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Gut, ISSN 0017-5749, 02/2012, Volume 61, Issue 2, pp. 172 - 178
  In 2006, it was reported that BM is a source of myofibroblast-like cells in fibrotic liver tissue, but the involvement of these cells in the progression of... 
FAT-STORING CELLS | PROGENITOR CELLS | CANCER CELLS | DUCTAL ADENOCARCINOMA | ALCOHOLIC CHRONIC-PANCREATITIS | MOUSE | RATS | LIVER DEVELOPMENT | IDENTIFICATION | GASTROENTEROLOGY & HEPATOLOGY | INDUCE FIBROSIS | Liver Regeneration | Pancreatic Diseases - metabolism | Pancreatic Neoplasms - metabolism | Signal Transduction | Humans | Pancreas - cytology | Pancreatic Neoplasms - pathology | Pancreas - pathology | Pancreatic Stellate Cells - physiology | Pancreatic Diseases - pathology | Pancreatitis, Chronic - pathology | Regeneration | Pancreatic Stellate Cells - cytology | Fibrosis | Liver Cirrhosis - metabolism | Liver Cirrhosis - pathology | Pancreas - physiology | Pancreatic Stellate Cells - pathology | Pancreatitis, Chronic - metabolism | Studies | Rodents | Morphology | Pancreatic cancer | Fibroblasts | Research | Gene expression | Index Medicus | Abridged Index Medicus | molecular biology | chronic pancreatitis | experimental pancreatitis | stellate cells | pancreatic cancer | cancer genetics | Helicobacter pylori | pancreas | cell migration | hepatic surgery | pancreatic surgery | pancreatic physiology | hepatic stellate cell | signalling | Leading | Abdominal surgery | alcohol | pancreatic stellate cells | stem cells | liver | signal transduction | pancreatic pathology | adenocarcinoma | endoscopy | pancreatic enzymes | consensus | pancreatic function | cell biology | pancreatic fibrosis | marker | acute pancreatitis | cancer | fibrosis | pancreatitis | adjuvant treatment | pancreatic disease | hepatic fibrosis | 1506 | extracellular matrix | gene expression
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 55, Issue 6, pp. 1353 - 1360
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 5, pp. 1319 - 1329.e11
Background & Aims The pathogenesis of cirrhosis, a disabling outcome of defective liver repair, involves deregulated accumulation of myofibroblasts derived... 
Gastroenterology and Hepatology | Gene Regulation | Signal Transduction | Liver Disease | Mouse Model | PARTIAL-HEPATECTOMY | ACTIVATION | TISSUE-REPAIR | SIGNALS | PATHWAY | LIVER-REGENERATION | IN-VIVO | MICE | GASTROENTEROLOGY & HEPATOLOGY | TUMOR-GROWTH | UP-REGULATION | Carbon Tetrachloride | Humans | Actins - metabolism | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Myofibroblasts - enzymology | Male | Cell Transdifferentiation - genetics | Gene Expression Profiling | RNA, Messenger - metabolism | Hepatic Stellate Cells - cytology | Hypoxia-Inducible Factor 1, alpha Subunit | Liver Cirrhosis - chemically induced | Actins - genetics | Glycolysis - genetics | Myofibroblasts - metabolism | Mitochondria | Ligation | Hedgehog Proteins - genetics | Time Factors | Liver Cirrhosis - metabolism | Liver Cirrhosis - genetics | Pyruvate Kinase - metabolism | Lactic Acid - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Hepatic Stellate Cells - enzymology | Rats | Signal Transduction - genetics | Animals | Bile Ducts | Mice | Pyruvate Kinase - genetics | Liver diseases | Gliomas | Actin | Analysis | Genes | Physiological aspects | Fluorescence | Drug resistance | Muscle proteins | Fructose | Lactates | Glucose metabolism | Liver | Cellular signal transduction | Liver cirrhosis | Index Medicus | Abridged Index Medicus | mouse model | liver disease | signal transduction | gene regulation
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 2011, Volume 412, Issue 1, pp. 74 - 79
► Expression of miR-29b was found to be down-regulated during the activation of hepatic stellate cells in primary culture. ► Transfection of a miR-29b... 
Extracellular signal-regulated kinase | c-fos | Focal adhesion kinase | Akt | Liver fibrosis | α-Smooth muscle actin | C-fos | MIGRATION | STIMULATION | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | INVOLVEMENT | I COLLAGEN | PROLIFERATION | BETA | BIOPHYSICS | alpha-Smooth muscle actin | PDGF | Mice, Inbred C57BL | Hepatic Stellate Cells - metabolism | Male | MicroRNAs - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Actins - genetics | RNA, Messenger - biosynthesis | Discoidin Domain Receptors | Animals | Collagen Type I - genetics | Receptor Protein-Tyrosine Kinases - genetics | Liver Cirrhosis - metabolism | Liver Cirrhosis - pathology | Fibronectins - genetics | Mice | MicroRNAs - genetics | Integrin beta1 - genetics | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Receptors, Mitogen - genetics | Fibronectins | Phosphates | Platelet-derived growth factor | Liver diseases | Sulfates | Transforming growth factors | Monosaccharides | Proteins | Messenger RNA | Surface active agents | Actin | Universities and colleges | Sugars | Integrins | 3' Untranslated regions | Index Medicus | FIBROSIS | SULFATES | SODIUM | COLLAGEN | ACTIN | PHOSPHORYLATION | CATTLE | ALBUMINS | CELL PROLIFERATION | 60 APPLIED LIFE SCIENCES | GROWTH FACTORS | INHIBITION | MESSENGER-RNA | LIVER | PHOSPHATES | MICE | GENE REGULATION | RECEPTORS | MORPHOLOGY | MUSCLES
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, pp. e0128745 - e0128745
Journal Article
Hepatology, ISSN 0270-9139, 09/2012, Volume 56, Issue 3, pp. 1150 - 1159
Interleukin (IL)‐22 is known to play a key role in promoting antimicrobial immunity, inflammation, and tissue repair at barrier surfaces by binding to the... 
APOPTOSIS | HEPATOCYTES | GASTROENTEROLOGY & HEPATOLOGY | IL-22 | INFLAMMATION | P53 | Liver Cirrhosis - immunology | Animals | Interleukins - physiology | Cellular Senescence | Hepatic Stellate Cells - physiology | Humans | Mice, Inbred C57BL | Mice | Senescence | Liver | Rodents | Hepatology | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2013, Volume 191, Issue 4, pp. 1835 - 1844
Liver fibrosis is a severe, life-threatening clinical condition resulting from nonresolving hepatitis of different origins. IL-17A is critical in inflammation,... 
NEUTROPHIL INFILTRATION | TGF-BETA | INTERLEUKIN-17 | PULMONARY INFLAMMATION | ALLERGIC-ASTHMA | DISEASE | SMOOTH-MUSCLE-CELLS | IMMUNOLOGY | NF-KAPPA-B | EXPRESSION | T-CELLS | MAP Kinase Signaling System - physiology | Humans | Middle Aged | Interleukin-17 - physiology | Hepatic Stellate Cells - metabolism | Male | Interleukin-17 - pharmacology | Transforming Growth Factor beta - biosynthesis | Hepatectomy | Receptors, Interleukin-17 - deficiency | Hepatitis, Animal - metabolism | Th17 Cells - metabolism | Carcinoma, Hepatocellular - chemistry | Collagen - biosynthesis | Cytokines - genetics | Hepatitis, Animal - pathology | Hemangioma - surgery | Liver Cirrhosis - complications | Interleukin-17 - genetics | Hemangioma - chemistry | Mice, Knockout | Chemical and Drug Induced Liver Injury - metabolism | Liver Cirrhosis - pathology | Th17 Cells - immunology | Mice | Interleukin-17 - analysis | Carbon Tetrachloride Poisoning - complications | Liver Neoplasms - chemistry | Actins - genetics | Liver Cirrhosis - metabolism | Adult | Female | Carcinoma, Hepatocellular - etiology | Collagen - genetics | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - etiology | Hepatic Stellate Cells - pathology | Hepatitis, Animal - chemically induced | Liver Cirrhosis - etiology | Actins - biosynthesis | Gene Expression Regulation - immunology | Neutrophils - physiology | Carcinoma, Hepatocellular - surgery | Liver Neoplasms - surgery | Interleukin-17 - biosynthesis | Recombinant Proteins - pharmacology | Gene Expression Regulation - drug effects | Animals | Hepatitis B, Chronic - complications | MAP Kinase Signaling System - drug effects | Transforming Growth Factor beta - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - analysis | Protein Kinase Inhibitors - pharmacology | Cytokines - biosynthesis | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article