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Gastroenterology, ISSN 0016-5085, 2010, Volume 139, Issue 3, pp. 953 - 964.e4
Background & Aims Hepatitis C virus (HCV) infection is a challenge to prevent and treat because of the rapid development of drug resistance and escape. Viral... 
Gastroenterology and Hepatology | Genetic Barrier | Antiviral | Treatment | Receptor | Host Factor | B TYPE-I | LIVER-TRANSPLANTATION | SOURCE OUTBREAK | ESCAPE | PROTECT | RESPONSES | BROADLY NEUTRALIZING ANTIBODIES | GASTROENTEROLOGY & HEPATOLOGY | BINDING | VIRAL ENTRY | Antibodies, Monoclonal - toxicity | Antibody Specificity | Cricetulus | Antiviral Agents - toxicity | Humans | Hepacivirus - genetics | Hepacivirus - pathogenicity | Antiviral Agents - metabolism | Virus Internalization - drug effects | Dose-Response Relationship, Drug | Hepatitis C - immunology | Hepatitis C - prevention & control | Hepatocytes - drug effects | CHO Cells | Binding, Competitive | Hepacivirus - drug effects | Cell Survival - drug effects | Antiviral Agents - pharmacology | Cricetinae | Antibodies, Monoclonal - pharmacology | Genotype | Membrane Proteins - immunology | Epitopes | Hepatocytes - immunology | Claudin-1 | Hep G2 Cells | Animals | Membrane Proteins - antagonists & inhibitors | Hepatocytes - virology | Antibodies, Monoclonal - metabolism | Binding Sites, Antibody | Prevention | Antiviral agents | Disease transmission | Gastrointestinal agents | Immunoglobulin G | Fluorescence | Monoclonal antibodies | Drug resistance | Hepatitis C | Hepatitis C virus | Health aspects | Infection control | Hépatology and Gastroenterology | Cell Survival | Antiviral Agents | Antibodies, Monoclonal | Virus Internalization | Membrane Proteins | Life Sciences | Human health and pathology | Hepatocytes | Hepacivirus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2010, Volume 107, Issue 19, pp. 8766 - 8771
Hepatitis B virus (HBV)-associated acute liver failure (ALF) is a dramatic clinical syndrome due to a sudden loss of hepatic cells leading to multiorgan... 
T lymphocytes | Hepatocytes | B lymphocytes | Hepatitis antigens | Liver | Kupffer cells | Plasma cells | Antibodies | Hepatitis B virus | Hepatitis B | Fulminant hepatitis B | Phage display fab library | B cell immunity | Gene expression profiling | Pathogenesis | ACTIVATION | PROTEIN | phage display Fab library | FULMINANT VIRAL-HEPATITIS | MEMBRANE | MULTIDISCIPLINARY SCIENCES | NEGATIVE REGULATOR | FAMILY | pathogenesis | fulminant hepatitis B | REJECTION | DISEASE | INFECTION | gene expression profiling | T-CELLS | Immunohistochemistry | Liver - virology | Liver - pathology | Humans | Hepatitis B - virology | Liver Failure, Acute - virology | Complement System Proteins - immunology | Gene Expression Profiling | Hepatitis B - blood | Necrosis - pathology | Necrosis - virology | T-Lymphocytes - virology | Necrosis - immunology | Young Adult | Hepatitis B - immunology | Liver - immunology | Immunoglobulin G - biosynthesis | Liver Failure, Acute - genetics | B-Lymphocytes - virology | Hepatitis B virus - immunology | Disease Progression | Hepatitis B Core Antigens - immunology | Cell Lineage | B-Lymphocytes - immunology | Liver Failure, Acute - immunology | T-Lymphocytes - immunology | Hepatitis B Antibodies - biosynthesis | Cluster Analysis | Immunoglobulin M - biosynthesis | Liver Failure, Acute - blood | Antigens | Care and treatment | Liver failure | Immunoglobulin G | Physiological aspects | Development and progression | Genetic aspects | B cells | Properties | Gene expression | Biological Sciences
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Journal Article