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Journal Article
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2008, Volume 135, Issue 1, pp. 111 - 121
Journal Article
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2010, Volume 54, Issue 4, pp. 795 - 809
The unfolded protein response (UPR) is activated upon the accumulation of misfolded proteins in the endoplasmic reticulum (ER) that are sensed by the binding... 
Gastroenterology and Hepatology | Hepatosteatosis | Endoplasmic reticulum stress | Unfolded protein response | Liver injury | MESSENGER-RNA TRANSLATION | UNFOLDED-PROTEIN RESPONSE | TRANSCRIPTION FACTOR XBP-1 | NECROSIS-FACTOR-ALPHA | ISCHEMIA-REPERFUSION INJURY | PROGRAMMED CELL-DEATH | GLUCOSE-REGULATED PROTEINS | HEPATITIS-C VIRUS | HUMAN HEPATOCELLULAR-CARCINOMA | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | Hepatitis C, Chronic - metabolism | Fatty Liver - metabolism | Cholestasis - metabolism | eIF-2 Kinase - metabolism | Humans | Liver Diseases - pathology | Stress, Physiological | Endoplasmic Reticulum - metabolism | Hyperhomocysteinemia - metabolism | Hepatocytes - pathology | Hepatocytes - metabolism | Unfolded Protein Response | Liver Diseases, Alcoholic - metabolism | Activating Transcription Factor 6 - metabolism | Inflammation - metabolism | Animals | Models, Biological | Liver Neoplasms - metabolism | Non-alcoholic Fatty Liver Disease | Protein Conformation | Liver Diseases - metabolism | Reperfusion Injury - metabolism | Apoptosis | Carcinoma, Hepatocellular - metabolism | Inositol | Immunoglobulins | Stress (Physiology) | Ribonuclease | Glucose | Genetic translation | Dextrose | Messenger RNA | Proteases | Proteolysis | Analysis | Hepatitis C | Adenylic acid | Hepatitis C virus | Health aspects | Protein binding | Tumors
Journal Article
Journal Article
Journal Article
European Journal of Immunology, ISSN 0014-2980, 09/2016, Volume 46, Issue 9, pp. 2204 - 2210
Immune exhaustion is a hallmark of chronic viral infections. However, pathogen eradication can result in reinvigorated immune responses. Indeed, this was... 
Chronic infection | HCV | Interferon‐free therapy | MAIT cells | Immune exhaustion | Interferon-free therapy | INVARIANT T-CELLS | ACTIVATION | DENDRITIC CELLS | PROLIFERATION | IMMUNOLOGY | NK CELLS | IMMUNE-RESPONSES | EXPRESSION | Hepatitis C, Chronic - metabolism | Follow-Up Studies | Antiviral Agents - therapeutic use | Hepacivirus - immunology | Humans | Gene Expression Regulation | Hepatitis C, Chronic - virology | Immunophenotyping | Treatment Outcome | Transcription Factors - genetics | Hepatitis C, Chronic - drug therapy | Case-Control Studies | Viral Load | Mucosal-Associated Invariant T Cells - immunology | Histocompatibility Antigens Class I - metabolism | Transcription Factors - metabolism | Lymphocyte Activation - immunology | Minor Histocompatibility Antigens - metabolism | Mucosal-Associated Invariant T Cells - metabolism | Phenotype | Hepatitis C, Chronic - immunology | Biomarkers | Virus diseases | Antigens | Interferon | Biological response modifiers | T cells | Hepatitis C virus | Health aspects | T cell receptors | Therapy | PD-1 protein | CD8 antigen | Mucosa | Immune clearance | Effector cells | Viruses | Lymphocytes T | Infections | Hepatitis | CD69 antigen | Granzyme B | Lymphocytes | Peripheral blood | Immune system | Pathogens | Immune response | Exhaustion | Histocompatibility antigen HLA | Hepatitis C
Journal Article