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Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2012, Volume 143, Issue 3, pp. 765 - 776.e3
... IL-17RA −/− mice and of bone marrow chimeric mice devoid of IL-17 signaling in immune and Kupffer cells ( IL-17RA... 
Gastroenterology and Hepatology | Immune Response | Myofibroblast | Bone Marrow-Derived Macrophages | Mouse Model | RAT-LIVER | TARGET | IL-17 RECEPTOR | HEPATOCYTES | FAMILY-MEMBERS | STAT3 | TRANSDUCTION | PATHWAY | DISEASE | GENE-EXPRESSION | GASTROENTEROLOGY & HEPATOLOGY | Kupffer Cells - pathology | Liver - pathology | Carbon Tetrachloride | Humans | Transforming Growth Factor beta1 - metabolism | Hepatic Stellate Cells - metabolism | Receptors, Interleukin-17 - genetics | Interleukin-23 - genetics | Receptors, Interleukin-17 - deficiency | Interleukins - genetics | Liver Cirrhosis, Experimental - prevention & control | Liver - immunology | Time Factors | Bone Marrow Transplantation | Liver Cirrhosis, Alcoholic - immunology | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | Interleukin-17 - administration & dosage | Liver Cirrhosis, Experimental - immunology | Signal Transduction | Liver Cirrhosis, Alcoholic - pathology | Liver - metabolism | Interleukin-17 - genetics | Genotype | Bile Ducts - surgery | Disease Progression | Mice, Knockout | Interleukin-17 - metabolism | Phenotype | Interleukins - deficiency | Mice | Liver Cirrhosis, Experimental - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation Mediators - administration & dosage | Liver Cirrhosis, Experimental - etiology | Interleukin-23 - deficiency | Ligation | Hepatic Stellate Cells - immunology | Inflammation Mediators - metabolism | STAT3 Transcription Factor - deficiency | Liver Cirrhosis, Experimental - pathology | Kupffer Cells - metabolism | Hepatic Stellate Cells - pathology | Cell Line | Interleukin-1 - metabolism | Collagen Type I - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Interleukin-17 - deficiency | Liver Cirrhosis, Experimental - genetics | Animals | Interleukins - administration & dosage | Kupffer Cells - immunology | Medical colleges | Liver diseases | Liver | Carbon tetrachloride | Transforming growth factors | Muscle proteins | Macrophages | Endothelium | Messenger RNA | Interleukins | Actin | Analysis | Collagen | Fibrosis | Liver cirrhosis | Index Medicus | Abridged Index Medicus
Journal Article
World journal of gastroenterology : WJG, ISSN 1007-9327, 2015, Volume 21, Issue 3, pp. 878 - 887
AIM:To determine the role of Notch1 and Hes1 in regulating the activation of hepatic stellate cells(HSCs... 
cells;Hepat | stellate | Hes1;Notch1;TGF-β/BMP;Hepatic | Hepatic fibrosis | TGF-β/BMP | Hepatic stellate cells | Hes1 | Notch1 | ANGIOGENESIS | INTEGRATION | TGF-beta/BMP | GASTROENTEROLOGY & HEPATOLOGY | Receptors, Transforming Growth Factor beta - genetics | Transcription Factor HES-1 | Homeodomain Proteins - metabolism | Actins - metabolism | Hepatic Stellate Cells - metabolism | RNA, Messenger - metabolism | Actins - genetics | Myofibroblasts - metabolism | Bone Morphogenetic Proteins - metabolism | Collagen Type I - genetics | Transfection | Basic Helix-Loop-Helix Transcription Factors - metabolism | Time Factors | Liver Cirrhosis - metabolism | Liver Cirrhosis - genetics | Genes, Reporter | Hepatic Stellate Cells - pathology | Protein-Serine-Threonine Kinases - metabolism | Biomarkers - metabolism | Myofibroblasts - pathology | Basic Helix-Loop-Helix Transcription Factors - genetics | Collagen Type I - metabolism | Signal Transduction | Cells, Cultured | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Bone Morphogenetic Protein Receptors, Type I - genetics | Rats | Receptor, Notch1 - metabolism | Bone Morphogenetic Protein Receptors, Type I - metabolism | Homeodomain Proteins - genetics | Animals | Receptors, Transforming Growth Factor beta - metabolism | Liver Cirrhosis - pathology | Receptor, Notch1 - genetics | Transforming Growth Factor beta - metabolism | BMP | Basic Study | TGF-β
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2013, Volume 110, Issue 6, pp. 2324 - 2329
.... In liver fibrogenesis, hepatic stellate cells (HSCs) are thought to transdifferentiate into myofibroblasts, but the origins of both HSCs and myofibroblasts remain elusive... 
Phenotypes | Hepatocytes | Epithelial cells | Liver | Fibrosis | Cell lines | Antibodies | Hepatic stellate cells | Myofibroblasts | Liver cells | Epithelial-mesenchymal transition | Alpha-smooth muscle actin | Glisson's capsule | Podoplanin | alpha-smooth muscle actin | PATHWAYS | ACID | RAT | MULTIDISCIPLINARY SCIENCES | MOUSE | RECEPTOR | POPULATIONS | podoplanin | SMOOTH-MUSCLE | fibrosis | LINEAGE | Liver - pathology | WT1 Proteins - metabolism | Membrane Glycoproteins - metabolism | Hepatic Stellate Cells - metabolism | RNA, Messenger - metabolism | Liver - injuries | Myofibroblasts - metabolism | Biliary Tract - metabolism | Cell Transdifferentiation - drug effects | Liver Cirrhosis - metabolism | Chemical and Drug Induced Liver Injury - pathology | WT1 Proteins - genetics | Hepatic Stellate Cells - pathology | Liver Cirrhosis - etiology | Myofibroblasts - pathology | Epithelium - pathology | Gene Expression | Liver Regeneration | Epithelium - metabolism | Signal Transduction | Liver - metabolism | RNA, Messenger - genetics | Cells, Cultured | Mice, Transgenic | Chemical and Drug Induced Liver Injury - genetics | Carbon Tetrachloride - toxicity | Biliary Tract - pathology | Nerve Tissue Proteins - metabolism | Cell Lineage | Transforming Growth Factor beta - pharmacology | Animals | Chemical and Drug Induced Liver Injury - metabolism | Liver Cirrhosis - pathology | Mesoderm - metabolism | Mice | Mesoderm - pathology | Transforming Growth Factor beta - metabolism | Biological Sciences
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 9, p. e24568
Background: In chronic liver disease, hepatic stellate cells (HSC) transdifferentiate into myofibroblasts, promoting extracellular matrix (ECM... 
FAT-STORING CELLS | RAT-LIVER | FIBROSIS | FACTOR SUPPRESSES | ACTIVATION | TGF-BETA | MULTIDISCIPLINARY SCIENCES | TRANSFORMING GROWTH-FACTOR-BETA-1 | BETA GENE-EXPRESSION | MICRORNAS | TRANSDIFFERENTIATION | Collagen Type IV - metabolism | Proto-Oncogene Proteins c-met - metabolism | Computational Biology - methods | Collagen Type I - metabolism | Extracellular Matrix - metabolism | Rats | Male | MicroRNAs - metabolism | RNA, Messenger - metabolism | Hepatic Stellate Cells - cytology | Hepatocyte Growth Factor - metabolism | Animals | Cell Differentiation | Fibrosis - pathology | 3' Untranslated Regions | Transforming Growth Factor beta - metabolism | Bile Ducts - pathology | Fibroblasts - metabolism | Liver diseases | MicroRNA | Collagen | Analysis | Liver | Bone morphogenetic proteins | Transforming growth factors | Cell culture | Collagen (type I) | Target recognition | Transforming growth factor-a | Immunoblotting | Stimulation | mRNA | Kinases | Western blotting | c-Met protein | Proteins | Signal transduction | Hepatitis | Reduction | Cell growth | Synthesis | Rodents | Hepatology | Gastroenterology | Fibroblasts | Extracellular matrix | miRNA | Bioinformatics | Growth factors | Stellate cells | Internal medicine | Hepatocyte growth factor | Gene expression | Real time | Pathology | Hepatocytes | 3' Untranslated regions | MicroRNAs | Fibrosis
Journal Article
Gut, ISSN 0017-5749, 02/2012, Volume 61, Issue 2, pp. 172 - 178
  In 2006, it was reported that BM is a source of myofibroblast-like cells in fibrotic liver tissue, but the involvement of these cells in the progression of liver fibrosis remains questionable... 
FAT-STORING CELLS | PROGENITOR CELLS | CANCER CELLS | DUCTAL ADENOCARCINOMA | ALCOHOLIC CHRONIC-PANCREATITIS | MOUSE | RATS | LIVER DEVELOPMENT | IDENTIFICATION | GASTROENTEROLOGY & HEPATOLOGY | INDUCE FIBROSIS | Liver Regeneration | Pancreatic Diseases - metabolism | Pancreatic Neoplasms - metabolism | Signal Transduction | Humans | Pancreas - cytology | Pancreatic Neoplasms - pathology | Pancreas - pathology | Pancreatic Stellate Cells - physiology | Pancreatic Diseases - pathology | Pancreatitis, Chronic - pathology | Regeneration | Pancreatic Stellate Cells - cytology | Fibrosis | Liver Cirrhosis - metabolism | Liver Cirrhosis - pathology | Pancreas - physiology | Pancreatic Stellate Cells - pathology | Pancreatitis, Chronic - metabolism | Studies | Rodents | Morphology | Pancreatic cancer | Fibroblasts | Research | Gene expression | molecular biology | chronic pancreatitis | experimental pancreatitis | stellate cells | pancreatic cancer | cancer genetics | Helicobacter pylori | pancreas | cell migration | hepatic surgery | pancreatic surgery | pancreatic physiology | hepatic stellate cell | signalling | Leading | Abdominal surgery | alcohol | pancreatic stellate cells | stem cells | liver | signal transduction | pancreatic pathology | adenocarcinoma | endoscopy | pancreatic enzymes | consensus | pancreatic function | cell biology | pancreatic fibrosis | marker | acute pancreatitis | cancer | fibrosis | pancreatitis | adjuvant treatment | pancreatic disease | hepatic fibrosis | 1506 | extracellular matrix | gene expression
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2012, Volume 142, Issue 4, pp. 938 - 946
Background & Aims The pathogenesis of liver fibrosis involves activation of hepatic stellate cells, which is associated with depletion of intracellular lipid droplets... 
Gastroenterology and Hepatology | Inflammation | Myofibroblasts | Energy Depletion | Mouse Model | RAT-LIVER | FIBROSIS | PATHWAY | DISEASE | MECHANISMS | RETINOL | GASTROENTEROLOGY & HEPATOLOGY | Liver - pathology | Kidney - pathology | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Carbon Tetrachloride | Humans | Hepatic Stellate Cells - metabolism | Autophagy - drug effects | Idiopathic Pulmonary Fibrosis - metabolism | Kidney - metabolism | Epoxy Compounds - pharmacology | Liver - drug effects | RNA Interference | Adenosine Triphosphate - metabolism | Liver Cirrhosis, Experimental - pathology | Lung - metabolism | Autophagy - genetics | Microtubule-Associated Proteins - deficiency | Hepatic Stellate Cells - pathology | Fibroblasts - metabolism | Hepatic Stellate Cells - drug effects | Cell Line | Lung - pathology | Oleic Acid - metabolism | Adenine - analogs & derivatives | Liver - metabolism | Mice, Inbred C57BL | Adenine - pharmacology | Liver Cirrhosis, Experimental - genetics | Fibroblasts - pathology | Mice, Knockout | Thioacetamide | Autophagy-Related Protein 7 | Animals | Autophagy-Related Protein 5 | Energy Metabolism | Fibroblasts - drug effects | Liver Cirrhosis, Experimental - chemically induced | Lipid Metabolism - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Mice | Liver Cirrhosis, Experimental - metabolism | Phosphates | Medical colleges | Neurosciences | Platelet-derived growth factor | Liver diseases | Albumin | Lipids | Muscle proteins | Fatty acids | Cells | Monosaccharides | Unsaturated fatty acids | Monounsaturated fatty acids | Actin | Analysis | Intermediate filament proteins | Sugars
Journal Article
Journal of food science, ISSN 0022-1147, 01/2016, Volume 81, Issue 1, pp. H240 - H245
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2012, Volume 143, Issue 4, pp. 1073 - 1083.e22
Journal Article
American journal of physiology: Gastrointestinal and liver physiology, ISSN 1522-1547, 2011, Volume 301, Issue 1, pp. G110 - G118
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 5, pp. 1319 - 1329.e11
Background & Aims The pathogenesis of cirrhosis, a disabling outcome of defective liver repair, involves deregulated accumulation of myofibroblasts derived from quiescent hepatic stellate cells (HSCs... 
Gastroenterology and Hepatology | Gene Regulation | Signal Transduction | Liver Disease | Mouse Model | PARTIAL-HEPATECTOMY | ACTIVATION | TISSUE-REPAIR | SIGNALS | PATHWAY | LIVER-REGENERATION | IN-VIVO | MICE | GASTROENTEROLOGY & HEPATOLOGY | TUMOR-GROWTH | UP-REGULATION | Carbon Tetrachloride | Humans | Actins - metabolism | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Myofibroblasts - enzymology | Male | Cell Transdifferentiation - genetics | Gene Expression Profiling | RNA, Messenger - metabolism | Hepatic Stellate Cells - cytology | Hypoxia-Inducible Factor 1, alpha Subunit | Liver Cirrhosis - chemically induced | Actins - genetics | Glycolysis - genetics | Myofibroblasts - metabolism | Mitochondria | Ligation | Hedgehog Proteins - genetics | Time Factors | Liver Cirrhosis - metabolism | Liver Cirrhosis - genetics | Pyruvate Kinase - metabolism | Lactic Acid - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Hepatic Stellate Cells - enzymology | Rats | Signal Transduction - genetics | Animals | Bile Ducts | Mice | Pyruvate Kinase - genetics | Liver diseases | Gliomas | Actin | Analysis | Genes | Physiological aspects | Fluorescence | Drug resistance | Muscle proteins | Fructose | Lactates | Glucose metabolism | Liver | Cellular signal transduction | Liver cirrhosis | mouse model | liver disease | signal transduction | gene regulation
Journal Article