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Journal Article
Drugs, ISSN 0012-6667, 2/2019, Volume 79, Issue S1, pp. 17 - 21
Neuropathology of hepatic encephalopathy (HE) in cirrhosis is primarily astroglial in nature characterized by Alzheimer type 2 astrocytosis together with... 
Pharmacotherapy | Internal Medicine | Medicine & Public Health | Pharmacology/Toxicology | PATHOGENESIS | AMMONIA | HYPERAMMONEMIA | LIVER | GABA | RATS | PHARMACOLOGY & PHARMACY | NECROSIS-FACTOR-ALPHA | TOXICOLOGY | BINDING-SITES | BRAIN | Liver Cirrhosis - drug therapy | Hepatic Encephalopathy - metabolism | Neurotransmitter Agents - antagonists & inhibitors | Ammonia - metabolism | Basal Ganglia - physiopathology | gamma-Aminobutyric Acid - metabolism | Humans | Hepatic Encephalopathy - physiopathology | Cerebellum - physiopathology | Hepatic Encephalopathy - drug therapy | Manganese - metabolism | Thalamus - physiopathology | Liver Cirrhosis - metabolism | Liver Cirrhosis - physiopathology | Inflammation - physiopathology | Cerebellum | Neuroimaging | Metabolic rate | Coma | Brain | Neurosciences | Basal ganglia | Dehydrogenases | Neuropathology | Pathogenesis | Liver | Systematic review | Antagonists | Activation | Reduction | γ-Aminobutyric acid | Encephalopathy | Hepatology | Thalamus | Movement disorders | Enzymes | Cortex (cingulate) | Medical imaging | Deactivation | Central nervous system diseases | Inflammation | Metabolism | Ganglia | Microglia | Neurology | Ammonia | Cirrhosis | Neurotransmitters | Gliosis | Medical prognosis | TNF inhibitors | Binding sites | Hepatic encephalopathy | Manganese | Review
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