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Journal Article
Nature, ISSN 0028-0836, 07/2011, Volume 475, Issue 7356, pp. 386 - 391
The generation of functional hepatocytes independent of donor liver organs is of great therapeutic interest with regard to regenerative medicine and possible... 
MURINE MODEL | HEPATIC-DYSFUNCTION | PANCREAS PROGENITORS | TYROSINEMIA TYPE-I | LIVER-FUNCTION | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | FACTOR NETWORK | DIRECT CONVERSION | GENE-EXPRESSION | ENRICHED TRANSCRIPTION FACTORS | Hydrolases - genetics | Liver - enzymology | Hepatocyte Nuclear Factor 1-alpha - genetics | Tail - cytology | Liver Diseases - pathology | Gene Expression Profiling | Hepatocyte Nuclear Factor 3-gamma - genetics | Hepatocytes - metabolism | Hepatocyte Nuclear Factor 1-alpha - metabolism | Liver - physiopathology | Regenerative Medicine - methods | DNA-Binding Proteins - deficiency | Cell Differentiation - genetics | Hepatocytes - cytology | Hepatocyte Nuclear Factor 3-gamma - metabolism | Liver Diseases - enzymology | Hepatocytes - transplantation | Hepatocytes - physiology | Fibroblasts - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | GATA4 Transcription Factor - metabolism | Tissue Engineering - methods | Transduction, Genetic | Cells, Cultured | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Liver Diseases - therapy | GATA4 Transcription Factor - genetics | Survival Rate | Hydrolases - deficiency | Mice, SCID | Cell Lineage | Animals | Liver - physiology | Mice, Inbred NOD | Fibroblasts - cytology | Liver - cytology | Mice | Liver Diseases - physiopathology | Physiological aspects | Fibroblasts | Transcription factors | Research | Embryonic stem cells | Liver cells | Proteins | Gene expression | Tissue engineering | Cell adhesion & migration | Index Medicus
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1533 - 1544
The cause of organ failure is enigmatic for many degenerative diseases, including end-stage liver disease. Here, using a CCl4-induced rat model of irreversible... 
LONG-TERM | PATHOGENESIS | MEDICINE, RESEARCH & EXPERIMENTAL | NUCLEAR | MECHANISM | CHRONIC LIVER-DISEASE | CIRRHOSIS | RATS | HEPATOCYTE | EXPRESSION | TRANSPLANTATION | Liver Cirrhosis, Experimental - therapy | Liver Failure - etiology | Dependovirus - genetics | Genetic Therapy | Hepatocyte Nuclear Factor 1-alpha - genetics | Hepatocyte Nuclear Factor 3-beta - genetics | Rats, Inbred Lew | Transcriptome | Hepatocyte Nuclear Factor 1-alpha - biosynthesis | Liver Failure - pathology | Hepatocytes - pathology | Male | Gene Expression Profiling | Genetic Vectors - therapeutic use | Hepatocytes - metabolism | Gene Regulatory Networks | Hepatocyte Nuclear Factor 4 - physiology | Recombinant Fusion Proteins - metabolism | CCAAT-Enhancer-Binding Protein-alpha - biosynthesis | Hepatocyte Nuclear Factor 4 - biosynthesis | Liver Cirrhosis, Experimental - pathology | Liver Cirrhosis, Experimental - complications | Hepatocyte Nuclear Factor 3-beta - biosynthesis | Transcription Factors - physiology | Transduction, Genetic | PPAR alpha - biosynthesis | Down-Regulation | Cells, Cultured | Gene Expression Regulation | Rats | PPAR alpha - genetics | Hepatocyte Nuclear Factor 4 - genetics | Liver Cirrhosis, Experimental - genetics | Liver Failure - genetics | Disease Progression | Animals | Carbon Tetrachloride Poisoning - therapy | Liver Failure - therapy | CCAAT-Enhancer-Binding Protein-alpha - genetics | Carbon Tetrachloride Poisoning - genetics | Cell Dedifferentiation - genetics | Transcription factors | Liver diseases | Genetic research | Development and progression | Genetic aspects | Genetic transcription | Research | Properties | Hypertension | Cytochrome | Transplants & implants | Rodents | Stem cells | Metabolism | Laboratory animals | Liver cirrhosis | Index Medicus | Abridged Index Medicus | Hepatology | Gastroenterology
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2006, Volume 103, Issue 11, pp. 4046 - 4051
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7516, pp. 110 - 114
Mutations in isocitrate dehydrogenase 1 (IDH1) and IDH2 are among the most common genetic alterations in intrahepatic cholangiocarcinoma (IHCC), a deadly liver... 
PROGENITORS | INTRAHEPATIC CHOLANGIOCARCINOMA | HOMEOSTASIS | LIVER-REGENERATION | MULTIDISCIPLINARY SCIENCES | MUTATION | GROWTH | MICE | PROLIFERATION | EXPRESSION | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Hepatocyte Nuclear Factor 4 - antagonists & inhibitors | Humans | ras Proteins - metabolism | Hepatocytes - pathology | Male | Hepatocytes - metabolism | Bile Duct Neoplasms - enzymology | Cell Differentiation - genetics | Neoplasm Metastasis | Hepatocyte Nuclear Factor 4 - biosynthesis | Female | Bile Duct Neoplasms - genetics | Cell Lineage - genetics | Cholangiocarcinoma - enzymology | Disease Models, Animal | Cell Division - genetics | Proto-Oncogene Proteins - metabolism | Hepatocyte Nuclear Factor 4 - metabolism | Bile Ducts, Intrahepatic - pathology | Mutant Proteins - genetics | Isocitrate Dehydrogenase - genetics | Mice, Transgenic | Mutant Proteins - metabolism | Proto-Oncogene Proteins - genetics | Hepatocyte Nuclear Factor 4 - genetics | Mutation - genetics | Bile Ducts, Intrahepatic - enzymology | Cholangiocarcinoma - pathology | Animals | Cholangiocarcinoma - genetics | Stem Cells - pathology | Isocitrate Dehydrogenase - metabolism | Glutarates - metabolism | Mice | Bile Duct Neoplasms - pathology | Hepatocytes - enzymology | Index Medicus | Càncer | Diferenciació cel·lular | Gallbladder diseases | Metabolisme | Cell diferentiation | Metabolism | Malalties de la vesícula biliar | Cancer
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2012, Volume 57, Issue 3, pp. 628 - 636
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2009, Volume 119, Issue 6, pp. 1537 - 1545
The forkhead box proteins A1 and A2 (Foxa1 and Foxa2) are transcription factors with critical roles in establishing the developmental competence of the foregut... 
TRANSCRIPTION FACTORS | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATED PROTEIN-KINASE | LIVER MORPHOGENESIS | HEPATOCYTE GROWTH-FACTOR | GENE FAMILY | INTERLEUKIN-6-DEFICIENT MICE | BILIARY EPITHELIAL-CELLS | NF-KAPPA-B | EXPRESSION | CHOLANGIOCYTE PROLIFERATION | Bile Ducts - growth & development | Cell Proliferation | Hepatocyte Nuclear Factor 3-beta - genetics | Bile Ducts - metabolism | Receptors, Glucocorticoid - metabolism | Hepatocytes - metabolism | Fibrosis - metabolism | Bile Ducts - cytology | Hepatocytes - cytology | Hyperplasia - genetics | Bile Duct Diseases - metabolism | Cell Differentiation | Interleukin-6 - metabolism | Hyperplasia - metabolism | Hepatocyte Nuclear Factor 3-beta - metabolism | Microscopy, Electron, Transmission | Fibrosis - genetics | Interleukin-6 - genetics | Hepatocyte Nuclear Factor 3-alpha - deficiency | Liver - metabolism | Mice, Inbred C57BL | Bile Duct Diseases - genetics | Hepatocyte Nuclear Factor 3-alpha - genetics | Hepatocyte Nuclear Factor 3-alpha - metabolism | Mice, Knockout | Hyperplasia - pathology | Animals | Liver - cytology | Mice | Fibrosis - pathology | Bile Duct Diseases - pathology | Hepatocyte Nuclear Factor 3-beta - deficiency | Common bile duct | Growth | Bile ducts | Genes | Physiological aspects | Genetic aspects | Research | DNA binding proteins | Index Medicus | Abridged Index Medicus
Journal Article
Development, ISSN 0950-1991, 08/2007, Volume 134, Issue 15, pp. 2761 - 2769
Journal Article
Science, ISSN 0036-8075, 2/2004, Volume 303, Issue 5662, pp. 1378 - 1381
The transcriptional regulatory networks that specify and maintain human tissue diversity are largely uncharted. To gain insight into this circuitry, we used... 
Beta cells | Transcription factors | Hepatocytes | RNA | Genes | Liver | Reports | Gene expression regulation | Islets of Langerhans | Regulator genes | Type 1 diabetes mellitus | DIABETES-MELLITUS | HEPATOCYTES | COMPLEX | YOUNG | UPSTREAM | HNF-4-ALPHA GENE | MULTIDISCIPLINARY SCIENCES | NETWORKS | DIFFERENTIATION | SPECIFICATION | BETA-CELL FUNCTION | Oligonucleotide Array Sequence Analysis | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Humans | Gene Expression Profiling | Hepatocytes - metabolism | RNA Polymerase II - metabolism | Phosphoproteins - metabolism | Islets of Langerhans - metabolism | Diabetes Mellitus, Type 2 - etiology | Transcription, Genetic | Hepatocyte Nuclear Factor 1-beta | DNA-Binding Proteins | Gluconeogenesis | Promoter Regions, Genetic | Carbohydrate Metabolism | Gene Expression Regulation | Hepatocyte Nuclear Factor 1-alpha | Lipid Metabolism | Precipitin Tests | Nuclear Proteins | Transcription Factors - metabolism | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors | Hepatocyte Nuclear Factor 6 | Trans-Activators - metabolism | Hepatocyte Nuclear Factor 4 | Genome, Human | Hepatocyte Nuclear Factor 1 | Genetic aspects | Diabetes | Genetic transcription | Chemical properties | Pancreas | HNF1^a protein | HNF6 protein | HNF4^a protein | Index Medicus
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 11/2017, Volume 21, Issue 11, pp. 2809 - 2822
The extracellular matrix ( ECM ) microenvironment is involved in the regulation of hepatocyte phenotype and function. Recently, the cell‐derived extracellular... 
endothelial cells | metabolism | hepatocyte | integrin | Src | extracellular matrix | MEDICINE, RESEARCH & EXPERIMENTAL | LIVER ORGANOGENESIS | PROLIFERATION | ESTABLISHMENT | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | ADHESION | STIFFNESS | FIBRONECTIN | EXTRACELLULAR-MATRIX | DIFFERENTIATION | EXPRESSION | RNA, Small Interfering - genetics | Integrin alpha5beta1 - genetics | Phosphorylation | Receptors, Steroid - metabolism | Hepatocyte Nuclear Factor 3-beta - genetics | Humans | Collagen Type I - chemistry | Adipose Tissue - cytology | Human Umbilical Vein Endothelial Cells - chemistry | Stem Cells - cytology | Hepatocytes - metabolism | Stem Cells - metabolism | Extracellular Matrix - chemistry | Adipose Tissue - metabolism | Hepatocytes - cytology | src-Family Kinases - metabolism | Cell Differentiation | Hepatocytes - drug effects | Hepatocyte Nuclear Factor 3-beta - metabolism | Hepatocyte Nuclear Factor 4 - metabolism | Signal Transduction | src-Family Kinases - antagonists & inhibitors | Gene Expression Regulation | Collagen Type I - isolation & purification | Rats | Hepatocyte Nuclear Factor 4 - genetics | Pyrimidines - pharmacology | Fibronectins - metabolism | Animals | Receptors, Steroid - genetics | Stem Cells - drug effects | Fibronectins - genetics | Primary Cell Culture | src-Family Kinases - genetics | Adipose Tissue - drug effects | Integrin alpha5beta1 - metabolism | RNA, Small Interfering - metabolism | Fibronectins | Stem cells | Integrins | Endothelium | Maturation | Src protein | Metabolism | Nuclear receptors | Fibronectin | Endothelial cells | Signaling | Receptors | Hepatocytes | Transcription activation | Forkhead protein | Extracellular matrix | Biocompatibility | Hepatocyte nuclear factor 4 | Inhibition | Index Medicus | Original
Journal Article