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Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 3/2006, Volume 103, Issue 11, pp. 4046 - 4051
Journal Article
Nature (London), ISSN 1476-4687, 07/2014, Volume 513, Issue 7516, pp. 110 - 114
Mutations in isocitrate dehydrogenase 1 (IDH1) and IDH2 are among the most common genetic alterations in intrahepatic cholangiocarcinoma (IHCC), a deadly liver... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Hepatocyte Nuclear Factor 4 - antagonists & inhibitors | Humans | ras Proteins - metabolism | Hepatocytes - pathology | Male | Hepatocytes - metabolism | Bile Duct Neoplasms - enzymology | Cell Differentiation - genetics | Neoplasm Metastasis | Hepatocyte Nuclear Factor 4 - biosynthesis | Female | Bile Duct Neoplasms - genetics | Cell Lineage - genetics | Cholangiocarcinoma - enzymology | Disease Models, Animal | Cell Division - genetics | Proto-Oncogene Proteins - metabolism | Hepatocyte Nuclear Factor 4 - metabolism | Bile Ducts, Intrahepatic - pathology | Mutant Proteins - genetics | Isocitrate Dehydrogenase - genetics | Mice, Transgenic | Mutant Proteins - metabolism | Proto-Oncogene Proteins - genetics | Hepatocyte Nuclear Factor 4 - genetics | Mutation - genetics | Bile Ducts, Intrahepatic - enzymology | Cholangiocarcinoma - pathology | Animals | Cholangiocarcinoma - genetics | Stem Cells - pathology | Isocitrate Dehydrogenase - metabolism | Glutarates - metabolism | Mice | Bile Duct Neoplasms - pathology | Hepatocytes - enzymology | Index Medicus | Càncer | Diferenciació cel·lular | Gallbladder diseases | Metabolisme | Cell diferentiation | Metabolism | Malalties de la vesícula biliar | Cancer
Journal Article
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1533 - 1544
The cause of organ failure is enigmatic for many degenerative diseases, including end-stage liver disease. Here, using a CCl4-induced rat model of irreversible... 
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Liver Cirrhosis, Experimental - therapy | Liver Failure - etiology | Dependovirus - genetics | Genetic Therapy | Hepatocyte Nuclear Factor 1-alpha - genetics | Hepatocyte Nuclear Factor 3-beta - genetics | Rats, Inbred Lew | Transcriptome | Hepatocyte Nuclear Factor 1-alpha - biosynthesis | Liver Failure - pathology | Hepatocytes - pathology | Male | Gene Expression Profiling | Genetic Vectors - therapeutic use | Hepatocytes - metabolism | Gene Regulatory Networks | Hepatocyte Nuclear Factor 4 - physiology | Recombinant Fusion Proteins - metabolism | CCAAT-Enhancer-Binding Protein-alpha - biosynthesis | Hepatocyte Nuclear Factor 4 - biosynthesis | Liver Cirrhosis, Experimental - pathology | Liver Cirrhosis, Experimental - complications | Hepatocyte Nuclear Factor 3-beta - biosynthesis | Transcription Factors - physiology | Transduction, Genetic | PPAR alpha - biosynthesis | Down-Regulation | Cells, Cultured | Gene Expression Regulation | Rats | PPAR alpha - genetics | Hepatocyte Nuclear Factor 4 - genetics | Liver Cirrhosis, Experimental - genetics | Liver Failure - genetics | Disease Progression | Animals | Carbon Tetrachloride Poisoning - therapy | Liver Failure - therapy | CCAAT-Enhancer-Binding Protein-alpha - genetics | Carbon Tetrachloride Poisoning - genetics | Cell Dedifferentiation - genetics | Transcription factors | Liver diseases | Genetic research | Development and progression | Genetic aspects | Genetic transcription | Research | Properties | Hypertension | Cytochrome | Transplants & implants | Rodents | Stem cells | Metabolism | Laboratory animals | Liver cirrhosis | Index Medicus | Abridged Index Medicus | Hepatology | Gastroenterology
Journal Article
Journal Article
Biochemical and biophysical research communications, ISSN 0006-291X, 09/2016, Volume 478, Issue 2, pp. 791 - 797
Hepatocytes from human bone marrow-derived mesenchymal stem cells (hBM-MSCs) are expected to be a useful source for cell transplantation. However, relatively... 
Hepatocyte-like cells | Hepatocyte nuclear factor 4 alpha | Mesenchymal stem cells | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Antibodies, Monoclonal - toxicity | Hepatocyte Nuclear Factor 3-beta - genetics | Chemical and Drug Induced Liver Injury, Chronic - metabolism | Humans | Male | Hepatocytes - metabolism | RNA, Messenger - metabolism | Cytochrome P-450 CYP2B6 - metabolism | Lentivirus - metabolism | Hepatocytes - cytology | Mesenchymal Stromal Cells - cytology | Glycogen - metabolism | Lentivirus - genetics | Urea - metabolism | Cell Differentiation | Hepatocytes - transplantation | Chemical and Drug Induced Liver Injury, Chronic - genetics | Hepatocyte Nuclear Factor 3-beta - metabolism | Hepatocyte Nuclear Factor 4 - metabolism | Transduction, Genetic | alpha 1-Antitrypsin - genetics | Genetic Vectors - chemistry | Signal Transduction | Bone Marrow Cells - cytology | Cytochrome P-450 CYP2B6 - genetics | RNA, Messenger - genetics | Gene Expression Regulation | Genetic Vectors - metabolism | Mesenchymal Stromal Cells - metabolism | Hepatocyte Nuclear Factor 4 - genetics | Chemical and Drug Induced Liver Injury, Chronic - pathology | Animals | Chemical and Drug Induced Liver Injury, Chronic - therapy | Mice | alpha 1-Antitrypsin - metabolism | Albumins - secretion | Albumins - genetics | Bone Marrow Cells - metabolism | Cell Line, Transformed | Index Medicus | TRANSCRIPTION FACTORS | CULTURE MEDIA | LIVER CELLS | BONE MARROW | STEM CELLS | INDUCTION | 60 APPLIED LIFE SCIENCES | SKELETON
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 11/2017, Volume 21, Issue 11, pp. 2809 - 2822
The extracellular matrix (ECM) microenvironment is involved in the regulation of hepatocyte phenotype and function. Recently, the cell‐derived extracellular... 
endothelial cells | metabolism | hepatocyte | integrin | Src | extracellular matrix | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | RNA, Small Interfering - genetics | Integrin alpha5beta1 - genetics | Phosphorylation | Receptors, Steroid - metabolism | Hepatocyte Nuclear Factor 3-beta - genetics | Humans | Collagen Type I - chemistry | Adipose Tissue - cytology | Human Umbilical Vein Endothelial Cells - chemistry | Stem Cells - cytology | Hepatocytes - metabolism | Stem Cells - metabolism | Extracellular Matrix - chemistry | Adipose Tissue - metabolism | Hepatocytes - cytology | src-Family Kinases - metabolism | Cell Differentiation | Hepatocytes - drug effects | Hepatocyte Nuclear Factor 3-beta - metabolism | Hepatocyte Nuclear Factor 4 - metabolism | Signal Transduction | src-Family Kinases - antagonists & inhibitors | Gene Expression Regulation | Collagen Type I - isolation & purification | Rats | Hepatocyte Nuclear Factor 4 - genetics | Pyrimidines - pharmacology | Fibronectins - metabolism | Animals | Receptors, Steroid - genetics | Stem Cells - drug effects | Fibronectins - genetics | Primary Cell Culture | src-Family Kinases - genetics | Adipose Tissue - drug effects | Integrin alpha5beta1 - metabolism | RNA, Small Interfering - metabolism | Fibronectins | Stem cells | Integrins | Endothelium | Maturation | Src protein | Metabolism | Nuclear receptors | Fibronectin | Endothelial cells | Signaling | Receptors | Hepatocytes | Transcription activation | Forkhead protein | Extracellular matrix | Biocompatibility | Hepatocyte nuclear factor 4 | Inhibition | Index Medicus | Original
Journal Article