | Emergency Medical Services | Cohort Studies
SURGERY | MULTICENTER TRIAL | NEUTROPHIL ACTIVATION | CONSENSUS CONFERENCE | INJURED PATIENTS | 7.5-PERCENT SODIUM-CHLORIDE | SALINE-DEXTRAN | OUTCOMES CONSORTIUM | IMPACT MANAGEMENT ERRORS | UNCONTROLLED HEMORRHAGIC-SHOCK | FLUID RESUSCITATION | Dextrans - adverse effects | Double-Blind Method | Hospital Mortality | Shock, Traumatic - therapy | Humans | Middle Aged | Male | Survival Rate | Combined Modality Therapy | Shock - mortality | Young Adult | Early Termination of Clinical Trials | Blood Transfusion | Saline Solution, Hypertonic - adverse effects | Adult | Female | Shock - therapy | Shock, Traumatic - mortality | Hypertonic Solutions - administration & dosage | Hypertonic Solutions - adverse effects | Saline Solution, Hypertonic - administration & dosage | Dextrans - administration & dosage | Emergency Medical Services | Cohort Studies
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Loading RightsCritical Care, ISSN 1364-8535, 10/2004, Volume 8, Issue 5, pp. 373 - 381
This review addresses the pathophysiology and treatment of hemorrhagic shock - a condition produced by rapid and significant loss of intravascular volume,...
Shock | Transfusion | Oxygen delivery | Blood loss | Oxygen consumption | Estimated blood volume | Hemorrhage | HEMOGLOBIN | estimated blood volume | blood loss | shock | transfusion | 2 PARTS | HYPOXIA | CRYSTALLOIDS | FLUID RESUSCITATION | MEDICAL PROGRESS | oxygen consumption | SKELETAL-MUSCLE PO2 | BLOOD-TRANSFUSION | oxygen delivery | VASODILATATION | hemorrhage | CRITICAL CARE MEDICINE | Cardiac Output - physiology | Critical Care - methods | Fluid Therapy | Intensive Care Units | Blood Transfusion | Emergency Treatment | Humans | Shock, Hemorrhagic - physiopathology | Hypovolemia - physiopathology | Shock, Hemorrhagic - therapy | Hypovolemia - etiology | Review
Shock | Transfusion | Oxygen delivery | Blood loss | Oxygen consumption | Estimated blood volume | Hemorrhage | HEMOGLOBIN | estimated blood volume | blood loss | shock | transfusion | 2 PARTS | HYPOXIA | CRYSTALLOIDS | FLUID RESUSCITATION | MEDICAL PROGRESS | oxygen consumption | SKELETAL-MUSCLE PO2 | BLOOD-TRANSFUSION | oxygen delivery | VASODILATATION | hemorrhage | CRITICAL CARE MEDICINE | Cardiac Output - physiology | Critical Care - methods | Fluid Therapy | Intensive Care Units | Blood Transfusion | Emergency Treatment | Humans | Shock, Hemorrhagic - physiopathology | Hypovolemia - physiopathology | Shock, Hemorrhagic - therapy | Hypovolemia - etiology | Review
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Loading RightsAnnals of Surgery, ISSN 0003-4932, 03/2016, Volume 263, Issue 3, pp. 565 - 572
OBJECTIVE:To determine the role of cell swelling in severe hemorrhagic shock and resuscitation injury. BACKGROUND:Circulatory shock induces the loss of...
osmotic effects | polyethylene glycol | resuscitation | Gluconate | ischemia | gluconate | LACTOBIONATE | ANIONS | SURGERY | IMPACT | TISSUE | PERFUSION | Resuscitation - methods | Mice, Inbred C57BL | Rats | Shock, Hemorrhagic - therapy | Liver - physiopathology | Rats, Sprague-Dawley | Microcirculation - physiology | Animals | Fluid Therapy - methods | Shock, Hemorrhagic - physiopathology | Isotonic Solutions | Edema - physiopathology | Mice | Hemodynamics | Regional Blood Flow - physiology | Disease Models, Animal
osmotic effects | polyethylene glycol | resuscitation | Gluconate | ischemia | gluconate | LACTOBIONATE | ANIONS | SURGERY | IMPACT | TISSUE | PERFUSION | Resuscitation - methods | Mice, Inbred C57BL | Rats | Shock, Hemorrhagic - therapy | Liver - physiopathology | Rats, Sprague-Dawley | Microcirculation - physiology | Animals | Fluid Therapy - methods | Shock, Hemorrhagic - physiopathology | Isotonic Solutions | Edema - physiopathology | Mice | Hemodynamics | Regional Blood Flow - physiology | Disease Models, Animal
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Loading RightsCritical Care Medicine, ISSN 0090-3493, 02/2013, Volume 41, Issue 2, pp. 573 - 579
Objective: To provide a conceptual and clinical review of the physiology of the venous system as it is related to cardiac function in health and disease. Data:...
obstructive shock | hemorrhagic shock | cardiovascular physiology | septic shock | cardiogenic shock | hemodynamics | CARDIAC-OUTPUT | BLOOD-VOLUME | HEMORRHAGIC-SHOCK | HEMODYNAMIC-RESPONSE | ANESTHETIZED DOGS | VASCULAR VOLUME | INOTROPIC AGENTS | NITRIC-OXIDE | SEVERE SEPSIS | CRITICAL CARE MEDICINE | Cardiac Output - physiology | Fluid Therapy | Vasoconstrictor Agents - therapeutic use | Catecholamines - metabolism | Coronary Circulation - physiology | Cytokines - metabolism | Vasodilator Agents - therapeutic use | Ventricular Dysfunction - therapy | Humans | Hypovolemia - physiopathology | Pneumothorax - physiopathology | Shock - physiopathology | Stroke Volume - physiology | Critical Illness | Positive-Pressure Respiration | Cardiotonic Agents - therapeutic use | Shock - therapy | Blood Pressure - physiology | Hypovolemia - therapy | Nitric Oxide - metabolism | Ventricular Dysfunction - physiopathology
obstructive shock | hemorrhagic shock | cardiovascular physiology | septic shock | cardiogenic shock | hemodynamics | CARDIAC-OUTPUT | BLOOD-VOLUME | HEMORRHAGIC-SHOCK | HEMODYNAMIC-RESPONSE | ANESTHETIZED DOGS | VASCULAR VOLUME | INOTROPIC AGENTS | NITRIC-OXIDE | SEVERE SEPSIS | CRITICAL CARE MEDICINE | Cardiac Output - physiology | Fluid Therapy | Vasoconstrictor Agents - therapeutic use | Catecholamines - metabolism | Coronary Circulation - physiology | Cytokines - metabolism | Vasodilator Agents - therapeutic use | Ventricular Dysfunction - therapy | Humans | Hypovolemia - physiopathology | Pneumothorax - physiopathology | Shock - physiopathology | Stroke Volume - physiology | Critical Illness | Positive-Pressure Respiration | Cardiotonic Agents - therapeutic use | Shock - therapy | Blood Pressure - physiology | Hypovolemia - therapy | Nitric Oxide - metabolism | Ventricular Dysfunction - physiopathology
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Loading RightsAmerican Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 04/2011, Volume 183, Issue 7, pp. 847 - 855
Shock occurs when failure of the cardiovascular system compromises tissue perfusion. When fluid administration fails to restore adequate arterial pressure and...
Shock | Vasopressor | Norepinephrine | Dopamine | Inotropic | norepinephrine | shock | MICROCIRCULATORY PERFUSION | LOW-DOSE DOPAMINE | BLOOD-FLOW | inotropic | SYSTEMIC HEMODYNAMICS | GOAL-DIRECTED THERAPY | SURVIVING SEPSIS CAMPAIGN | VENOUS OXYGEN-SATURATION | SEVERE SEPTIC SHOCK | RESPIRATORY SYSTEM | vasopressor | NOREPINEPHRINE INFUSION | DECOMPENSATED HEART-FAILURE | dopamine | CRITICAL CARE MEDICINE | Critical Care - methods | Prognosis | Humans | Shock, Cardiogenic - mortality | Norepinephrine - administration & dosage | Male | Shock, Hemorrhagic - diagnosis | Shock, Hemorrhagic - drug therapy | Vasopressins - administration & dosage | Shock - diagnosis | Shock, Hemorrhagic - mortality | Female | Intensive Care Units | Shock, Cardiogenic - diagnosis | Vasoconstrictor Agents - administration & dosage | Risk Assessment | Treatment Outcome | Dopamine - administration & dosage | Randomized Controlled Trials as Topic | Shock - mortality | Shock, Cardiogenic - drug therapy | Cardiotonic Agents - administration & dosage | Shock - drug therapy | Survival Analysis | Hemodynamics - drug effects
Shock | Vasopressor | Norepinephrine | Dopamine | Inotropic | norepinephrine | shock | MICROCIRCULATORY PERFUSION | LOW-DOSE DOPAMINE | BLOOD-FLOW | inotropic | SYSTEMIC HEMODYNAMICS | GOAL-DIRECTED THERAPY | SURVIVING SEPSIS CAMPAIGN | VENOUS OXYGEN-SATURATION | SEVERE SEPTIC SHOCK | RESPIRATORY SYSTEM | vasopressor | NOREPINEPHRINE INFUSION | DECOMPENSATED HEART-FAILURE | dopamine | CRITICAL CARE MEDICINE | Critical Care - methods | Prognosis | Humans | Shock, Cardiogenic - mortality | Norepinephrine - administration & dosage | Male | Shock, Hemorrhagic - diagnosis | Shock, Hemorrhagic - drug therapy | Vasopressins - administration & dosage | Shock - diagnosis | Shock, Hemorrhagic - mortality | Female | Intensive Care Units | Shock, Cardiogenic - diagnosis | Vasoconstrictor Agents - administration & dosage | Risk Assessment | Treatment Outcome | Dopamine - administration & dosage | Randomized Controlled Trials as Topic | Shock - mortality | Shock, Cardiogenic - drug therapy | Cardiotonic Agents - administration & dosage | Shock - drug therapy | Survival Analysis | Hemodynamics - drug effects
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Loading RightsJournal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2006, Volume 40, Issue 1, pp. 185 - 194
Although 17β-estradiol (E2) administration following trauma-hemorrhage (T-H) improves cardiac function in male rodents, it is not known whether the salutary...
Shock | Agonists | Receptors | Hormones | Estrogen | APOPTOSIS | estrogen | CARDIAC & CARDIOVASCULAR SYSTEMS | receptors | hormones | NITRIC-OXIDE SYNTHASE | shock | ISCHEMIA | ALPHA | CELL BIOLOGY | VASCULAR INJURY | agonists | ESTRADIOL | INFLAMMATORY RESPONSE | STRESS | GENDER-DIFFERENCES | EXPRESSION | Propionates - pharmacology | Up-Regulation | Estradiol - analogs & derivatives | Nitriles - pharmacology | Shock, Hemorrhagic - metabolism | Male | Shock, Hemorrhagic - drug therapy | Heat Shock Transcription Factors | Transcription Factors - drug effects | DNA-Binding Proteins - metabolism | Wounds and Injuries - metabolism | Estrogen Receptor alpha - agonists | Estrogen Receptor beta - agonists | Protective Agents - pharmacology | Myocardium - metabolism | Estradiol - pharmacology | Pyrazoles - pharmacology | Heat-Shock Proteins - drug effects | Heat-Shock Proteins - metabolism | DNA-Binding Proteins - drug effects | Wounds and Injuries - complications | Rats | Rats, Sprague-Dawley | Transcription Factors - metabolism | Animals | Phenols | Selective Estrogen Receptor Modulators - pharmacology
Shock | Agonists | Receptors | Hormones | Estrogen | APOPTOSIS | estrogen | CARDIAC & CARDIOVASCULAR SYSTEMS | receptors | hormones | NITRIC-OXIDE SYNTHASE | shock | ISCHEMIA | ALPHA | CELL BIOLOGY | VASCULAR INJURY | agonists | ESTRADIOL | INFLAMMATORY RESPONSE | STRESS | GENDER-DIFFERENCES | EXPRESSION | Propionates - pharmacology | Up-Regulation | Estradiol - analogs & derivatives | Nitriles - pharmacology | Shock, Hemorrhagic - metabolism | Male | Shock, Hemorrhagic - drug therapy | Heat Shock Transcription Factors | Transcription Factors - drug effects | DNA-Binding Proteins - metabolism | Wounds and Injuries - metabolism | Estrogen Receptor alpha - agonists | Estrogen Receptor beta - agonists | Protective Agents - pharmacology | Myocardium - metabolism | Estradiol - pharmacology | Pyrazoles - pharmacology | Heat-Shock Proteins - drug effects | Heat-Shock Proteins - metabolism | DNA-Binding Proteins - drug effects | Wounds and Injuries - complications | Rats | Rats, Sprague-Dawley | Transcription Factors - metabolism | Animals | Phenols | Selective Estrogen Receptor Modulators - pharmacology
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