X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (19654) 19654
Dissertation (8) 8
Book Chapter (5) 5
Magazine Article (5) 5
Data Set (2) 2
Conference Proceeding (1) 1
Publication (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (18927) 18927
hep g2 cells (18531) 18531
animals (7721) 7721
mice (4980) 4980
biochemistry & molecular biology (3732) 3732
male (3560) 3560
cell line, tumor (3528) 3528
apoptosis (3515) 3515
cancer (3202) 3202
expression (2759) 2759
liver neoplasms - pathology (2749) 2749
pharmacology & pharmacy (2607) 2607
cell survival - drug effects (2483) 2483
liver (2481) 2481
carcinoma, hepatocellular - pathology (2458) 2458
female (2399) 2399
liver neoplasms - metabolism (2344) 2344
apoptosis - drug effects (2244) 2244
analysis (2171) 2171
cell proliferation - drug effects (2161) 2161
carcinoma, hepatocellular - metabolism (2159) 2159
proteins (2115) 2115
hepatocellular carcinoma (2038) 2038
cell biology (2004) 2004
cells (1948) 1948
antineoplastic agents - pharmacology (1947) 1947
gene expression (1921) 1921
liver neoplasms - genetics (1895) 1895
liver cancer (1837) 1837
carcinoma, hepatocellular - genetics (1753) 1753
oncology (1720) 1720
chemistry, medicinal (1714) 1714
cytotoxicity (1709) 1709
activation (1682) 1682
liver - metabolism (1675) 1675
dose-response relationship, drug (1612) 1612
signal transduction (1527) 1527
oxidative stress (1524) 1524
in-vitro (1489) 1489
metabolism (1448) 1448
research (1424) 1424
transfection (1382) 1382
rats (1379) 1379
liver neoplasms - drug therapy (1372) 1372
hela cells (1360) 1360
cell line (1351) 1351
cell proliferation (1346) 1346
toxicology (1336) 1336
hepatoma (1315) 1315
hepatocytes - metabolism (1305) 1305
molecular structure (1280) 1280
multidisciplinary sciences (1253) 1253
phosphorylation (1239) 1239
gene expression regulation, neoplastic (1225) 1225
inhibition (1217) 1217
mice, nude (1207) 1207
nanoparticles (1204) 1204
hek293 cells (1183) 1183
hepatocytes (1181) 1181
carcinoma, hepatocellular - drug therapy (1159) 1159
chemistry, multidisciplinary (1144) 1144
mice, inbred c57bl (1117) 1117
antineoplastic agents - chemistry (1111) 1111
signal transduction - drug effects (1099) 1099
health aspects (1094) 1094
toxicity (1068) 1068
research article (1067) 1067
mice, inbred balb c (1045) 1045
structure-activity relationship (1041) 1041
reactive oxygen species - metabolism (1031) 1031
physiological aspects (1022) 1022
medicine (1014) 1014
genes (1005) 1005
liver - drug effects (999) 999
gene-expression (991) 991
middle aged (991) 991
hepatocytes - drug effects (976) 976
growth (933) 933
protein binding (932) 932
protein (931) 931
tumors (918) 918
rna, messenger - metabolism (916) 916
hepatocellular-carcinoma (913) 913
rna interference (909) 909
mcf-7 cells (894) 894
plant extracts - pharmacology (878) 878
identification (870) 870
hepg2 cells (852) 852
dna (850) 850
kinases (843) 843
proliferation (836) 836
down-regulation (835) 835
metastasis (832) 832
enzymes (823) 823
cell cycle (815) 815
medicine, research & experimental (815) 815
cells, cultured (812) 812
gene (812) 812
biology (810) 810
drug screening assays, antitumor (802) 802
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (19356) 19356
Chinese (669) 669
Japanese (171) 171
German (30) 30
Russian (28) 28
Korean (15) 15
Spanish (6) 6
French (4) 4
Dutch (2) 2
Czech (1) 1
Persian (1) 1
Portuguese (1) 1
Serbian (1) 1
Turkish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Particle and fibre toxicology, ISSN 1743-8977, 2013, Volume 10, Issue 1, pp. 27 - 27
Journal Article
Journal of Immunology, ISSN 0022-1767, 11/2012, Volume 189, Issue 10, pp. 4748 - 4758
The TNF superfamily ligands APRIL and BAFF bind with different affinity to two receptors, BCMA and TACI, and induce cell survival and/or proliferation, whereas BAFF also binds specifically to BAFFR... 
Forkhead Transcription Factors - immunology | MAP Kinase Kinase 7 - genetics | G2 Phase Cell Cycle Checkpoints - immunology | Humans | M Phase Cell Cycle Checkpoints - genetics | MAP Kinase Kinase 7 - immunology | DNA-Binding Proteins - metabolism | MAP Kinase Kinase 7 - metabolism | Cell Cycle Proteins - immunology | Liver - immunology | Phosphorylation - genetics | Proliferating Cell Nuclear Antigen - genetics | Forkhead Transcription Factors - metabolism | Cell Cycle Proteins - genetics | B-Cell Activating Factor - immunology | Proliferating Cell Nuclear Antigen - immunology | Phosphorylation - immunology | Nuclear Proteins - genetics | Transcription Factors - immunology | B-Cell Maturation Antigen - immunology | DNA-Binding Proteins - immunology | Cell Cycle Proteins - metabolism | M Phase Cell Cycle Checkpoints - immunology | Nuclear Proteins - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Forkhead Transcription Factors - genetics | Nuclear Proteins - immunology | Hep G2 Cells | Transcription Factors - metabolism | G2 Phase Cell Cycle Checkpoints - genetics | B-Cell Activating Factor - metabolism | B-Cell Maturation Antigen - metabolism | B-Cell Activating Factor - genetics | Protein Binding | Transcription, Genetic - immunology | Liver - cytology | B-Cell Maturation Antigen - genetics | Transcription, Genetic - genetics | Forkhead Box Protein O3 | Proliferating Cell Nuclear Antigen - metabolism
Journal Article
Molecular Medicine Reports, ISSN 1791-2997, 11/2017, Volume 16, Issue 5, pp. 6156 - 6161
.... The aim of the present study was to explore the mechanism by which Bmi-1 may promote invasion and migration of hepatocellular carcinoma Hep G2 cells... 
Hep G2 cells | Epithelial-mesenchymal transition | BMI1 proto-oncogene | CD133 | MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | METASTASIS | DOWN-REGULATION | PROLIFERATION | E-CADHERIN | INVASION | ONCOLOGY | CANCER STEM-CELL | epithelial-mesenchymal transition | PROGRESSION
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 5, p. e0125727
.... We demonstrated that ARG could induce apoptosis in Hep G2 and SMMC7721 cells but not in normal hepatic cells, and its apoptotic effect on Hep G2 was stronger than that on... 
CANCER-CELLS | TRANSCRIPTION FACTORS | IN-VITRO | INDUCED APOPTOSIS | MULTIDISCIPLINARY SCIENCES | MITOCHONDRIA | DEATH | NF-KAPPA-B | BCL-2 FAMILY-MEMBERS | KINASE ACTIVATION | P53 | Tumor Necrosis Factor-alpha - metabolism | Fas Ligand Protein - metabolism | Caspase 9 - genetics | Caspase 9 - metabolism | Furans - pharmacology | Humans | Tumor Necrosis Factor-alpha - genetics | bcl-2-Associated X Protein - agonists | Caspase 3 - metabolism | Gene Expression Regulation, Neoplastic | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Membrane Potential, Mitochondrial - drug effects | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | fas Receptor - metabolism | Lignans - pharmacology | Proto-Oncogene Proteins c-akt - genetics | Tumor Necrosis Factor-alpha - agonists | Proto-Oncogene Proteins c-bcl-2 - metabolism | Caspase 3 - genetics | Fas Ligand Protein - agonists | fas Receptor - genetics | Proto-Oncogene Proteins c-akt - metabolism | bcl-2-Associated X Protein - genetics | NF-kappa B - antagonists & inhibitors | Signal Transduction | Cytochromes c - metabolism | bcl-2-Associated X Protein - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Organ Specificity | Hep G2 Cells | fas Receptor - agonists | Phosphatidylinositol 3-Kinases - genetics | Poly(ADP-ribose) Polymerases - metabolism | Poly(ADP-ribose) Polymerases - genetics | NF-kappa B - genetics | Cell Line, Tumor | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Antineoplastic Agents, Phytogenic - pharmacology | Fas Ligand Protein - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Apoptosis | Antiviral agents | Mitochondrial DNA | Liver diseases | Hepatoma | Tumor proteins | Cytochrome | Veterinary colleges | Bax protein | Bcl-2 protein | Toxicity | p53 Protein | Cytotoxicity | Hepatocellular carcinoma | AKT protein | Activation | Kinases | Caspase-3 | Liver cancer | Mitochondria | Ribose | Tumor necrosis factor-TNF | Membrane potential | Translocation | NF-κB protein | Deactivation | Caspase | Adenosine diphosphate | Inflammation | Tumor cell lines | Tumor necrosis factor-α | Nuclear transport | 1-Phosphatidylinositol 3-kinase | Cytochrome c | Polymerase | Caspase-8 | FasL protein | Caspase-9
Journal Article
Molecules, ISSN 1420-3049, 01/2017, Volume 22, Issue 1, pp. 124 - 124
...) on olaquindox-induced DNA damage and cell cycle arrest in HepG2 cells. The results showed that olaquindox could induce reactive oxygen species (ROS... 
JNK/p38 pathways | GADD45a | Olaquindox | DNA damage | S-phase arrest | QUINOXALINE 1,4-DIOXIDES | OXIDATIVE STRESS | GENOTOXIC STRESS | CANCER CELLS | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | olaquindox | G1 ARREST | CHEMISTRY, MULTIDISCIPLINARY | IN-VITRO | HEPG2 CELLS | MITOCHONDRIAL DYSFUNCTION | HUMAN HEPATOCELLULAR-CARCINOMA | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Reactive Oxygen Species - agonists | Quinoxalines - pharmacology | MAP Kinase Kinase 4 - metabolism | Animal Feed - analysis | Cell Cycle Proteins - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | DNA Fragmentation - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Nuclear Proteins - genetics | Cell Survival - drug effects | Nuclear Proteins - agonists | Signal Transduction | Cell Cycle Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - genetics | Nuclear Proteins - metabolism | Imidazoles - pharmacology | Cell Cycle Proteins - agonists | Comet Assay | Anthracenes - pharmacology | Food Additives - pharmacology | Hep G2 Cells | MAP Kinase Kinase 4 - genetics | Anti-Bacterial Agents - pharmacology | Cell Proliferation - drug effects | Pyridines - pharmacology | S Phase - drug effects | Phosphorylation | Toxicity | Genotoxicity | JNK protein | Activation | Kinases | Gene expression | Signal transduction | Pathways | Cell cycle | Damage | Deoxyribonucleic acid--DNA
Journal Article
Cell stress & chaperones, ISSN 1355-8145, 7/2014, Volume 19, Issue 4, pp. 541 - 548
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 04/2013, Volume 268, Issue 2, pp. 106 - 112
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 150, Issue 1, pp. 194 - 205
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 01/2014, Volume 443, Issue 4, pp. 1286 - 1290
....•The oxidized Hep G2 cells were subjected to subcellular fractionation.•The obtained results suggest that the ATM activation occurs in mitochondria... 
Peroxisome | Oxidative stress | Mitochondria | ATM | Hydrogen peroxide | PHOSPHORYLATION |