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1. Full Text PNPLA3 I148M polymorphism and progressive liver disease
by Dongiovanni, Paola and Donati, Benedetta and Fares, Roberta and Lombardi, Rosa and Mancina, Rosellina Margherita and Romeo, Stefano and Valenti, Luca and Institute of Medicine, Department of Molecular and Clinical Medicine and Wallenberg Laboratory and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy and Sahlgrenska akademin and Institutionen för medicin, avdelningen för molekylär och klinisk medicin and Wallenberglaboratoriet
World journal of gastroenterology : WJG, ISSN 1007-9327, 11/2013, Volume 19, Issue 41, pp. 6969 - 6978
Liver disease | Patatin-like phospholipase domain-containing 3 | Chronic hepatitis C virus hepatitis | Genetics | Hepatocellular carcinoma | Nonalcoholic fatty liver disease | Single nucleotide polymorphism | Fibrogenesis | Alcoholic liver disease | Steatosis | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Cholangitis, Sclerosing - enzymology | Hepatitis C, Chronic - pathology | Fatty Liver - pathology | Humans | Cholangitis, Sclerosing - genetics | Fatty Liver - complications | Hepatitis B, Chronic - enzymology | Hepatitis C, Chronic - complications | Liver Cirrhosis - enzymology | Hemochromatosis - pathology | Fatty Liver, Alcoholic - genetics | Hepatitis B, Chronic - genetics | Liver Cirrhosis, Alcoholic - enzymology | Hepatitis C, Chronic - enzymology | Fatty Liver - enzymology | Non-alcoholic Fatty Liver Disease | Liver Neoplasms - pathology | Fatty Liver, Alcoholic - pathology | Liver Neoplasms - enzymology | Fatty Liver, Alcoholic - enzymology | Hepatitis B, Chronic - pathology | Liver Cirrhosis - genetics | Hemochromatosis - genetics | Fatty Liver - genetics | Genetic Predisposition to Disease | Liver Neoplasms - genetics | Liver Cirrhosis, Alcoholic - pathology | Membrane Proteins - genetics | Risk Factors | Hemochromatosis - enzymology | Disease Progression | Polymorphism, Genetic | Cholangitis, Sclerosing - pathology | Fatty Liver, Alcoholic - complications | Liver Cirrhosis, Alcoholic - genetics | Phenotype | Liver Cirrhosis - pathology | Carcinoma, Hepatocellular | Hepatitis C, Chronic - genetics | Lipase - genetics | Index Medicus | Review | Gastroenterologi | CHRONIC HEPATITIS-C | NONALCOHOLIC STEATOHEPATITIS | Hepatocellular | GREATER-THAN-G | B-VIRUS INFECTION | HEPATOCELLULAR-CARCINOMA | INSULIN-RESISTANCE | FATTY LIVER | DOMAIN-CONTAINING 3 | GENETIC VARIANT | Gastroenterology and Hepatology | GENOME-WIDE ASSOCIATION
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2. Full Text Inhibition of Hepatitis B Virus Replication by APOBEC3G
by Priscilla Turelli and Bastien Mangeat and Stephanie Jost and Sandrine Vianin and Didier Trono
Science (American Association for the Advancement of Science), ISSN 0036-8075, 3/2004, Volume 303, Issue 5665, pp. 1829 - 1829
Polymerase chain reaction | RNA | Plasmids | DNA | Cell lines | Viral DNA | Viruses | Cellular immunity | Hepatitis B virus | HIV 1 | Brevia | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Replicative cycle, interference, host-virus relations, pathogenicity, miscellaneous strains | Biological and medical sciences | Microbiology | Virology | Cytidine Deaminase | Liver - enzymology | Humans | Repressor Proteins | DNA, Viral - metabolism | Hepatitis B - virology | Hepatitis B virus - physiology | Nucleoside Deaminases | Hepatitis B - enzymology | APOBEC-3G Deaminase | Proteins - metabolism | Transfection | Virus Replication | Hepatitis B virus - genetics | Polymerase Chain Reaction | Cell Line, Tumor | RNA, Viral - metabolism | Viral antibodies | Physiological aspects | Antibodies | Chemical properties | Hepatitis B | Hepatitis | Immunology | Immune system | Index Medicus
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3. Full Text MicroRNA‐1271 functions as a potential tumor suppressor in hepatitis B virus–associated hepatocellular carcinoma through the AMPK signaling pathway by binding to CCNA1
by Chen, Yang and Zhao, Zhen‐Xian and Huang, Fei and Yuan, Xiao‐Wei and Deng, Liang and Tang, Di
Journal of cellular physiology, ISSN 0021-9541, 04/2019, Volume 234, Issue 4, pp. 3555 - 3569
CCNA1 | invasion | proliferation | hepatitis B virus–associated hepatocellular carcinoma | AMPK signaling pathway | microRNA‐1271 | migration | apoptosis | microRNA-1271 | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Hepatitis B virus - growth & development | AMP-Activated Protein Kinases - metabolism | Cell Proliferation | DNA, Viral - biosynthesis | Humans | Middle Aged | Hepatitis B - virology | Male | MicroRNAs - metabolism | Cyclin A1 - metabolism | Hepatitis B - complications | Carcinoma, Hepatocellular - genetics | Female | Liver Neoplasms - enzymology | Liver Neoplasms - virology | Liver Neoplasms - genetics | Signal Transduction | Neoplasm Invasiveness | Cyclin A1 - genetics | DNA Replication | Carcinoma, Hepatocellular - virology | Carcinoma, Hepatocellular - enzymology | Disease Progression | Hep G2 Cells | Host-Pathogen Interactions | Virus Replication | Hepatitis B virus - genetics | MicroRNAs - genetics | DNA, Viral - genetics | Apoptosis | Cell Movement | Cell proliferation | Flow cytometry | Viruses | Hepatocellular carcinoma | Kinases | Tissues | Assaying | Proteins | Hepatitis | Liver cancer | Signal transduction | Cell growth | Reporter gene | miRNA | Hepatitis B virus | Bioinformatics | Deoxyribonucleic acid--DNA | Binding | Adenosine monophosphate | AMP | siRNA | Ribonucleic acid--RNA | DNA biosynthesis | Signaling | Protein kinase | Adenosine kinase | MicroRNAs | Tumor suppressor genes | Replication | Cell migration | Scratch tests | Hepatitis B | Tumors | Index Medicus
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4. Full Text Incidence and Risk Factors for Chronic Elevation of Alanine Aminotransferase Levels in HIV-Infected Persons without Hepatitis B or C Virus Co-Infection
by Helen Kovari and Bruno Ledergerber and Manuel Battegay and Andri Rauch and Bernard Hirschel and Alain Kenfak Foguena and Pietro Vernazza and Enos Bernasconi and Nicolas J. Mueller and Rainer Weber and Swiss HIV Cohort Study
Clinical infectious diseases, ISSN 1058-4838, 2/2010, Volume 50, Issue 4, pp. 502 - 511
Antiretrovirals | Body mass index | Coinfection | HIV | Liver | Mortality | Predisposing factors | Infections | Hepatitis B virus | ARTICLES AND COMMENTARIES | Alcohols | Infectious Diseases | Immunology | Life Sciences & Biomedicine | Microbiology | Science & Technology | Immunopathology | Viral hepatitis | Infectious diseases | Viral diseases | Viral diseases of the lymphoid tissue and the blood. Aids | Biological and medical sciences | Medical sciences | Human viral diseases | Immunodeficiencies. Immunoglobulinopathies | Immunodeficiencies | Multivariate Analysis | Liver - enzymology | Prospective Studies | Humans | Middle Aged | Hepatitis B - virology | Male | Alanine Transaminase - blood | HIV Infections - enzymology | Viral Load | Sensitivity and Specificity | Adult | Female | Alcohol Drinking - metabolism | Anti-Retroviral Agents - therapeutic use | HIV Infections - virology | Risk Factors | Anti-Retroviral Agents - adverse effects | Hepatitis C - enzymology | Hepatitis B - enzymology | Hepatitis C - virology | HIV Infections - drug therapy | Poisson Distribution | Alanine Transaminase - metabolism | Longitudinal Studies | Measurement | Comorbidity | Physiological aspects | Alanine aminotransferase | Research | HIV infection | Hepatitis B | Hepatitis | Medical diagnosis | Human immunodeficiency virus--HIV | Hepatology | Risk factors | Index Medicus
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5. Full Text C‐terminal truncated hepatitis B virus x protein is associated with metastasis and enhances invasiveness by c‐jun/matrix metalloproteinase protein 10 activation in hepatocellular carcinoma
by Sze, Karen M.F and Chu, Glanice K.Y and Lee, Joyce M.F and Ng, Irene O.L
Hepatology (Baltimore, Md.), ISSN 0270-9139, 01/2013, Volume 57, Issue 1, pp. 131 - 139
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Liver Neoplasms - virology | Gene Expression | Liver - pathology | Neoplasm Invasiveness | Humans | Middle Aged | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Carcinoma, Hepatocellular - virology | Carcinoma, Hepatocellular - enzymology | Hep G2 Cells | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Adult | Female | Liver Neoplasms - pathology | Matrix Metalloproteinase 10 - metabolism | Trans-Activators - metabolism | Aged | Enzyme Activation | Liver Neoplasms - enzymology | Proteins | Hepatitis | Liver cancer | Hepatology | Index Medicus
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6. Full Text Polo‐like‐kinase 1 is a proviral host factor for hepatitis B virus replication
by Diab, Ahmed and Foca, Adrien and Fusil, Floriane and Lahlali, Thomas and Jalaguier, Pascal and Amirache, Fouzia and N'Guyen, Lia and Isorce, Nathalie and Cosset, François‐Loïc and Zoulim, Fabien and Andrisani, Ourania and Durantel, David
Hepatology (Baltimore, Md.), ISSN 0270-9139, 12/2017, Volume 66, Issue 6, pp. 1750 - 1765
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Proto-Oncogene Proteins - metabolism | Amino Acid Sequence | Proto-Oncogene Proteins - antagonists & inhibitors | Phosphorylation | Pteridines - pharmacology | Pteridines - therapeutic use | Humans | Cell Cycle Proteins - metabolism | Hepatitis B virus - physiology | Cell Cycle Proteins - antagonists & inhibitors | Host-Pathogen Interactions | Animals | Virus Replication | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Viral Core Proteins - metabolism | Mice | Enzyme Activation | Primary Cell Culture | Drug Evaluation, Preclinical | Protein-Serine-Threonine Kinases - metabolism | Hepatocytes - enzymology | Animal models | Threonine | Polo-like kinase 1 | Chronic infection | Hepatocellular carcinoma | Infections | Biosynthesis | siRNA | Kinases | Carcinogenesis | DNA biosynthesis | Hepatitis | RAG2 protein | Hepatocytes | Mutagenesis | Core protein | Site-directed mutagenesis | Replication | Interferon | Hepatitis B | Index Medicus | Life Sciences | Cancer | capsid-associated reverse transcription | core protein | BI-2536 | polo-like-kinase 1 | Hepatitis B virus
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7. Full Text Suppression of hepatitis B virus DNA accumulation in chronically infected cells using a bacterial CRISPR/Cas RNA-guided DNA endonuclease
by Kennedy, Edward M and Bassit, Leda C and Mueller, Henrik and Kornepati, Anand V.R and Bogerd, Hal P and Nie, Ting and Chatterjee, Payel and Javanbakht, Hassan and Schinazi, Raymond F and Cullen, Bryan R
Virology (New York, N.Y.), ISSN 0042-6822, 2014, Volume 476, pp. 196 - 205
Infectious Disease | Antiviral | DNA editing | Lentiviral vector | cccDNA | HBV | CRISPR/Cas | CccDNA | Life Sciences & Biomedicine | Science & Technology | Virology | Gene Targeting - methods | Hepatitis B - therapy | Down-Regulation | Humans | Bacterial Proteins - genetics | DNA, Viral - metabolism | Hepatitis B - virology | Hepatitis B virus - physiology | Streptococcus pyogenes - genetics | Deoxyribonuclease I - metabolism | Virus Replication | CRISPR-Associated Proteins - metabolism | Streptococcus pyogenes - enzymology | CRISPR-Cas Systems | Clustered Regularly Interspaced Short Palindromic Repeats | Hepatitis B virus - genetics | Bacterial Proteins - metabolism | DNA, Viral - genetics | Nucleases | Molecular genetics | RNA | Analysis | DNA | Hepatitis B virus | Health aspects | Hepatitis B | Index Medicus | CRISPR | Cas | antiviral | lentiviral vector
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8. Full Text Hepatitis B virus upregulates host expression of α-1,2-mannosidases via the PPARα pathway
by Song Hu Li-Bin Jiang Xiao-Jing Zou Wei Yi De-Ying Tian
World journal of gastroenterology : WJG, ISSN 1007-9327, 2016, Volume 22, Issue 43, pp. 9534 - 9543
Glycosylation | Dendritic cells | α-Mannosidase | Hepatitis B | Pattern recognition receptors | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Hepatitis B Surface Antigens - genetics | Liver - virology | Up-Regulation | PPAR alpha - antagonists & inhibitors | Liver - enzymology | Hepatitis B - genetics | Humans | alpha-Mannosidase - metabolism | Hepatitis B - virology | Hepatitis B Surface Antigens - metabolism | Hepatitis B - enzymology | Hep G2 Cells | Host-Pathogen Interactions | Hepatitis B e Antigens - genetics | Transfection | Liver - drug effects | Hepatitis B virus - genetics | Hepatocytes - virology | Indoles - pharmacology | Hepatitis B virus - metabolism | PPAR alpha - metabolism | Hepatocytes - drug effects | Hepatitis B e Antigens - metabolism | Hepatocytes - enzymology | Index Medicus | Basic Study
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9. Full Text Structure and Function of Viral Deubiquitinating Enzymes
by Bailey-Elkin, Ben A and Knaap, Robert C.M and Kikkert, Marjolein and Mark, Brian L
Journal of molecular biology, ISSN 0022-2836, 11/2017, Volume 429, Issue 22, pp. 3441 - 3470
viruses | viral deubiquitinating enzyme | papain-like protease | innate immune evasion | ubiquitin | Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Host-Pathogen Interactions | Deubiquitinating Enzymes - chemistry | Viruses - enzymology | Immune Evasion | Viral Proteins - chemistry | Viruses - immunology | Deubiquitinating Enzymes - metabolism | Immunity, Innate | Viral Proteins - metabolism | Hemorrhagic fever | Microbiology | RNA | Sarcoma | Vaccination | Gastroenteritis | Severe acute respiratory syndrome | Papain | Monkeys | Biological response modifiers | Genetic translation | Proteins | Ligases | Adenoviruses | Analysis | Index Medicus | DUB, deubiquitinating enzyme | AdV, adenovirus | MHV, mouse hepatitis virus | OTU, ovarian tumor protease | ISG, interferon-stimulated gene | EVG, Enterovirus species G | TGEV, porcine transmissible gastroenteritis | PLP, papain-like protease | PRRSV, porcine reproductive and respiratory syndrome virus | IBV, avian infectious bronchitis virus | NSDV, Nairobi sheep disease virus | ISG15, interferon-stimulated gene 15 | AMC, 7-amino-4-methylcoumarin | TLR, toll-like receptor | HAUSP, herpesvirus-associated USP | MDA5, melanoma differentiation factor 5 | TNF, tumor necrosis factor | RR, ribonucleotide reductase | BL2, blocking-loop 2 | SARS, severe acute respiratory syndrome | LDV, lactate dehydrogenase-elevating virus | TANK, TRAF family member-associated activator | Ub, ubiquitin | cGAS, cyclic-GMP-AMP synthase | RIG-I, retinoic acid-inducible gene I | EAV, equine arteritis virus | MERS, Middle East respiratory syndrome | EBV, Epstein–Barr virus | MERS-CoV, Middle East respiratory syndrome CoV | IRF, IFN regulatory factor | NEMO, NF-κB essential modulator | UBL, Ub-like | STING, stimulator of IFN genes | HSV-1, herpes simplex virus 1 | USP, Ub-specific protease | cGAMP, cyclic-GMP-AMP | MAVS, mitochondrial antiviral signaling protein | TYMV, turnip yellow mosaic virus | ssRNA, single-stranded RNA | TRAF, TNF receptor-associated factor | PEDV, porcine epidemic diarrhea virus | CCHFV, Crimean-Congo hemorrhagic fever virus | DUGV, Dugbe virus | IRF3, IFN regulatory factor 3 | KSV, Kaposi's sarcoma virus | ERVV, Erve virus | FMDV, foot-and-mouth disease virus | eIF4G, eukaryotic translation initiation factor 4 gamma | IFN, interferon | SHFV, simian hemorrhagic fever virus | MCMV, murine cytomegalovirus | ORF, open reading frame | MDV, Marek's disease virus | HCMV, human cytomegalovirus | PRR, pattern-recognition receptor | CRL, Cullin-RING Ub ligase | UbV, Ub variant | TF, trans frame | CoV, coronavirus | SARS-CoV, severe acute respiratory syndrome CoV | nsps, non-structural proteins | TBK1, TANK-binding kinase 1
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