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Nature Medicine, ISSN 1078-8956, 05/2011, Volume 17, Issue 5, pp. 589 - 595
Hepatitis C virus (HCV) is a major cause of liver disease, but therapeutic options are limited and there are no prevention strategies. Viral entry is the first... 
MEDICINE, RESEARCH & EXPERIMENTAL | B TYPE-I | NEUTRALIZING ANTIBODIES | CD81 | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEPATOMA-CELLS | FACTOR RECEPTOR | CELL BIOLOGY | CELL LUNG-CANCER | EPIDERMAL-GROWTH-FACTOR | HUMAN LIVER | INFECTION | TRANSFORMING-GROWTH | Erlotinib Hydrochloride | RNA, Small Interfering - genetics | Receptor, Epidermal Growth Factor - genetics | Hepatitis C - therapy | Humans | Virus Internalization - drug effects | RNA Interference | Membrane Proteins - physiology | Base Sequence | Hepacivirus - physiology | Hepatitis C - prevention & control | Receptor, Epidermal Growth Factor - physiology | Hepacivirus - drug effects | Cell Line | Antiviral Agents - pharmacology | Receptor, EphA2 - physiology | Tetraspanin 28 | Claudin-1 | Animals | Hepatitis C - virology | Receptor, EphA2 - genetics | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Antigens, CD - physiology | Hepatitis C - physiopathology | Ligands | Mice | Protein Kinase Inhibitors - pharmacology | Receptor, EphA2 - antagonists & inhibitors | Quinazolines - pharmacology | Host-Pathogen Interactions - physiology | Ephrin A | Antiviral agents | Care and treatment | Patient outcomes | Physiological aspects | Research | Hepatitis C | Hepatitis | Viruses | Drug therapy | Antiviral drugs | Index Medicus | Receptor, EphA2 | Hépatology and Gastroenterology | Antigens, CD81 | Antiviral Agents | Quinazolines | Virus Internalization | Membrane Proteins | Host-Pathogen Interactions | Life Sciences | Human health and pathology | Protein Kinase Inhibitors | RNA, Small Interfering | Hepacivirus | Antigens, CD | Receptor, Epidermal Growth Factor | Liver | HCV escape variants | Cell-cell transmission | genetics | pharmacology | Antiviral | therapy | physiology | drug effects | prevention & control | antagonists & inhibitors | physiopathology | Phosphotyrosine kinase | virology
Journal Article
Gut, ISSN 0017-5749, 04/2014, Volume 63, Issue 4, pp. 665 - 673
Background IL-7 and IL-15 are produced by hepatocytes and are critical for the expansion and function of CD8 T cells. IL-15 needs to be presented by IL-15Rα... 
NATURAL-KILLER | IN-VITRO | EPITHELIAL-CELLS | BIOLOGY | GENE-EXPRESSION | IL-15 | RECEPTOR | INFECTION | IRF-1 | GASTROENTEROLOGY & HEPATOLOGY | CD8(+) T-CELLS | Interleukin-15 - physiology | Hepatitis C, Chronic - metabolism | Humans | Hepatitis C, Chronic - physiopathology | Hepacivirus - genetics | Hepatitis C, Chronic - virology | Interleukin-7 - physiology | Gene Expression Regulation, Viral - genetics | Hepatocytes - metabolism | Interleukin-15 - biosynthesis | Hepacivirus - metabolism | Interferon Regulatory Factor-2 - biosynthesis | Interferon Regulatory Factors - physiology | Hepacivirus - physiology | Real-Time Polymerase Chain Reaction | Hepatocytes - physiology | Interferon Regulatory Factor-1 - biosynthesis | Gene Expression Regulation, Viral - physiology | Interleukin-15 Receptor alpha Subunit - physiology | Interferon Regulatory Factor-1 - physiology | Genotype | Interferon Regulatory Factor-2 - physiology | Blotting, Western | CD8-Positive T-Lymphocytes - physiology | Interleukin-7 - biosynthesis | Hepatocytes - virology | Interleukin-15 Receptor alpha Subunit - biosynthesis | Virus Replication - physiology | Influence | Interferon | Research | Gene expression | Hepatitis C | Liver cells | Cell culture | Genotype & phenotype | Hepatitis | Cytokines | Biopsy | Lymphocytes | Liver | Homeostasis | Tumor necrosis factor-TNF | Infections | Viral infections | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2011, Volume 108, Issue 35, pp. 14590 - 14595
Journal Article
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 56, Issue 4, pp. 803 - 809
Background & Aims Hepatitis C virus (HCV) causes progressive liver disease and is a major risk factor for the development of hepatocellular carcinoma (HCC).... 
Gastroenterology and Hepatology | Hepatitis C | Hypoxia | Invasion | ANGIOGENESIS | HEPATOCELLULAR-CARCINOMA | HYPOXIA-INDUCIBLE-FACTOR-1-ALPHA | MESENCHYMAL TRANSITIONS | GROWTH | INFECTION | RECURRENCE | GASTROENTEROLOGY & HEPATOLOGY | CELL ENTRY | EXPRESSION | Carcinoma, Hepatocellular - physiopathology | Humans | Transforming Growth Factor beta - physiology | Glycoproteins - physiology | Cell Movement - physiology | Disease Progression | Tight Junctions - physiology | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - physiopathology | Cell Line, Tumor | Cell Polarity - physiology | Hepacivirus - physiology | Hepatitis C - physiopathology | Liver Neoplasms - pathology | Hepatitis C - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - physiology | Vascular Endothelial Growth Factor A - physiology | Virus Replication - physiology | Index Medicus | TGFβ, transforming growth factor-beta | PHH, primary human hepatocytes | VEGF, vascular endothelial growth factor | EMT, epithelial to mesenchymal transition | TNFα, tumor necrosis factor alpha | BC, bile canaliculi | JFH-1, Japanese fulminant hepatitis-1 | MRP-2, multidrug resistant protein-2 | HIF-1α, hypoxia inducible factor 1 alpha | HCVcc, hepatitis C virus cell culture | HCC, hepatocellular carcinoma | SR-BI, scavenger receptor class B member 1 | CMFDA, 5-chloromethylfluorescein diacetate | VSV-G, vesicular stomatitis virus glycoprotein
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2009, Volume 50, Issue 5, pp. 872 - 882
Background/Aims The hepatitis C virus (HCV) structural core and non-structural NS5A proteins induce in liver cells a series of intracellular events, including... 
Gastroenterology and Hepatology | Calcium signalling | NS5A protein | Actin reorganization | Core protein | Hepatitis C virus | Quercetin | PANCREATIC ACINAR-CELLS | RAT HEPATOCYTES | TYROSINE PHOSPHORYLATION | HUMAN PLATELETS | NITRIC-OXIDE PRODUCTION | HYDROGEN-PEROXIDE | ENDOPLASMIC-RETICULUM | ANTIOXIDANT THERAPY | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | CA2+ OSCILLATIONS | Reactive Oxygen Species - metabolism | Hepatitis C - drug therapy | Hepatitis C - metabolism | Calcium - metabolism | Calcium Signaling - physiology | Humans | Oxidative Stress - physiology | Actins - metabolism | Viral Core Proteins - physiology | Hepatocytes - cytology | Hepacivirus - physiology | Homeostasis - drug effects | Cell Survival - physiology | Hepatocytes - drug effects | Hepatocytes - physiology | Cell Line | Virus Replication - drug effects | Cytokines - metabolism | Reactive Nitrogen Species - metabolism | Antioxidants - pharmacology | Viral Nonstructural Proteins - physiology | Quercetin - pharmacology | Calcium Signaling - drug effects | Cysteine Proteinase Inhibitors - pharmacology | Homeostasis - physiology | Calcium-Binding Proteins - pharmacology | Oxidative Stress - drug effects | Virus Replication - physiology | Synthesis | Actin | Physiological aspects | Lipids | Hepatitis C | Muscle proteins | Adenosine triphosphatase | Index Medicus
Journal Article
Hepatology, ISSN 0270-9139, 01/2008, Volume 47, Issue 1, pp. 17 - 24
Hepatitis C virus (HCV) infection of Huh‐7.5 hepatoma cells results in focal areas of infection where transmission is potentiated by cell‐cell contact. To... 
IN-VITRO | ENTRY | ASSAY | RNA REPLICATION | HUMAN CYTOMEGALOVIRUS | TISSUE | INFECTION | SPREAD | GASTROENTEROLOGY & HEPATOLOGY | CULTURE | EXPRESSION | Antibodies, Viral - physiology | Hepacivirus - immunology | Membrane Proteins - physiology | Humans | Antigens, CD - physiology | Hepacivirus - physiology | Tetraspanin 28 | HeLa Cells | Claudin-1 | Receptors, Virus - physiology | Index Medicus
Journal Article